Major trauma Flashcards

1
Q

What forms the basis for the standard of trauma care in the UK?

A

Advanced trauma life support

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2
Q

What forms the initial assessment of a major trauma patient?

A

Primary survey + resuscitation of vital organs

A-E assessment

The pririties of the primary survery are always the same, regardless of what has caused the injury

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3
Q

What are the 5 key components of the primary survey in major trauma?

A
  1. Airway mantenance + c-spine protection
  2. Breathing + ventilation
  3. Circulation + haemorrhage control
  4. Disability (neurological status)
  5. Exposure and environmental control: completely undress patient whilst avoiding hypothermia

>>This is generally not done as a sequence, if staffing and direction permits, all done simultaneously<<

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4
Q

Important to remember whilst doing a primary survey?

A

Talk to your patient

Deal with any encountered problem before moving on ‘FIND the bleeding STOP the bleeding’

After any intervention, return to the start of the primary survey

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5
Q

What does airway maintenance with c-spine control involve?

A

All major trauma patients have an unstable cervical spine fracture until proven otherwise - high cervical injury can lead to loss of respiratory drive

Ensure airway is patent

Simple airway maneuvres: jaw thrust, chin lift

Suction should be available

Be aware of obstructions: blood, swelling, obstruction due to injury

Crepitus: crunchy feeling in neck suggests direct laryngeal injury

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6
Q

What is an oropharyngeal airway?

A

Rigid plastic tube that sits along the top of the mouth and ends at the base of the tongue

Prevents tongue occluding epiglottis in patients with reduced GCS

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7
Q

Patient gags when you insert oropharyngeal airway - what to do?

A

The gagging indicaes the patient will not tolerate to OP airway - remove it and try a nasopharyngeal airway

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8
Q

Which airway adjunct is used for more alert patients?

A

Nasopharyngeal

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9
Q

What is a nasopharyngeal airway?

A

Flexible rubber tube inserted through the nose. ends at the base of the tongue

Prevents tongue covering epiglottis

Better tolerated that OP airway

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10
Q

Why are patients with a reduced conscious level at higer risk of airway obstruction?

A

Relaxation of smooth muscle causing:

  • occlusion of the oropharynx by the tongue
  • occlusion of the laryngopharynx by the epiglottis
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11
Q

Causes of airway obstruction

A

Lumen: vomit, secretions, blood, foreign body

In wall: infection e.g. tonsilitis, epiglottitis, trauma to larynx, tumour, anaphylaxis, angioedema

From outside airway: penetrating neck injury, tumour, oesophageal foreign body

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12
Q

How is airway compromise identified?

A

Conscious patients + airway compromise

  • Patients usually sit up and look distressed

Look for: swollen tongue, sooty sputum (thermal injury), neck haematoma, rashes (anaphylaxis), wheeze/ laboured breathing (asthma), facial fractures, crepitus (laryngeal trauma)

Unconscious patients + airway compromise

Examine for:

  • snoring/ added airway noises (indicating partial airway obstruction), abnormal chest and abdo wall movement (suggesting obstruction), lack of fogging of oxygen mask
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13
Q

What are the simple airway manoeuvres?

A
  1. Suction: vomit, blood, secretions
  2. Chin-lift
  3. Place pillow under patients head (unless obese) - flexes neck
  4. Jaw thrust - use this on its own if you suspect your patient has a c-spine injury
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14
Q

What are the simple airway adjuncts?

A

Oropharyngeal (OP) and nasopharyngeal (NP) adjuncts

  • Designed to address airway obstruction and free the airway practitioner
  • Both generally only tolerate by unconscious patients esp. OP

- If patient is tolerating airway adjuncts consider the need for intubation

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15
Q

How to insert an OP airway adjunct

A
  • Insert OPA ‘upside down’
  • Twist 180 once inserted halfway (behind the tongue)
  • The flanged front end should sit just in front of the teeth (See image)

If this causes vomiting, gagging or laryngospasm - remove immediately

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16
Q

When should NP aoirways absolutely be avoided?

A

If patient has facial injuries - particularly mid-face as there is risk the tube can enter the brain

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17
Q

Once a patent airway is secured, meaning air can enter and exit the lungs, what question should we ask?

A

Does the patient require

a) passive ventilation: oxygen mask
b) assisted ventilation

Determined by: depth of chest wall movement, rate of chest wall movement, coordination of breaths, o2 sats, pco2 via ABG

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18
Q

What is done if ventilation is needed?

A

Get correct size face mask and a self inflating bag

Check airway doesn’t need suctioning

Apply mask

Jaw thrust

Squeeze bag firmly at 10 breaths per min

*Ideal if two people do this: one does bag squeeze, other does jaw thrust

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19
Q

If a patient needs ventilating and they wear dentures - what do we do?

A

Keep dentures in or pack cheeky with gauze

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20
Q

Why might bag-mask ventilation be difficult?

A

Dentures:keep them in or put gauze in cheeks

Unstable fractures: consider early intubation

Beard: apply gel to improve seal

Stiff/ immobilised neck: no option availble - do not force elderly patients neck

COPD/ astha: aggressive medical therapy

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21
Q

You’ve tried to ventilate the patient but failed - next step?

A

Call for sensior airway help - patient may need intubating

Optimise patient positioning

Try 2x NPA + OPA

Try a laryngeal mask airway (type of supraglottic airway device)

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22
Q

How can we assess breathing?

A

Resp rate

Breath sounds

Chest movement

Air entry

Sats

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23
Q

Problem with breathing - what could the cause be?

A

An airway problem: air cannot get in despite efforts

A ventilation problem: problem with the process of breathing itself

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24
Q

How can circulation be assessed

A

BP

Skin colour + temperature

Pulse rate and character

Cap refill

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25
Q

Patient is haemorrhaging - what do we do to restore volume?

A

Blood products, packed red cells

  • Used to be cyrstalloid but this is no longer recommended
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26
Q

Important to consider when assessing circulation in a young/ healthy patient?

A

They often have a lot of physiological reserve and maintain their BP before suddently dropping off

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27
Q

Management of poor circulation

A

2 Large bore cannulas to allow for fluid resus

Constantly reassess

Blood not crystalloid

Find a bleed, stop a bleed

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28
Q

Disability assessment

A

Baseline neurological evaluation

GCS/ AVPU

Pupillary response for ICP

If GCS <8 - intubate

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29
Q

GCS <8?

A

INTUBATE 🫁 😵

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30
Q

ABCD

Exposure - what to do?

A

Completely undress patient

Look for any other injuries

Avoid hypothermia

Head to toe exam

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31
Q

What are the adjuncts to the primary survey?

A
  • ECG
  • Vital sgns
  • ABGs
  • Pulse oximeter
  • Urinary/ gastric catheters
  • Urinary output > sign of end organ damage > insert catheter to monitor (beware of urethral injuries)

FAST scan to look for bleeding in: hepatorenal recess (Morrison’s pouch), splenorenal recess, pelvis, paricardium

CT: done within 30 mins

Analgesia

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32
Q

Main mode of investigation in major trauma?

A

CT - guidelines say this should be avai;able within 30mins of patient arriving

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33
Q

What is a secondary survey?

A

Systematic review of the back and front of the patient looking for all injuries

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34
Q

What is involved in the history taking of a secondary survey?

A

AMPLE

Allergies: do they have any allergies?

Medication: are they taking any?

Past medical hx: any medical conditions, epilepsy, heart disease, injuries, surgery

Last meal: when did they last eat and drink

Event/ environment: what happened and where, ask people nearby

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35
Q

Components of the secondary survey

A

Head

Neuro: GCS + pupil response - compare w/ primary survey, CN assessment, fundoscopy, evaluate for spinal cord injury

Scalp: palpate for haematoma/ fracture, look for wounds

Maxillofacial: signs of fracture/ crepitus, basal skull fracture

Neck: neck bruit (carotid dissection), palpate spine

Thorax

Respiratory exam

Rib fractures (if first rib is fractures, suspect vascular damage)

Life threatening injuries: ATOM PD (aortic, tracheal, oesophageal, myocardial, pulmonary, diaphragmatic) - see subsequent cards

Abdomen

Abdo examination

  • most common site of significant bleeding from blunt trauma = spleen
  • most common stab wound site = liver

If any trauma to abdomen - CT regardless of whether patient is stable

Pelvis

Fracture: pain on palpation, unequal leg length, instability

Always suspect palevic injury when inability to void

Lower GU tract: bladder/ urehtral injuries

Genital region: vagina (blood/ lacerations), rectum, perineum

Extremities

Full MSK examination

Long bones have potential for significant blood loss

Neurovascular impairment

Spinal column

  • Full in-line immobilisation if: under influence of drugs/ alcohol/ confused, spinal pain, hand/ foot weakness/ numbness, priapism, hx of spinal problems

😳 **Don’t let embarrassment stop you from checking everywhere** 😳

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36
Q

Aortic injuries

A

Tears of aorta or pulmonary arteries associated with blunt or deceleration injuries e.g. car crash/ fall from height

Aorta is fixed in position at three points

  1. Aortic valve
  2. Ligamentum arteriosum (most common site of shearing injury)
  3. Dipahragm

>> Sudden deceleration causing shearing at attachment points <<

Shearing of the ligamentum arteriosum, which attaches to pulmonary artery, is fatal in 90%

Management: Surgical repair, control hypertension (SBP no more than 110), control tachycardia (labetalol)

37
Q

What is the investigation of choice in aortic injuries?

A

Contrast CT thorax

38
Q

What is the most reliable sign of aortic injury on CXR?

A

Widened mediastinum

Other:

  • Fractures of 1st and 2nd rib
  • Obliteration of aortic knob
  • Deviation of trachea
  • Presence of pleural cap
39
Q

Tracheal injuries

A

Uncommon but can be caused by penetrating/ blunt trauma

Can result in free air in neck, chest wall, mediastinum

Bronchoscopy can confirm diagnosis

40
Q

Oesophageal injuries

A

Occur following penetrating & (rarely) blunt injury

May cause gastric contents to be forced into oesophagus causing tearing and leaking of acid into mediastinum or pleural space - can cause mediastinitis which rapidly causes sepsis

Clinical picture is identical to post-emetic oesophageal rupture (Boerhaave syndrome)

41
Q

Most commonly undiagnosed fatal thoracic injury?

A

Blunt myocardial injury (contusion)

Occurs when there is direct compression of the heart due to blunt trauma or rapid deceleration

Often associated with sternal fractures

Diagnosis: troponin, ECG changes, echo

Normal ECG on admission virtually eliminates this problem

42
Q

Pulmonary contusion

A

Injury to lung tissue associated with blunt trauma - a bruise of the lung

Leads to blood and oedema within alveoli

SUSPECT IN ALL WITH FLAIL CHEST

Causes impaired gas exchange

Clinical picture: increasing resp. distress + hypoxia

Diagnosis: CXR or CT

Management: supportive while contusion resolves, if severe patients need early intubation and ventilation

Important not to fluid overload because increased capillary leakage can lead to pulmonary oedema

43
Q

Which is the most commonly injured abdominal organ?

A

Spleen

44
Q

What is crush syndrome?

A

Systemic manifestation of muscle cell damage/ rhabdomyolysis

Causes: direct injuries, severe burns, compartment syndrome, myositis, grand mal fitting

Usually due to crush injuries >> tissue ischaemia >> necrosis + rhamdomyloysis >> cellular components re-enter circulation e.g. myoglobin, K+ uric acid and CK >> arrhythmia, AKI, metabolic acidosis

Management: fluids, dialysis if renal failure occurs

45
Q

Hard and soft signs of arterial injury

A

Hard signs = immediate transfer to theatre >90% risk of arterial injury

  1. Pulsatile haemorrhage
  2. Rapidly expanding haematoma
  3. Palpable thrill/ bruit
  4. Absent distal pulses/ signs on ischaemia (palor, paraesthesia, paralysis, perishingly cold)

Soft signs = further investigation 30% risk arterial injury

  1. Hx of arterial bleeding which has since ceased
  2. Small, non-expanding haematoma
  3. Subjectively decreased pulse
  4. Unexplained hypotension
  5. Neurologic deficit originating in a nerve adjacent to a named artery
  6. High risk orthopaedic injury e.g. fracture, doslocation, penetration
46
Q

What are the three peaks of trauma

A
  1. Seconds - mins: e.g. laceration of brain
  2. Mins - hrs: e.g. haemothorax
  3. Days - weeks: e.g. sepsis
47
Q

Discuss tension pneumothorax

A

Air trapped in the pleural space under positive pressure due to one way valve

>> Compresses lung

>> Impaired venous return

>> Trachea deviates away

>> Heart can be compressed

Features: chest pain + respiratory compromise, tachypnoea, raised JVP

Examination: decreased breath sounds on affected side, hyper-resonance and tracheal deviation away from affected side

Management: needle thoracentesis - 2nd intercostal space, mid-clavicular line OR if skills permit - finger thoracostomy in 5th intercostal space >> both followed by chest drain

Pitfalls of thoracentesis: tends to get overused, lack of hiss/ bubble used as evidence of no tension pneumothorax, standard cannula might not reach pleural space, can cause pneumothorax itself

48
Q

What is an open pneumothorax?

A

AKA a communicating pneumothorax or sucking chest wound

Hole in the chest

Rare - usually due to shot gun injury

Unlikely to be missed clinically

As patient breathes in, the hole in the chest competes with the trachea for delivery of air

Diagnosis: resp distress, bubbling wound on expiration

Management: high flow oxygen, sterile dressing taped down at 3 sides to allow air out but not in, insert drain, repair wound

49
Q

What is a massive haemothorax?

A

Blood in pleural space with volume of >1500mL or 1/3 patients blood volume

  • Uncommon
  • Due to blunt/ penetrating trauma
  • Causes hypovolaemic shock and decreased ventilation

Features: shock, dullness to percussion, reduced/ absent breath sounds, can be seen on x-ray

Management: restore circulating volume, drain blood (AFTER CIRCULATING VOLUME RESTORED), thoracotomy may be required if bleeding is >1500ml or ongoing loss of >200ml every 2hrs

50
Q

Flail chest

A

Series of rib fractures in >1 place causing a section of free-floating chest wall

>> Minumum of 2 fractures in 2 ribs <<

Fairly common

Significant force is required so look for other injuries too

Potential source of significant haemorrhage

Diagnosis: abnormal chest movement - inwards on expiration & outwards on inspiration , respiratory distress, painful (very)

Investigations: O2 sats, ABG, CXR

Management: high flow o2, manage any haem thoraces/ pneum thoraces, pain management is important, surgery to stabilise fractures rarely indicated

51
Q

Features of sternal fracture

A

Frequently occurs following RTAs due to steering wheel/ seatbelt

Anterior chest pain + localised tenderness over sternum + bruising/ swelling

Investigations: ECG to rule out arrhythmias, STEMI or myocardial contusion

If ECG changes, check tropnonin

Request CXR and lateral sternal x-ray

Management: admit ig signs of myocardial contusion/ other injuries

Ig sternal fracture occurred in isolation, ECG normal and no other injuries consider discharge with NSAIDs+ co-codamil + GP follow up

52
Q

Cardiac tamponade

A

Collection of fluid in pericadial sac causing haemodynamic compromise >> compression of heart >> inadequate filling >> reduced CO

Exclude/ confirm using FAST scan

50-200ml blood is enough to cause pulseless electrical activity cardiac arrest in which the ECG shows a rhythm which should produce a pulse but it doesn’t

Diagnosis: Beck’s triad (low BP, muffled heart sounds, raised JVP), Kussmaul’s sign (JVP rises on inspiration instead of falls), FAST scan

Management: thoracotomy is preferred choice as blood is oftn clottes so has to be scooped out, consider pericardiocentesis if patient is peri-arrest to buy time

53
Q

What is Kussmaul sign?

A

JVP rises on inspiration instead of falling as would be appropriate

This occurs when there is fluid in the pericardial sac that impaires venous return and inspiration causes further compression of the heart and blood shoots back up jugular vein

54
Q

What is Beck’s triad?

A

Seen in cardiac tamponade 🫀🫀🫀

  1. Low BP
  2. Muffled heart sounds
  3. Raised JVP

*Only a small number of cases of cardiac tamponade present with all 3 features*

55
Q

What is used at the bed-side to diagnose cardiac tamponade?

A

FAST scan

Focussed assessment with sonography for trauma

56
Q

Which 4 areas is a FAST scan used to look at?

A
  1. Pericardium
  2. Right flank: perihepatic view/ Morrison’s pouch - DONE FIRST AS BLOOD COLLECTS HERE FIRST
  3. Left flank: perisplenic
  4. Pelvis

Blood appears as a black, echo free area

Visible free fluid in the abdomen implies a minimum volume of ~500mL

57
Q

Can a patient have a normal ECG in cases of cardiac contusion?

A

No - normal ECG effectively rules out cardiac contusion

58
Q

What is ALI and ARDS?

A

Complication of trauma

ALI = acute lung injury

ARDS = acute respiratory distress syndrome

Can be caused by trauma

Features: acute onset (within 1 week), bilateral opacities on CXR, severe hypoxaemia

59
Q

Pathophysiology of ARDS

A

Interstitial fluid in lungs despite normal pulmonary arterial and venous pressures

Fluid causes lung stiffness which impaires gas exchange >> hypoxia

Fluid can also promote cytokine release which can promote fibrosis

Stages

  1. Exudative: oedema
  2. Repair and fibrotic change >> causes loss of elasticity, emphysema formation and damage to vasculature

Common after blunt trauma affecting the thorax

Other causes: Sepsis (most common), acute pancreatitis, pneumonia, aspiration of gastric contents , fat embolism, inhalation injury

60
Q

How might ARDS present?

A

1st sign is often an unexplained tachypnoea leading to hypoxaemia, central cyanosis and dyspnoea

Fine crackles heard throughout lungs

61
Q

Management of ARDS

A

ICU: supportive ventilation with PEEP

Prone position

Fluid resus + diuretics

NO to improve ventilation of unaffected lung

62
Q

Coagulopathy following trauma

A

Following trauma, coagulation, anti-coagulation and fibrinolysis are disproportionately affected >> impaired haemostasis

Clinical features: Bleeding, prothrombotic state, disseminated intravascular coagulation

Management: control primary cause, manage clotting abnormalities, multi-organ support

63
Q

Fat embolisation following trauma

A

Fat particles within blood stream

Symptoms generally begin within 24hrs

Systemic manifestations

Respiratory distress: inflammatory response in lungs - tachycardia, tachypnoea, hypoxia

Neurological problems: due to hypoxia/ emboli lodging in cerebral circulation - mild headache >> comatose

Petechial rash: emboli promotes local platelet aggregation >> widespread inflammatory reaction in the skin, also seen in eyes + retina

>> Can also cause renal failure

Aetiology: long bone fractures, massive soft tissue injury, severe burns, orthopaedic procedures

64
Q

Skin rash, tachypnoea + hypoxia + tachycardia, headache/ confusion following trauma - thoughts?

A

Fat emboli

65
Q

Investigations for fat emboli following trauma

A

Fat in: urine + sputum

CXR: snow storm but often normal

May show up on CTPA

  • Usually clinicians rely on physical examination
66
Q

What are Gurd’s criteria?

A

Used to assess presence of fat emboli

Major criteria: petechiae, CNS depression, pulmonary oedema

Minor criteria: tachycardia, low grade temperature, retinal emboli, fat in urine/ sputum, thrombocytopenia, increased ESR

>>Diagnosis requires at least 1 major criteria + at least 4 minor criteria<<

67
Q

How is are fat emboli managed?

A

Supportive

  • Immobilise fracture
  • Optimise oxgenation
  • DVT prophylaxis

Mortality = 5-15%

Persistent neurological deficits may occur

68
Q

How soon after trauma would you expect to see signs of fat emboli?

A

24-72 hrs

69
Q

What % of trauma patients develop PE/ DVT?

A

50%

3rd most common cause of death in the 24hrs following major trauma

70
Q

When to suspect major trauma

A

High speed collisions, vehicle ejection, rollover and prolonged extraction

Death of another individual in same collision

Pedestrians thrown uo or run over by a vehicle

Falls of >2m

71
Q

When would you suspect c-spine injury?

A

Neck pain

Loss of consciousness

Assume all major trauma patients has a c-spine injury until proven otherwise

72
Q

Use of TXA in major trauma

A

Give 1g IV over 10mins within 3hr of injury

Followed by 1g IVI over 8hrs

73
Q

What are the phases of treatment of patients with major trauma?

A
  1. Primary survey
  2. Resuscitation phase
  3. Secondary survey
  4. Definitive care phase

Key feature = re-evaluation throughout

74
Q

We start with airway management unless…

A

Catastrophic haemorrhage

75
Q

What is the resuscitation phase?

A

Treatment continues for the problems identified during the primary survery

Adjuncts to primary survey e.g. airway adjunct, chest drain, urinary catheter

Sometimes surgery is required for haemorrhage control before the secondary survey is done

76
Q

What is the definitive care phase?

A

Early management of all injuries is addressed e.g. fracture stabilisation and emergency operative intervention

77
Q

Important to have available, regarding airway management, when treating seriously injured patient

A
  • O2
  • Suction
  • Airway equipment
  • Senior ED/ ICU anaesthetic help if serious airway problem arrives/ is expected
78
Q

How can a patient’s breathing be assessed

A

Talk to patient: lucid reply shows airway is patent, patient is breathing and blood is reaching brain

Look and listen to breathing

Partial obstruction: gurgling, snoring, stridor

Total obstruction: patient trying to breathe but unable, paradoxical chest movements but no breath sounds

79
Q

Managing the obstructed airway

A
  1. Look in mouth for obstruction and remove with suction or Magill’s forceps
  2. Basic airway manoeuvres - lift chin and jaw thrust but do not flex or extend neck
  3. After any intervention look, listen and feel to reassess airway
  4. Use airway adjunct (OP/NP as appropriate)
    5a. Airway patent + patient breathing: 15L o2 via non-rebreathe mask
    5b. Airway pateint but breathing inadequate: ventilate with o2 bag and mask and prepare for tracheal intubation (ideally a 2 person job)
80
Q

How is tracheal intubation confirmed?

A
  1. See tube pass through vocal cords
  2. Observe symmetrical chest movement
  3. Listen over axillae for symmetrical chest movement
  4. Confirm placement with end-tidal CO2 monitoring
81
Q

Discuss surgical airways

A

Needed if the airway is obstructed by trauma, oedema or infection and tracheal intubation is not possible

1. Surgical cricothyroidotomy

  • Feel thyroid and cricoid cartilages and cricothyroid membrane between them
  • Clean area, give LA
  • Hold thyroid cartilage and make transverse incision
  • Slide a bougie tube into trachea, remove scalpel and railroad a 6.0mm cuffed tracheal tube into trachea
  • Remove bougie tube, inflate cuff and connect tube to a catheter mount and ventilation bag
  • Ventilate with o2 and secure tracheal tube
  • Examine chest and check for adequate ventilation
    2. Needle cricithyroidotomy - temporary measure while preparing for surgical cricothyroidotomy
  • Needle placed through cricoid membrane at a 45 degree angle with syringe attached
  • Aspirate via syringe whilst advancing needle - aspiration of air confirmes you are in trachea - remove needle and keep cannula in
  • Connect cannula to o2 at 15L/min with a side port/ hole in tube: occlude port/ hole for 1 second to allow air in, open for 4 seconds to allow air out
  • Can be tolerate/ function for 45mins - proceed immediately to definitive airway
82
Q

Site for insertion of surgical airway

A

Cricothyroid membrane/ ligament

Between thyroid cartilage and cricothyroid cartilage

83
Q

Site for needle decompression in tension pneumothorax

A

2nd intercostal space, midclavcular line

84
Q

Which side of the diaphragm most commonly ruptures?

A

Left (75%)

Liver tends to protect the right diaphragm

85
Q

Patient has a major diaphragmatic rupture - what do you do?

A

Usually associated with herniation of stomach contents

Call surgeon and an anaesthetist as patient requires urgent intubation and IPPV (intermittent positive pressure ventilation)

Can result in abdominal contents in thorax

86
Q

Chest drain insertion

A

Give IV opioids if patient conscious

Abduct arm fully, sterile gown and goggles/ face shield, clean skin, find 5th inercostal space mid axillary line

Give LA (lidocaine + adrenaline), make 2-3cm incision

Use blunt dissection with forceps to open tissues down to pleural space

Puncture pleura with forceps, insert gloved finger into cavity to ensure there are no adhesions, insert drain and connect to underwater seal

Suture drain in place and cover with dressive

Ensure underwater seal is swinging in the tube with respiration

Listen for air entry

>Refer to surgeon if drains >1500mL blood or 200mL every hr for 2hr

87
Q

What are the reversible causes of cardiac arrest?

A

Hypoxia: secure airway and ventilate

Hypovolaemia: give blood and plasma e.g. 4U O-neg warmed packed RBCs stat

Tension pneumothorax: perform bilateral thoracostomies

Cardiac tamponade: if fluid seen on FAST scan do a clam shell thoracotomy - pericardiocentesis often fails because clots form in pericardial sac

88
Q

Advantages of FAST scan

A
  • Can be done in ED
  • Quick: 2-3mins
  • Non-invasive
  • Repeatable
89
Q

Disadvantages of FAST scan

A
  • Operator dependant
  • Doesn’t define injured organ - only presence of blood or fluid in abdomen or pericardium
  • Looks at 4 areas only