Lecture 20 3/24/25 Flashcards

1
Q

What prevents the facial lymph nodes of a cow from being palpable?

A

the large amount of salivary tissue

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2
Q

What is the tooth eruption pattern for cattle?

A

-first pair of incisors erupts by 1.5 years
-second pair of incisors erupts by 2.5 years
-third pair of incisors erupts by 3.5 years
-fourth pair of incisors erupts by 4.5 years

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3
Q

What is the tooth wear pattern for cattle?

A

-all teeth are in wear by 5 years
-first pair of incisors has root exposure around 6 years
-second pair of incisors has root exposure around 7 years
-third pair of incisors has root exposure around 8 years
-fourth pair of incisors has root exposure around 9 years

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4
Q

What is the tooth eruption and wear pattern for small ruminants?

A

first pair: erupts at 1 year
second pair: erupts at 2 years
third pair: erupts at 3 years
fourth pair: erupts at 4 years
all pairs: in wear at 5 years, root exposure by 6 to 8 years

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5
Q

What can exacerbate dental attrition?

A

-sandy soils
-acidic feed

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6
Q

What are the clinical signs of dental attrition?

A

-chronic weight loss
-quidding/dropping partially chewed food
-choke

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7
Q

Which teeth are commonly affected by tooth root abscesses in llamas and alpacas?

A

cheek teeth

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8
Q

What causes tooth root abscesses?

A

-damage to the gingiva leading to ascending infections
-occasionally hematogenous or tooth fractures

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9
Q

What is the presentation of tooth root abscesses?

A

-often seen in spring when animals are sheared
-animals typically 4+ years old
-hard bony swelling over affected tooth
-possible draining fistula
-painful mastication (if osteomyelitis)
-weight loss (if osteomyelitis)

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10
Q

How are tooth root abscesses diagnosed?

A

-clinical exam
-radiographs
-CT

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11
Q

What is the medical treatment for tooth root abscesses?

A

-florfenicol every 48 hrs for 10 treatments (penetrates bone)
-NSAIDs
-gastroprotectants to combat NSAID effects

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12
Q

What is the surgical treatment for tooth root abscesses?

A

-burred drilling of affected root
-extraction
-thorough curettage of alveolus
-aftercare including daily packing, lavage, and supportive care

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13
Q

What are the characteristics of ruminant saliva?

A

-high in HCO3 and PO4
-daily production is in excess of 50 L/day in cattle
-critical buffering source for the rumen
-animals losing saliva are at risk of dehydration and ruminal acidosis

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14
Q

What is ptyalism?

A

excessive salivation

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15
Q

What are the etiologic categories of ptyalism?

A

-inability to swallow
-excessive production

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16
Q

What are the characteristics of sialoceles?

A

-cyst-like collections of mucoid saliva
-occur when ducts or glands rupture and release saliva into SQ
-typically occur with trauma
-causes immune response, inflammation, and necrosis

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17
Q

How are sialoceles diagnosed?

A

-clinical exam
-aspiration

18
Q

What is the main clinical sign of sialoceles?

A

soft flocculent oral or pharyngeal mass

19
Q

What are the treatment options for sialoceles?

A

-surgical: extirpation of sialocele, duct, and gland
-medical: drainage and chemical cauterization

20
Q

What are the characteristics of Actinobacillus lignieresii?

A

-causes wooden tongue
-gram-neg. commensal of oral cavity
-infections arise following damage to oral mucosa

21
Q

What are the clinical signs of wooden tongue?

A

-dysphagia
-ptyalism
-large, inflexible tongue
-disease of lips, nose, or lymph nodes
-nodular granulomatous abscesses

22
Q

What is the treatment for wooden tongue?

A

*sodium iodide:
-1 bottle per cow
-treatment repeated every week until signs of iodinism
-premedication with NSAIDs and dexamethasone to prevent anaphylaxis
*other antibiotics as needed

23
Q

What are the signs of iodine toxicity?

A

-dandruff
-alopecia
-excessive tearing
-diarrhea
-coughing
-inappetence

24
Q

What are the characteristics of Actinomyces bovis?

A

-causes lumpy jaw
-infects bony tissue
-gram-positive commensal
-infections arise following damage to oral mucosa and bony tissues

25
What are the clinical signs of lumpy jaw?
-hard, painless, immovable mass on jaw (typically mandible) -possible draining fistula -dysphagia -weight loss -disease of esophagus, forestomaches, trachea, and lungs
26
What is the treatment for lumpy jaw?
-sodium iodide weekly until signs of iodinism -isoniazid -curettage
27
What is the prognosis for lumpy jaw?
-infection can only be arrested if it encompasses bone; not cured -permanent jaw deformation likely -soft tissue lesions typically resolve with therapy
28
What are the characteristics of Fusobacterium necrophorum?
-anaerobic, opportunistic pathogen -normally found in oral and GI tract -causes disease in the face of oral mucosa damage -causes oral necrobacillosis/calf diphtheria/necrotic laryngitis
29
What are the clinical signs of oral necrobacillosis?
-swollen cheeks -fetid breath -deep ulcers in oral mucosa -dysphagia/ptyalism -stertor
30
What is the treatment for oral necrobacillosis?
-procaine penicillin G -oxytetracycline -NSAIDs (decreases inflammation) -dexamethasone (decreases resp. distress and inflammation) -indwelling NG tube for feeding during healing
31
How is oral necrobacillosis prevented?
-good hygiene -minimizing trauma
32
What are the characteristics of bovine papular stomatitis?
-caused by parapoxvirus of same name -most infections are subclinical -clinical signs are induced by stress or shipment -morbidity can be very high, up to 100% -zoonotic
33
What are the clinical signs of bovine papular stomatitis?
-raised papules on muzzle, nares, hard palate, and esophagus -papules eventually transition into erosions -stomatitis -ptyalism -loss of hair on tail
34
What are the histologic features of bovine papular stomatitis virus?
-epitheliotrophic -attacks keratinocytes -induces a progressive focal necrosis of the epithelium
35
What are the characteristics of contagious ecthyma/orf?
-caused by parapoxvirus of same name -just like bovine papular stomatitis but more proliferative -causes proliferative lesions at mucocutaneous junctions of nose and mouth -very zoonotic -also known as sore mouth
36
What are the clinical signs of orf?
-seen in young animals -lesions at the nose, mouth, gums, tongue, conjunctiva, coronary band, interdigital area, genitalia, teats, and udder -reluctance to eat, nurse, walk, or be nursed -self-limiting lesions that persist for 3 to 6 weeks
37
What are the histologic features of orf virus?
-induces proliferation -causes crusting of the epidermis
38
How is orf prevented?
-orf free herds -quarantine of new additions -vaccination via scarification to attenuate clinical disease and shorten duration of infection
39
What are the characteristics of esophageal obstruction/choke?
-acute obstruction of esophagus -complete obstruction prevents eructation and leads to rumen gas build up/bloat -acute bloat can be fatal; distended rumen compresses thoracic cavity and causes high pressure; causes CV collapse -partial obstructions cause dysphagia and temporary bloat -often seen in small ruminants that choke on pelleted feed
40
What are the clinical signs of choke?
-anxiety -yawning -dysphagia -ptyalism -collapse -bloat -acute death
41
What are the long term sequelae of choke?
-esophageal damage -stricture -cellulitis -diverticula -recurrent bloat
42
What is the treatment for bloat?
-gently pass OG tube to push obstruction into rumen -massage of externally palpable obstructions -temporary rumenostomy -bloat whistles for chronic recurrence -trocarization in emergencies