Midterm 3 (4) Flashcards

1
Q

Borna disease causes dementia/CNS signs

A

True

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2
Q

Borna disease occurs mainly in the Far East

A

False

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3
Q

Borna disease virus is spreading from cell to cell

A

True

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4
Q

Borna disease appears in horses only

A

False

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5
Q

Mainly central nervous clinical signs can be seen in the case of Borna disease

A

True

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6
Q

The lethality of Borna disease is low

A

False

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7
Q

Borna disease replicate in the nucleus of cells and is called Joest Degen bodies

A

True

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8
Q

Borna disease can be seen all over the world

A

False

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9
Q

Respiratory signs are the most typical ones in the case of Borna disease

A

False

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10
Q

Only ruminants are susceptible to Borna disease virus

A

False

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11
Q

Infective prions replicate in the cytoplasm of the neurons

A

True

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12
Q

Infective prions are taken per os

A

True

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13
Q

Encephalitis is typical in the case of transmissible encephalopathies

A

False

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14
Q

Protease breaks down prions

A

False

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15
Q

Transmissible encephalopathies are acute or per-acute diseases

A

False

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16
Q

Antibodies to prions cannot be detected in the case of transmissible encephalopathies

A

True

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17
Q

Allergy tests are widely used to diagnose transmissible encephalopathies

A

False

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18
Q

Scrapie is seen only in adult sheep

A

False

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19
Q

Genotype can predispose animals to scrapie

A

True

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20
Q

Scrapie is seen in sheep and goats

A

True

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21
Q

Goat are resistant to scrapie

A

False

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22
Q

Scrapie is spread within the flock from animal to animal

A

True

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23
Q

Scrapie prion can infect susceptible animals per os

A

True

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24
Q

Itching can be seen in the case of typical scrapie

A

True

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25
Q

Itching can be seen in the case of atypical scrapie

A

False

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26
Q

Certain sheep can be resistant to scrapie

A

True

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27
Q

Scrapie can be prevented with inactivated vaccines

A

False

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28
Q

Scrapie prion is shed by the infected animals

A

True

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29
Q

Scrapie can be prevented by using attenuated vaccines

A

False

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30
Q

Scrapie in goat, sheep and cattle could be infected

A

False

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31
Q

In case of spongiform encephalopathies micro abscesses are in the brain stem

A

False

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32
Q

Spongiform encephalopathies are mainly acute diseases

A

False

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33
Q

In the case of spongiform encephalopathies there is encephalitis

A

False

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34
Q

Spongiform encephalopathies can be diagnosed by detecting circulating antibodies

A

False

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35
Q

In the case of spongiform encephalopathies the behavior of the animal is generally changes

A

True

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36
Q

In the case of spongiform encephalopathies encephalitis is the main post mortem lesion

A

False

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37
Q

In the case of spongiform encephalopathies high levels of antibodies is produced

A

False

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38
Q

Spongiform encephalopathies are caused by prions

A

True

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39
Q

Bovine spongiform encephalopathy cannot infect humans

A

False

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40
Q

Bovine spongiform encephalopathy causes aggression

A

True

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41
Q

You can diagnose bovine spongiform encephalopathy with ELISA

A

True

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42
Q

Bovine spongiform encephalopathy causes an immune response

A

False

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43
Q

Bovine spongiform encephalopathy is a contact infection

A

False

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44
Q

Bovine spongiform encephalopathy is spreading fast in the infected herd

A

False

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45
Q

Clinical signs of BSE can be seen mainly in 1-1.5 years old cattle

A

False

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46
Q

Cattle with bovine spongiform encephalopathy become aggressive

A

True

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47
Q

BSE can be seen in calves from the age of 6 months

A

False

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48
Q

Hypersensitivity is a clinical sign of BSE

A

True

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49
Q

Focal necrosis in the liver is a typical post mortem lesion of BSE

A

False

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50
Q

Antibodies to BSE can be detected with ELISA in infected animals

A

False

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51
Q

2-6 months old calves having BSE are frequently aggressive

A

False

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52
Q

Bovine spongiform encephalopathy is seen only in beef cows

A

False

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53
Q

Cattle with bovine spongiform encephalopathy have movement difficulties

A

True

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54
Q

Only capsulated strain of B. anthracis is virulent

A

True

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55
Q

Toxin is a virulence factor of B. anthracis

A

True

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56
Q

Lethal factor is a virulence factor of B. anthracis

A

True

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57
Q

Cell wall antigen is a virulence factor of B. anthracis

A

False

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58
Q

Oxygen is needed to the spore production of B. anthracis

A

True

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59
Q

Spore is a virulence factor of B. anthracis

A

False

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60
Q

B. anthracis can cause blackleg

A

False

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61
Q

Anthrax is generally seen as a chronic disease in cattle

A

False

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62
Q

In case of anthrax, febrile animals have to be separated and vaccinated

A

False

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63
Q

Animals with anthrax can be treated with penicillin

A

True

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64
Q

Anthrax spreads rapidly in a herd from animal to animal

A

False

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65
Q

B. anthracis can only be diagnosed by bacterial culture

A

False

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66
Q

B. anthracis cannot form spores in the infected animal

A

True

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67
Q

B. anthracis can only be diagnosed by Ascoli test

A

False

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68
Q

Animals suspected of being infected with anthrax should be vaccinated

A

False

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69
Q

Animals infected with anthrax should be treated with antibiotics

A

True

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70
Q

Virulence factors of anthrax: capsule, toxin, protective

A

True

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71
Q

Virulence factors of anthrax: capsule, toxin, cilia

A

False

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72
Q

Virulence factors of anthrax: capsule, toxin, edema factor

A

True

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73
Q

Virulence factors of anthrax: capsule, toxin, cell wall antigen

A

False

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74
Q

Anthrax is an epidemic disease that rapidly develops

A

False

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75
Q

Anthrax is a quickly spreading, contagious infectious disease

A

False

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76
Q

For lab examination of Anthrax you always have to send a spleen sample

A

False

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77
Q

Animals can only be infected by anthrax on the pasture

A

False

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78
Q

Sheep, cattle, and goats are the most sensitive animals to anthrax infection

A

True

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79
Q

Capsule is a virulence factor of B. anthracis

A

True

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80
Q

Flagella is a virulence factor

A

False

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81
Q

Cell wall antigen is a virulence factor of B. anthraci

A

False

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82
Q

Edema factor is a virulence factor of B. anthraci

A

True

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83
Q

Anthrax spore is a virulence factor

A

False

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84
Q

Anthrax capsule is a virulence factor

A

True

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85
Q

Cattle are infected with B. anthracis mainly from the soil

A

True

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86
Q

Pigs are more susceptible to B. anthracis than sheep

A

False

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87
Q

Anthrax is a per-acute or acute disease in cattle

A

True

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88
Q

Colic is a typical clinical sign of anthrax in horses

A

True

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89
Q

Incomplete clotting of the blood is a typical post mortem lesions of anthrax

A

True

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90
Q

Anthrax can be diagnosed with microscopic examination of blood

A

True

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91
Q

Inactivated vaccines are used for the prevention of anthrax

A

False

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92
Q

The source of anthrax infection on animals is generally the soil

A

True

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93
Q

Anthrax appears generally in the form of a local infection in pigs

A

True

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94
Q

Fever is a typical sign of acute anthrax

A

True

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95
Q

Anthrax can be prevented by using a live vaccine

A

True

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96
Q

Europe is free from Anthrax

A

False

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97
Q

Anthrax is caused by Clostridium chauvoei

A

False

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98
Q

Anthrax is not spreading from animal to animal

A

True

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99
Q

Horses are resistant to anthrax

A

False

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100
Q

Anthrax is an epidemic disease that rapidly develops

A

False

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101
Q

Ruminants are the most sensitive to anthrax

A

True

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102
Q

Animals suffering from anthrax should be treated with antibiotics and hyperimmune sera, they should not be slaughtered

A

True

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103
Q

Sheep, cattle and goat are the most sensitive animals to anthrax infection

A

True

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104
Q

Causative agent of anthrax is spore-forming bacterium in air

A

True

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105
Q

Anthrax spreads in a herd by direct contact

A

False

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106
Q

In order to diagnose anthrax all carcasses have to be dissected

A

False

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107
Q

Anthrax is an acute disease in cattle with high fever

A

True

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108
Q

Swine is highly susceptible in anthrax

A

False

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109
Q

Splenic fever causes suffocation

A

True

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110
Q

Splenic fever in cattle is a per-acute/acute disease

A

True

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111
Q

Splenic fever is similar in every species

A

False

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112
Q

Swine anthrax is generally seen in the form of local lesions

A

True

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113
Q

Carnivorous animals are resistant to Bacillus anthracis

A

False

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114
Q

Incomplete clotting of the blood is a post mortem lesion of anthrax

A

True

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115
Q

Fibrinous pneumonia is a common post mortem lesion of anthrax

A

False

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116
Q

Animals showing clinical signs of anthrax have to be treated with antibiotics immediately

A

True

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117
Q

Horses are more susceptible to Bacillus anthracis than pigs

A

True

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118
Q

Only vaccinated animals are allowed to graze on pastures infected with Bacillus anthracis

A

False

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119
Q

Gastric juice can kill Bacillus anthracis in the meat, so per os infection does not occur in humans

A

False

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120
Q

Clostridium novyi is the causative agent of bacillary hemoglobinuria

A

False

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121
Q

Coccidiosis is a predisposing factor of ulcerative enteritis in poultry

A

True

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122
Q

Ulcerative enteritis can occur in 4-12-week-old chickens

A

True

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123
Q

Clostridium perfringens is the causative agent of ulcerative enteritis in poultry

A

False

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124
Q

Clostridium difficile can be treated with metronidazole

A

True

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125
Q

Clostridium difficile is seen in foal and piglets

A

True

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126
Q

Ulcerative enteritis of poultry is generally prevented with vaccination

A

False

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127
Q

Infectious necrotic hepatitis is caused by Clostridium septicum

A

False

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128
Q

Infectious necrotic hepatitis is caused by Clostridium novyi

A

False

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129
Q

Many Clostridium species have flagella

A

False

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130
Q

Infectious necrotic hepatitis is spread by tick

A

False

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131
Q

Clostridium species are only found in the subtropics

A

False

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132
Q

Clostridium can cause severe contagious diseases

A

False

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133
Q

Clostridium are obligate pathogens

A

False

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134
Q

Infectious necrotic hepatitis is caused by Clostridium novyi type B

A

True

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135
Q

Infectious necrotic hepatitis is found worldwide

A

True

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136
Q

Infectious necrotic hepatitis can be transmitted by liver flukes

A

Flase

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137
Q

Infectious necrotic hepatitis occurs mostly in young sheep

A

False

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138
Q

Malignant edema occurs in ruminants and pigs

A

True

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139
Q

Malignant edema is seen as a consequence of a wound infection

A

True

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140
Q

Malignant edema is an acute fatal disease

A

True

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141
Q

Malignant edema can be treated with antibiotics

A

False

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142
Q

Malignant edema usually develop following a wound infection

A

True

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143
Q

Malignant edema can occur in any warm-blooded animal

A

True

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144
Q

Malignant edema is caused by C. chauvoei

A

False

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145
Q

Once an area is infected with gas gangrene re-occurrence is common

A

True

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146
Q

There is no vaccine to prevent malignant edema

A

False

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147
Q

Malignant edema cannot occur in swine

A

False

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148
Q

Malignant edema can be prevented by using attenuated vaccine

A

False

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149
Q

Malignant edema occurs in cattle mainly after endogenous infection

A

False

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150
Q

Malignant edema usually develop following an endogenous infection

A

False

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151
Q

Malignant edema is well treated with long-term antibiotics therapy

A

False

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152
Q

Malignant edema is well treated with long-term antibiotics therapy

A

False

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153
Q

Malignant edema can be treated with polymyxin

A

False

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154
Q

Malignant edema can be prevented by using anaculture or anatoxin vaccines

A

False

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155
Q

Malignant edema can be well treated with antibiotics over a long period

A

False

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156
Q

Is gas gangrene (malignant oedema) a regional illness

A

False

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157
Q

The lesions of malignant edema are mainly seen in the lungs

A

False

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158
Q

Lamb dysentery occurs in 2-6 weeks old lambs

A

False

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159
Q

For diagnosis of lamb dysentery, the pathogen should be cultured from the intestine

A

True

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160
Q

Pathological symptoms of lamb dysentery can be found in the large intestines

A

False

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161
Q

Lamb dysentery can be prevented by vaccinating pregnant ewes

A

True

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162
Q

Lamb dysentery can be successfully treated with penicillin when clinical signs appear

A

False

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163
Q

Lamb dysentery occurs in a week-old animal

A

True

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164
Q

Lamb dysentery can be diagnosed by culturing the bacteria

A

True

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165
Q

Newborn lambs have to be vaccinated in order to prevent lamb dysentery

A

False

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166
Q

Toxoid vaccines can be used in the prevention of the disease

A

True

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167
Q

In order to diagnose lamb dysentery the agent has to be cultured from intestinal sample

A

False

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168
Q

Infection of lamb by secretion in the milk

A

False

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169
Q

Lamb dysentery occurs in 1-2 weeks old lambs

A

True

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170
Q

Lesions of lamb dysentery can be seen in the large intestine

A

False

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171
Q

Overeating can predispose to struck

A

True

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172
Q

Struck is an acute disease in horses

A

False

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173
Q

Struck is a zoonotic disease

A

False

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174
Q

Struck is caused by Clostridium perfringens C

A

True

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175
Q

Struck is a slow disease of older sheep

A

False

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176
Q

Struck is a worldwide common disease with great economic impact

A

False

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177
Q

Bacillary Hemoglobinuria occurs mainly in cattle

A

True

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178
Q

Bacillary Hemoglobinuria is caused by Clostridium hemolyticum

A

True

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179
Q

Bacillary hemoglobinuria causes severe haemorrhages

A

True

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180
Q

Bacillary hemoglobinuria are caused by infection from the soil

A

True

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181
Q

Bradsot occurs only in suckling lambs

A

False

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182
Q

Bradsot is typically a chronic disease

A

False

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183
Q

Bradsot is caused by Clostridium septicum

A

True

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184
Q

Bradsot is common in the summer out on the pasture

185
Q

Bradsot is an acute disease resulting in sudden death in many cases

186
Q

We can use anaculture strain vaccine against Bradsot

187
Q

Bradsot causes edema of the legs and necrosis

188
Q

Köves disease is caused by Clostridium chavoei

189
Q

Post mortem lesions of bradsot can be seen in the stomach (rennet)

190
Q

Necrotic enteritis of piglets is seen in piglets around weaning

191
Q

Clostridium enterotoxaemia of piglets is caused by Clostridium perfringens C strains

192
Q

Necrotic enteritis of piglets can be prevented by vaccination the sow with anatoxin

193
Q

Pig enterotoxaemia has to be diagnosed by detecting antibodies in the piglets

194
Q

Pig enterotoxaemia causes abdominal contractions in sows

195
Q

Pig enterotoxaemia occurs in 2-4 days old piglets

196
Q

Clostridium enterotoxaemia of piglets occurs in 2-4 days old piglets

197
Q

Clostridium enterotoxaemia of piglets is more frequent in the case of first farrowing sows

198
Q

Lesions of clostridium enterotoxaemia of piglets are seen in the large intestine

199
Q

Mesenteric lymph node is congested in case of pig enterotoxaemia

200
Q

Clostridium enterotoxaemia can be cultured from mesenteric lymph nodes or gut

201
Q

Enteritis in piglets are caused by Clostridium perfringens D

202
Q

Enteritis in piglets can be avoided by anatoxin vaccination

203
Q

Enteritis in piglets cannot be diagnosed by post-mortem, only by bacteriology

204
Q

Pig enterotoxaemia is caused by β-toxin production in 1st week of life

205
Q

Pig enterotoxaemia can cause a high mortality

206
Q

Necrotic enteritis of piglets cannot be diagnosed by isolating the agent from the gut

207
Q

Enterotoxaemia is mainly seen in piglets after weaning

208
Q

Pig enterotoxaemia is not present in Europe.

209
Q

Pig enterotoxaemia cannot be prevented by using vaccines

210
Q

Pulpy kidney disease is seen in piglets in the first week of life

211
Q

Pulpy kidney disease is a worldwide common disease

212
Q

Enterotoxaemia of sheep is also called pulpy kidney disease

213
Q

Pulpy kidney disease is caused by Clostridium perfringens D

214
Q

Cattle are not susceptible to this disease

215
Q

Vaccination are possible against pulpy kidney disease

216
Q

Necrotic enteritis mostly occurs in chicken

217
Q

Waterfowl are not susceptible to necrotic enteritis

218
Q

Necrotic enteritis occurs in 1-3 weeks of age

219
Q

Lesions of ulcerative enteritis are mostly seen in the small intestines

220
Q

Ulcerative enteritis is a common disease in large scale farms

221
Q

Prevention of coccidiosis can help lower the incidence of ulcerative enteritis

222
Q

Gangrenous dermatitis is caused by Clostridium septicum and Clostridium perfringens A

223
Q

Gangrenous dermatitis is caused by obligate pathogens

224
Q

Gangrenous dermatitis causes muscle edema

225
Q

Vaccines are the primary way of prevention of gangrenous dermatitis

226
Q

Clostridium tetani produced endotoxin

227
Q

C. tetani needs anaerobic conditions for propagation

228
Q

Dogs are susceptible to tetanus

229
Q

Tetanus can be prevented with vaccines containing inactivated bacteria

230
Q

Tetanus can cause spasms

231
Q

Spasms are typical clinical signs in tetanus

232
Q

Horses are resistant to tetanus

233
Q

Tetanus can only develop after deep wounds

234
Q

Wounds can predispose to tetanus

235
Q

Tetanus can be prevented by using toxoid vaccines

236
Q

The paralysis usually starts at the place of the wound

237
Q

Clostridium tetani toxin is produced in the feed

238
Q

Horses are most sensitive to tetanus

239
Q

Tetanus can be prevented by anatoxin vaccination

240
Q

Tetanus causes rigid paralysis

241
Q

There is no vaccine for tetanus

242
Q

Dogs have high resistance to tetanus

243
Q

Clostridium tetani produces neurotoxins

244
Q

Clostridium botulinum generally causes wound infection

245
Q

Spasms are the main signs of botulism

246
Q

Clostridium botulinum can produce toxins outside the hosts

247
Q

No characteristic post mortem lesions can be seen in the case of botulism

248
Q

Botulism doesn’t occur in Europe

249
Q

Clostridium botulinum cannot tolerate air at all

250
Q

Botulism usually develops following a wound infection

251
Q

Birds are resistant to botulism

252
Q

Clostridium botulinum propagates in rotten materials

253
Q

Botulism causes flaccid paralysis

254
Q

In Hungary, botulism is seen most commonly in birds

255
Q

Clostridium botulinum spores are extremely resistant to heat

256
Q

In Hungary, botulism occurs in winter and early spring

257
Q

Necrotic foci in the liver are typical post mortem lesions of botulism

258
Q

Botulism is eradicated in Europe

259
Q

Clostridium botulinum can produce toxin, some of which are activated by proteases

260
Q

Botulism is seen mainly during summer

261
Q

Spasms are the typical clinical sign of botulism

262
Q

Focal necrosis in the liver is a typical post mortem lesion of botulism

263
Q

Paralysis is the main sign of botulism

264
Q

Flaccid paralysis is the main clinical sign botulism

265
Q

Toxins of botulism are produced generally in the food

266
Q

Botulism happen generally through wound infection

267
Q

Animals are mostly sensitive to C and D types of Clostridium botulinum

268
Q

Blackleg disease occurs only in ruminants

269
Q

Blackleg can usually be treated with antibiotics successfully

270
Q

Blackleg in cattle is mainly endogenous between 6 months-3 years old

271
Q

The disease caused by Clostridium chauvoei occurs mainly in cattle and sheep

272
Q

The disease caused by Clostridium chauvoei is primarily the result of endogenous infection in cattel

273
Q

Blackleg has four toxins

274
Q

Blackleg can be prevented by using vaccine

275
Q

We use neomycin and polymyxin to treat disease caused by Clostridium chauvoei

276
Q

Blackleg in cattle is mainly endogenous between 2 months-2 years old

277
Q

Blackleg in bovine is caused by wound infections

278
Q

Endotoxins are virulence factors of Staphylococci

279
Q

Some extracellular enzymes are virulence factors of Staphylococci

280
Q

Hemolysins are virulence factors of Staphylococci

281
Q

Some species of Staphylococcus are obligate pathogens

282
Q

Protein A is a virulence factor of Staphylococci

283
Q

Staphylococcus are epiphytes

284
Q

Staphylococcus can produce EC enzymes

285
Q

Staphylococcus can be found on healthy animals’ mucous membranes

286
Q

Staphylococcus are gram negative cocci

287
Q

Coagulase positive Staphylococcus species are less pathogenic than Coagulase negative

288
Q

Staphylococcosis of rabbits is caused by Staphylococcus hyicus

289
Q

Rabbit Staphylococcus can be prevented/treated by vaccination

290
Q

The main sign in rabbit staphylococcosis is bronchopneumonia

291
Q

Staphylococcus aureus subsp. aureus can cause septicemia in day old chicken

292
Q

Staphylococcus aureus subsp. aureus can cause arthritis in poultry

293
Q

Staphylococcus aureus subsp. aureus can cause frequent pneumonia in chicken

294
Q

Staphylococcus aureus subsp. aureus can cause dermatitis in poultry

295
Q

Poultry staphylococcus is caused by S. aureus

296
Q

In poultry staphylococcus there is a septicaemic form, giving generalized disease

297
Q

Poultry staphylococcus can infect eggs

298
Q

Poultry staphylococcus is a rare disease nowadays

299
Q

Rabbit staphylococcus occurs in 4-16 weeks old rabbits

300
Q

Rabbit staphylococcus are caused by S. aureus subsp. piriformes

301
Q

Rabbit staphylococcus causes severe respiratory signs in rabbits

302
Q

We can use antibiotic treatment to cure rabbit staphylococcus

303
Q

Rabbit staphylococcus is an obligate pathogen

304
Q

Overcrowding is a predisposing factor of rabbit staphylococcosis

305
Q

Rabbit staphylococcosis typically occurs in suckling rabbits

306
Q

Pneumonia is a typical sign of rabbit staphylococcosis

307
Q

Staphylococcus aureus subsp. anaerobius is the causative agent of rabbit staphylococcosis

308
Q

Exudative dermatitis is generally seen in fattening pigs

309
Q

Exudative dermatitis can be prevented by attenuated vaccines

310
Q

Exudative dermatitis is sometimes seen on the udder of sows

311
Q

Exudative dermatitis of pigs is caused by Staphylococcus hyicus

312
Q

Exudative dermatitis of pigs is caused by Streptococcus hyicus

313
Q

Itching is the major clinical sign of exudative dermatitis in pigs

314
Q

Exudative skin inflammation occur usually in pigs 1-4-week-old

315
Q

Exudative dermatitis can be treated with antibiotics

316
Q

Vaccination is widely used in order to prevent exudative dermatitis

317
Q

Exudative skin inflammation is caused by Staphylococcus aureus

318
Q

Exudative dermatitis cannot occur in adult pigs

319
Q

Exudative dermatitis can be spread by lice and ticks

320
Q

Morel’s disease is caused by Staphylococcus aureus subsp. aureus

321
Q

Morel’s disease is seen mainly seen in cattle

322
Q

Abscess formation is the main clinical sign of Morel’s disease

323
Q

Morel’s disease occurs in sheep and goats

324
Q

Morel’s disease affects only lymph nodes in the head

325
Q

Morel ́s disease is caused by Streptococcus zooepidemicus

326
Q

In case of several clinical sign in Morel ́s disease, antibiotics should be given through drinking water

327
Q

Morel disease causes lymph node enlargement

328
Q

Morel disease is not a zoonosis

329
Q

Morel’s disease is caused by Staphylococcus hyicus

330
Q

Ataxia is an important sign of the Morel ́s disease

331
Q

In the case of Morel disease per oral antibiotic treatment is used

332
Q

Morel’s disease causes subcutaneous abscesses.

333
Q

Streptococcus can be divided according to their antigens

334
Q

Streptococcus are epiphytes

335
Q

Streptococcus are obligate aerobic

336
Q

Streptococcus suis can cause generalized septicemia in 1-4 weeks old piglets

337
Q

Calcium deficiency can predispose suckling piglets to streptococcosis

338
Q

Neurological signs are frequent in the case of porcine streptococcosis

339
Q

Abscesses in the liver are frequently seen in the case of porcine streptococcosis

340
Q

Streptococcus in swine can be caused by S. suis serotype II

341
Q

Streptococcus in swine can cause acute purulent encephalomyelitis

342
Q

All ages are susceptible in case of S. suis

343
Q

S. porcinus can cause disease and is an epiphyte

344
Q

S. porcinus is a contagious disease

345
Q

The causative agent of strangles is Streptococcus equi subsp. equi

346
Q

The causative agent of strangles has to be introduced in the herd

347
Q

Strangles is treated with

348
Q

Strangles can be successfully treated with penicillin

349
Q

When abscesses develop in strangles, the prognosis is poor

350
Q

The causative agent of strangles is obligate pathogen

351
Q

In strangles, morbidity is high

352
Q

Fever is an important sign of strangles

353
Q

Strangles has disappeared, due to extensive vaccination of the foals

354
Q

Mortality of strangles is high

355
Q

Strangles disappeared because of widespread vaccination of the foals

356
Q

Carriage of the agent of strangles can be confirmed by isolation from the tonsils

357
Q

Strangles has a morbidity of 100 %

358
Q

Strangles pathogen is usually present on mucous membranes

359
Q

The causative agent of strangles are an epiphyte

360
Q

Prognosis of strangles is bad if an abscess rupture

361
Q

Strangles can be treated with penicillin

362
Q

For the occurrence of strangles, predisposing factors are needed

363
Q

Strangles can be diagnosed by serology

364
Q

The causative agent of strangles is present in all horses

365
Q

Strangles is mainly seen in horses aged 6 months-21⁄2 years

366
Q

Penicillin is an effective antibiotic for the treatment of strangles

367
Q

The agent of strangles is carried by the majority of horses on the mucous membranes

368
Q

Diarrhea is a typical sign of strangles

369
Q

Animals with strangles generally do not have fever

370
Q

Erysipelothrix rhusiopathiae is facultative pathogen

371
Q

Turkeys are susceptible to Erysipelothrix rhusiopathiae

372
Q

There is a serotype-specific protection against swine erysipelas

373
Q

Erysipelas often appears in a septicemia form

374
Q

Erysipelas has to be introduced into a herd

375
Q

In erysipelas, small vessels in the skin become inflamed, causing erythema

376
Q

Erysipelothrix rhusiopathiae is a facultative pathogenic bacterium

377
Q

In the case of acute erysipelas high fever is an important sign

378
Q

The swine erysipelas bacterium is an obligate pathogen

379
Q

Erysipelas can be prevented by inactivated vaccine

380
Q

The main sign in acute erysipelas is fever

381
Q

Geese are susceptible to Erysipelothrix rhusiopathiae

382
Q

Acute erysipelas causes

383
Q

Endocarditis is seen in acute erysipelas

384
Q

Erysipelothrix rhusiopathiae is not resistant, it cannot survive in the environment

385
Q

Some extracellular enzymes are virulence factors of Erysipelothrix rhusiopathiae

386
Q

Erysipelas can be well treated by penicillin

387
Q

Warm weather can predispose pigs to erysipelas

388
Q

The causative agent of swine erysipelas is an epiphyte

389
Q

“Strong” erysipelas comes together with mild fever

390
Q

There is a serotype specific protection in case of erysipelas

391
Q

Listeriosis causes septicemia in lambs

392
Q

Listeriosis mainly occurs at the end of winter

393
Q

Listeriosis causes mainly abortion in cattle

394
Q

Listeriosis can be isolated from the brain stem

395
Q

Overcrowding is a predisposing factor of listeriosis

396
Q

Listeria can be found in soil

397
Q

Abortion is the most frequent clinical sign in bovine listeriosis

398
Q

Listeria are not resistant, they cannot survive in the environment

399
Q

Listeriosis can be a septicemic disease

400
Q

Pneumonia is a frequent clinical sign of listeriosis

401
Q

Listeriosis is a zoonotic disease

402
Q

Silage can contain large

403
Q

Encephalitis is the most frequent clinical sign of listeriosis in sheep

404
Q

Listeriosis is the most common neurological disease in cattle

405
Q

Listeriosis occurs more frequently during the summer, at time of silage-making

406
Q

In the case of listeriosis of cattle, signs of the nervous system are the most frequently seen

407
Q

Listeriosis occurs only in tropical areas

408
Q

Neurological symptoms are the most common clinical sign of listeriosis in cow

409
Q

Listeriosis occurs in the summer

410
Q

Listeriosis occurs only in

411
Q

Main symptoms of listeriosis in sheep is encephalitis, abortion and septicaemia

412
Q

In cases with encephalitis, abscesses can be found in the medulla oblongata

413
Q

Phospholipase D is a virulence factor of C. pseudo tuberculosis

414
Q

In Corynebacterium pseudotuberculosis edema of the chest is common

415
Q

Corynebacterium pseudotuberculosis can be transmitted between goats and horses

416
Q

Pseudotuberculosis does not occur in Hungary

417
Q

In pseudotuberculosis, only submandibular lymph nodes of sheep are affected

418
Q

In pseudotuberculosis edema of the limbs is common

419
Q

Corynebacterium pseudotuberculosis causes caseous lymphadenitis

420
Q

Corynebacterium pseudotuberculosis forms due to dipping of sheep

421
Q

Corynebacterium pseudotuberculosis is present in tropical regions, not in cold environemnts

422
Q

Caseous lymphadenitis is caused by Corynebacterium pseudotuberculosis

423
Q

Caseous lymphangitis is nitrate negative

424
Q

Caseous lymphadenitis occurs only in young lambs

425
Q

Corynebacterium pseudotuberculosis causes caseous lymphadenitis in goats

426
Q

Abortion can be a clinical sign of caseous lymphangitis

427
Q

Caseous lymphadenitis of sheep occur only in the tropics

428
Q

Caseous lymphangitis is seen mostly in sheep

429
Q

Caseous lymphangitis does not occur in Hungary

430
Q

Cross section of the lymph node with caseous lymphangitis shows an onion-like pattern

431
Q

Caseous lymphangitis can cause abortion in waves

432
Q

Vaccination can be used in prevention against caseous lymphangitis

433
Q

Abscess formation in the lymph nodes is typical in the case of caseous lymphadenitis in goats

434
Q

Caseous lymphadenitis is caused by Corynebacterium equi

435
Q

Sheep with caseous lymphadenitis can infect horses

436
Q

The agent of caseous lymphadenitis causes bacteremia

437
Q

Pyelonephritis ooccurs mostly some weeks after parturition

438
Q

Pyelonephritis is caused by C. renale, C. pilosum, C. cystiditis

439
Q

Pyelonephritis mostly occurs in horses

440
Q

Pyelonephritis ooccurs mostly some weeks after parturition

441
Q

Pyelonephritis can cause positive pain probes of skin area above spine

442
Q

Corynebacterium renale is the causative agent of bovine purulent nephritis

443
Q

Penicillin is used for the treatment of bovine purulent nephritis

444
Q

Hematuria can occur in bovine purulent nephritis

445
Q

Bovine purulent nephritis is mainly seen in suckling calves

446
Q

Equine ulcerative lymphadenitis is caused by C. pseudotuberculosis

447
Q

Equine ulcerative lymphadenitis is an acute disease with high fever

448
Q

Equine ulcerative lymphadenitis occurs only in tropical countries

449
Q

Corynebacterium pseudotuberculosis causes ulcerative lymphangitis in horses

450
Q

Ulcerative lymphangitis is caused by nitrate negative C. pseudotuberculosis

451
Q

Ulcerative lymphangitis does not occur in Hungary

452
Q

Causative agent of ulcerative lymphangitis in horses is C. pseudotuberculosis

453
Q

Symptoms of ulcerative lymphangitis in horses are seen in pectoral region, legs and ventral abdomen

454
Q

Ulcerative lymphangitis may evolve during navel infection

455
Q

Animals with clinical signs of ulcerative lymphangitis have good prognosis

456
Q

Best way of prevention for ulcerative lymphangitis is toxoid vaccine

457
Q

Clinical signs of equine ulcerative lymphangitis can be seen in the lymphatic vessels

458
Q

Equine ulcerative lymphangitis is a chronic disease of horses

459
Q

Equine ulcerative lymphangitis is not zoonotic