MSK Flashcards

1
Q

3 types of equine muscle disorders

A

primary - traumatic/metabolic/infectious - diffuse or focal pain

secondary - using muscles differently because of pain - localised, muscle enzymes often normal

neuromuscular - muscle atrophy - myogenic or neurogenic

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2
Q

muscle strain - equine

A

acute pain
localised pain and swelling
hard to isolate - large muscle mass

risk factors -
type of activity
surface terrain
poor warm up

mildly elevated muscle enzymes
Ultrasound - fluid accumulation and disrupted fibre pattern

treatment -
cold hosing/icing
NSAIDs
rest
gentle mobilisation and exercises

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3
Q

exertional myopathies - equine

A

very prevalent

signs -
poor exercise tolerance
muscle stiffness
shortened hind limb stride
reluctance to move
firm, painful, hindquarter muscles
anxiety
pain
sweating
increased resp rate
colic type signs - pawing, trying to lie down
myoglobinuria
elevated CK and AST

sporadic or recurrant
sporadic - one off extrinsic factors - overexercising, dietary imbalance, exhausted horse syndrome
recurrant - intrinsic factors - heritable factos, breed related, recurrent exertional rhabdomyolysis, polysaccharide storage myopathy

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4
Q

recurrent exertional rhabdomyolysis (RER) - equine

A

abnormal regulation of muscle contraction
issue in calcium kinetics
light or hot breeds - thoroughbreds
suggested heritability
more often in nervous females

risk factor - high grain diet

diagnosis -
signs and history
serum CK and AST increase
muscle histology - used to rule out concurrent conditions

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5
Q

polysaccharide storage myopathy (PSSM) - equine

A

accumulation of glycogen in muscle fibres

type 1 - mutation in glycogen synthesis gene
can test for mutation

type 2 - looks similar on biopsy but no gene identified

mutation most common in draft breeds, warmbloods, appaloosas, cobs, and ponies

diagnosis -
signs
muscle enzymes
muscle biopsies - only see changes over 2yo
genetic testing - on blood or hair roots

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6
Q

myofibrillar myopathy - equine

A

exercise intolerance
intermittent RER

warmbloods and arabs
warmbloods can have normal muscle enzymes - confirm on biopsy
arabs - high enzymes after exercise, myoglobinuria - tend to be less painful

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7
Q

exertional myopathies - treatment - equine

A

relieve pain
correct fluids
protect kidneys from effects of NSAIDs
stabling for acute stages then keep them working - days off can make it worse

diet -
low starch, high fat
adequate electrolytes
vitamin E supplementation
amino acid supplementation - less evidence

medications - dantrolene sodium (muscle relaxant)

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8
Q

clostridial myositis - equine

A

rare but nasty
usually iatrogenic - recent IM injection
necrotising infection of muscle
systemic illness - severe toxemia
swollen, painful muscle at injection site
subcutaneous emphysema

antimicrobials
multiple surgical debridements

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9
Q

delayed onset muscle soreness - equine

A

poor performance
diffuse pain
couple of days after unusual exercise
eccentric muscle contractions - contraction when muscle under tension

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10
Q

equine motor neurone disease

A

not common
generalised atrophy
oxidative damage to motor neurons

risk factors -
vitamin E/selenium deficiency
prolonged pasture access with high CHO diet

signs -
happy enough
normal appetitie
muscle weakness and atrophy
trembling
weight shifting on standing
walk better than they stand
low head carriage
exercise intolerance
“elephant on tub” stance
retinal changes - pigmentation at back of retina

diagnosis -
low vitamin E in plasma
elevated muscle enzymes
muscle biopsy of tailhead - definitive

usually need euthanising

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11
Q

vitamin E deficiency - equine

A

presents similar to motor neuron disease
responsive to treatment

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12
Q

immune mediated myositis - equine

A

rapid atrophy
moderate muscle enzymes elevation

usually quarter horses
MYH1 mutation - homozygous horses more affected

triggered by exposure to episode of strangles or other respiratory disease

diagnosis -
biopsy - epxial or gluteal muscles
genetic testing

treatment - steroids, antibiotics if concurrent infection

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13
Q

hip dysplasia - risk factors

A

weight
types of exercise
overexercising when young
breed - big dogs

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14
Q

ddx - hip dysplasia

A

arthritis
legg-calves perthes - avascular necrosis of femoral head
psoas injury
elbow dysplasia
cruiciate injury
luxated patella
nervous conditions - sciatic pathology
neoplasia - bone or joint
sepsis

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15
Q

hip dysplasia - pathophysiology

A

joint laxity –> femoral head subluxation
osteoarthritis changes - wear, cartilage thinning, fluid, osteophytes

most crucial time for hip development - before 8 weeks

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16
Q

hip scoring

A

each hip scored between 0-52 (overall out of 106)
lower score better

VD radiograph under GA
very straight
both limbs straight out and tied together
labelled
ID number and kennel club number

reviewed by 2 experts

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17
Q

patellar luxation - pathophysiology

A

bad conformation - muscle action pulls patellar ligament and displaces patella

not congenital - born normal, other abnormalities/deformities in limb causing them to turn inwards

grading 1-4

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18
Q

hip dysplasia - signs

A

difficulty rising
abnormal gait
bunny hopping
pelvic limb lameness
clicking of hips
assymetric muscle mass
sensitivity on hind quarters - esp on extension and abduction
reduced range of motion
crepitus
asymmetric pad wear

all non specific

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19
Q

hip dysplasia - testing

A

ortolani test
barden test
radiography - VD legs extended, VD frogleg, lateral (hip series) - measures of dorsal acetabular cover, norberg angle, distraction index

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20
Q

hip dysplasia - management

A

pain management
exercises - range of motion, muscle mass, strength
controlled activity level
weight management
nutraceuticals
physiotherapy

surgery - excision arthroplasty (joint fills with soft tissue to create false joint), total hip replacement, joint fusion (not hips but other joints)

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21
Q

cruciate injury - signs

A

frog sitting - external rotation of opposing stifle
sit test
loss of muscle mass
cranial draw

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22
Q

meniscal injury

A

seen secondary to cranial cruciate rupture (in most cases)
usually medial meniscus
heals poorly - usually needs taken out
if taken out - always end up with degenerative joint disease

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23
Q

cruciate injury - management

A

suture - loop round to create rotating force and hold in place
osteotomy - TPLO, TTA - good outcomes, expensive

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24
Q

cruciate injury

A

cruciate function - prevents cranial translation and internal rotation of tibia and prevents hyperextension of stifle
most common cause of hindlimb lameness

often eventually bilateral
changes can exist before ruptures
partial tears common
usually not a contact injury - different from in people

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25
elbow dysplasia - pathopahysiology
osteochondrosis of humeral condyle - failure of endochondrial ossification ununited anconeal process - failure of fusion of growth plate allows anconeal process to detach fragmented coronoid process - medial coronoid fragments due to fissuring under the surface radio-ulnar incongruity - step between radius and ulna instead of smooth gap can have any of these concurrently - complicated joint
26
elbow dysplasia - signs
lameness choppy forelimb gait effusion pain crepitus reduced range of motion
27
patellar luxation - surgery
wedge recession block recession abrasion tibial tubercule transposition distal femoral osteotomy - dramatic
28
angular limb deformity - equine
vargus - limb bending inwards valgus - limb bending outwards limbs turned in, knees out happens as they grow mild deformities - stall confinement more dramatic - may need surgery - perioteal stripping or transphyseal bridging corrective foot trimming prognosis affected by - age at intervention, degree of deformity, joint affected and time that growth plate usually fuses, what the horse needs to eventually do
29
growth plate closure times - equine
P1 - long pstern - 2-3 months MC3 - cannon bone - 6-9 months distal radius - 9-12 months
30
osteochondrosis - equine
2 types - osteochondrosis dessicans (OCD) - non loaded margin of high impact joints sub chondral bone cysts (SBC) - high load margins, cysts develop within the joint usually genetic with environmental factors circulation not getting to cartilage as well as it should in rapidly growing young animals cartilage doesn't develop how it should
31
fracture classification
cause - intrinsic or extrinsic open or closed - is skin breached extent of damage number of fractures position direction of fracture lines location forces acting on the fracture - affects stability after fixed involvement of other tissues - trapped nerves, vessels, soft tissues, puncture of organs age of fracture fracture geometry transverse - straight across oblique - line of break offset from right angle comminuted - multiple peices segmental - large segment blown out
32
femoral fracture
most common small animal appendicular fracture access for surgery lateral - less vessels
33
radius/ulna fracture
usually distal of mid diaphyseal usually just repair radius little dogs jumping off things can sometimes just use external fixator if sides of fracture well opposed
34
tibial fracture
high velocity injury access for surgery from medial side - tension side, skin tighter over bone, less muscles to go through external fixator may work if comminuted and open
35
salter-harris fractures
physeal (growth plate) fractures S - straight across A - above (along plate then up) L - lower (along plate then down) T - through (cutting down through plate) ER - erasure of plate (crush) always damage to growth plate avoid putting in devices that compress or restrict bone lengthening - smooth k wires preferred
36
femoral head fracture
usually young dogs (3-10 months) and cats fracture across growth plate operate early, delayed action worsens prognosis
37
proximal tibial fracture
main growth plate is a complression plate but also a tension plate at tibial tuberosity (attachment for patellar ligament) need a tension wire so tibial tuberosity doesn't just pull off
38
mandibular symphyseal separation
common in cats wire it shut - occlude sides but don't overtighten and crush tissues take wire out after about 6 weeks can use heavy gauge PDS instead of wire so will absorb check no other jaw fractures before fixing
39
pelvic fractures
referral needs a significant impact, not easy to break multiple fractures check spine, bladder, sciatic nerve
40
MSK cancer types
primary - osteosarcoma - most common - make bone myeloma, lymphoma, chrondrosarcoma - don't make bone secondary - metastasis to bone - squamous cell cancinoma, lung-digit - more common than primary
41
osteosarcoma
agreesive osteoblast tumour large and giant breeds more in male than female middle aged to older usually appendicular poor prognosis needs amputation and chemo - amputation alone not enough 90% have micro mets at time of diagnosis
42
arthrodeisis
used in cases of untreatable fractures, chronic pain from degenerative joint disease, chronic luxation fuse the joint outcome dependent on joint
43
extensor tendon lacteration
not too serious - can sever whole way and still go back to work usually from wound on front of leg unable to extend joint usually common digital and long digital extensor tendon robert jone bandage if knuckling forwards standard wound care good prognosis
44
flexor tendon laceration
potentially life threatening - major support structure for distal limb over reach injury severe lameness weight bearing can give indication which tendon risk of synovial sepsis Ultrasound to see which tendon SDFT - fetlock drops on weight bearing DDFT - toe off ground when weightbearing SDFT, DDFT and SL - toe off ground and fetlock drop treatment - immobilise limb debride wound treat synovial sepsis suture tendon ends - repair prognosis good for life but bad for function complete laceration - very poor prognosis synovial sepsis - very poor prognosis
45
causes of recurrent abscess - equine
foreign body insufficient drainage sequestrum keratoma laminitis quittor3
46
sequestrum and foreign body in foot - equine
persistently draining tract at coronary band radiograph - classic sequestrum appearance (dead bone, black bit where there should be bone surround by involcrum) can lead to infection in pedal bone remove inciting cause for antibiotics to work
47
keratoma - equine
benign tumour - in keratinised tissue in hoof wall noticable circular structure (keratinisation) in white line on base of foot radiograph - circular radiolucent in P3 needs surgical removal good prognosis but can reoccur
48
quittor - equine
mostly draught breeds draining above coronary band non healing wound infection of collateral cartilages --> necrosis needs surgical debridment antibiotics will hold it but if stop taking them draining will start again
49
white line disease - equine
sometimes pus separation of horn sections quite lame crumbling of hoof wall multifactorial - environmental, nutritional (biotin, calcium, vitamin A), mechanical (hoof maintenance), infection can usually diagnose from separation of white line aggressive debridement of hoof wall - remove enough but keep stable manage triggering factors
50
canker - equine
hobby breeds moist environment - predisposes to anaerobic bacterial infection (so bacterial but management related) hypertrophy of dermis of horn of foot shite soft moist tissue around heel and frog usually smells bad aggressive debridement needed will bleed a lot - needs lots of packing topical metronidazole astringents - dry out tissue address management issues - otherwise will reoccur
51
causes of foot pain - equine
foot balance hoof wall joint collateral ligament digital cushion DDFT navicular bursa distal sesamoid ligament
52
food pain - presentation - equine
lameness - chronic and intermittent inconsistent performance loss of suppleness - not moving as usual tripping or stumbling unwillingness to go forwards - more likely to canter than trot temporary improvement with rest
53
bilateral lameness signs - equine
worse on hard surfaces worse when circling mild to moderate usually one limb worse decreased cranial stride
54
unilateral lameness signs - equine
minimal external signs insidious or acute onset usually no digital pulse or swelling improves with rest then gets worse when back to work
55
poor foot balance - equine
usually a contributing factor address before looking to other ddx foot trimming
56
navicular syndrome - equine
associated with navicular bone, bursa and soft tissues in that region middle aged competition horses bilateral shortened stride worse on hard surface resolves on rest then comes back insidious onset risk factors - upright conformation jumping working on hard surfaces radiograph - once signs visible usually quite advanced - abnormal synovial fossae, cysts in bone, osteophytes, decreased cortical meduallry definition
57
navicular syndrome - treatment - equine
change foot balance - decrease heel pressure, maximise surface area for weight NSAIDs steroids into bursa stem cells - repair DDFT damage polyacrimide hydrogel - support and cushioning for navicular bone bisphosphonates surgery - remove section of palmar digital nerve so can't feel foot progressive, not curable manage pain and progression
58
DDFT tears - equine
unilateral forelimb lameness acute onset often can block with palmar digital nerve block usually only shows on radiograph if very chronic tear MRI best for diagnosis
59
articular pain - equine
DIP and PIP joints mild lameness loss of performance reluctance to move forwards nerve blocks to diagnose acute joint damage --> synovitis (rapid) --> osteoarthritis (ongoing damage)
60
DIP joint - collateral ligament lameness - equine
worse when cicrcling will block with abaxial sesamoid nerve block usually acute MRI to diagnose
61
side bone - equine
ossification of collateral cartilages --> extra bone formation at side of foot rarely causes lameness unless fracture causing inflammation or concurrent cause of lameness cob type horses
62
synovial disease - treatment - equine
NSAIDs - chronic lameness only effective if inflammation bute - cheap, effective for synovitis, possible renal and GI side effects flunixin meglumine - most effective for visceral pain, anti endotoxic, not usually first line for orthopedic pain fibrocoxib - osteoarthritis, more expensive than bute but less side effects paracetamol - human kind, analgesia for laminitis bisphosphonates - stop bone breakdown, alleviate pain and reduce lameness good for some arthritis significant side effects - renal, not with NSAIDs, colic (then can't give flunixin with it) tested for in doping - 30 day detection intra-articular steroids - reduced local inflammation short acting or long acting shorter acting - tramcinolone - high motion joints longer acting - methyprednisolone - low motion joints hyaluronic acid - improved viscosity of synovial fluid anti-inflammatory polyacrimide hydrogel - improved joint lubrication and cellular growth high motion joints - more space to fit gel in regenerative therapies - stem cells - made from horse or off the shelp reduction of mild to moderate degenerative joint disease grow new cartilage
63
SDFT tendonitis - types
intrinsic - strain extrinsic - injury displacement
64
SDFT tendonitis - stages of damage
tendon matrix degeneration - cumulative, associated with aging fibrillar spillage - crosslinks breakage fibril rupture complete rupture most common injury of event horses acute overload or degenerative
65
SDFT tendonitis - risk factors
previous inflammation uneven surfaces muscular exhaustion foot imbalance long sloping pasterns reduced blood supply to mid SDFT region
66
SDFT tendonitis - signs
swelling lameness thickening of tendon pain on palpation oedema heat sinking fetlock stance ultrasound - oedema, varying echogenicity, fibre allignment (scar tissue)
67
SDFT tendonitis - treatment
acute phase - days after injury - limit inflammation cold hosing icing NSAIDs steroids bandage - reduce oedema supportive bandage, box rest - prevent further damage subacute phase - reparative stage, some reduction in lameness and swelling box rest stem cells - promote angiogenesis, and quality repair platelet rich plasma - promote quality repair physio - therapeutic ultrasound, low level laser, hydrotherapy - reduce oedema and inflammation chronic phase - remodelling (months) controlled exercise programme - small amounts of hand walking then progressive increase rescanning before and after changes in exercise level
68
mesenchymal stem cells
expensive and time consuming - have to harvest and grow them off the shelf probably will be available eventually autologous stem cells for tendon injuries form tendon tissue instead of scar tissue reduced re injury rate - better quality repair
69
platelet rich plasma
cheaper than stem cells increased speed of healing ultrasound guided injection lacking long term studies around rate of reinjury
70
check ligament injury
common - forelimb swelling in proximal 1/3 metacarpal ultrasound - enlargement, focal abnormalities, fibre pattern disruption, concurrent SDFT tendonitis on margins treatment - acute - box rest, NSAIDs, cold hosing, bandaging chronic - controlled exercise, NSAIDs, corrective foot trimming
71
suspensory ligament desmitis
proximal, body or branch types proximal forelimb - just below knee, some pain on deep palpation, some enlargement treat with controlled exercise, box rest, stem cells prognosis good hindlimb - more chronic, bilateral, poor perofrmance associated with striaght hindlimbs, worse on soft surface and with exercise radiography - sclerosis, avulsion fracture US - fibrosis, loss of striation, new bone laid down - US hard on hindlimbs body older event horses variable lameness further down leg associated with the new bone laid down on splint bone after injury diagnose on palpation treat - controlled exercise, shockwave branch any limb poor-fair prognosis
72
tenosynovitis
inflammation of tendon sheaths mild to moderate lameness minimal response to rest intrasynovial analgesia to confirm radiography - with contrast can see manica flexoria
73
manica flexoria
tendon ring that hold SDFT in place marginal tears can drive chronic cases of tenosynovitis tears caused by compression of tendon in overextension
74
capped hock
synovial fluid in cacaneal bursa (point of hock) cosmetic not usually lame
75
windgalls
fetlock or tendon sheath distension with or without lameness
76
thoroughpin
synovial distension of tarsal sheath cosmetic
77
tenosynovitis - treatment
acute - rule out sepsis rest ice/cold hosing NSAIDs intrathecal steroids hyaluronic acid tenoscopy - if lack of response to conservative treatment chronic - tenoscopy - early exercise post surgery to reduce post op adhesions rest and controlled exercise address underlying issues palmar annular ligament desmotomy - cut ligament to reduce constriction, good prognosis if desmitis has been controlled
78
carpal canal syndrome - equine
tenosynovitis of carpal sheath (carpal tunnel) secondary to tendonitis of SDFT and DDFT, or fracture in that region, or osteochondroma need to remove the osseous impingment
79
Wooden tongue
actinobacillus lignaressi - gram -ve, oral commensal, gains entry through breaks in buccal mucosa sudden onset salivation dysphagia protrusion of tongue firm, swollen, painful tongue - not moving as much as usual enlarged lymph nodes submandibular swelling diagnosis - exam and bacterial c&s
80
wooden tongue - ddx
stomatitis lumpy jaw dental disease oral foreign body pharyngeal trauma
81
wooden tongue - management
isolate animal - makes sure get enough food an water ensure adequate food and water antibiotics oral potassium iodide or IV sodium iodide - not licensed in UK food animals
82
lumpy jaw
actinomycosis pyogranulomatous osteitis seen when young cattle teething - entry through mucosa when teeth erupting infects bone usually bright seeming enlargement of horizontal ramus of jaw soft tissue swelling irregular swelling and bone remodelling tooth displacement fractures discharging sinus tracts pain and inability to eat --> weight loss diagnosis - exam, impression smear from discharging tracts, radiography (extent of bone remodelling, doesn't change treatment options)
83
lumpy jaw - ddx
tooth root abscess fracture of mandible neoplasia foreign body feed impaction
84
lumpy jaw - treatment
food and water to reduce weight loss antibiotics
85
mandibular fracture - farm
usually due to trauma - kick, hit by tractor, dystocia dysphagia weight loss excess salivation swelling at fracture site protruding tongue diagnosis - palpate misalignment at site, radiography management - slight displacement - will heal over time wire external fixator euthanasia/emergency slaughter (if don't have time for aftercare then just euthanise) ramus in calf will heal better than mandibular symphysis
86
dental disease - farm
not commonly reported leads to inefficient chewing --> decreased intake --> decreased productivity tooth root abscess - from infection from periodontal disease or penetration of crown of tooth by commensal bacteria jaw swelling salivation halitosis discharging sinus tracts inappetence weight loss diagnosis - inside mouth exam (use a gag), external palpation, radiography
87
tooth root abscess - treatment - farm
remove infected tooth (could flush or suture after but hard to get it clean enough will cause overgrowth or malocclusion of opposing tooth antibiotics - long term if bone infection, can have negative effects on gut microflora NSAIDs dietary change - high energy concentrate that doesn't need as much chewing
88
choke
oesophageal obstruction can be proximal cervical, at thoracic inlet or in thoracic oesophagus usually due to access to large lumps of root vegetables distress extended neck and lowered head salivation regularly trying to swallow lower obstruction - large amounts of clear viscous saliva over time - rumen bloat from not being able to ruminate may be able to feel or see blockage if very cranial pass stomach tube - won't go down if blocked
89
choke - ddx
frothy bloat gassy bloat tetanus
90
choke - management
relieve bloat - trocar or needle hyoscine - relaxes oesophagus massage obstruction further cranial manually remove if very cranial pass form stomach tube to push block into reumen probang - device that dislodges or slices obstruction if not too distressed can just leave it until it degrades a bit and can be swallowed
91
bovine papular stomatitis
parapoxvirus zoonotic usually calves under 1yo papules in mouth anorexia salivation mild pyrexia lesions most severe - raised lesions ddx - foot and mouth BVD vesicular stomatitis bovine herpes blue tongue
92
foot and mouth
picornavirus notifiable very contagious lesions in mouth and on feet anorexia depression pyrexia salivation milk drop vesicles on tongue and dental pad and hard palate - rupture to leave shallow ulcers lesions on coronary band later - pathopneumonic
93
calf diptheria
fusobacterium necrophorum necrosis in mouth young calves - see outbreaks associated with poor hygiene lesions caused by trauma to mucosa wet lower jaw - excess saliva firm swellings in cheeks necrosis in mouth mucosa halitosis swelling in submandibular lymph nodes pyrexia if infection in larynx - anorexia, pyrexia, coughing inspiratory stridor (roaring), dyspnoea death - due to asphyxiation/respiratory distress treatment - penicillin steroids if in respiratory distress NSAIDs tracheotomy - if really struggling to breathe
94
laryngeal chondritis
texel throat (sheep) unable to breath properly more in tups infection of mucosal abrasions --> abscess in larynx --> exacerbates existing issue with larynx in predisposed breeds acute respiratory distress inspiratory effort and stertor - snoring sound neck extended, head lowered, nostril flare open mouth breathing diagnose by signs and breed predisposition
95
laryngeal chondritis - management
steroids - reduce swelling NSAIDs - more long term antibiotics - long term, prevent abscessation euthanasia tracheotomy - in severe cases usually doesn't fully resolve and often recurs
96
hypocalcemia - signs
loss of smooth muscle tone recumbant s shaped neck
97
hypocalcemia - management
calcium sub cut - takes a few hours to work because impaired peripheral circulation calcium borogluconate (with magnesium) - into jugular slow calcium if heart irregularities don't give too much calcium - spike thick bedding with lots of grip food and water in easy reach remove calf for 24 hours lift and turn every 6 hours if recumbent - so not laying on muscle groups too long bloods - test if need also magnesium or phosphorus may need further calcium - bolus, electrolyte fluid, oral gels prevention - high calcium concentrate at calving assess forage component in diet prophylactic calcium for at risk coews - 3rd lactation on, twins keep in calving box for an extra day remove calf not milk for first few days hand feed colostrum and don't milk out whole udder D3 injection before calving control BCS calcium restriction last 3 weeks of gestation magnesium supplementation calcium binding products last 2 weeks gestation diet cation:anion balance manipulation - low sodium and potassium, high chloride and sulphates - monirot using urine pH straw or maize silage better low calcium
98
hypocalcemia - sheep
late pregnancy/early lactation older sheep associated with stress passive reflux of ruminal contents down nose when lying down blood sample pre treatment - calcium under 1mmol/litre treatment - slow IC calcium borogluconate prevention - feed adequate but not excessive calcium (5-10g/day), avoid stressful conditions/events
99
hypomagnesia
majority magnesium locked in bone, can only use the part from rumen and omasum (or intestines in calf) needed for milk and tissue production spring and autumn lactating cows without enough supplementation types - peracute acute subclinical chronic calf milk tetany
100
hypomagnesia - signs
nervous and aggressive hyperaesthesia convulsions death
101
hypomagnesia - management
emergency - treat ASAP calm - surprises can cause convulsions keep in lateral while treating then move to sternal calcium borogluconate - has magnesium in, IV in tail vein control seizures - xylazine, ACP, pentobarbitone food and water nearby food with magnesium ongoing clinical cases usually tip of iceberg, check herd for subclinical lick blocks bolus magnesium concentrated magnesium in water spray pasture check soil magnesium levels good grasses that translate magnesium to their leaves ensure good daily dry matter intake reduce stress
102
hypomagnesia - sheep
first 6 weeks after lambing ewes with multiple lambs convulsion and sudden death ddx - acute mastitis (also presents as sudden death) treatment - IV calcium borogluconate, reduced stress (provide shelter)
103
hypophosphatemia
down cows high production cows in periparturient period or if reduced feed intake around calving decreased GI motility young cows - slow growth, rickets adult - lethargy, weight loss, anorexia, muscle weakness, muscle and bone pain, rhabdomyolysis, intravascular hemolysis later stages - pica usually seen with milk fever diagnosis - blood sample for phosphorus, watch reaction to calcium treatment (try and get up but just shuffle about) oral or injectable phosphorus
104