Interpretation of an ECG Flashcards

1
Q

calculating Bp from an ECG

A

300 divide by the

number of big squares per R – R interval.

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2
Q

QRS complex

A

<120 ms (0.12s) or wide >120 ms (0.12s)?

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3
Q

P waves

A

<0.25mV, and upright in II III and AVF

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4
Q

Arrhythmia - ATRIAL FIBRILATION

A

No discernible P waves and irregular QRS complexes

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5
Q

Arrhythmia - Atrial flutter

A

p waves can be seen as a rate of 300 per minute giving a sawtooth appearance, 4 p waves per QRS complex

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6
Q

nodal tachycardia

A

normal QRS but absent p waves

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7
Q

ventricular tachycardia

A

after two sinus beats the rate increases to 150 per minute, the QRS complexes become broad a t waves are difficult to identify

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8
Q

abnormal p wave - p-mitrale

A

Bifid P wave = left atrial hypertrophy

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9
Q

abnormal p wave - p-pulmonale

A

Peaked P wave = right atrial

hypertrophy

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10
Q

P-R Interval (Time Between Atrial and Ventricular Depolarisation)

A

Measure from the beginning of P to the beginning of Q wave

Normal range 120-200 ms (0.12-0.2 s)
Prolonged >200 ms (0.2 s) implies delayed AV conduction

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11
Q

If>120ms(0.12s) in QRS interval

A

Ventricular Conduction Defects
 Bundle Branch Block
 Left and Right Bundle Branch Block

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12
Q

Lowvoltage<5mm: in QRS complex

A

Hypothyroidism
 COAD
 Myocarditis
 Pericarditis and Pericardial effusion

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13
Q

Left Ventricular Hypertrophy:

A

 RwaveinV5 >25mm or
 SumoftheS
wave in V1 and R wave in V5 or V6 >35mm
 Sokolow-Lyon index

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14
Q

 Right Ventricular Hypertrophy:

A

 Dominant R wave in V1
 T wave inversion in V1 – V3 or V4
 Deep S wave in V6

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15
Q

Significant Q Wave

A

> 40ms(0.04s)
 Depth>2mm.
 Present couple of hours/days after acute MI
 If present in lead III consider PE.

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16
Q

QT interval

A

 Measure from start QRS to end of T wave – varies with rate.
 Corrected QTc Interval  QTc=QT/√RR
Normal 380-420 ms (0.38-0.42 sec)

17
Q

Prolonged QT Interval

A
 Acute Myocardial Ischaemia
 Myocarditis
 Bradycardia
 Head Injury
 Hypothermia
 U&amp;E Imbalance (K+ Ca2+ Mg2+ )
 Congenital
 Drugs (Quinidine, Antihistamines, Macrolides, Amiodarone, Phenothiazines)
18
Q

ST Segment

A

 Time from the end of ventricular
depolarisation to the start of ventricular repolarisation
 Usually Isoelectric

19
Q

T Wave (Ventricular Repolarisation)

A

Abnormal if inverted in I, II and V4 -V6 (Ischaemia /Infarction)

20
Q

T Wave Digoxin effects

A

 T Wave inversion

 ST segment sloping depression

21
Q

Axis

A

(normal between -30° and +90°)

22
Q

Left Axis Deviation (-30°to -90°)

A

 Negative QRS deflections in II and III

 LV Hypertrophy, MI

23
Q

Right Axis Deviation

A

 Negative QRS deflections in I

 RV Hypertrophy, PE, MI

24
Q

MI ECG Changes

A

T wave peaking followed by T wave inversion
 ST segment elevation
 Appearance of new Q waves

25
Q

MI ECG Localizing the Infarct

A
 AnteriorInfarction:
 Any of precordial leads (V1 through V6)
 LateralInfarction:
 Leads I, AVL, V5 and V6
 InferiorInfarction:
 Leads II, III, and AVF
 PosteriorInfarction:
 Reciprocal changes in lead V1 (ST- segment depression, tall R wave)