Head injury Flashcards

1
Q

what is an extradural haematoma?

A

Bleeding into the space between the dura mater and the skull.

Often results from acceleration-deceleration trauma or a blow to the side of the head.

The majority of extradural haematomas occur in the temporal region where skull fractures cause a rupture of the middle meningeal artery.

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2
Q

features of extradural haematoma

A

Raised intracranial pressure

Some patients may exhibit a lucid interval

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3
Q

what is a subdural haematoma?

A

Bleeding into the outermost meningeal layer.

Most commonly occur around the frontal and parietal lobes.

May be either acute or chronic.

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4
Q

risk factors of subdural haematoma?

A

old age

alcoholism

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5
Q

onset of subdural haematoma in comparison to extradural haematoma

A

Slower onset of symptoms than a extradural haematoma.

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6
Q

what is a subarachnoid haemorrhage?

A

Usually occurs spontaneously in the context of a ruptured cerebral aneurysm, but may be seen in association with other injuries when a patient has sustained a traumatic brain injury.

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7
Q

when does a secondary brain injury occur?

A

cerebral oedema, ischaemia, infection, tonsillar or tentorial herniation exacerbates the original injury.

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8
Q

what is the cushings effect

A

(hypertension and bradycardia) often occurs late and is usually a pre terminal event

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9
Q

Mx of life threatening rising of ICP such as in an extradural haematoma

A

whilst theatre is prepared or transfer arranged use of IV mannitol/ frusemide

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10
Q

Mx of diffuse cerebral oedema

A

decompressive craniotomy

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11
Q

how are open depressed skull fractures managed

A

formal reduction and debridement

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12
Q

how are closed depressed skull fractures managed

A

non-operatively

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13
Q

when is ICP monitoring appropriate?

A

GCS 3-8

Normal CT scan

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14
Q

when is ICP monitoring mandatory

A

GCS 3-8

abnormal CT scan

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15
Q

what is the minimum cerebral perfusion pressure in adults?

A

70 mmHg

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16
Q

what is the minimum cerebral perfusion pressure in kids?

A

40-70 mmHg

17
Q

3rd nerve compression secondary to tentorial herniation pupils and light response

A

unilaterally dilated

sluggish or fixed

18
Q

poor CNS perfusion and bilateral 3rd nerve palsy pupils and light response

A

bilaterally dilated

sluggish or fixed

19
Q

optic nerve injury pupils and light reflex

A

unilaterally dilated or equal

cross reactive (Marcus-Gunn)

20
Q

OPiates, pontine lesions and metabolic encephalopathy pupils and light reflex

A

bilaterally constricted

maybe difficult to assess

21
Q

sympathetic pathway disruption pupils and light reflex

A

unilaterally constricted

preserved

22
Q

CT head with in 1 hour

A

GCS < 13 on initial assessment

GCS < 15 at 2 hours post-injury

suspected open or depressed skull fracture

any sign of basal skull fracture (haemotympanum, ‘panda’ eyes, cerebrospinal fluid leakage from the ear or nose, Battle’s sign).

post-traumatic seizure.

focal neurological deficit.

more than 1 episode of vomiting

23
Q

CT head with in 8 hours

A

age 65 years or older

any history of bleeding or clotting disorders including anticogulants

dangerous mechanism of injury (a pedestrian or cyclist struck by a motor vehicle, an occupant ejected from a motor vehicle or a fall from a height of greater than 1 metre or 5 stairs)

more than 30 minutes’ retrograde amnesia of events immediately before the head injury

23
Q

CT head with in 8 hours

A

age 65 years or older

any history of bleeding or clotting disorders including anticogulants

dangerous mechanism of injury (a pedestrian or cyclist struck by a motor vehicle, an occupant ejected from a motor vehicle or a fall from a height of greater than 1 metre or 5 stairs)

more than 30 minutes’ retrograde amnesia of events immediately before the head injury

24
Q

ct head and warfarin

A

perform a CT head scan within 8 hours of the injury