Mobility Flashcards

1
Q

Fracture are often a result of

A

Accidental trauma at home or at school (falls, climbing, running into immovable objects
•Motor vehicle-related injuries
•Recreational activities (skateboards, bicycles, ATVs, snowboards, motorcycles, and sports)

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2
Q

Fracture during infancy

A
  • Typically caused by MVAs or falls from heights.
  • Injuries causing fracture are uncommon.

•Bony injuries warrants further investigation to rule out nonaccidental trauma.
-Skeletal Surveys (radiologic evidence of fractures at various stages of healing).

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3
Q

Fractures suspicious for abuse include

A
  • Any fracture in a non-ambulatory infant
  • Mid-shaft humeral or femoral fractures in children less than 18 months old
  • Rib fractures in children less than 3 years of age
  • Unusual fractures such as:
  • Scapula
  • Sternum
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4
Q

BONE HEALING AND REMODELING

A

Fractures heal much faster in children. The younger the child, the more quickly the bone heals

  • Children’s bones have a thick, strong periosteum with an abundant blood supply which contributes to a more rapid formation of callous strong enough to heal the fracture more rapidly than adults
  • With pediatric fractures less callus formation is required to achieve a clinically stable or healed fracture than with adults.
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5
Q

Stages of bone healing

A

1) hematoma formation
2) granulation tissue formation (inflammation)
3) callus formation
4) consolidation
5) bone remodeling

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6
Q

Nursing care for fractures

A
  • Assess NV status of the distal extremity
  • Move injured part as little as possible.
  • Inspect skin for bruising, erythema, or swelling.
  • Observe the extremity for deformity.
  • Cover open wounds with a sterile or clean dressing.
  • Immobilize limb (above and below fx site)
  • Do not attempt to push protruding bone under the skin.
  • Apply manual traction if circulatory compromise is present.
  • Elevate the injured limb if possible.
  • Apply cold (no longer than 20 minute intervals).
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7
Q

What is SCOLIOSIS

A
  • Spinal deformity, usually involving a lateral curvature than exceeds 10 degrees, and spinal rotation causing rib asymmetry.
  • Classified according to age of onset: congenital, infantile, juvenile, or adolescent.
  • Most are idiopathic (no known cause).
  • May occur alone or in association with neuromuscular disease
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8
Q

ADOLESCENT IDIOPATHIC SCOLIOSIS (AIS)

A
  • Develops during the growth spurt of early adolescence, and may become progressively worse with growth.
  • Predominant in females, and may be familial.
  • AIS screening usually starts at age 10 years during well-child visits.
  • Screenings are also commonly done by school nurses
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9
Q

Scoliosis screening

A

Early screening/detection and management results in improved outcomes

  • Shoulder height
  • Hip height
  • Scapular predominance
  • Distance between elbows and waist (flank area)
  • Asymmetry of ribs and flanks (during Adam’s forward bend test)
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10
Q

SCOLIOSIS DIAGNOSIS

A

Degree of Cobb Angle
Classification:

< 10° Normal

10° to 25°Mild

25° to 45° Moderate

> 45° Severe

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11
Q

Risser grade

A

Used to determine when child is done growing(at stage5)

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12
Q

SCOLIOSIS SURGERY (SPINAL FUSION)

Pre‐Op Patient/Family Education

A

•Bathe daily with CHG cloths starting 3 days prior to surgery.

•Miralax 17gm daily,
one day prior to surgery. (If
patient is constipated, start
2 days prior to surgery.)

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13
Q

POSTERIOR SPINAL FUSION POSTOPERATIVE CARE

A

Patient log-rolled every 2 hours

  • Ambulation started as soon as possible (2nd post-op day) with physical therapy assistance
  • Indwelling foley catheter is typically removed prior to ambulation.
  • Priority assessments: wound, VS, NV, neurologic, pain
  • Pain management (PCA MSO4 initially and then oral narcotics once tolerating fluids
  • Dressing changes daily
  • In some cases, brace or cast may be needed.
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14
Q

CEREBRAL PALSY (CP)

A

Characterized by abnormal muscle tone and coordination

  • Caused by damage to the motor areas of the brain, resulting in a neurological lesion.
  • The lesion disrupts the brain’s ability to control movement and posture.
  • The lesion does not get worse (non-progressive); however, the symptoms can change.
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15
Q

Spastic CP

A

Stiff muscles

Damage to cerebral cortex

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16
Q

Athetoid/ dyskinetic CP

A

Uncontrollable movement

Damage to basal ganglia

17
Q

Ataxic CP

A

Poor balance and coordination

Damage to cerebellum

18
Q

Mixed CP

A

Combo of two or more

19
Q

Congenital CP

A

related to brain damage before or during birth. Specific cause is often not known

•Risk factors: low birthweight, premature birth, multiple births, assistive reproductive technology, infections during pregnancy, jaundice and kernicterus, medical conditions of the mother such as thyroid disease or seizures, and birth complications.

20
Q

Acquired CP

A

caused by brain damage that occurs more than 28 days after birth.

Usually caused by infection (meningitis or encephalitis), injury (motor vehicle injuries or child abuse), or cerebrovascular accidents.

21
Q

Associated Problems with CP

A
  • Cognitive and learning difficulties
  • Speech and language disorders
  • Behavioral and mental health disorders
  • Sensory impairments
  • Impaired vision/hearing
  • Seizures
  • Growth and nutrition problems
  • Drooling and feeding problems
  • Dental problems
  • Hydrocephalus
22
Q

Complications of CP

A
  • Pressure ulcers
  • UTIs
  • Constipation
  • Bowel and bladder incontinence
  • GERD
  • Sleep disorders
  • Failure to thrive
  • Joint contractures
  • Scoliosis
  • Pulmonary problems
23
Q

MANAGEMENT OF COMPLICATIONS

A

•Pulmonary problems are the leading cause of hospitalization and death for children with CP

  • Multifactorial
    • Aspiration
    • Ineffective airway clearance
      * Airway obstruction
    • Chronic lung disease
24
Q

PREVENTION OF COMPLICATIONS with CP

A
  • Immunizations
  • Regular dental care
  • Screen for aspiration (Feeding evaluation and/or video swallow study)
  • Screen for sleep-disordered breathing
  • Mucous clearance
  • Management of constipation
25
Q

CP therapeutic interventions

A
PT
OT
Speech
Special education 
Surgical interventions 
Medication therapy 
Care coordination
26
Q

COMMON PHARMACOLOGIC AGENTS for CP

A
  • Stool softeners (Colace)
  • Osmotic laxatives (MiraLax - polyethylene glycol PEG)
  • Baclofen – may be administered orally, g-tube, or via intrathecal pump
  • Diazepam
  • Dantrolene sodium