Midterm - ophthalmoscopic exam of internal eye Flashcards

1
Q

What are the 4 steps of an internal eye exam?

A
  1. Check the red reflex
  2. Evaulate the optic disk
  3. Evaluate the retina/vessels
  4. Assess anterior structures
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2
Q

The red reflex is formed by reflection of illuminating light off the _________. Examination is started at ____ degrees lateral to the patient’s line of vision

A

Fundus (back of the eye)

15 degrees

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3
Q

What is a cataract?

A

Any opacity in the lens causing the lens to lose transparency or scatter light

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4
Q

____% of 65-74 yr olds, and ____% of 75+ yr olds have cataracts

A

50, 70

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5
Q

What is the most common cause of cataracts?

A

Age

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6
Q

What risks are associated with cataracts?

A

Cigarette smoking, ocular UV-B light exposure, diabetes, long-term systemic steroids

*when cataracts are due to these caucuses (rather than aging) formation is more rapid

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7
Q

What vision changes occur with cataracts?

A

Gradual loss of visual acuity, foggy vision, increased glare

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8
Q

What are three different cataract types?

A
  1. Subcapsular - at back of lens
  2. Nuclear - gradual clouding of central vision, usually bilaterally
  3. Cortical - “spoke-like” appearance, less impact on vision
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9
Q

What does a normal optic disc look like?

A

Round, yellowish-orange to creamy pink, pale central cup, distinct margins (nasal aspect is naturally slightly more blurred)

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10
Q

What does a blurred disc margin suggest? What causes it?

A
  • Swelling of the optic nerve (papilledema)

- increased intracranial pressure

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11
Q

Papilledema is usually (unilateral/bilateral), causes (early/ no early) vision loss, and (no eye pain/eye pain) ?

A
  • bilateral
  • no early vision loss
  • no eye pain
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12
Q

True or false: a pigment crescent, sclera crescent or myelinated nerve fibers are all normal variants seen?

A

True

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13
Q

The normal cup-to-disc ratio is ______

A

O.5 or 1:2

The cup diameter is half of the optic diameter

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14
Q

Increased cup:disc ratio suggests

A

Glaucomatous cupping (ex: end-stage open angle glaucoma

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15
Q

Primary open-angle glaucoma is related to abnormal function of ____________ and results in (anterior/posterior?) intraocular pressure?

A

Trabecular meshwork

Posterior

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16
Q

What is the leading cause of preventable blindness?

A

Primary open-angle glaucoma

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17
Q

What risks are associated with primary open-angle glaucoma?

A
  • African-Americans are 3-5 times more likely and the risk begins earlier
  • > 35 years old in African-Americans
  • > 50 years old in caucasians
  • family history, severe myopia (near-sightedness), diabetes, cardiovascular disease, medications are all other risks
18
Q

Why are corticosteroids dangerous for the eye?

A

They increase the risk of intraocular pressure elevation - especially if patient has other risks.

  • IOP lowers after stopping steroid treatment but vision damage may be permanent
19
Q

In primary open-angle glaucoma, IOP increase is (gradual/sudden), often (bilateral/unilateral), (painless/painful), and (symptomatic/asymptomatic)* until end-stages?

A
  • gradual
  • bilateral
  • painless
  • asymptomatic
20
Q

In primary open-angle glaucoma, why do patients turn their head to see objects?

A

They gradually lose peripheral vision

21
Q

How do you typically assess primary open-angle glaucoma?

A
  • “air-puff” tonometry screening exam
  • cup:disc ratio
  • peripheral vision testing
22
Q

How is primary open-angle glaucoma treated?

A
  • prescription eye drops
  • aerobic exercise may decrease IOP
  • trabeculoplasty
23
Q

What does the normal macula look like?

A
  • temporal to the optic disc
  • darker than the surrounding retina
  • focal absence of surrounding retinal vessels
24
Q

What does an abnormal macula look like?

A
  • drusen (yellowish-colored sub-retinal deposits)

- can be due to normal aging but may also occur with dry macular degeneration

25
Macular degeneration can cause __________ in elderly patients
Central vision loss and blind spot (scotoma)
26
What are risk factors of macular degeneration?
- >55 years old - female - Caucasian - family history of AMD - cigarette smoking
27
Both dry and wet age-related macular degeneration have _________, but only wet has ________
Drusen | Choroidal (subretinal) neovascularization
28
What is the treatment for age-related macular degeneration?
1. Zinc& antioxidants (Vit C, E, lutein, & zeaxanthin beta-carotene): - Reduces the risk of worsening vision in intermediate or advanced AMD 2. Referral to ophthalmologist to discuss laser, surgical, and/or medical injection treatment options
29
What do normal arteries in the eye look like?
- light red - smaller - bright central light reflection
30
What do normal veins in the eye look like?
- darker red - larger - minimal or absent light reflex
31
What are 2 main causes of abnormal retinal vessels?
- hypertensive retinopathy | - diabetic retinopathy
32
What are floaters and flashers a symptom of? Is it dangerous?
- Posterior vitreous detachment (associated with aging) - Usually harmless Can indicate retinal tear or detachment
33
Why is abnormal vitreous?
Hemorrhages or floaters (dark specks or strands between the fundus and the lens
34
Why is proliferative diabetic retinopathy a big threat to vision?
- neovascularization occurs in response to ischemic changes | - the new vessels can grow into the vitreous increasing risk of retinal detachment and vitreous hemorrhage
35
How does non-proliferative diabetic retinopathy lead vision issues?
- microaneurysms cause weakened vessel walls > vessels leak lipids and blood > hard exudates, hemorrhages and macular edema - small vessel ischemia to the superficial nerve fiber layer of the retina > cotton wool spots
36
How does diabetes lead to vision problems?
- increased risk of cataract formation - increased risk of glaucoma - diabetic retinopathy (non-proliferative and proliferative)
37
Diabetic retinopathy is associated with ____ years of diabetes
20+
38
What is the difference between cotton-wool and hard exudates? Are they white or red spots?
- both are white spots (exudates) found in hypertensive retinopathy and diabetic retinopathy - cotton = soft exudates, fuzzy borders, irregularly shaped, caused by infractions of the nerve fiber layer of the retina - hard = aka lipoid or fatty exudates, more well defined borders, smaller and in deeper layer than cotton-wool spots
39
What is the difference between flame shaped hemorrhages and round microaneurysms? Are they red or white spots?
- both are red spots - flame = occur in superficial retinal layer and is a classic hypertensive retinopathy finding - microaneurysm = "dots and blots", small round with smooth borders, earliest sign of diabetic retinopathy
40
Chronic hypertensive retinopathy can cause ______
1. Arteriovenous crossing (AV nicking and/or tapering) - veins appears to stop abruptly but is actually due to wide, invisible artery walls that have become thickened 2. Copper wire deformity - some arteries become full, tortuous and with increased light reflex
41
What are early signs of hypertensive retinopathy?
- narrowing Artie's with narrowed light reflex | - occasionally silver wire deformity (narrowed artery develops an opaque wall)