head injury Flashcards

1
Q

what is a primary brain injury

A

occurs at the moment of impact

target prevention, not treatable

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2
Q

what is secondary brain injury

A

effects of trauma on the brain that occur later, e.g. lack of cerebral perfusion, blood glucose, CO2, temp
secondary processes which occur at the cells and molecular level to exacerbate neurological damage

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3
Q

what processes can cause secondary brain injury

A
glutamate release
free radical generation 
calcium mediated damage
inflammatory response
mitochondrial dysfunction 
early gene activation
oedema 
opening of BBB
increased interstitial fluid pressure
neuronal death cascades
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4
Q

describe a graph of intracranial volume against intracranial pressure

A

as intercranial pressure rises pressure slowly increases too until the critical volume where pressure rises exponentially

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5
Q

how do you manage head injury

A

assessment of pt

investigations -CT head

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6
Q

who should be sent to hospital

A
extremes of age
amnesia
LoC
high energy injury 
vomiting 
seizure
bleeding/clotting disorder
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7
Q

describe the eye component of GCS

A

1 eyes do not open
2 open to pain
3 open to speech
4 open spontaneously

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8
Q

what is the verbal component of GCS

A
1 no response
2 incomprehensible sounds
3 innapropriate words
4 confused
5 oriented
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9
Q

what is the motor component of GCS

A
1 no response
2 extension to pain
3 abnormal flexion to pain 
4 normal flexion to pain 
5 localizes pain 
6 obeys command
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10
Q

what GCS constitutes a mild head injury

A

13-15

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11
Q

what GCS constitutes a moderate head injury

A

9-12

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12
Q

what GCS constitutes a severe head injury

A

8 or less

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13
Q

when should you request a ct head

A

GCS is less than 13 on initial assessment

gcs is less than 15 2 hours after injury

suspected open or depressed skull

basal skull fracture

vomiting

amnesia for events more than 30 mins before impact

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14
Q

what are red flags`

A
LoC
painful headache
vomiting 
visual disturbance
clear fluid from ear or nose
bleeding from ears
deafness
difficulty understanding 
loss of balance
weakness
difficulty walking
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15
Q

how do you treat convulsions in head injury

A

phenytoin

treat pyrexia

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16
Q

what PaCO2 is ideal

A

4.5-5 kPa

17
Q

what is CPP

A

cerebral perfusion pressure

18
Q

what is MAP

A

mean arterial pressure

diastolic pressure + 1/3 pulse pressure

19
Q

what is ICP

A

intra cranial pressure

20
Q

how do you calculate CPP

A

MAP - ICP

21
Q

what is cerebral autoregulation

A

maintains constant blood flow to brain between various MAP

this may be compromised in trauma/ ischaemia

22
Q

what parameters should be maintained after head injury

A

CPP above 60-70mmHg

systolic should be above 90mmHg

ICP less than 20 mmHg

23
Q

what should you do if the patient is hypotensive

A

not due to head injury

could be chest trauma
pelvic fracture

stop bleeding and give fluids

24
Q

what features suggest a risk of intracranial mass

A

high impact injury
retrograde amnesia
coagulopathy
seizure

gcs <12
signs of skull fracture

25
Q

what does periorbital bruising suggest

A

anterior cranial fossa fracure

26
Q

what does Battle’s sign suggest (bruising behind the ear

A

petrous temporal bone fracture

27
Q

what is an extradural haematoma associated with

A

skull fracture

middle meningeal artery or venous bleeding

good outcome if treated

28
Q

subdural haematoma

A

complicates 25% of head injuries

rupture of veins travelling from the brain to the saggital sinus

worse prognosis

29
Q

subarachnoid haemorrhage

A

ruptured aneurysm or head injury

30
Q

intracerebral head injury

A

contre coup injury

impact on side of skull

31
Q

what should you say when you contact the neurosurgeons

A
  • Mechanism of injury
  • Age of patient
  • Respiratory and cardiovascular status
  • GCS score & pupil response
  • Alcohol/drugs
  • Associated injuries
  • Results of CT scan
32
Q

what are the signs of herniation

A
dilated or unreactive pupils
extensory posturing (decerebrate injury)

decrease in gcs of 2 or more

33
Q

what does mannitol do

A

decreases blood viscosity
osmotic duretic
decrease icp

34
Q

what is the importance of glucose

A

tight control improves outcome
hypoglycaemia is dangerous

enteral nutritional support