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Felder 3rd semester > Module 5 Musculoskeletal > Flashcards

Module 5 Musculoskeletal Flashcards

1
Q

Opiates and Opioids are used for what level of arthritis

A

Moderate to Severe arthritis

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2
Q

Inhibits prostaglandin synthesis, cyclooxygenase, arachidonic conversion

A

Salicylates

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3
Q

Aspirin is an example of a

A

Salicylates

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4
Q

arachidonic conversion + cyclooxygenase =

A

Prostaglandin

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5
Q

Where do Salicylates come from

A

bark of willow tree

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6
Q

Avg dose of Aspirin is

A

81mg-650mg

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7
Q

What is the max dose for Aspirin as an anti-inflammatory and what is it used for

A

3g/day Arthritis

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8
Q

What is the max dose for Aspirin as an Non-narcotic analgesic and what is it used for

A

650mg 1 4-6 hrs Moderate mild pain

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9
Q

What is the max dose for Aspirin as an Antipyretic

A

650mg q 4 hrs

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10
Q

What is the max dose for Aspirin as an Antiplatelet

A

81mg or 325mg / day

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11
Q

Viral infection causes fatty liver

A

Reyes Syndrome

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12
Q

Acute poisoning in Salicylates can lead to

A

Rhabdomyolysis, acute renal failure

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13
Q

If poisoning occurs in Salicylates what can we use to absorb substances

A

Activated charcoal

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14
Q

A non-Salicylates is also called

A

NSAIDS

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15
Q

Ibuprofen max dose is (for arthritis)

A

1200mg-2400mg q 4-6 hrs

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16
Q

Ibuprofen max dose for pain is

A

400-800mg q 4-6 hrs

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17
Q

Naproxen max dose is

A

500-1000mg / day

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18
Q

How should Indocin be administered

A

50-200 mg/day in 2-3 doses

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19
Q

Diclofenac should be administered

A

100-200 mg/day

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20
Q

Misoprostol is a combination drug that includes

A

Cytotec

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21
Q

Celecoxib is used to treat

A

Osteoarthritis and Rheumatoid arthritis

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22
Q

What is the max dose for Acetaminophen

A

4 grams a day

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23
Q

What is the max dose for Acetaminophen for a patient with ETOH

A

2 grams a day

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24
Q

Drug used to lower the effects of Acetaminophen

A

Mucomyst

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25
Q

If meds are +10days for Acetaminophen what should patient do

A

consult prescriber

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26
Q

What should we monitor for a patient taking DMARDS

A

Blood sedimentation rate

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27
Q

Gold salts are used in

A

Chrysotherapy

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28
Q

Chrysotherapy is used to

A

Decrease tenderness, swelling, pain

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29
Q

How long should it take for full therapeutic effect in Chrysotherapy

A

6 months

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30
Q

Auranofin is a drug used in …. and can only be given

A

Chrysotherapy .. PO LARGE MUSCLE MASS

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31
Q

If protein gets in blood during Chrysotherapy what should be done

A

d/c tx.

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32
Q

4 side effects of Chrysotherapy

A

Stomatitis
loose stools
Bone marrow suppression
Bluish green skin discoloration

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33
Q

We should report any use of gold salts prior to what test

A

TB test

34
Q

Chrysotherapy causes what type of reaction

A

Nitroid reaction

35
Q

Cytokine produced by macrophages, results in inflammation and cartilage erosion

A

Tumor Necrosis Factor

36
Q

Decreases immune system response, used for psoriasis and arthritis

A

TNF inhibitors

37
Q

4 side effects of TNF inhibitors

A

Leukemia
Lymphoma
Hepatitis B infection
TB

38
Q

Etanercept, Adalimumab, and Golimumab are examples of what kind of drug class

A

TNF inhibitor

39
Q

How do we infuse TNF inhibitors

A

over 2 hours, 2 weeks - 6 - 8

40
Q

Penicillamine should be taken how … and not with what

A

on an empty stomach … not with antacids

41
Q

Chelator med

A

Penicillamine

42
Q

How long does it take to see improvement with Methotrexate

A

3-6 weeks

43
Q

Triple therapy with Methotrexate includes what other 2 drugs

A

Hydroxychloroquine and sulfasalazine

44
Q

Hydroxychloroquine can be toxic to

A

eyes

45
Q

sulfasalazine should be given how

A

With food, Full glass of water

46
Q

Sulfasalazine can cause (2)

A

Male infertility

yellow orange skin discoloration

47
Q

Patients with gout should decrease foods that contain

A

Purine

48
Q

Uric acid in gout should be what #

A

less than 6mg/dl

49
Q

Used to treat/prevent acute flares of gout

A

Colchicine

50
Q

When should we d/c Colchicine

A

when patient reaches 8mg

51
Q

In gout patient can use what uric acid inhbitor

A

Allopurinol

52
Q

Allopurinol can also be called a

A

Hypouricemic

53
Q

4 side effects of Allopurinol

A

alopecia
SJS
purpuric rash
Bone marrow supression

54
Q

This drug is used to enhance uric acid excretion

A

Probenecid

55
Q

Probenecid can also be called a

A

Uricosuric

56
Q

How should we give Probenecid

A

With food OR antacid

57
Q

What is the action of a Bisphosphonate

A

Increases bone density

58
Q

How should we give Bisphosphonates

A

Full glass of water on empty stomach.

59
Q

What is something a person should not do after taking a Bisphosphonate

A

Do not lay down for 30 minutes

60
Q

What are 3 side effects of Bisphosphonates

A

Esophageal Ulcerations
Bone pain
Headache

61
Q

What is something a person should not consume if taking Bisphosphonates

A

Milk

62
Q

Alendronate, Risedronate, Zoledronic Acid, and Ibandronate are examples of what drug class

A

Bisphosphonates

63
Q

Raloxifene is an example of what drug

A

Selective Estrogen Receptor Modulator (SERM)

64
Q

Selective Estrogen Receptor Modulator (SERM) drugs are used for

A

Bone, Uterine cancer

65
Q

Forteo (Teriparatide) is what kind of drug

A

Bone Formation Agent

66
Q

Forteo contributes what hormone and what can it cause

A

Contributes parathyroid hormone and can cause hypercalcemia

67
Q

What is the major adverse effect of Central Acting drugs

A

Drowsiness, Dizziness, Ataxia

68
Q

Define Ataxia

A

inability to control muscle

69
Q

Inhibits skeletal muscle contraction by blocking conduction within the spinal cord

A

Central Acting drugs

70
Q

Muscle spams, epilepsy, electrolyte imbalance can all be treated with what drug group

A

Central Acting drugs

71
Q

Central Acting drugs should be given

A

With food

72
Q

Titrate means

A

Start slow, gradually increase

73
Q

Taper means

A

Decrease slowly

74
Q

Methocarbamol a central acting drug can cause what

A

Greenish, Brownish, Black, urine discoloration

75
Q

Neuromuscular blockers are different from central acting drugs because

A

Acts on receptor site, peripheral acting

76
Q

interrupts transmission at skeletal neuromuscular junction

A

Neuromuscular blockers

77
Q

Agonist Depolarizing Neuromuscular blockers, differ from Competitive Non Depolarizing Neuromuscular blockers because

A

Competitive Non Depolarizing Neuromuscular blockers REQUIRE MED TO REVERSE ACTIVITY

78
Q

Muscle spasms require Peripheral or Central

A

Peripheral

79
Q

It is important to tell patient that Neuromuscular blockers

A

Have no effect on pain or consciousness

80
Q

Dantrolene is an example of a

A

Direct Acting drug

81
Q

A major adverse effect of Dantrolene is

A

Malignant hyperthermia

82
Q

Skeletal muscle stimulants can cause (2)

A

increase in saliva

increase in sweat

Felder 3rd semester (10 decks)

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