Higher Cortical Function Flashcards

1
Q

What provides most of the inputs to the dense collection of neurones in the cortex?

A

Mostly Thalmic Inputs

cortex also communicates with other cortical areas

(spinothalamaic, visual etc…)

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2
Q

Which cell type (strucutre) provides most of the outputs from the cortex?

A

Pyramidal Cells

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3
Q

What are 3 broad functions of the frontal lobe?

A

Motor Output

Expression of Speech

Behavioural Regulation

Cognition (e.g. mental arythmetic)

Continence

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4
Q

What does the frontal lobe co-ordinate via the corticonuclear projections?

A

Eye Movements

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5
Q

Which hemisphere of the frontal lobe is usually responsible for expression of speech?

A

Left Hemisphere

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6
Q

TBC

How may a patient present with a frontal lobe lesion?

A
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7
Q

What are 3 major functions of the parietal lobes?

A

Sensory Perception

Comprehension of Speech

Body Image

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8
Q

Which visual pathway passes through the parietal lobe?

A

Superior Optic Radiation

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9
Q

Which lobe of the brain may be damaged when a patient develops neglect syndrome and outline how it may manifest?

A

Parietal Lobe

patient neglects half of their body / environment

  • only eating half a plate of food*
  • ignoring limbs on one side of the body*
  • only copying half a picuture of a clock face*
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10
Q

TBC

How may a patient present with a parietal lobe lesion?

A
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11
Q

What are 3 broad functions of the temporal lobe?

A

Hearing

Olfaction

Memory

Emotion

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12
Q

Which visual pathway passes through the temporal lobe?

A

Inferior Optic Radiations

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13
Q

Which specific part of the temporal lobe is responsible for laying down long term memories?

A

Hippocampus

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14
Q

TBC

How may a patient present with a temproal lobe lesion?

A
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15
Q

What is cerebral dominance and how can this be useful clinically?

A

certain functions are over-represented in one hemisphere of the brain

left hemisphere

language / mathematics

right hemisphere

body image / emotion / visuospatial awareness

a patient with difficulty speaking may have a left hemisphere lesion

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16
Q

What is the corpus callosum?

A

a large bundle of white matter connecting the two hemispheres

17
Q

What is the function of the Broca’s Area and Wernicke’s Area?

A

Broca’s Area

production of speech

Wernicke’s Area

interpretation of language

18
Q

Which structure connects the Broca’s and Wernicke’s area?

A

Arcuate Fasiculus

19
Q

Outline the pathway for repeating a heard word

A

Olfactory Nerve > Primary Auditory Cortex > Wernike’s Area > via arcuate fasiculus > Broca’s Area > Primary Motor Cortex

20
Q

Outline the pathway for repeating a written word

A

Optic Nerve > Primary Visual Cortex > Wernike’s Area > via arcuate fasiculus > Broca’s Area > Primary Motor Cortex

21
Q

Outline the pathway for speaking a ‘thought’

A

Multiple Cortical Inputs > Wernicke’s Area > via arcuate fasiculus > Broca’s Area > Primary Motor Cortex

22
Q

How would a patient present with Wernicke’s aphasia (dysphasia is more accurate)?

A

able to speaks fluently as Broca’s area functioning

speech is nonsensical as the Wernicke’s area is not functioning and therefore the patient cannot understand the question

23
Q

How would a patient present with Broca’s aphasia (dysphasia is more accurate)?

A

unable to speak fluently, speech is slow as Broca’s area not functioning

patient is able to understand the question and can write down answers

24
Q

What are the two classifications of memory?

(not short and long term)

A

Declarative

explicit/fact

Nondeclarative

implicit/motor skills/emotions

25
Where are memories stored within the brain?
distributed throughout large areas of the brain
26
In which two distinct regions of the brain are declarative and nondeclarative memories store?
**Declarative** cerebral cortex **Nondeclarative** Cerebellum
27
What factors can help to consolidate memories from the short term to the long term?
Emotional Context Rehersal Association
28
What type of memories is consolidated by the hippocampus and how is it believed to perform this role?
Consolidates Declarative Memories increases amount of neurotransmitter released, increases number of receptors at post-synpaptic juncton and increases the number of pre-synaptic branches to release neurotransmitter *"neuroplasticity"*