Lecture 17: Dissociative Disorders Flashcards

1
Q

Dissociation gets used in many ways in psychiatry and psychology eg, (10)

A
  1. Depersonalization
  2. Derealization
  3. Flashbacks
  4. Hypnosis
  5. Trance states
  6. Absorption
  7. Divided attention
  8. Non-organic amnesia
  9. Multiple identities
  10. Conversion symptoms
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2
Q

Dissociation may or may not be a sign of _______

A

Psychopathology

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3
Q

Dissociations can be (3)

List examples of all

A
  1. Benign eg. Highway hypnosis i.e., driving a familiar route and having no memory of how you got there
  2. Helpful eg. Meditation blunts stress response/improves immune function
  3. Harmful eg. Flashbacks of trauma
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4
Q

Evolutionary hypothesis (5)

A
  1. Absorption
  2. Tonic immobility
  3. Dissociative amnesia
    4.Conversion of stress into somatic symptoms that can help elicit aid from kin/reduce the groups demands on the afflicted individual
  4. Dissociative trances are used in shamanistic practices that are experienced as cathartic/healing/promote group cohesion
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5
Q

Absorption AKA: ??
What does it facilitate? (2)

A

Highly selective attention that can be plausibly understood as a type of dissociation

Learning for
1. Implicit knowledge/skills
2. Explicit knowledge/skills

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6
Q

Tonic immobility AKA:??
What can it prevent

A

“Playing dead” - can prevent further attack from predators

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7
Q

Dissociative amnesia functions (3)

A
  1. Reduce stress response
  2. Produce adaptive self-deception
  3. Promote self-interest
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8
Q

How does dissociative amnesia reduce stress response?

A

Keeping anxiety-inducing knowledge of memories out of awareness

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9
Q

How does dissociative amnesia produce adaptive self-deception

A
  1. Creates positive illusions about ourselves —> promotes well-being
  2. Influence other’s evaluations to be more favorable
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10
Q

How does dissociative amnesia promote self-interest

A

People who are self-deceived are better able to deceive others to promote their own self-interest

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11
Q

In dissociative amnesia there is an evolutionary hypothesis (no.4)

Conversion of stress into somatic symptoms can help _____ ____ ___ ___ and ____ ___ ___ ______ on the affected individual

A
  1. Elicit aid from kin
  2. Reduce the group’s demands
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12
Q

In dissociative amnesia there is an evolutionary hypothesis (no.5)

Dissociative trances are used in shamanistic practices that are experienced as ______ and ______. These practices can also promote _______ _______.

A
  1. Cathartic
  2. Healing
  3. Group cohesion
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13
Q

Dissociation definition: A disturbance to the ______ _______ __ that control ______ ______, which results in pathological ______ or _________

A
  1. Basic mental processes
  2. Adaptive actions
  3. Detachment
  4. Compartmentalization
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14
Q

Detachment

A

Alteration in consciousness resulting in SEPARATION from yourself, the world, and or emotions

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15
Q

Detachment examples (3)

A
  1. Depersonalization
  2. Derealization
  3. Emotional detachment
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16
Q

Depersonalization

A

Alienation from self eg. Not recognizing yourself in a mirror

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17
Q

Derealization

A

Alienation from the world eg. Feeling as if the world is a simulation

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18
Q

Emotional detachment

A

Absence of emotion activation under conditions that would ordinarily produce emotions eg. Feeling “numb” when someone close to you dies

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19
Q

Is Depersonalization more prevalent in women or men?

A

Gender ratio is equal

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20
Q

When does the onset of depersonalization usually occur?

A

adolescence

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21
Q

Duration of episodes of depersonalization can range from _____ to _____

A

Minutes to months

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22
Q

Risk factors of depersonalization (6)

A
  1. Childhood trauma
  2. High levels of harm-avoidance
  3. Primitive defense machinists eg.denial
  4. Depression
  5. Anxiety - especially oanic attacks
  6. Drug use
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23
Q

Neurobiology of depersonalization (6)

A
  1. Blunted Autonomic nervous system
  2. Overactive baseline levels of HPA, blunted HPA when responding to stressful stimuli
  3. Impaired functioning in somatosensory cortex
  4. Overactive prefrontal cortical regions
  5. Limbic structures inhibited by frontal cortex
  6. Overactive serotonergic transmission
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24
Q

When the autonomic nervous system is blunted, this means that there are _____ levels of sympathetic nervous system arousal compared to stressful stimuli

25
When the prefrontal cortical regions are overactive this leads to excessive inhibition of the _____ _____
Limbic systems
26
When Limbic structures (eg. Amygdala/stiatum) are inhibited by frontal cortex, this leads to the absence of ____ _________ or often described as “_____”
Felt emotion - “numbness”
27
Serotonergic transmission might be overactive in DPDR. Administration of serotonin ________ can induce depersonalization
Agonists
28
Treatments of DPDR (5)
1. CBT 2. Insight-oriented therapy 3. Transcranial magnetic stimulation 4. Trauma-informed therapy 5. SSRI’s for comorbidities anxiety and depression
29
Trauma informed therapy - therapy that provides clients with a sense of (3)
Therapy that provides clients with a sense of 1. Safety/trust 2. Choice 3. Empowerment
30
Superordinate types of dissociation (2)
1. Detachment 2. Compartmentalization
31
Compartmentalization - failure of ______ _____ over mental processes that are ordinarily under control. Typically, the mental processes that are no longer under ______ ______ continue to be _______ or ________ ________
Volitional control Volitional control Partially Entirely functional
32
Compartmentalization examples (3)
1. Hypnosis 2. Conversion disorder 3. Dissociative amnesia
33
Hypnosis - a state of (3)
1. Relaxation 2. Focused attention 3. Suggestibility
34
Conversion disorder (name an example too!)
Inability to control motor behavior to attend to some set of sensations eg. Psychogenic blindness
35
Dissociative Amnesia
The inability to retrieve memories that were successfully encoded Inaccessible memories can often be retrieved under hypnosis
36
The two superorder types of dissociation (2) co-occur in people with __________ ________ ________
Dissociative identity disorder
37
Multiple personality disorder
Where there is one primary “host” personality and one or more non-primary personalities often referred to as “alters” or “part”
38
39
Common characteristics of parts (9)
Different 1. Names - symbolic quality common 2. Ages - typically one part is a child 3. Genders 4. Vocal quality/accents 5. Posture/gait 6. Abilities/skills 7. Emotional states (differences in anger/fear) 8. Access to memories (including ordinary/traumatic 9. Some report hearing voices of their parts when experiencing their primary personality and some don’t
40
Switching
The transition form one personality to another
41
Can people with DID control their switching? What factors in their ability to control?
Some can, some only partially, some not at all Ability to control switching is associated with severity
42
Dissociative amnesia can present as
1. Not being able to remember what you did for some part of the day 2. Not being able to recall any part of specific periods of your life 3. Not remembering how old you are 4. Discovering things at home you don’t remember buying 5. Encountering people who know you but you don’t remember meeting them 6. People calling you by another name 7. Dissociative fugues
43
Dissociative fugues
Switching into a part, traveling somewhere, then switching back to the primary personality, who can’t recall how they got their
44
Is DID only in western countries?
No it’s in wide range of cultures
45
DID is typically caused by ______, ______. ______ ______, particularly ______ and ______ _____
Recurrent, severe childhood abuse, particularly physical and sexual abuse
46
Some people with DID have “_______” identities i.e they experience the part as a spirit, ghost, demon, or other supernatural being
Possession
47
Common comorbidities of DID (10)
1. Depression 2. Anxiety 3. Substance use 4. Self injury 5. Non-epileptic seizures 6. Dissociative flashbacks 7. Suicidal thoughts/behaviors 8. Transient psychosis 9. Conversion symptoms 10. PTSD
48
Neurobiological explanations of DID (2)
1. Smaller amygdala volume 2. Smaller hippocampus volume
49
Smaller amygdala and hippocampus volume explanations for DID (2)
1. People with smaller amygdala/hippocampus are at elevated risk of DID 2. People with TRAUMA have small amygdala/hippocampus due to excitotoxicity
50
Excitotoxicity
Cell death from extreme neuronal firing
51
1. Hippocampus mediates consolidation of ____ ____ ____ into ______ ____ ____ 2. Hippocampus is sensitive to ________ ________ i.e., Time and place 3. Hippocampus is sensitive relational properties between stimuli, helping to bind various stimuli into a ______ ________
1. STM into LTM 2. Context information 3. Unitary representation
52
During stress ______ is released from the adrenal glands. High levels of _______, ________ hippocampal functioning
Cortisol, cortisol, decrease
53
Decreased hippocampal functioning = (3)
1. Impaired LTM consolidation - why people with PTSD/DID recall traumatic events with difficulty/not at all 2. Impaired context information - why people with PTSD/DID have “flashbacks” where they experience a traumatic past as thought it were happening in the present 3. Impairment of binding sensory experiences into unitary representations - why people with PTSD/DID often recall fragments of a traumatic event
54
In DID it’s not just memories that are fragmented but also ___ _____.
The self
55
Treatment of DID is ______ _______ _______
Trauma-informed psychotherapy
56
Trauma informed psychotherapy emphasizes (8)
1. Emotion awareness and regulation 2. Impulse awareness and control 3. Grounding/mindfulness to reduce dissociation 4. Identifying/reducing high-risk behaviors 5. Mapping of personality states 6. Identifying/modifying cognitive disortions due to trauma 7. Processing trauma (controversial - some do it some don’t) 8. Integration of parts into primary personality (controversial - some do it some don’t)
57
Iatrogenic treatment
Treatment that causes harm to people
58
The validity of the diagnosis of DID, and the stories of abuse disclosed by people with DID is denied by some mental health professionals —> Treatment becomes ______
Iatrogenic