Heart Failure Flashcards

1
Q

cardiac output equation

A

SVxHR (5L/min)

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2
Q

heart failure

A

progressive disease from a condition that stops adequate cardiac output
risk factors: myocardial ischemia (most common), CAD, hypertension, cardiomyopathy
-involves multiple systems

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3
Q

s1 heart sound

A

mitral and tricuspid valve closing

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4
Q

s2 heart sound

A

aortic and pulmonic valve closing

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5
Q

s3 heart sound

A

ventricular filling during diastole

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6
Q

s4 heart sound

A

BAD

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7
Q

systolic dysfunction

A

less blood ejected/pumped out of the heart during systole (EF<45)
-decreased force, contraction, weakened and enlarged ventricles
-myocardial infarction from decreased contractibility and lost myocytes (heart cells) that don’t regenerate-high mortality

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8
Q

diastolic dysfunction

A

less blood filling during diastole (EF>50)
-CAD, hypertension, stiff and weak ventricles
-normal EF

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9
Q

ECHOS

A

show ejection fractions

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10
Q

BNP labs

A

found in the blood with stretching of arterial walls

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11
Q

heart failure manifestations

A

inadequate supply to brain, forward or backward failure

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12
Q

right-sided failure

A

inadequate RV contractions>systemic congestion since blood isn’t pumped to lungs
-usually from back flow of blood from left heart failure
-can be from infarctions or pulmonary embolism>makes RV pump harder to get blood through, or from pulmonary hypertension>makes RV pump harder from more resistance/after-load
-manifestations: JVD, edema, increased venous pressure, cor pulmonale, GI distress

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13
Q

left-sided heart failure

A

inadequate LV contractions>blood stays in pulmonary circulation and causes pulmonary congestion, edema, and decreased output
-causes: LV MI, hypertension, valve stenosis/insufficiency, nocturnal dyspnea, tachycardia, fatigue, cyanosis/hypoxia
-can cause right-sided heart failure from pulmonary congestion

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14
Q

pressures of the left heart

A

equal forces of COP, hydrostatic pressure
-hydrostatic “push” into the lungs
-COP “pull” fluid back into capillary
-COP pressure is from albumin

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15
Q

abnormal pressure of the left heart

A

decreased hydrostatic pressure/push causes too much fluid in the lung/pulmonary edema
-COP “pull” stays the same so not enough fluid is in the capillaries

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16
Q

biventricular heart failure

A

from LV failure that regresses to the RV
-decreased cardiac output, pulmonary congestion, systemic congestion

17
Q

chronic heart failure

A

decreased cardiac function from volume overload
-pulmonary and systemic congestion

18
Q

acute heart failure

A

rapid change in heart failure signs/symptoms
-pulmonary congestion from high LV filling

19
Q

compensation in heart failure

A

decreased output to renal system>RAAS activation>aldosterone, Na+, H20 retention>increased vascular volume>increased preload>Frank-Starling mechanism (increased force, contraction)
decreased output>sympathetic reflexes activated>increased BP from increased HR and contractility

20
Q

myocardial hypertrophy and remodeling

A

less affective compensatory mechanism
-from increased work
-remodeling=concentric, symmetric, eccentric

21
Q

HF diagnosis

A

FACES
-fatigue, activity limitation, congestion, edema, shortness of breath
chest X-ray, ECG, BNP and BNP plasma detection

22
Q

HF classification

A

classes 1-4:
1-no limitations/symptoms/SD
2-symptoms from activity/small SD
3-some symptoms all around/SD
4-severe symptoms all around/lot of SD

23
Q

HF treatment

A

ACEIs, aldosterone antagonists, B1 blockers
-diuretics help congestion and workload by reducing VV/preload
-pacemakers
-improvement of risk factors: hypertension, atherosclerosis

24
Q

heart failure in children

A

usually in infants and from structural (congenital) heart defects
-surgery of heart defects can cause heart failure

25
Q

aging and cardiac function

A

contributes to HR, doesn’t cause
-hard to diagnose
-increased vascular stiffness, LV hypertrophy