Fundamentals 29 Flashcards

1
Q

Vital Signs

A

Indicators of health status, these measures indicate the effectiveness of circulatory, respiratory, neural, and endocrine body functions.

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2
Q

Assessment of Vital Signs

A

provides data to identify nursing diagnoses, implement planned interventions, and evaluate outcomes of care.An alteration in vital signs signals a change in physiological function and the need for medical or nursing intervention

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3
Q

Vital sign ranges for an adult

A

Acceptable Ranges for Adults

Temperature Range: 36° to 38° C (96.8° to 100.4° F)

Respirations

12 to 20 breaths/min

Average oral/tympanic: 37° C (98.6° F)

Average rectal: 37.5° C (99.5° F)

Average axillary: 36.5° C (97.7° F)

Blood Pressure

Pulse

Average: <80mmHg

Pulse pressure: 30 to 50mmHg

60 to 100 beats/min

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4
Q

When do you measure vital signs?

A
  • On admission to a health care facility
  • When assessing a patient during home care visits
  • In a hospital on a routine schedule according to the health care provider’s order or hospital standards of practice
  • Before and after a surgical procedure or invasive diagnostic procedure
  • Before, during, and after a transfusion of blood products
  • Before, during, and after the administration of medication or therapies that affect cardiovascular, respiratory, or temperature-control functions
  • When a patient’s general physical condition changes (e.g., loss of consciousness or increased intensity of pain)
  • Before and after nursing interventions influencing a vital sign (e.g., before a patient previously on bed rest ambulates or before a patient performs range-of-motion exercises)
  • When a patient reports nonspecific symptoms of physical distress (e.g., feeling “funny” or “different”)
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5
Q

Use the following guidelines to incorporate measuring vital signs in the nursing practice.

A

The nurse caring for the patient is responsible for measurement of vital signs. Although you sometimes delegate measurement of selected vital signs (i.e., in stable patients), as a nurse you need to analyze them to interpret their significance and make decisions about interventions.

  • Ensure that equipment is functional and appropriate for the size and age of the patient. Equipment used to measure vital signs (e.g., a thermometer) needs to work properly to obtain accurate findings.
  • Select equipment based on the patient’s condition and characteristics (e.g., do not use an adult-size BP cuff for a child).
  • Know the patient’s usual range of vital signs. These values can differ from the acceptable range for that age or physical state. The patient’s usual values serve as a baseline for comparison with later findings. Thus you are able to detect a change in condition over time.
  • Determine the patient’s medical history, therapies, and prescribed medications. Some illnesses or treatments cause predictable changes in vital signs. Some medications affect one or more vital signs.
  • Control or minimize environmental factors that affect vital signs. For example, assessing the patient’s temperature in a warm, humid room may yield a value that is not a true indicator of his or her condition.
  • Use an organized, systematic approach when taking vital signs. Each procedure requires a step-by-step approach to ensure accuracy.
  • Based on the patient’s condition, collaborate with health care providers to decide the frequency of vital sign assessment. In the hospital, health care providers order a minimum frequency of vital sign measurements for each patient. Following surgery or treatment intervention you measure vital signs more frequently to detect complications. In a clinic or outpatient setting you take vital signs before the health care provider examines the patient and after any invasive procedures. As a patient’s physical condition worsens, it is often necessary to monitor vital signs as often as every 5 to 10 minutes. The nurse is responsible for judging whether more frequent assessments are necessary (Box 29-2).
  • Use vital sign measurements to determine indications for medication administration. For example, give certain cardiac drugs only within a range of pulse or BP values. Administer antipyretics when temperature is elevated outside of the acceptable range for the patient. Know the acceptable ranges for your patients before administering medications.
  • Analyze the results of vital sign measurement. Vital signs are not interpreted in isolation. You need to also know related physical signs or symptoms and be aware of the patient’s ongoing health status.
  • Communicate significant changes in vital signs to the patient’s health care provider or the charge nurse. Document findings and compare with baseline measurements to identify significant changes. When vital signs appear abnormal, have another nurse or health care provider repeat the measurement to verify readings.
  • Instruct the patient or family caregiver in vital sign assessment and the significance of findings
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6
Q

Core Temperature

A

(temperature of the deep tissues) relatively constant, despite extremes in environmental conditions and physical activity

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7
Q

Thermoregulation

A

Physiological and behavioral mechanisms regulate the balance between heat lost and heat produced

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8
Q

Hypothalamus

A

located between the cerebral hemispheres, controls body temperature the same way a thermostat works in the home. Rise in temperature shuts the system down.

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9
Q

Anterior Hypothalamus

A

controls heat loss, and when nerve cells in the anterior hypothalamus become heated beyond the set point, impulses are sent out to reduce body temperature. Mechanisms of heat loss include sweating, vasodilation (widening) of blood vessels, and inhibition of heat production. The body redistributes blood to surface vessels to promote heat loss

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10
Q

Posterior Hypothalamus

A

senses that body temperature is lower than the set point, the body initiates heat-conservation mechanisms. Vasoconstriction (narrowing) of blood vessels reduces blood flow to the skin and extremities. Compensatory heat production is stimulated through voluntary muscle contraction and muscle shivering. When vasoconstriction is ineffective in preventing additional heat loss, shivering begins. Disease or trauma to the hypothalamus or the spinal cord, which carries hypothalamic messages, causes serious alterations in temperature control.

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11
Q

Basal Metabolic Rate(BMR)

A

accounts for the heat produced by the body at absolute rest and depends on the body surface area.

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12
Q

How do thyroid hormones affect BMR?

A

Thyroid hormones increase the rate of chemical reactions in almost all cells of the body by promoting the breakdown of body glucose and fat.

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13
Q

What happens when large amounts of thyroid hormones are secreted?

A

BMR can increase 100% above normal

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14
Q

What happens in the absence of thyroid hormones?

A

The BMR is reduced by half, causing a decrease in heat production.

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15
Q

Radiation

A

the transfer of heat from the surface of one object to the surface of another without direct contact between the two. As much as 85% of the surface area of the human body radiates heat to the environment.

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16
Q

Peripheral Radiation

A

increases blood flow from the internal organs to the skin to increase radiant heat loss.

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17
Q

Peripheral Constriction

A

minimizes radiant heat loss.

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18
Q

Conduction

A

the transfer of heat from one object to another with direct contact. Solids, liquids, and gases conduct heat through contact

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19
Q

How does the body gain heat?

A

when it makes contact with materials warmer than skin temperature (e.g., application of an aquathermia pad).

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20
Q

Convection

A

the transfer of heat away by air movement. A fan promotes heat loss through convection. Convective heat loss increases when moistened skin comes into contact with slightly moving air.

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21
Q

Evaporation

A

the transfer of heat energy when a liquid is changed to a gas. The body continuously loses heat by evaporation. Approximately 600 to 900mL a day evaporates from the skin and lungs, resulting in water and heat loss.

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22
Q

What happens when the body temperature rises?

A

the anterior hypothalamus signals the sweat glands to release sweat through tiny ducts on the surface of the skin. Sweat evaporates, resulting in heat loss. During physical exercise over 80% of the heat produced is lost by evaporation

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23
Q

Diaphoresis

A

visible perspiration primarily occurring on the forehead and upper thorax, although you can see it in other places on the body.

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24
Q

How much fluid is lost for each hour of exercise in hot conditions?

A

approximately 1L of body fluid is lost in sweat

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25
Q

What happens if the core temperature is too high?

A

the hypothalamus inhibits vasoconstriction. As a result, blood vessels dilate, and more blood reaches the surface of the skin.

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26
Q

What happens if the core temperature is too low?

A

the hypothalamus initiates vasoconstriction, and blood flow to the skin lessens to conserve heat

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27
Q

What does the ability of a person to control body temperature depend on

A

(1) the degree of temperature extreme, (2) the person’s ability to sense feeling comfortable or uncomfortable, (3) thought processes or emotions, and (4) the person’s mobility or ability to remove or add clothes. Individuals are unable to control body temperature if any of these abilities is lost.

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28
Q

Why are older adult particularly sensitive to temperature extremes?

A

because of deterioration in control mechanisms, particularly poor vasomotor control (control of vasoconstriction and vasodilation), reduced amounts of subcutaneous tissue, reduced sweat gland activity, and reduced metabolism.

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29
Q

Fever or pyrexia

A

occurs because heat-loss mechanisms are unable to keep pace with excessive heat production, resulting in an abnormal rise in body temperature. A fever is usually not harmful if it stays below 39° C (102.2° F)

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30
Q

Pyrogens

A

such as bacteria and viruses elevate body temperature. Pyrogens act as antigens, triggering immune system responses. The hypothalamus reacts to raise the set point, and the body responds by producing and conserving heat.

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31
Q

Afebrile

A

When the fever “breaks.”

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32
Q

Fever is an important defense mechanism.

A

Mild temperature elevations as high as 39° C (102.2° F) enhance the immune system of the body. During a febrile episode white blood cell production is stimulated. Increased temperature reduces the concentration of iron in the blood plasma, suppressing the growth of bacteria. Fever also fights viral infections by stimulating interferon, the natural virus-fighting substance of the body.

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33
Q

Cellular Hypoxia

A

Inadequate oxygen.

34
Q

Myocardia Hypoxia

A

produces angina (chest pain)

35
Q

Cerebral Hypoxia

A

Produces confusion.

36
Q

Hyperthermia

A

An elevated body temperature related to the inability of the body to promote heat loss or reduce heat production.Whereas fever is an upward shift in the set point, hyperthermia results from an overload of the thermoregulatory mechanisms of the body.

37
Q

Heatstroke is 40° C (104° F)

A

Heatstroke is a dangerous heat emergency with a high mortality rate. Patients at risk include the very young or very old and those who have cardiovascular disease, hypothyroidism, diabetes, or alcoholism. Also at risk are those who take medications that decrease the ability of the body to lose heat (e.g., phenothiazines, anticholinergics, diuretics, amphetamines, and beta-adrenergic receptor antagonists) and those who exercise or work strenuously (e.g., athletes, construction workers, and farmers).

38
Q

Signs and symptoms of Heatstroke:

A

giddiness, confusion, delirium, excess thirst, nausea, muscle cramps, visual disturbances, and even incontinence. Vital signs reveal a body temperature sometimes as high as 45° C (113° F), with an increase in heart rate (HR) and lowering of BP. The most important sign of heatstroke is hot, dry skin. Victims of heatstroke do not sweat because of severe electrolyte loss and hypothalamic malfunction. If the condition progresses, the patient with heatstroke becomes unconscious, with fixed, nonreactive pupils. Permanent neurological damage occurs unless cooling measures are rapidly started.

39
Q

Heat exhaustion

A

occurs when profuse diaphoresis results in excess water and electrolyte loss. Caused by environmental heat exposure, the patient exhibits signs and symptoms of fluid volume deficit (see Chapter 41). First aid includes transporting him or her to a cooler environment and restoring fluid and electrolyte balance.

40
Q

Hypothermia

A

Heat loss during prolonged exposure to cold overwhelms the ability of the body to produce heat, causing hypothermia. skin temperature drops below 35° C (95° F), the patient suffers uncontrolled shivering, loss of memory, depression, and poor judgment. As the body temperature falls below 34.4° C (94° F), HR, respiratory rate, and BP fall. The skin becomes cyanotic. Patients experience cardiac dysrhythmias, loss of consciousness, and unresponsiveness to painful stimuli if hypothermia progresses.

41
Q

Frostbite

A

Ice crystals form inside the cell, and permanent circulatory and tissue damage occursThe injured area becomes white, waxy, and firm to the touch. The patient loses sensation in the affected area. Interventions include gradual warming measures, analgesia, and protection of the injured tissue.

42
Q

Assessment

A

During the assessment process thoroughly assess each patient, explore his or her beliefs and experiences, and critically analyze findings to ensure that you make patient-centered clinical decisions.

43
Q

Nursing Diagnosis

A

After concluding your assessment, cluster defining characteristics to form a nursing diagnosis.Implement actions to minimize or eliminate the risk factors.

44
Q

Planning

A

Integrate the knowledge gathered from assessment and the patient history to develop an individualized plan of care. Match the patient’s needs with interventions that are supported and recommended in the clinical research literature. Teach the patient and caregiver the importance of thermoregulation and actions to take during excessive environmental heat

45
Q

Implementation

A

Initiate and complete the nursing actions necessary to help the patient achieve health care goals.

46
Q

Acute Care /Fever

A

When an elevated body temperature develops, initiate interventions to treat fever. The objective of therapy is to increase heat loss, reduce heat production, and prevent complications. The choice of interventions depends on the cause; adverse effects; and the strength, intensity, and duration of the temperature elevation. Nurses are essential in assessing and implementing temperature-reducing strategies.

47
Q

Nursing interventions for Patients with a fever:

A

Obtain blood cultures (before beginning antibiotics) if ordered. Obtain blood specimens to coincide with temperature spikes when the antigen-producing organism is most prevalent.

  • Minimize heat production: reduce the frequency of activities that increase oxygen demand such as excessive turning and ambulation; allow rest periods; limit physical activity.
  • Maximize heat loss: reduce external covering on patient’s body without causing shivering; keep clothing and bed linen dry.
  • Satisfy requirements for increased metabolic rate: provide supplemental oxygen therapy as ordered to improve oxygen delivery to body cells; provide measures to stimulate appetite and offer well-balanced meals; provide fluids (at least 8 to 10 8-oz glasses for patients with normal cardiac and renal function) to replace fluids lost through insensible water loss and sweating.
  • Promote patient comfort: encourage oral hygiene because oral mucous membranes dry easily from dehydration; control temperature of the environment without inducing shivering; apply damp cloth to patient’s forehead.
  • Identify onset and duration of febrile episode phases: examine previous temperature measurements for trends.
  • Initiate health teaching as indicated.
  • Control environmental temperature to 21° to 27° C (70° to 80° F)
48
Q

Antipyretics

A

are medications that reduce fever. Nonsteroidal antiinflammatory drugs such as acetaminophen, salicylates, indomethacin, and ketorolac reduce fever by increasing heat loss. Corticosteroids reduce heat production by interfering with the immune system and mask signs of infection. They are not used to treat a fever. However, they can suppress fever in response to a pyrogen.

49
Q

Medications such as meperidine or butorphanol:

A

Reduce shivering.

50
Q

Cardiac Output

A

volume of blood pumped by the heart during 1 minute which is the product of HR and the stroke volume (SV) of the ventricle.

51
Q

Apical pulse

A

If the radial pulse is abnormal or intermittent resulting from dysrhythmias or if it is inaccessible because of a dressing or cast, assess the apical pulse. When a patient takes medication that affects the HR, the apical pulse provides a more accurate assessment of heart function. The brachial or apical pulse is the best site for assessing an infant’s or young child’s pulse because other peripheral pulses are deep and difficult to palpate accurately.

52
Q

Acceptable ranges of Heart Rate

A

AGE

HEART RATE (BEATS/MIN)

Infant

120-160

Toddler

90-140

Preschooler

80-110

School-age child

75-100

Adolescent

60-90

Adult

60-100

53
Q

Tachycardia

A

abnormally elevated HR, above 100 beats/min in adults.

54
Q

Bradycardia

A

is a slow rate, below 60 beats/min in adults.

55
Q

Pulse deficit

A

An inefficient contraction of the heart that fails to transmit a pulse wave to the peripheral pulse site

56
Q

How to assess a pulse deficit:

A

you and a colleague assess radial and apical rates simultaneously and then compare rates. The difference between the apical and radial pulse rates is the pulse deficit. For example, an apical rate of 92 with a radial rate of 78 leaves a pulse deficit of 14 beats. Pulse deficits are often associated with abnormal rhythms.

57
Q

Dysrhythmia

A

interval interrupted by an early or late beat or a missed beat indicates an abnormal rhythm. A dysrhythmia threatens the ability of the heart to provide adequate cardiac output, particularly if it occurs repetitively.

58
Q

Strength or amplitude of pulse

A

reflects the volume of blood ejected against the arterial wall with each heart contraction and the condition of the arterial vascular system leading to the pulse site. Normally the pulse strength remains the same with each heartbeat. Document the pulse strength as bounding (4+); full or strong (3+); normal and expected (2+); diminished or barely palpable (1+); or absent (0). Include assessment of pulse strength in the assessment of the vascular.

59
Q

Respiration

A

the mechanism the body uses to exchange gases between the atmosphere and the blood and the blood and the cells.

60
Q

Ventilation

A

the movement of gases in and out of the lungs

61
Q

Perfusion

A

(the distribution of red blood cells to and from the pulmonary capillaries.

62
Q

Hypoxemia

A

low levels of arterial O2.

63
Q

Tidal Volume

A

The diaphragm moves approximately 1cm ( inch), and the ribs retract upward from the midline of the body approximately 1.2 to 2.5cm ( to 1 inch). During a normal, relaxed breath, a person inhales 500mL of air.

64
Q

Drop in respirations occurring in a patient after head trauma.

A

often signifies injury to the brainstem

65
Q

A longer expiration phase

A

s evident when the outward flow of air is obstructed (e.g., asthma)

66
Q

Acceptable Ranges of Respiratory Rate

A

AGE

RATE (BREATHS/MIN)

Newborn

35-40

Infant (6 months)

30-50

Toddler (2 years)

25-32

Child

20-30

Adolescent

16-20

Adult

12-20

67
Q

Pulse assessment determines the general state of cardiovascular health and the response of the body to other system imbalances. Tachycardia, bradycardia, and dysrhythmias are defining characteristics of many nursing diagnoses, including the following:

A

Activity intolerance

  • Anxiety
  • Decreased cardiac output
  • Fear
  • Deficient/excess fluid volume
  • Impaired gas exchange
  • Hyperthermia
  • Hypothermia
  • Acute pain
  • Ineffective peripheral tissue perfusion
68
Q

Alterations in Breathing Pattern:

A

Bradypnea

Rate of breathing is regular but abnormally slow (less than 12 breaths/min).

Tachypnea

Rate of breathing is regular but abnormally rapid (greater than 20 breaths/min).

Hyperpnea

Respirations are labored, increased in depth, and increased in rate (greater than 20 breaths/min) (occurs normally during exercise).

Apnea

Respirations cease for several seconds. Persistent cessation results in respiratory arrest.

Hyperventilation

Rate and depth of respirations increase. Hypocarbia sometimes occurs.

Hypoventilation

Respiratory rate is abnormally low, and depth of ventilation is depressed. Hypercarbia sometimes occurs.

Cheyne-Stokes respiration

Respiratory rate and depth are irregular, characterized by alternating periods of apnea and hyperventilation. Respiratory cycle begins with slow, shallow breaths that gradually increase to abnormal rate and depth. The pattern reverses; breathing slows and becomes shallow, climaxing in apnea before respiration resumes.

Kussmaul’s respiration

Respirations are abnormally deep, regular, and increased in rate.

Biot’s respiration

Respirations are abnormally shallow for two to three breaths followed by irregular period of apnea.

69
Q

Oxygen Saturation

A

The amount of hemoglobin fully saturated with oxygen, given as a percent value.

70
Q

The percent of saturation of hemoglobin (or SaO2)

A

Is the percent of hemoglobin that is bound with oxygen in the arteries.It is usually between 95% and 100%.

71
Q

SaO2 is affected by:

A

factors that interfere with ventilation, perfusion, or diffusion (see Chapter 40). The saturation of venous blood (SvO2) is lower because the tissues have removed some of the oxygen from the hemoglobin molecules. Factors that interfere with or increase tissue oxygen demand affect the usual value for SvO2, which is 70%

72
Q

Measurement with the Pulse oximeter:

A

The LED emits light wavelengths that the oxygenated and deoxygenated hemoglobin molecules absorb differently. The photodetector detects the amount of oxygen bound to hemoglobin molecules, and the oximeter calculates the pulse saturation (SpO2). SpO2 is a reliable estimate of SaO2 when the SaO2 is over 70%. Values obtained with pulse oximetry are less accurate at saturations less than 70%

73
Q

Respiratory assessment data are defining characteristics of many nursing diagnoses, including the following:

A

Activity intolerance

  • Ineffective airway clearance
  • Anxiety
  • Ineffective breathing pattern
  • Impaired gas exchange
  • Acute pain
  • Ineffective peripheral tissue perfusion
  • Dysfunctional ventilatory weaning response
74
Q

Blood Pressure

A

is the force exerted on the walls of an artery by the pulsing blood under pressure from the heart. Blood flows throughout the circulatory system because of pressure changes. It moves from an area of high pressure to one of low pressure. Blood pressure reflects the interrelationships of cardiac output, peripheral vascular resistance, blood volume, blood viscosity, and artery elasticity.

75
Q

Systolic Pressure

A

The peak of maximum pressure when ejection occurs

76
Q

Diastolic Pressure

A

When the ventricles relax, the blood remaining in the arteries exerts a minimum.Diastolic pressure is the minimal pressure exerted against the arterial walls at all times.

77
Q

Diuretics

A

Furosemide (Lasix), spironolactone (Aldactone), metolazone, polythiazide, benzthiazide. These Lowers blood pressure by reducing resorption of sodium and water by the kidneys, thus lowering circulating fluid volume.

78
Q

Beta-adrenergic blockers

A

Atenolol (Tenormin), nadolol (Corgard), timolol maleate (Blocadren), propranolol (Inderal). These Combines with beta-adrenergic receptors in the heart, arteries, and arterioles to block response to sympathetic nerve impulses; reduces heart rate and thus cardiac output.

79
Q

Vasodilators

A

Hydralazine hydrochloride (Apresoline), minoxidil (Loniten). Acts on arteriolar smooth muscle to cause relaxation and reduce peripheral vascular resistance.

80
Q

Calcium channel blockers

A

Diltiazem (Cardizem, Dilacor XR), verapamil hydrochloride (Calan SR), nifedipine (Procardia), nicardipine (Cardene).Reduces peripheral vascular resistance by systemic vasodilation.

81
Q

Angiotensin-converting enzyme (ACE) inhibitors

A

Captopril (Capoten), enalapril (Vasotec),Lowers blood pressure by blocking the conversion of angiotensin I to angiotensin II, preventing vasoconstriction; reduces aldosterone production and fluid retention, lowering circulating fluid volume lisinopril (Prinivil, Zestril), benazepril (Lotensin).

82
Q

Angiotensin-II receptor blockers (ARBs)

A

Losartan (Cozaar), olmesartan (Benicar). Lowers blood pressure by blocking the binding of angiotensin II, which prevents vasoconstriction.