Images Flashcards

1
Q
A

Aphthous Stomatitis

(Canker sore)

  • Tx: Good oral hygiene, symptom relief (topical lidocaine), and/or avoidance of exacerbating factors (e.g. braces or habitual cheek biting)
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2
Q
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Allergic Rhinitis

(Transverse Nasal Crease)

  • 2/2 β€œallergic salute”
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3
Q
A

HSV-1 Gingivostomatitis

  • Orolabial herpes
  • Often the first presentation of primary HSV-1 infection
  • Inflammation of the oral muscoa & gingiva
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4
Q
A

Leukoplakia

(precancerous SCC)

  • Smoking, drinking, +/- other SCC risk factors
  • Does not scrape off like candida
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5
Q
A

Lymphedema

(Disruption of lymphatics)

  • Tx: Weight loss, elevation & compression, & physiotherapy (e.g. lymphatic drainage by massage)
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6
Q
A

Oral Candidiasis

(oral thrush)

  • DM
  • HIV/imcx
  • Glucocorticoids (e.g. long term asthma tx)
  • Abx
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7
Q
A

PJP

  • Patches of ground-glass opacity
  • Cystic lesions
  • Reticulation
  • Septal thickening
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8
Q
A

PJP

  • Patches of ground-glass opacity
  • Cystic lesions
  • Reticulation
  • Septal thickening
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9
Q
A

Pancoast tumor

(SST–Superior Sulcus Tumor)

  • P/w Horner syndrome (ptosis, miosis, anhydrosis; sympathetic nerve fiber injury)
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10
Q
A

ARDS

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11
Q
A

Asbestos

(Pleural plaques on CXR)

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12
Q
A

Aspergilloma

  • Chronic pulmonary aspergillosis - usually superimposed onto apical cavitary TB
  • Halo sign (surrounding ground-glass opacities) on CXR
  • Invasive aspergillosis - occurs in imcx setting (e.g. neutropenia, glucocorticoids, HIV)
  • +galactomannan, +ß-D-glucan
  • Tx: Voriconazole Β± casopofungin (an echinocandin)
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13
Q
A

Lung cancer

(Left)

  • Central: SCC or SCLC
  • Peripheral: Adenocarcinoma or LCC
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14
Q
A

PJP

  • Small pneumatocoeles and/or subpleural blebs (random scattered black pockets overlaying diffuse reticular pattern on CXR)
  • Fine, reticular interstitial pattern
  • Often perihilar distribution
  • Ground-glass on HRCT
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15
Q
A

Pleural Effusion

(2/2 Empyema in HIV pt)

  • Exudative = high protein, high LDH
  • Complicated = low glucose, low pH
  • Empyema = Exudative & Complicated parapneumonic effusion
  • Causes of exudative: IMP: Infection, Malignancy, PE
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16
Q
A

Aspergilloma

  • ​Chronic pulmonary aspergillosis - usually superimposed onto apical cavitary TB
  • Halo sign (surrounding ground-glass opacities) on CXR
  • Invasive aspergillosis - occurs in imcx setting (e.g. neutropenia, glucocorticoids, HIV)
  • +galactomannan, +ß-D-glucan
  • Tx: Voriconazole Β± casopofungin (an echinocandin)
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17
Q
A

Bacterial PNA

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18
Q
A

Aspergilloma

  • ​Chronic pulmonary aspergillosis - usually superimposed onto apical cavitary TB
  • Halo sign (surrounding ground-glass opacities) on CXR
  • Invasive aspergillosis - occurs in imcx setting (e.g. neutropenia, glucocorticoids, HIV)
  • +galactomannan, +ß-D-glucan
  • Tx: Voriconazole Β± casopofungin (an echinocandin)
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19
Q
A

Pancoast tumor

(SST–Superior Sulcus Tumor)

  • P/w Horner syndrome (ptosis, miosis, anhydrosis; sympathetic nerve fiber injury)
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20
Q
A

Lung Abscess

(Cavity w/ Air-Fluid Level; Thick-walled)

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21
Q
A

PE

(CT w/ contrast)

  • Shows pulmonary artery filling defect, which indicates acute PE
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22
Q
A

TB

(Reactivation TB)

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23
Q
A

PE

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24
Q
A

COPD

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25
Q
A

Mesothelioma

(Pleural thickening w/ effusion)

  • Adeno- still more common w/ asbestos exposure
26
Q
A

Alveolar bleb rupture from COPD

(SSP: Secondary Spontaneous PTX)

  • UL decreased breath sounds in pts with known COPD or CF
  • Hyper-resonance on percussion
  • Smoking
  • Destruction of alveolar sacs > formation of large alveolar blebs > rupture, leaking air into pleural space
27
Q
A

Cryptogenic Organizing PNA

(BL ground glass infiltrates)

28
Q
A

Lung Cancer

(Malignancy)

29
Q
A

PE

(Wedge-shaped, pleural-based opacification)

30
Q
A

Pulmonary Fibrosis

(ILD)

  • Honeycombing
  • If 2/2 Rx: ABBMNR
    • Amiodarone
    • Busulfan
    • Bleomycin
    • MTX
    • Nitrofurantoin
    • Radiation
31
Q
A

Lobar PNA

32
Q
A

Asbestosis

(Pleural Plaques)

33
Q
A

PTX

34
Q
A

PTX

35
Q
A

Superior Sulcus Tumor

(Pancoast Tumor)

  • P/w Horner syndrome (ptosis, miosis, anhidrosis)
36
Q

50M presents to ED w/ fevers, cough, hypoxia

A

Severe CAP

(Left hemithorax white-out)

  • Tx: CAP CAM = CTX + Azithromycin
  • or: Moxifloxacin
37
Q

55F

A

Lobar PNA (RLL)

38
Q

48F

A

Lobar PNA (RLL)

39
Q
A

Lobar PNA (RUL)

40
Q
A

Lobar PNA (RML)

41
Q
A

Lobar PNA (RML)

42
Q

1

A

SVC

43
Q

2

A

Ascending Aorta

44
Q

3

A

Main Pulmonary Artery

45
Q

4

A

L Pulmonary Artery

46
Q

5

A

Descending Aorta

47
Q

6

A

Thoracic Duct

48
Q

7

A

Azygos Vein

  • Runs up thoracic vertebral column from lumbar region and drains into SVC just above RA
49
Q

8

A

Esophagus

50
Q

9

A

R Pulmonary Artery

51
Q

A

A

Stomach

52
Q

C

A

Gallbladder

53
Q

E

A

Duodenum

54
Q

F

A

Portal Vein

  • Always next to IVC
  • Drains INTO liver from small intestine (SMV), large intestine (IMV), and spleen (splenic vein)
  • Formed by the superior mesenteric vein, inferior mesenteric vein, and splenic vein
55
Q

H

A

Descending Colon

56
Q
A

GBM

(Grade IV Astrocytoma)

  • Classic appearance on MRI imaging
  • Risk factors: Neurofibromatosis, Li Fraumeni syndrome, previous radiation therapy
  • Tx: Surgery, HD Steroids, Temozolomide (CTX)
  • High recurrence rate, low survival
  • Differs from Grade III in that it has:
    • necrotizing tissue
    • anaplastic cells (no differentiation)
    • hyperplastic blood vessels (proliferative)
57
Q
A

GBM

(Grade IV Astrocytoma)

  • Classic appearance on MRI imaging
  • Risk factors: Neurofibromatosis, Li Fraumeni syndrome, previous radiation therapy
  • Tx: Surgery, HD Steroids, Temozolomide (CTX)
  • High recurrence rate, low survival
  • Differs from Grade III in that it has:
    • necrotizing tissue
    • anaplastic cells (no differentiation)
    • hyperplastic blood vessels (proliferative)
58
Q
A

Epidural Hematoma

(Biconvex)

  • Middle Meningeal Artery rupture
  • Often w/ skull fracture
59
Q
A

Subdural Hematoma

(Bridging Vein Rupture)

  • Elderly, hx of fall, delayed sx
60
Q
A

Subarachnoid Hemorrhage (SAH)

(Aneurysm rupture 2/2 HTN or vascular disease)

  • Suspect SAH if HA reaches peak, severe intensity within seconds of onset or causes LOC
  • If CT negative but still suspect, and low suspicion for IIP, do LP
  • CSF:
    • Xanthochromia
    • Numerous RBCs
    • Elevated Pressure