Lecture 22 3/25/25 Flashcards

1
Q

What are the characteristics of rumen chloride measurement as a diagnostic tool for vagal indigestion?

A

-normal rumen chloride levels is < 30 mEq/L
-types 1 and 2 have normal chloride
-type 3 has chloride levels > 30 mEq/L due to Cl sequestration

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2
Q

Why is there elevated rumen chloride in type 3 vagal indigestion?

A

-abomasum is not functioning properly; HCl produced in abomasum cannot move into the small intestine for absorption
-HCl backs up into the rumen fluid rather than entering the bloodstream; leads to high rumen chloride and low blood chloride

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3
Q

What are the clin path findings in vagal indigestion?

A

-dehydration
-inflammatory leukogram
-lymphocytosis if patient has lymphoma

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4
Q

What is the most common metabolic derangement in adult cattle?

A

hypochloremic, hypokalemic metabolic alkalosis

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5
Q

What are the characteristics of hypochloremic metabolic alkalosis?

A

-seen in mature, dehydrated bovids
-decreased GI motility and type 3 vagal indigestion can lead to low blood chloride
-body expands bicarb. production to make up for low blood Cl

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6
Q

Which conditions lead to development of acidosis rather than alkalosis?

A

-late stage strangulating lesions
-enteritis
-young animals

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7
Q

How is vagal indigestion treated?

A

*based on inciting cause
-relieve distention; kingman tube or rumenotomy
-prokinetic agents
-antibiotics if infectious cause
-restrict water
-good quality grass hay
-fistulation long-term

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8
Q

What are the characteristics of rumen putrefaction?

A

-abnormal accumulation of milk in rumen of calves
-milk becomes putrefied and promotes gram-pos. anaerobic overgrowth
-primary pathogen is Clostridium perfringens

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9
Q

How can milk get into the rumen?

A

-failure of esophageal-omasal groove
-abomasal reflux due to overfeeding or inflammation
-feeding milk to calves older than 4 months of age

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10
Q

What are the clinical signs of rumen putrefaction?

A

-mild bloat
-pot belly
-unthriftiness
-rough hair coat
-poor appetite
-signs associated with feedings

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11
Q

What are the findings on rumen fluid analysis in rumen putrefaction?

A

-alkaline pH (greater than 7)
-sour, rancid milk odor

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12
Q

What is the treatment for rumen putrefaction?

A

-siphon off the rumen
-oral antibiotics (PPG)
-transfaunation
-slow the rate of milk feeding; small, frequent meals
-wean

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13
Q

What is ruminal tympany?

A

accumulation of gas or froth in the rumen

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14
Q

What can cause free gas bloat?

A

-esophageal obstruction; something inside or outside esophagus blocks eructation
-ruminal motility dysfunction; poor rumen contraction prevents eructation

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15
Q

What can lead to rumen motility dysfunction?

A

-vagal indigestion
-traumatic reticuloperitonitis
-grain overload

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16
Q

What are the characteristics of frothy bloat?

A

-dietary factors promote production of small gas bubbles that cannot merge
-no ping when testing rumen
-must be treated as an emergency

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17
Q

What type of feed causes frothy bloat?

A

lush, high protein forage that is metabolized to produce viscous ruminal fluid; legumes are the primary culprit

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18
Q

What are the clinical signs of bloat?

A

-mild to severe bloat
-reduced appetite
-weight gain
-abdominal discomfort/colic
-respiratory distress
-sudden death

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19
Q

How is the pressure of bloat relieved with free gas bloat?

A

OG intubation

20
Q

How is frothy bloat alleviated?

A

anti-foaming agents or surfactants, including:
-poloxalene
-docusate sodium
-laundry detergent in emergency cases

21
Q

How is bloat ruminal tympany prevented?

A

-remove offending feed
-exercise
-supplemental grass hay
-poloxalene blocks, tubs, or top dress
-ionophores such as monensin that will select for microbiota that produce less gas

22
Q

What are the characteristics of traumatic reticuloperitonitis/hardware disease?

A

-perforation of reticulorumen with an ingested sharp metal foreign body
-rhythmic contractions push object through the wall
-leakage of ruminal contents results in infection, fibrin deposition, and adhesion

23
Q

What are the commonly affected sites in hardware disease?

A

-peritoneum
-pleura
-pericardial sac

24
Q

What are the clinical signs of acute peritonitis in hardware disease?

A

-fever
-tachycardia
-abdominal pain
-bruxism/grinding teeth/clenching jaw
-toxemia

25
Q

What are the clinical signs of chronic peritonitis in hardware disease?

A

-localized pain and abscess
-similar signs to acute peritonitis, but less severe

26
Q

Which tests are used to determine if cows are experiencing abdominal pain?

A

grunt test or withers pinch

27
Q

Which other diagnostics are done for hardware disease besides grunt test/withers pinch?

A

-ultrasound
-compass
-1 view radiograph

28
Q

What are the signs of pleuritis due to hardware disease?

A

-pleural friction rubs
-thoracic pain
-pulmonary abscesses

29
Q

What are the signs of pericarditis due to hardware disease?

A

-right-sided congestive heart failure
-venous distention
-jugular pulses
-brisket edema

30
Q

What are the characteristics of hardware dz treatment?

A

-unrewarding
-typically culled, may need to condemn
-can try long-term antibiotics
-may be able to extract foreign body and/or treat abscesses via laparotomy/rumenotomy
-magnets for prevention is best method

31
Q

What are the characteristics of omasal impaction?

A

-obstruction of omasal outflow by fibrous or stringy materials
-presents similarly to type 2 vagal indigestion
-diagnosed and treated like vagal indigestion
-can do oral or IV fluids and macrolide antibiotics; cannot due surgery

32
Q

What is an abomasal ulcer?

A

erosion of gastric mucosa that may involve bleeding and may cause peritoneal perforation

33
Q

What can cause abomasal ulcers in calves?

A

-stress
-mineral deficiencies
-Clostridium perfringens type A
-trichobezoars
-NSAIDs

34
Q

What can cause abomasal ulcers in adult cattle?

A

-stress
-parturition
-peak milk time
-concurrent disease
-diets high in starches
-NSAIDs
-lymphosarcoma (most common cause)

35
Q

What are the four classifications of ulcers?

A

-type 1: non-perforating, no signs
-type 2: non-perforating, bleeding
-type 3: perforating, local peritonitis
-type 4: perforating, diffuse peritonitis

36
Q

What are the clinical signs of abomasal ulcers?

A

-possibly asymptomatic
-melena/dark tarry manure with lots of bleeding
-mild to severe pain
-poor production
-rough hair coat
-bruxism
-groaning
-localized to severe/diffuse peritonitis

37
Q

How are abomasal ulcers diagnosed?

A

-fecal occult blood
-abdominocentesis
-ultrasound

38
Q

What is the treatment for abomasal ulcers?

A

-acid reducers
-gastroprotectants
-blood transfusion if anemic
-systemic antibiotics

39
Q

What can help to reduce acid for ulcer treatment?

A

-antacids
-H2 antagonists
-proton pump inhibitors
-reductions in high energy feed rations

40
Q

Which gastroprotectants can be used in ulcer treatment?

A

-sucralfate
-misoprostol (not in alpacas)

41
Q

What are the characteristics of abomasal impaction?

A

-failure of abomasal emptying
-can be primary or secondary
-seen in calves and adult cattle
-look for nutritional deficiency
-early stages of type 3 vagal indigestion

42
Q

What can cause secondary abomasal impaction?

A

-poor quality forage
-foreign body
-sand
-trichobezoars
lymphoma

43
Q

What are the clinical signs of abomasal impaction?

A

early:
-reduced feed intake
-reduced manure production
-mild bloat
late:
-vagal indigestion

44
Q

How is abomasal impaction diagnosed?

A

-clinical exam
-hypochloremia
-L-shaped rumen on rectal exam

45
Q

What is the treatment for abomasal impaction?

A

-laxatives
-aggressive oral and IV fluids
-surgery to remove obstruction
-better quality ration and forages