Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

ABOS Part I > Misc > Flashcards

Misc Flashcards

1
Q

Treatment of symptomatic Watanabe I and II discoid menisci.

A

Saucerization only.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

MSK malignancy associated with Rothmund-Thompson syndrome

A

Osteosarcoma.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Indication for ORIF for ASIS avulsion fractures.

A

Displacement greater than 3cm or painful nonunion.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What factor most significantly contributes to development of pseudotumor formation for MoM implants?

A

Edge loading.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Antoni A.

A

Cellular area in benign schwannoma.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Antoni B.

A

Less cellular area in benign schwannoma.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Serratus anterior innervation.

A

Long thoracic.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Rhomboids innervation.

A

Dorsal scapular.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Supraspinatus innervation.

A

Suprascapular n.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Infraspinatus innervation.

A

Suprascapular n.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Horner syndrome indicates preganglionic injury to these nerve roots.

A

C8, T1.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

CC ligaments.

A

Conoid and trapezoid.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Most medial CC ligament.

A

Conoid.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Most lateral CC ligament.

A

Trapezoid.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

These ligaments are torn with type 3 AC separation.

A

CC ligaments.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

This ligament prevents anterosuperior escape.

A

Coracoacromial.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Hornblower’s sign tests this

A

Teres minor.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Teres minor innervation.

A

Axillary nerve.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Delayed infection after pediatric PSF.

A

p. acnes.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Pediatric spondylolisthesis indications.

A

Progression, grade 3+

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Type of surgery for pediatric spondylolisthesis.

A

PSF L4-S1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Highest risk of progression of congenital scoliosis.

A

Hemivertebra with contralateral bar.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

EOS progression risk (3).

A
  1. RVAD > 20
  2. Phase 2 ribs
  3. Curve > 30 deg
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Phase I versus phase II ribs in infantile scoliosis.

A

Phase I – no rib overlap with vertebral body

Phase – rib overlap with vertebral body

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Palpable mass in SCM.

A

Congenital muscular torticollis.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Algorightm for C1-C2 pediatric rotator subluxation.

A

CT scan, traction.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Most important risk factor for DDH.

A

Breech presentation.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Ortolani positive.

A

Reducible.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

Barlow positive.

A

Dislocatable.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

This column intact with PAO.

A

Posterior.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Boys age maturity.

A

16

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

Female age maturity.

A

14

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

Perthes more common in boys or girls.

A

Boys.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

Bilateral perthes in ___%.

A

12-15%.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

Congital dislocation of knee associated with ____.

A

DDH.

36
Q

Treat DDH or CDK first?

A

Treat knee dislocation first with casting.

37
Q

For long bone fx, OI treated with.

A

IMN.

38
Q

Medical treatment for OI.

A

Pamidronate.

39
Q

CH17Q21.

A

Neurofibromatosis.

40
Q

Achondroplasia physeal defect in this region of physis.

A

Proliferative zone.

41
Q

DTD gene.

A

Diastrophic dwarfism.

42
Q

Aperts syndrome gene mutation.

A

FGFR2.

43
Q

FGFR3 encodes this type of protein.

A

Tyrosine kinase receptor.

44
Q

Camptomelic dysplasia gene mutation.

A

Sox9.

45
Q

Spondyloepiphyseal dysplasia gene mutation.

A

COL2A1

46
Q

Multiple epiphyseal dysplasia type I mutation.

A

COMP.

47
Q

Diastophic dysplasia gene mutation.

A

DTDST.

48
Q

Pynknodysostosis gene mutation.

A

CTSK.

49
Q

Gene product disrupted in pynknodysostosis.

A

Cathepsin K.

50
Q

Urinary excretion in Morquio’s syndrome.

A

Keratan sulfate.

51
Q

Sprengel’s deformity is a failure of this.

A

Descent.

52
Q

Congenital pseudoarthrosis of the clavicle occurs in this clavicle.

A

Right side.

53
Q

Congenital speudoarthrosis of the clavicle is associated with neurofibromatosis true or false.

A

FALSE.

54
Q

This must be functional in radial deficiency to perform centralization.

A

Biceps.

55
Q

Treatment of thumb hypoplasia dependent on this.

A

CMC stability.

56
Q

Congential radioulnar synostosis is a failure of this.

A

Separation.

57
Q

Fibrous dysplasia with soft tissue myxomas.

A

Mazabraud syndrome.

58
Q

Polyostotic fibrous dysplasia and cafe au lait spots.

A

McCune-Albright syndrome.

59
Q

Multiple non-ossifiying fibromas with cafe au lait skin patches.

A

Jaffe-Campanacci syndrome.

60
Q

Penicillin mechanism of action.

A

Cell wall synthesis inhibitor.

61
Q

Cephalosporin mechanism of action.

A

Cell wall synthesis inhibitor.

62
Q

Vancomycin mechanism of action.

A

Cell wall synthesis inhibitor.

63
Q

Aminoglycoside mechanism of action.

A

Protein synthesis inhibition (30S ribosome).

64
Q

Clindamycin/erythromycin mechanism of action.

A

Protein synthesis inhibition (50S ribosome).

65
Q

Tetracycline mechanism of action.

A

Protein synthesis inhibition (tRNA).

66
Q

Rifampin mechanism of action.

A

RNA synthesis inhibition (RNA polymerase).

67
Q

Cipro/quinolone mechanism of action.

A

Inhibit DNA gyrase.

68
Q

Amphotericin mechanism of action.

A

Fungal cell membrane disruption.

69
Q

Polymyxin mechanism of action.

A

Cell membrane disruptor.

70
Q

Diastophic dysplasia site of physis defect.

A

Reserve zone.

71
Q

Psuedoachondroplasia site of physis defect.

A

Reserve zone.

72
Q

Multiple hereditary exostoses site of physis defect.

A

Proliferative zone.

73
Q

Gigantism (HGH) site of physis action.

A

Proliferative zone.

74
Q

Achondroplasia site of physis defect.

A

Proliferative zone.

75
Q

Rickets physis defect site.

A

Provisional calcification zone fo hypertrophic zone of physis.

76
Q

Site of physeal defect of endchondromas.

A

Hypertrophic zone.

77
Q

Mucopolysacharide diseases site of physis defects.

A

Hypertrophic zone.

78
Q

Zones of the physis from proximal to distal.

A
  1. Reserve zone.
  2. Proliferative zone
  3. Hypertrophic zone
  4. Primary spongiosa
  5. Secondary spongiosa (metaphysis)
79
Q

Three phases of hypertrophic zone of physis.

A
  1. maturation zone
  2. degenerative zone
  3. provisional calcification zone
80
Q

Non-renal SCFE occurs in this physeal zone.

A

Hypertrophic.

81
Q

Renal SCFE occurs in this physeal zone.

A

Secondary spongiosa

82
Q

Metaphyseal corner fractures occur in this physeal zone.

A

Primary spongiosa.

83
Q

Scurvy has effects in this physeal zone.

A

Primary spongiosa.

84
Q

Most abundant non-collagenous protein in bone matrix.

A

Osteocalcin.

85
Q

Function of osteocalcin.

A

Promotes mineralization.

86
Q

Osteocalcin is produced by these cells.

A

Osteoblasts.

ABOS Part I (16 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions