Introduction to Ortho Flashcards

1
Q

Orthopedics is the study of

A

The study of the musculoskeletal system including bone, joints, ligaments, tendons, muscles, and supporting neurological and vascular structures

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2
Q

Sprain
Common where?
Uncommon in who?
Treatment of sprains

A

Sprain is injury to ligaments (connects 2 or more bones) and typically occurs in the ankle, knee, or wrist as result of trauma.

Uncommon in children or those with osteoporosis dt bone being more fragile than ligament.

I: Partial tear, no instability, symptomatic tx
II: Partial tear, instability, immobilize to protect.
III: Complete tear, immobilize/repair

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3
Q

strain:
Common where?

Grades I -IV

A

Strain is injury to tendon (connects muscle to bone).

Commonly seen with gastrocnemius, hamstring, and quadriceps.

I: Tear of a few muscle fibers
II: Tear of moderate amount of muscle fibers
III: Tear of all muscle fibers
IV: Tear of all muscle fibers – “rupture”

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4
Q

what is the clinical presentation of sprain or strain?

A

With sprain and strain, have hx of traumatic event with reports of snapping, popping, tearing sensation at time of injury

Associated with pain, swelling, stiffness, difficulty bearing weight. Ecchymosis may appear.

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5
Q

what are you PE findings of sprains and strains

A

Compare exam findings of swelling/tenderness w/contralateral side & examine for joint instability. Able to contract affected musculature- if they can’t its a higher grade strain

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6
Q

what is used for diagnosing strains and sprains?

A

X-ray to r/o fracture.
Consider w/II, III, IV strain or II, III sprain

MRI to evaluate soft tissue injury

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7
Q

what are the Ottawa Ankle Rules

A

guidelines of when to get an X-ray

if 1 or more of the following is +
pain at lateral malleolus
pain at medial malleolus
cannot bear weight for 4 steps
pain at navicular 
Pain in the mid foot at the base of the 5th metatarsal
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8
Q

Treatment of sprains and strains

A

PRICE
Minor: sprains can be treated with elastic compression bandage, bracing or brief periods of immobilization

Strains should be immobilized where muscle is stretched to minimize bleeding of injured muscle

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9
Q

What types of strains or sprains do you reef?

A

grade III or severe grade II

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10
Q

Contusion

Result of:
Presents with:

A

-Result of direct, blunt trauma causing bleeding & soft tissue damage

Presents with activity related pain, swelling, ecchymosis, possible hematoma

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11
Q

Diagnostic evaluation of a contusion?

A

X-ray to rule out fracture

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12
Q

Treatment of a contusion?

A

Conservative treatment, drainage if large hematoma

Monitor for compartment syndrome and join instability

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13
Q

Fracture
describe:
Often a/w:

A

Described as a Disruption in continuity or structural integrity of bone that occurs when stress applied to bone is greater than bone’s intrinsic strength

Often time is a/w injury to surrounding tissue (muscle, blood vessels, neurological structures)

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14
Q

Closed v Open Fractures

A

Closed: Skin over and near the fracture is intact

Open: Skin over or near fracture is lacerated or abraded by injury & requires expedient care & evaluation by specialist.

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15
Q
types of fractures
1:
2:
3:
4:
5:
A

Non displaced: In alignment

Displaced: Not in alignment

Angulated: Fractures are malaligned and angulated

Bayonetted: Distal fragment overlaps proximal fragment

Distracted: Distal fragment separated from proximal by gap

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16
Q

Treatment of fractures:

A

Specialist to determine if manipulation, reduction, stabilization is needed

Depends on bone involved- in general involves immobilization, reduction and rehab

Immobilize, reduction, rehab. Open fractures → URGENT tx

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17
Q

Open fractures are

A

URGENT treatment!

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18
Q

What are some factors that improve stability/ increase likelihood of good prognosis?

A

Young,
1 fracture in forearm/lower leg,
nondisplaced,
Thoracic spine fracture

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19
Q

What factors that worsen tability/ increase likelihood of poor prognosis?

A

Older, displaced, compartment syndrome, osteonecrosis, oblique, neurologic/vascular injury

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20
Q

Stress Fractures:
Occurs as a result of:

May occur with hx of:

Present:

A

Occurs as a result of repetitive stress loads to healthy bone

May occur with hx of prior stress fracture, < level of fitness, > volume/intensity of activity, female, menstrual irregularity, poor diet of calcium, poor biomechanics, poor bone health

Present insidiously compared to acute fracture

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21
Q

What will a stress fracture present as?

A

May report > in activity level, gradual onset of localized, activity related pain that progresses to pain at rest

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22
Q

what is the Diagnosis of stress fractures?

A

It will take 2-3 weeks to show up on Radiograph, it is recommended as initial study, but if it is negative and highly suggestive MRI is the diagnostic study of choice

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23
Q

what is the treatment of stress fractures?

early intervention

Low risk

High risk

Re - evaluate

A

Early intervention : < pain, promote healing, prevent future damage

Conservative management is recommended for low risk: 2nd-4th metatarsal shaft, posteriormedial tibial shaft, fibula, proximal humerus or shaft, ribs, sacrum, pubic rami

Specialist consult for high risk or complete or want to return quickly: Pars interarticularis (between vertebrae), femoral head or neck, patella, anterior tibia, medial malleolus, talus, proximal 5th metatarsal, sesamoids on great toe, base of 2nd metatarsal

Reevaluate every 1-3 weeks, Possible >12 weeks for return of activity

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24
Q

Dislocations

A

Joint injury that forces ends of bones out of position, commonly dt trauma. May involve ankle, knee, shoulder, hip, elbow, jaw, and finger.

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25
Q

what is the clinical presentation of a dislocation?

A

Pain, swelling, deformity, < ROM, numb/tingling if nerve involved

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26
Q

what is the treatment of a dislocation?

A

Depends on severity and joint involved – Emergent, Analgesia, Manipulation and/or surgical intervention followed by immobilization & subsequent rehab

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27
Q

Subluxation

A

Occurs when a joint begins to dislocate (partial dislocation) and can occur d/t trauma or underlying conditions that predispose to join laxity

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28
Q

Arthritis
cause:
manifestations:
Most common types:

A

Degenerative process a/w aging or acute infections & inflammation

Disability can range from minimal to crippling

Most common are osteoarthritis and rheumatoid arthritis

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29
Q

Septic Arthritis
aka:
caused by:
Commonly affected joints:

A

define: an infection of the going space

Also called pyogenic or suppurative arthritis

caused by infection to the joint space by either direct inoculation, hematogenous spread, or extension from adjoining bone infection.

Common joints include knee, hip, shoulder, elbow, wrise

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30
Q

Risk factors for septic arthritis?

A

> 80 y.o., DM, RA, gout, prosthetic joint, skin infection, > alcohol

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31
Q

Bacteria in septic arthritis?

A

Virus, bacteria, or fungi.

Staph Aureus is the most common in those 2 y.o. and older

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32
Q

Presentation of septic arthritis?
Early on:
Children:
Systemic Symptoms:

A

Symptoms early on are typically mild

join pain, swelling, < ROM.

Children may refuse to bear weight or move affected joint.

Systemic symptoms such as fever & tachycardia

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33
Q

PE findings of Septic Arthritis?

A

Red hot swollen joint that doesn’t move

Watch for possible primary infection – skin defect, penetrating injuries, skin abscess, tooth abscess

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34
Q

Dx of septic arthritis?

A
  • > WBC, > ESR, > CRP, Blood culture
  • If you suspect gonococcus, need cultures from other than mouth, cervical or utrethra

Joint aspiration: crystal analysis, gram stain, culture, cell count (>50,000)

Radiographs are often normal, may see soft tissue swelling

US, CT, MRI may reveal joint effusion

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35
Q

Treatment of septic arthritis

A

Empiric IV ABX for 14 days, then 14 days of oral abx

Surgical decompression, splinting of joint

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36
Q

Septic arthritis due to a periprostetic joint

can result form:
consider in:
dx:
tx:

A

can result from intraoperative contamination, local infection or hematogenous spread

consider if pt with prosthetic joint who presents with new onset of pain

Dx: similar to systemic

Treatment
Generally attempt surgical debridement.

37
Q

Septic Arthritis due to gonococcal infection

can lead to:

common cause:

dx:

tx:

A

can lead to a vartiey of clinical manifestation including arthritis, tenosynovitis and or dermatitis

Common cause of acute poly arthalgia, poly arthritis or oligoarthritis in young healthy patient

DX: Hx and PE and cultures

Treatment: Cefriaxone x 7-14 days and a single dose of azithomycin.

38
Q

Bursitis
define:
may follow:
fluid can be:

A

Inflammation of synovial tissue lining bursa resulting in increased fluid production and subsequent pain/swelling

May be seen following injury or repetitive motion

Sterile (acute – > trauma, crystal disease, bleeding) or infectious

39
Q

Presentation of Bursitis

A

Pain on motion and at rest with < ROM.
Swelling, local tenderness, hx of trauma or repeat injury
if infection/fever → septic bursitis

40
Q

Diagnostic evaluation of Bursitis

A

Aspiration of bursal fluid for gram stain, culture, cell count, crystal eval

Imaging studies not helpful if superficial. If deep, x-ray/MRI may help. If superficial – inflammation. If deep – Unexplained pain w/motion.

41
Q

Treatment of bursitis

A

Modify activity, analgesia (NSAIDS), splinting, corticosteroids, and surgical excision if relapsing.

If infectious, use broad spectrum abx and/or surgical drainage

42
Q

Tendinopathy
define:
d/t:
risk factors:

A

clinical syndrome characterized by tendon thickening and chronic localized tendon pain

can occur d/t: Acute trauma or more commonly d/t overuse

Risk factors:include > age, > BMI, biomechanical abnormalities, prior tendon lesion, fluoroquinolone use, training errors or poor equipment

43
Q

Presentation of Tendinopathy

A

Pain > with palpation of affection tendon and tendon loading.

Palpable tendon thickening may be present.

44
Q

how do you dx Tendinopathy

A

US and MRI

45
Q

What is the treatment of Tendinopathy?

A

Conservative measures (rest, correction of biomechanical factors, rehab, gradual return to activities), NSAIDS, sx if refractory cases

AVOID glucocorticoids due to > risk of tendon rupture

46
Q

what is the prognosis of tendiopathy?

A

o Slow, chronic disorder requiring months for complete healing
o Symptoms worsen initially with rehab

47
Q

Tenosynovitis
Define:
Occurs most freq in:
Due to:

A

inflammation of a tendon and its synovial sheath

Occurs most frequently in hands and wrist on extensor & flexor side

Infectious or non-infectious

48
Q
Presentation of tenosynovitis?
Infection most commonly involves the:
Cardinal Signs:
1:
2:
3:
4:
5:
-
-
-Fingers look like
A

o Infections most commonly involve flexor tendon sheath.

Cardinal Signs:

1: Tenderness along flexor sheath
2: symmetric enlargement of affected digit
3: slight flexed finger at rest
4: pain at tendon w/passive Extension

Fever may be present

Cutaneous sign of ischemia - skin necrosis may be present

Fingers are “sausage like”

49
Q

How do you diagnose tenosynovitis?

A

o Gram stain/culture
o Radiograph is usually normal but r/o bony involvement or foreign body
o MRI/US to confirm presence of tendon sheath abnormalities

50
Q

Treatment of tenosynovitis?

A

Surgical intervention, empiric abx therapy

51
Q

Osteomyelitis
define:

-
D/t

A

Infection of bone most commonly occurring in long bones of extremity in children and vertebrae in adults.

Classified according to duration of illness & mechanism of infection

Acute: period prior to development of complications
Chronic: period after development of complications

Occur dt hematogenous seeding, contiguous spread, direct inoculation

52
Q

Osteomyelitis in children is seen in

in adults?

A

in long bones of extremity in children

vertebrae in adults.

53
Q

Risk Factors for osteomyelitis

A

Injury, sx, circulation problems, invasive medical tubing, IVDU, splenectomy, immunosuppressed, malignancy, bacteremia

54
Q

Microbiology cause of osteomyelitis

A

o Staph aureus, coag (-) staph, arerobic gram (-) bacilli most common.
o Hematogenous osteomyelitis is usually mono microbial
o Contiguous may be poly or monomicrobial

55
Q

Clinical Presentation of osteomyelitis
acute:
subacute:
chronic:

A

Acute: Gradual onset, dull pain @ involved site, worsens at night & exacerbated w/movement. < mobility & possible systemic symptoms
-Recent broken bones, immunosuppressed, use of street drugs

Subacute: Mild pain over several weeks, minimal fever

Chronic: Pain, erythema, swelling, ulcers & fractures that fail to heal

56
Q

PE of osteomyelitis?

A

Tenderness & warmth, < ROM, spasms of muscle, draining sinus tract

57
Q

Diagnosing of osteomyelitis?

A

o > WBC, > ESR, > CRP
o Blood culture, sinus tract culture, sputum and urine culture to identify primary site of infection. May extend into joint (septic arthritis)
o Gold Standard: Isolate bacteria from bone biopsy
o X-ray may show osteopenia, soft tissue swelling
o CT can detect bony changes earlier that x-ray
o ECHO may determine endocarditis

58
Q

Treatment of osteomyelitis

A

Analgesia, abx (must debride first!), possible bony reconstruction

Serial inflammatory markers to assess treatment response

59
Q

Prognosis of osteomyelitis?

A

Acute > chronic. Chronic may relapse over many years → amputation

60
Q
Animal Bites
most common in who?
what type of animal is most common?
what animal are you most concerned about?
vaccination?
A

Most commonly in children.
Incidence is higher in dogs than cats however infection more common w/cats (Pasteurella multocida)

Must consider rabies!

61
Q

Animal Bite Presentation

A

Irregular, jagged wound, fever, erythema, swelling, tender, draining, lymphadenopathy, streaking up the arm (wound infection)

62
Q

Diagnosing of animal bites

A

o Gram stain/culture, blood culture
o X-ray AP and lateral to r/o presence of foreign body (cat tooth)
o US to identify abscess formation

63
Q

what is the treatment of animal bites?

A

o Wound irrigation and debridement, let wound heal by itself.
o ABX prophylaxis if high risk
o Tetanus prophylaxis

64
Q

Human Bites
Most commonly found on the:

Clenched fist injury concern:

Infectious agents:

A

Most commonly found on face, upper extremities, trunk of young kids

Clenched fist injury – teeth of one person come in contact with knuckles of another person during fight. When you open hand blood leaves and goes to heart → > chance for infection.

Eikenella corrodens, aerobic gram positive cocci and anaerobes

65
Q

Human bite Presentation

A

Semicircular or oval area of erythema/bruising

Skin may or may not be intact

Tenderness, erythema, swelling, drainage, streaking/fever = infection

66
Q

Human bite Diagnose

A

X-ray if bite is close to bone

67
Q

treatment of human bite

A

Irrigation w/debridement

Abx prophylaxis if through dermis, especially if hand involved

Do not suture, immobilize for clenched fist injury, and follow up!

Tetanus prophylaxis, hepatitis B if not immunized or other person is +

68
Q

Tumors
Can arise from:

types

Metastatic is more common than:

Commonly found:

A

Can arise from bone, cartilage, marrow, vascular structures, synovium

Benign, malignant, inflammatory, traumatic

Metastatic is more common than primary bone tumors!

Commonly found incidentally

69
Q

tumor Presentation

A

Asymptomatic oftentimes. If symptoms, dull/aching pain, fever and malaise, weight loss → Metastatic disease!

70
Q

Diagnosis of tumor?

A

bone biopsy for definitive diagnosis

X-ray initially, with CT to further assess lesions.

MRI to see if soft tissue involved.

Bone scan to identify multiple skeletal lesions

US to see if lesion is cystic or solid

CBC, ESR, CRP,

71
Q

tumor treatment
Benign:
Metastatic

A

Benign: Monitor, resect

Metastatic: < pain and preserve function. Radiation, chemo, sx

72
Q

Avascular necrosis
define:
Types:
Most common location:

A

Death of bone tissue due to lack of blood supply

Traumatic or non-traumatic (corticosteroid use & > alcohol)

Most common location: anterolateral femoral head but can occur elsewhere

73
Q
Avascular Necrosis Presentation
-
-
-
-
A

Occasionally its are Asymptomatic & diagnosis is incidental

If symptomatic – presents late in disease course

Unilateral or bilateral

Pain - most common symptom occurring w/ weight bearing exercise

74
Q

Avascular Necrosis PE

A

Nonspecific, < ROM, pain w/ROM

Altered gait

Antalgic: Shorter time on affected foot
Trendelenberg: Wide base gait to shift hip over

75
Q

Avascular Necrosis Diagnosing

A

X-ray – can be normal for months after symptom onset. → Bone scan

Pathognomic crescent sign shows subchrondral collapse

MRI IS GOLD STANDARD FOR DIAGNOSIS

76
Q

Compartment Syndrome

A

Group of 1> muscles and their associated nerves and vessels as well as surrounding fascia has > pressure within a compartment. Circulation and function is impaired.

77
Q

Compartment Syndrome patho ACUTE

A

Insult occurs (trauma, infection) resulting in > compartment pressure.

Venous pressure is elevated and cannot drain.

Decreased arterial flow to the muscle
Muscle ischemia develops → irreversible damage in 8 hours!

Medical emergency! Can be due to constrictive dressing, thermal injury, and penetrating trauma.

Anterior leg and anterior (volar) arm is most common.

78
Q

Compartment Syndrome Presentation ACUTE

A

Pain out of proportion to apparent injury or PE findings

Rapid progression of symptoms

Sensory hyoesthsia distal to involved compartment

Parasthesia
Weakness

79
Q

PE findings in acute compartment syndrome

A

Increased Pain with passive stretching of the muscle in the involved compartment

palpation reveals tense compartment

< sensation, muscle weakness, pallor is uncommon!

80
Q

Diagnosis of Acute Compartment Syndrome?

A

Only way to definitely dx is to measure the compartment pressure.

No specific measurement, surgeons assess this.

Can measure acute compartment syndrome delta pressure: diastolic – measure compartment pressure

Acute compartment syndrome pressure: <20-30mmHg → fasciotomy

81
Q

Treatment of acute compartment syndrome:

A

o IMMEDIATE surgical fasciotomy
o Wounds left open with delayed closure

o HBO (hypobaric oxygen) may be used as adjunct therapy. Must be done inpatient for insurance to pay, 14 days!!!!

82
Q

Chronic Exertional Compartment Syndrome

A

Occurs when muscles that are metabolically active during exercise swell pathologically, leading to compression of neurovascular structures within the same muscular compartment.

Most common in anterior and lateral compartments

Occurs when pt’s have major change in activity level

83
Q

Chronic Exertional Compartment Syndrome Presentation

A

Gradually increasing pain in specific region – aching, squeezing, cramp

Begins after starting activity, resolves with rest.

Bilaterally

Neurologic symptoms may develop – paraesthesia, numb, weakness

84
Q

Chronic Exertional Compartment Syndrome Diagnosis

A

Measure compartment pressure for diagnosis

Must do > 1x, look for change → pre and post exercise

85
Q

Chronic Exertional Compartment Syndrome Treatment

A

Initiate conservative measure, cessation of provocative activity if able, refer for surgical muscle compartment release

86
Q

Peripheral arterial disease
Associated with
Risk Factors:
Presentation:

A

Associated with lower extremity disease

Risk Factors: Hyperlipidemia, smoking, HTN, DM, age

Presentation: Asymptomatic, atypical leg pain, claudication, critical limb ischemia

87
Q

Peripheral arterial disease PE

May be]

Diminished or absent

May hear

_______ test: (positive!)

A

May be normal

Diminished or absent pulses below level of stenosis, may have normal groin pulses but decreased pulses distally.

May hear bruits over stenotic lesion, may have evidence of poor wound healing, signs of ischemia (cool extremity, prolonged venous filling time, shiny atrophied skin, nail changes)

Buerger test: Foot pallor with leg elevation (positive!)

88
Q

Peripheral arterial disease Dx:

A

ABI – ankle brachial index

Resting systolic blood pressure at ankle is compared to systolic brachial pressure. Ratio of the equals ABI

Segmental pressure, volume recordings, US, CT angiography, MRA

89
Q

Treatment of peripheral Arterial Disease:

A

oExercise (claudication exercise rehab)
o Cilostazol suppresses platelet aggregation and vasodilates
o Antiplatelet agents are superior to ASA, ASA still preferred bc cardioprotective.
o Pentoxiflylline – relieves claudication
o Angioplasty
o Surgery
o Lifestyle therapy first!