FNP - Concept of Stress, Adaptation, and Homeostasis Flashcards

1
Q

An experience a person is exposed to,
through a stimulus; A condition in which a person experiences changes in the normal
balanced state

A

Stress

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2
Q

Characteristics of Stress (5)

A

Not a nervous energy (not from the nerves)
Man acts as a unified whole
Stress not always results to feeling
of distress (distress - damaging to health)
Stress not always due to tissue injury
Stress not always to be avoided at entirely (bc of eustress)

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3
Q

Models of Stress

A

Stimulus-based (Holmes, Rahe)
Response-based (Selye)
Transaction-based (Lazarus, Folkman)
Adaptation Model of Stress (Mechanic)

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4
Q

who made the stimulus-based model of stress

A

Holmes and Rahe

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5
Q

model of stress; Disturbing or disruptive characteristics within the environment, anything in environment that disrupts our balance

A

Stimulus-based

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6
Q

who made the response-based model of stress

A

Selye

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7
Q

model of stress; a nonspecific response of the
body to any demand made upon it, our body (including organs) are
the only ones affected

A

response-based

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8
Q

who made the transaction-based model of stress

A

Lazarus and Folkman

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9
Q

model of stress; perceptual response rooted in psychological and cognitive process, includes the mind, emotions, and way of thinking

A

transaction-based

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10
Q

model of stress; Stress is an anxiety provoking stimuli

A

Adaptation Model of Stress

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11
Q

difference of transaction base to adaptation model of stress

A

transaction based - mind, emotions, and way of thinking
adaptation model - emotions only

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12
Q

model of stress; ex. change in weather, pollution

A

Stimulus-based

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13
Q

model of stress; ex. The heat of the sun stimulates sweating or sunburn

A

response-based

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14
Q

model of stress; ex. When you’re taking a test (mental blocks)

A

Transaction-based

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15
Q

model of stress; ex. Anxiousness in test

A

Adaptation Model of Stress

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16
Q

A mechanism in which deviation from normal are sensed and counteracted, Balance/ Equilibrium or constancy while continually changing

A

homeostasis

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17
Q

A process of modifying to meet new/changing or different conditions, Alteration in functioning to maintain balance

A

Adaptation

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18
Q

Characteristics of the Adaptive Mechanisms (8)

A

Automatic
Requires time and energy
Tiring and Egocentric
Entire body is involved
With limits
Varies from person to person
May be inadequate or excessive
Attempts to maintain homeostasis

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19
Q

Factors affecting Stress Response (4)

A

Intensity
Scope
Duration
Number and Nature of stressors

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20
Q

PHYSIOLOGICAL ADAPTATION

A

Local Adaptive Syndrome (LAS)
General Adaptive Syndrome (GAS)

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21
Q

purpose of LAS

A

Prevent spread of infection
Localize the injury
Promotes healing

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22
Q

4 stages of LAS

A

Vascular stage
Cellular Stage
Exudative Stage
Reparative stage

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23
Q

Vascular stage (3)

A

Vasoconstriction
Vasodilation
Increase capillary permeability

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24
Q

Vascular stage; Platelet and thromboxane (cell) at the site of injury to stop bleeding

A

Vasoconstriction

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25
Vascular stage; Histamine (hormone) will be produced at the site of injury (redness or rubor and color or calor)
Vasodilation
26
Vascular stage; Tumor (swelling at the site of injury), dolor or pain, temporary loss of function
Increase capillary permeability
27
Cellular stage
Margination Diapedesis Emigration Chemotaxis
28
cellular stage; because of capillary permeability, solid will enter tight capillary walls in order to get to the site of injury
Diapedesis
29
cellular stage; proteins secreted in site of injury to signal cells
Chemotaxis
30
WBC will have to go to the site of injury
Positive
31
WBC moves away from the site of injury (retreats as it is unable to deal with the microorganisms) causing an extension of injury
Negative
32
Exudative stage (5)
Serous Sanguinous Purulent Serosanguinous Purosaguinous
33
Exudative stage; Watery fluid secretions; blister formation
serous
34
Exudative stage; blood clotting; red in color;
sanguineous
35
Exudative stage; pus formation; yellow in color (Example: Acne)
purulent
36
Exudative stage; Watery red
Serosanguinous
37
Exudative stage; yellow with red fluid
Purosaguinous
38
Reparative Stage (3)
Primary Intention or Regeneration Secondary Intention or Scar Formation Tertiary Intention or Intentional Delay in Wound Closure
39
Reparative Stage; damaged cells replaced with original cells
Primary Intention or Regeneration
40
Reparative Stage; damaged cells replaced with fibrous tissues
Secondary Intention or Scar Formation
41
Reparative Stage; Insertion of latex for drainage ex. Example: Ruptured appendicitis is placed with a tube for drainage, Jackson Pratt Drain
Tertiary Intention or Intentional Delay in Wound Closure
42
increase in WBC count in the blood
Leukocytosis
43
swelling or enlargement of the lymph nodes
Lymphadenopathy
44
Organs Primarily Involved in GAS
Medulla Oblongata Reticular Formation Endocrine Glands
45
controls vital functions for survival
medulla oblongata
46
clusters of neuron in the brainstem and spinal cord; connections with sensory and motor parts
reticular formation
47
supplies hormones functions
Endocrine Glands
48
stages of GAS
alarm resistance exhaustion
49
alarm stage of GAS
Awareness of stressor Mobilization of defenses Increased hormones Increased Vital Sign Decreased resistance SNS predominates
50
resistance stage of GAS
Adaptation occurs - balance as hormones circulate Normalization of hormones and Vital Sign Homeostasis occurs Increased resistance PNS predominates
51
exhaustion stage of GAS
Decreased energy Breakdown in (-) feedback mechanism Damaged organ Decreased physiological function
52
Sympatho-Adreno-Medulary Response
Epinephrine and norepinephrine increased body activities (e.g., increased VS depending on what is affected)
53
Effect of SNS and adrenal medulla on () - Heightened alertness restlessness because of increased blood flow
Heightened alertness restlessness because of increased blood flow
54
Effect of SNS and adrenal medulla on () - Increased blood flow in deeper organs; there will be peripheral vasoconstriction (cold and clammy feeling of skin)
blood vessels
55
Effect of SNS and adrenal medulla on () - Increased rate of pump of blood (heart rate) to accommodate increase in blood flow needed by different organs in response to stress
heart
56
Effect of SNS and adrenal medulla on () -Dilation of blood vessels will result in the increase of respiratory rate (RR)
lungs
57
Effect of SNS and adrenal medulla on () -Increase in blood flow (where nutrients, glucose and hormones are found)
skeletal muscles
58
Effect of SNS and adrenal medulla on () - Increased activity but less utilization of glucose (patient in alarm stage is in a state of hyperglycemia)
pancreas
59
Anterior Pituitary gland will release adrenocorticotropic hormone (ACTH) stimulating the adrenal cortex to release aldosterone and cortisol
Adrenocortical response
60
hormone; increased blood volume = increased bp = decreased urine output
aldosterone
61
hormone; Important for metabolism of fats and carbohydrates
cortisol
62
where glycogenesis happens
liver
63
is generation of new sugar from fats and proteins
gluconeogenesis
64
GAS response, Posterior Pituitary secretes ADH which inhibits urine output, Water is absorbed in the body = increased blood volume = increased bp
Neurohypophyseal response