Motor Speech Flashcards
Main features of apraxia (5)
- Difficulty transitioning between sounds (turning voice on and off)
- Issues with prosody
- Articulatory groping
- Difficulty with initiation
- Increased sound errors with increase in length
Apraxia of speech is due to a defecit in:
a) programming
b) planning
c) execution
d) all of the above
e) a and b only
d) a and c only
e) programming and planning
Main features of dysarthria
Abnormality in strength, speed, range, steadiness, and tone of movement required for speech
What is oral apraxia?
Difficulty planning volitional movement of oral structures for non-speech tasks
Ideomotor apraxia
Unable to follow commands or imitate gestures
Ideational apraxia
Able to do individual tasks but can’t plan out a longer sequence/action (e.g.
What is the most common cause of AOS?
Degenerative disease + stroke
If a person with AOS complained of chewing/swallowing difficulties, what should you do?
Assess for dysarthria
Protocol for AOS when it comes to potential aphasia
All AOS patients should be considered aphasic until comprehensive language ax has been completed because AOS frequently occurs with Broca’s
AOS is nearly always the result of abnormality in the _____ cerebral
hemisphere
Left (dominant)
T/F: Dysarthria is often associated with aphasia
False - AOS is very often associated wth aphasia but Dysarthria is NOT
Differential dx of dysarthria and AOS
Dysarthria vs AOS
- All speech systems vs. artic + prosody
- Consistent errors vs. inconsistent
- More distortions (makes speech easier) vs. more substitutions (making it harder)
- Weakness vs. no change in strength/tone
- Keeps rhythm of SMRs but reduced range/speed/force vs. trouble with the rhythm/sequence
Ataxic dysarthria and AOS SIMILARITIES
- Poor motor control/coordination
- Irregular artic errors
- Most prominent difficulties are with articulation and prosody
- Oral mec findings can be normal
Differential dx of ataxic dysarthria and AOS
- AMRs worse in ataxic while SMRs worse in AOS
- Don’t usually grope to correct vs. do
- Substitutions not as frequent in ataxic compared to AOS
- Dysarthria doesn’t improve with automaticity while AOS can
What are some tx goals for RESTORATION when confronted with MSDs?
- Increase maximum phonation time
- Strengthen muscles of exhalation
- Increase intelligibility
- Increase participation/comm. effectiveness
What are some tx goals for COMPENSATION when confronted with MSDs?
- Use extra effort to increase loudness
- Use palatal lift/nasal obturator
What are some tx goals for SUPPLEMENTATION when confronted with MSDs?
- Alphabet board to indicate first letter of word
- AAC device when natural speech profoundly affected
Flaccid dysarthrias come from an injury to the:
a) Upper Motor Neuron
b) Lower Motor Neuron
c) Both
B) Lower Motor Neuron
Why do bilateral UMN lesions cause spastic dysarthria?
UMN sends the signal to muscles to relax; when impaired, it’s not giving that signal and so ++ stretch reflex, ++tonicity
In spastic dysarthria, the bias of the muscle tone in the larynx is usually toward
a) hyperadduction
b) hypoadduction
a) hyperadduction
Perceptual voice/speech characteristics of spastic dysarthria
- Slow, deliberate pace
- Equal + excess stress; monotone
- Strained/strangled vocal quality
- Hypernasal
- Imprecise artic (sluggish tongue mvmt)
What does a person with PD having mixed spastic-ataxic dysarthria indicate?
Either they don’t have PD or they have something else going on as well because PD shouldn’t be associated with ataxia