Lecture 20 3/24/25 Flashcards

1
Q

What prevents the facial lymph nodes of a cow from being palpable?

A

the large amount of salivary tissue

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2
Q

What is the tooth eruption pattern for cattle?

A

-first pair of incisors erupts by 1.5 years
-second pair of incisors erupts by 2.5 years
-third pair of incisors erupts by 3.5 years
-fourth pair of incisors erupts by 4.5 years

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3
Q

What is the tooth wear pattern for cattle?

A

-all teeth are in wear by 5 years
-first pair of incisors has root exposure around 6 years
-second pair of incisors has root exposure around 7 years
-third pair of incisors has root exposure around 8 years
-fourth pair of incisors has root exposure around 9 years

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4
Q

What is the tooth eruption and wear pattern for small ruminants?

A

first pair: erupts at 1 year
second pair: erupts at 2 years
third pair: erupts at 3 years
fourth pair: erupts at 4 years
all pairs: in wear at 5 years, root exposure by 6 to 8 years

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5
Q

What can exacerbate dental attrition?

A

-sandy soils
-acidic feed

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6
Q

What are the clinical signs of dental attrition?

A

-chronic weight loss
-quidding/dropping partially chewed food
-choke

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7
Q

Which teeth are commonly affected by tooth root abscesses in llamas and alpacas?

A

cheek teeth

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8
Q

What causes tooth root abscesses?

A

-damage to the gingiva leading to ascending infections
-occasionally hematogenous or tooth fractures

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9
Q

What is the presentation of tooth root abscesses?

A

-often seen in spring when animals are sheared
-animals typically 4+ years old
-hard bony swelling over affected tooth
-possible draining fistula
-painful mastication (if osteomyelitis)
-weight loss (if osteomyelitis)

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10
Q

How are tooth root abscesses diagnosed?

A

-clinical exam
-radiographs
-CT

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11
Q

What is the medical treatment for tooth root abscesses?

A

-florfenicol every 48 hrs for 10 treatments (penetrates bone)
-NSAIDs
-gastroprotectants to combat NSAID effects

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12
Q

What is the surgical treatment for tooth root abscesses?

A

-burred drilling of affected root
-extraction
-thorough curettage of alveolus
-aftercare including daily packing, lavage, and supportive care

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13
Q

What are the characteristics of ruminant saliva?

A

-high in HCO3 and PO4
-daily production is in excess of 50 L/day in cattle
-critical buffering source for the rumen
-animals losing saliva are at risk of dehydration and ruminal acidosis

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14
Q

What is ptyalism?

A

excessive salivation

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15
Q

What are the etiologic categories of ptyalism?

A

-inability to swallow
-excessive production

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16
Q

What are the characteristics of sialoceles?

A

-cyst-like collections of mucoid saliva
-occur when ducts or glands rupture and release saliva into SQ
-typically occur with trauma
-causes immune response, inflammation, and necrosis

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17
Q

How are sialoceles diagnosed?

A

-clinical exam
-aspiration

18
Q

What is the main clinical sign of sialoceles?

A

soft flocculent oral or pharyngeal mass

19
Q

What are the treatment options for sialoceles?

A

-surgical: extirpation of sialocele, duct, and gland
-medical: drainage and chemical cauterization

20
Q

What are the characteristics of Actinobacillus lignieresii?

A

-causes wooden tongue
-gram-neg. commensal of oral cavity
-infections arise following damage to oral mucosa

21
Q

What are the clinical signs of wooden tongue?

A

-dysphagia
-ptyalism
-large, inflexible tongue
-disease of lips, nose, or lymph nodes
-nodular granulomatous abscesses

22
Q

What is the treatment for wooden tongue?

A

*sodium iodide:
-1 bottle per cow
-treatment repeated every week until signs of iodinism
-premedication with NSAIDs and dexamethasone to prevent anaphylaxis
*other antibiotics as needed

23
Q

What are the signs of iodine toxicity?

A

-dandruff
-alopecia
-excessive tearing
-diarrhea
-coughing
-inappetence

24
Q

What are the characteristics of Actinomyces bovis?

A

-causes lumpy jaw
-infects bony tissue
-gram-positive commensal
-infections arise following damage to oral mucosa and bony tissues

25
Q

What are the clinical signs of lumpy jaw?

A

-hard, painless, immovable mass on jaw (typically mandible)
-possible draining fistula
-dysphagia
-weight loss
-disease of esophagus, forestomaches, trachea, and lungs

26
Q

What is the treatment for lumpy jaw?

A

-sodium iodide weekly until signs of iodinism
-isoniazid
-curettage

27
Q

What is the prognosis for lumpy jaw?

A

-infection can only be arrested if it encompasses bone; not cured
-permanent jaw deformation likely
-soft tissue lesions typically resolve with therapy

28
Q

What are the characteristics of Fusobacterium necrophorum?

A

-anaerobic, opportunistic pathogen
-normally found in oral and GI tract
-causes disease in the face of oral mucosa damage
-causes oral necrobacillosis/calf diphtheria/necrotic laryngitis

29
Q

What are the clinical signs of oral necrobacillosis?

A

-swollen cheeks
-fetid breath
-deep ulcers in oral mucosa
-dysphagia/ptyalism
-stertor

30
Q

What is the treatment for oral necrobacillosis?

A

-procaine penicillin G
-oxytetracycline
-NSAIDs (decreases inflammation)
-dexamethasone (decreases resp. distress and inflammation)
-indwelling NG tube for feeding during healing

31
Q

How is oral necrobacillosis prevented?

A

-good hygiene
-minimizing trauma

32
Q

What are the characteristics of bovine papular stomatitis?

A

-caused by parapoxvirus of same name
-most infections are subclinical
-clinical signs are induced by stress or shipment
-morbidity can be very high, up to 100%
-zoonotic

33
Q

What are the clinical signs of bovine papular stomatitis?

A

-raised papules on muzzle, nares, hard palate, and esophagus
-papules eventually transition into erosions
-stomatitis
-ptyalism
-loss of hair on tail

34
Q

What are the histologic features of bovine papular stomatitis virus?

A

-epitheliotrophic
-attacks keratinocytes
-induces a progressive focal necrosis of the epithelium

35
Q

What are the characteristics of contagious ecthyma/orf?

A

-caused by parapoxvirus of same name
-just like bovine papular stomatitis but more proliferative
-causes proliferative lesions at mucocutaneous junctions of nose and mouth
-very zoonotic
-also known as sore mouth

36
Q

What are the clinical signs of orf?

A

-seen in young animals
-lesions at the nose, mouth, gums, tongue, conjunctiva, coronary band, interdigital area, genitalia, teats, and udder
-reluctance to eat, nurse, walk, or be nursed
-self-limiting lesions that persist for 3 to 6 weeks

37
Q

What are the histologic features of orf virus?

A

-induces proliferation
-causes crusting of the epidermis

38
Q

How is orf prevented?

A

-orf free herds
-quarantine of new additions
-vaccination via scarification to attenuate clinical disease and shorten duration of infection

39
Q

What are the characteristics of esophageal obstruction/choke?

A

-acute obstruction of esophagus
-complete obstruction prevents eructation and leads to rumen gas build up/bloat
-acute bloat can be fatal; distended rumen compresses thoracic cavity and causes high pressure; causes CV collapse
-partial obstructions cause dysphagia and temporary bloat
-often seen in small ruminants that choke on pelleted feed

40
Q

What are the clinical signs of choke?

A

-anxiety
-yawning
-dysphagia
-ptyalism
-collapse
-bloat
-acute death

41
Q

What are the long term sequelae of choke?

A

-esophageal damage
-stricture
-cellulitis
-diverticula
-recurrent bloat

42
Q

What is the treatment for bloat?

A

-gently pass OG tube to push obstruction into rumen
-massage of externally palpable obstructions
-temporary rumenostomy
-bloat whistles for chronic recurrence
-trocarization in emergencies