Intro & SCD Flashcards

1
Q

What is the OT process?

A
  1. Evaluation (which starts with referral)
    - You evaluate their patient’s occupations and learn about them through the occupational profile.
    - Based on the setting and your skills, ask yourself if you should accept the patient.
    - You need background knowledge about the diagnosis to use clinical reasoning to develop an intervention plan that will help the patient function.
  2. Intervention plan
    - based on evaluation, diagnosis, and patient desires
  3. Outcomes
    - based on the patient’s current performance, did they achieve the appropriate outcome or should you reevaluate
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2
Q

What are the core values of OT?

A
  • altruism
  • dignity
  • equality
  • freedom
  • justice
  • truth
  • prudence
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3
Q

What is altruism?

A

unselfish concern for the welfare of others (understanding)

ex: listening to the patient’s story even though you’re busy

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4
Q

What is dignity?

A

valuing worth and uniqueness of each person (empathy and respect)

EX: asking patient about their hobbies

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5
Q

What is equality?

A

individuals have the same rights and opportunities (fairness and impartiality)

EX: treating all people with no bias

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6
Q

What is freedom?

A

Allowing clients to have a choice, independence, initiative, and self direction

EX: allowing patient to choose between tasks

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7
Q

What is justice?

A

upholding moral and legal principles (equity, truth, objectivity)

EX: easy wheelchair access

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8
Q

What is truth?

A

faithfulness to facts and reality (accountable, honest, accurate)

EX: being honest with patient about likely outcomes

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9
Q

What is prudence?

A

ability to self govern and discipline

EX: supervising an unstable patient from sitting to standing

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10
Q

What are the 5 domains?

A
  1. Occupations
  2. Performance skills
  3. Client factors
  4. Performance patterns
  5. Contexts and environment
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11
Q

What is clinical reasoning?

A

the combination of knowledge of body systems and diagnoses, the OT profile, and the setting, combines with the core values of OT

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12
Q

Illness vs Disease

A

Disease: medical classification; something an organ has

Illness: personal factors, perception of suffering; something a man has

Cassell uses illness to mean “what a patient feels on the way to the doctor” and disease to mean “what a patient has on the way home.”

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13
Q

What are predisposing factors?

A
  1. heredity
    - CF, some cancers, sickle cell
  2. age
    - young: tonsillitis, otitis media (earache)
    - old: arthritis, senile dementia
  3. gender
    - prostate cancer, osteoporosis, CMC arthritis
  4. environment
    - rural vs city, noise, toxins
  5. lifestyle
    - diet, alcohol, drugs, stress
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14
Q

Epidemiology

A

study of the cause and distribution of the disease

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15
Q

Etiology

A

the cause of disease, agent, or event
- genetics
- acquired
- multifactorial

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16
Q

Pathophysiology

A

mechanism - what does the disease do? does it destroy or weaken the muscles?

biology

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17
Q

Clinical manifestations

A

physical signs, patient reports

what the patient tells you

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18
Q

Outcome

A

expected course of the disease
- prognosis: is the patient going to die in 6 weeks
- actual outcome
- remission: disease is still present but not progressing

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19
Q

Clinical intervention

A

healthcare professional

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20
Q

Prevention

A

Identify risk factors and reduce them

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21
Q

Treatment

A

optimal outcome, often therapy

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22
Q

Precautions

A

discomfort, injury, death

contraindications: risk outweighs the benefit
- hot pack on shoulder of patient with breast cancer might make shoulder feel better, but can cause the cancer to metastasize

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23
Q

What are the components of the disease process?

A
  1. epidemiology
  2. etiology
  3. pathophysiology
  4. clinical manifestations
  5. outcome
  6. clinical intervention
  7. prevention
  8. treatment
  9. precautions
24
Q

How do diseases/disorders occur?

A
  1. Trauma
  2. Hereditary
  3. Chromosomal
  4. Idiopathic
  5. Iatrogenic
  6. Nosocomial
  7. Multifactorial
  8. Infection and inflammation related
  9. Physical agents
  10. Cancers/ neoplasm
  11. Immunity related
25
Q

Inflammation

A

Natural response to tissue damage necessary for the healing process
- changes pressure to slow internal bleeding
- causes pain to tell the body to stop

May or may not come with infection

Lack of inflammatory response can indicate a disease such as AIDS

Occurs with trauma, allergy, heat, and bacteria

Acute or chronic

For the most part, if you are hurting the patient, you are doing more harm than good.

Classic signs:
- redness, edema, heat, pain

26
Q

Infection

A

An invasion of a pathogenic microorganism that disturbs homeostasis
A foreign substance has invaded and is attacking. Infection is always a problem.

27
Q

Trauma

A

an injury or wound caused by external force or violence

28
Q

Physical and chemical agents

A

extremities of heat and cold

Types:
1. burns
- 1st degree: suburn
- 2nd degree: blister
- 3rd degree: damage to epidermis and dermis
- 4th degree: damage to underlying bone and or muscle

  1. frostbite
  2. radiation
  3. electric shock - runs through arteries, veins, and muscle
  4. bites
29
Q

Neoplasia and cancer

A

Neoplasm - means new growth or formation
- may form a tumor (clump of tissue)
- can be in blood (leukemia)

Benign - small to large, but stays within its margins
Malignant - spreads to other cells or tissues close by
Metastasis - spread to other unrelated parts by way of blood or lymphatic stream

30
Q

Describe immunity related diseases/disorders and the different types.

A

the body’s ability to fight off invasion

Types:
1. Natural and acquired
- natural: genetic - race, sex, genetics
- acquired: when the body develops an immunity
- active (when you got it)
- passive (immunization; maternal immunity occurs until baby is 8 weeks)

  1. Allergy
    - malfunction of the immunity system
    • anaphylaxis: life threatening
  2. Autoimmunity
    - the body attacks itself
    • IDDM (type I diabetes) and lupus
  3. Immunodeficiency
    - impaired immune system
    • AIDS
31
Q

What is sickle cell disease?

A

A group of inherited (genetic) disorders that impact red blood cells
- abnormal hemoglobin (carries O2)
- normal RBCs live 120 days, are round, soft, and flexible; but with SCD they life 20 days, are sticky, brittle, and crescent shaped which leads to O2 loss, pain, and stoke

32
Q

Sickle cell inheritance

A

Each person has a hemoglobin A trait from EACH parent
- If you have one that is the SCT, you are a carrier
- If you have TWO- you have SCD
- If you have NONE- you do not have the trait and cannot have or pass on the disease

33
Q

What are the different types of SCD?

A

Type depends on the type of hemoglobin
Most common is hemoglobin (SS)
- most severe
- inherit one sickle gene from each parent

Other types:
Hemoglobin SB
Hemoglobin SB+
Hemoglobin SB 0
Hemoglobin SD, SE, SO

34
Q

Where is SCD most common and why?

A

Most common in Africa, India, Latin America, the Middle East, and the Mediterranean

There is a link between SCD and malaria

Roughly 100,000 in the US
- 1 million carriers
- Genetic counseling can help stop the spread
7% of the world carries the trait

35
Q

Signs, symptoms, and complications of SCD

A

Slowing or blockage of blood to any part of the body

Pain crisis: miss 1-14 days of work/school, but are experiencing symptoms everyday

Dactylitis: inflammation of digits
- blood vessels are smaller so it’s easier for a sickle cell to turn and blood blood and lymph flow

Wounds: if an area doesn’t receive enough blood it will rot

Spleen (infection): spleen helps with immune system, infection, and RBCs

Respiratory and cardio systems: asthma and chest pain

Strokes
- Silent stroke: not noticeable, but seen on MRI
- Overt stokes:
- ischemic: lack of oxygen; SNAPCHAT blocks blood flow
- hemorrhagic: occurs after ischemic due to aneurism
* Thrombosis is a blood clot stuck to the vessel wall. If a piece breaks off, the piece is called an embolism. If the thrombosis keeps collecting RBCs, it will cause an ischemic stroke. The walls of the vessel will expand. This expansion is called an aneurism. If the aneurism ruptures, a hemorrhagic stroke can occur.

Visual deficits: retinopathy
- retina is affected, can cause blindness

Avascular necrosis
- bone loses blood, bone dies
- often in femur and lunate

36
Q

How does SCD impact function?

A

Slower to hit milestones
- higher resting energy expenditure
- smaller and slower to hit puberty

Miss opportunities
- 4-6 days per year in hospital

Intellectual deficits from stroke

Social impact
- ADHD and behavioral issues
- psychosocial factors (depression)
- labeled as “sick kid” and often the outcast among peers

Long term impact
- diminished executive function (planning, decision making, organization)
- memory showed decline faster than adults without SCD
- higher unemployment since they have to take sick days more often

37
Q

Systemic issues for those with SCD

A

Access to medical care
- health care and disparity

Testing
- went wrong in the 70s
- became mandatory in 1989

Less funding for research

Come from lower education levels and lack healthcare literacy

Job discrimination

Drug seekers
- often have medication for pain; if they go to a new doctor, they could be accused of drug seeking

38
Q

Mortality and management of SCD

A

In the US, most live to adulthood, but shortened by 10 years.
Medications:
* prophylactic penicillin: for those under 5 because they can’t communicate how they’re feeling
* opioids: during pain crisis
* hydroxyurea: cancer drug to build up RBCs
* contraception: progestin only are safe
* immunizations
* supplements (non iron based)

Worldwide die by the age of 5
Pediatric hematologists should be established in the first year and seen regularly. The issue with that is there are only 1,900 to 2,100 total in the US.

39
Q

Interventions for SCD

A

Ultrasound/MRI
- problem is they are expensive and hard to come by

Blood transfusions
- gold standard to prevent CVAs
- hemosiderosis (iron overload) is a possible side effect

Splenectomy
- removal after recurring splenomegaly (spleen inflammation)

Joint replacement
- after avascular necrosis

Bone marrow transplant
- experimental and has many risks

Gene therapy and stem cell options

39
Q

Interventions for SCD

A

Ultrasound/MRI
- problem is they are expensive and hard to come by

Blood transfusions
- gold standard to prevent CVAs
- hemosiderosis (iron overload) is a possible side effect

Splenectomy
- removal after recurring splenomegaly (spleen inflammation)

Joint replacement
- after avascular necrosis

Bone marrow transplant
- experimental and has many risks

Gene therapy and stem cell options

40
Q

Impact of SCD of occupational performance

A

ADLs - mostly independent
- impacted by acute pain, strokes, wounds, and avascular necrosis

IADLs
- executive functions
- supporting the client and parents

Spirituality and prayer
- patients who are spiritual have fewer hospitalizations
- area of acceptance

Health and management
- medication adherence routines and habits
- exercise and activity that fits them (avoid strenuous due to higher resting energy expenditure)

Sleep management

Education

Work

Play and leisure

41
Q

What are occupations?

A
  • ADLs, BADLs, IADLs
  • sleep, work, play, leisure, social participation
41
Q

What are performance skills?

A
  • motor (ROM, strength), process and social skills (comfort, anxiety)
  • building blocks of occupation
42
Q

What are client factors?

A

body function, structure, values, beliefs, personality

43
Q

What are performance patterns?

A

habits, routines, roles

44
Q

What are contexts and environments? (domain)

A

physical, personal, social, temporal, and virtual

45
Q

What is hereditary?

A
  • dominant, recessive, sex linked genetic material
  • sickle cell
  • happens before conception
46
Q

What is chromosomal?

A
  • defect in a chromosome (after conception)
  • trisomy of chromosome 21 (downs syndrome)
  • fragile x (worse in boys)
47
Q

What is idiopathic?

A

random

48
Q

What is iatrogenic?

A
  • caused by an intervention
  • pt with cancer does radiation and gets leukemia
49
Q

What is nosocomial?

A
  • get it from hospital
  • in hospital for heart attack, gets COVID
50
Q

What is multifactorial?

A
  • partly genetic, partly environmental
  • arthritis
51
Q

What are physical agents?

A
  • trauma
  • chemical burns
52
Q

What are the various methods of transmission of infections?

A
  1. fungi - through contact
    - histoplasmosis, thrush, ringworm
  2. rickettsial - bites from live, ticks, unsanitary conditions
    - rocky mountain spotted fever, lyme disease
  3. protozoa - single celled microorganism
    - malaria, trichomoniasis
  4. viruses
    - smallest microorganism (hardest to deal with)
    - can be dormant for long period of time
    - chicken pox, hepatitis, flu
  5. bacteria - single celled
    - can be useful
    - TB, whooping cough, syphilis
  6. parasites
    - requires a host
    - internal and external
    - lice, pin worms
53
Q

Types of trauma

A

Types:
1. head trauma
- TBI or CHI (closed head injury)
- concussion, cerebral contusion, skull, nose, jaw, or eardrum

  1. chest trauma
    - penetrating injuries
    - nonpenetrating injuries (rib fracture, aorta rupture)
  2. abdominopelvic trauma
    - penetrating or nonpenetrating
    - any trauma to the guts
  3. neck and spine
    - fracture, dislocation, contusion, compression
    - occurs at levels: osteological (what’s broken), functional, neurological (where you feel it)
  4. Extremities
    - fractures
    - tendon (muscle to bone), ligament (bone to bone), nerves (supply electricity for muscles to move), cartilage (lines articular surfaces and acts as shock absorber)