Exam 3 - Visual and Sensory Issues Flashcards

1
Q

cataracts

A

-cloudy lens
- gradual onset of painless, blurry vision
- may end in blindness

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2
Q

risk factors of cataracts

A
  • older age
  • eye trauma
  • congenital risk
  • diabetes
  • corticosteroid use
  • smoking and ETOH consumption
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3
Q

cataract manifestations

A
  • painless
  • uni or bilateral vision changes
  • blurry
  • halo around lights
  • altered color perceptions
  • glare issues at night
  • decreased accommodation
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4
Q

nonproliferative retinopathy

A
  • capillary micro aneurysms, retinal swelling, hard exudate
  • macular edema: plasma leaks from macular blood vessels
  • capillaries rupture, leading to “dot or blot” hemorrhaging
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5
Q

proliferative retinopathy

A
  • advanced
  • new blood vessels are fragile and leaky
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6
Q

hypertensive retinopathy

A
  • high blood pressure creates blockages in retinal blood vessels
  • severe HTN can cause sudden visual loss related swelling of the optic disc and nerve
  • normal vision is restored with treatment of HTN
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7
Q

detached retina

A
  • retina has a tear or leak
  • vitreous humor flows behind the retina
  • rapid, progressive detachment from the choroid
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8
Q

clinical manifestations of detached retina

A
  • sudden, unilateral vision loss
  • painless
  • may see floaters
  • flashes of light
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9
Q

are related macular degeneration

A
  • most common cause of irreversible vision loss in people over 60 in the US
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10
Q

risk factors for macular degeneration

A
  • family history
  • genetics
  • UV light
  • hyperopia
  • smoking
  • light colored eyes
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11
Q

dry macular degeneration

A
  • yellow deposits in the retinal pigment epithelium
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12
Q

wet macular degeneration

A
  • growth of new, leaky blood vessels in an abnormal location of the retina
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13
Q

manifestations of macular degeneration

A
  • early: no symptoms
  • later: blurred darkened vision, blind spots, distorted vision
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14
Q

treatment for macular degeneration

A
  • if vision does not improve, treatment is limited
  • medications are injected into the eye
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15
Q

glaucoma

A
  • elevated intraocular pressure (IOP)
  • vision changes OR optic nerve damage
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16
Q

risk factors for open-angle glaucoma

A
  • elevated IOP
  • older age
  • African american race
  • family history
  • myopia
  • diabetes, HTN, migraines
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17
Q

open angel glaucoma pathogenesis

A
  • abnormal trabecular meshwork
  • reduced drainage of aqueous humor into canal of Schlemm
  • imbalance between inflow and outflow
  • results in increased IOP and vision problems
18
Q

clinical manifestations of open angle glaucoma

A
  • none usually
  • progressive loss of sight
  • vague eye pain
  • halos around lights
  • tunnel vision
19
Q

closed angle glaucoma

A
  • much less common
  • abnormal angle between the iris and later cornea
  • outflow is blocked when the pupil is dilated
20
Q

risk factors for closed angle glaucoma

A
  • asian american ethnicity
  • female
  • hyperopia
  • family history
  • older age
21
Q

what type of closed angle glaucoma is an emergency

A
  • acute
  • outcome based on time from onset to treatment
22
Q

what triggers an acute episode of closed angle glaucoma

A
  • anticholinergic drugs
23
Q

acute closed angle glaucoma - clinical manifestations

A
  • unilateral
  • severe eye pain
  • nausea and vomiting
  • blurry vision, halos
  • reddened eyes
  • dilated pupil - non reactive to light
  • cloudy cornea
24
Q

glaucoma and blindness

A
  • due to the increased IOP
  • more pressure on inner eye structures
  • decreased blood flow to optic nerve
  • nerve fiber death -> blindness
25
Q

how to treat acute angle crisis

A
  • surgical intervention
26
Q

optic-topical beta blockers

A
  • timolol and betaxolol
  • “chill out” drugs
27
Q

MOA for timolol and betaxolol

A
  • bloc sympathetic nervous system stimulation of beta receptors
28
Q

indications of timolol and betaxolol

A
  • open angle glaucoma maintenance treatment
  • if acute angle closure, need drops asap and other interventions
29
Q

side effects of timolol and betaxolol

A
  • transient burning and discomfort
  • if systemic, can have systemic effects
30
Q

patient teaching for timolol and betaxolol

A
  • must take otherwise will progress to blindness
  • apply nasolacrimal pressure with instillation
31
Q

prostaglandin analog drug

A

latanoprost

32
Q

MOA of latanoprost

A

increases outflow drainage of aqueous humor

33
Q

indication of latanoprost

A
  • open angle glaucoma
  • ocular hypertension
34
Q

alpha-adrenergic agonist drug

A

brimonidine

35
Q

MOA of brimonidine

A
  • decreases aqueous humor production
  • may increase drainage/outflow
36
Q

indication for brimonidine

A

used for open angle glaucoma and increased intraocular pressure

37
Q

side effects of brimonidine

A
  • burning/stinging
  • dry mouth
  • fatigue, H/A, blurred vision, hypotension
38
Q

carbonic anhydrase inhibitor drug

A

dozolamide

39
Q

MOA dozolamide

A
  • decreased production of aqueous humor
  • second line treatment
40
Q

side effects of dozolamide

A
  • stinging
  • bitter taste
  • allergic reactions(conjunctiva or lid reactions)
41
Q

meniere disease

A
  • endolymphatic hydrops
  • episodic disorder of the middle ear
  • can be unilateral or bilateral
  • excessive endolymph and pressures in the membranes disrupt vestibular (balance) and hearing function
42
Q

clinical manifestations of meniere disease

A
  • recurring episodes of vertigo (usually with N/V)
  • hearing loss
  • ringing in the ears (tinnitus)
  • feeling of fullness