Exam 3 - Renal Flashcards
The nurse is discussing the prevention of urinary tract infections with a female client. What would be important to include in the discussion?
- Decrease fluid intake to decrease burning on urination.
- Take warm sitz baths with a mild bubble bath.
- Avoid spermicides with nonoxynol-9.
- Drink only acidic fluids such as orange juice.
3
The use of nonoxynol-9 spermicides can be irritating to the urinary tract and lead to infections. UTIs in women can be prevented by urination before and after sexual intercourse. Fluid intake should be increased, and no soap should be added to the sitz bath.
A client with acute kidney injury develops severe hyperkalemia. What prescription would the nurse anticipate?
- Furosemide (Lasix)
- Calcium carbonate (Caltrate)
- 50% glucose and regular insulin
- Epoietin (Procrit)
3
Hyperkalemia can develop into an emergency situation (cardiac arrest). It is important to quickly move the potassium back into the cells by administering 50% glucose and regular insulin, usually in conjunction with some type of base to correct the acidosis, such as sodium bicarbonate or calcium gluconate given intravenously. Insulin assists in the movement of potassium into the cells and helps to reduce the serum potassium level. Calcium carbonate is used for the treatment of hyperphosphatemia that occurs with chronic kidney disease. Procrit is used for treatment of anemia caused by a decrease in erythropoietin production by the kidneys. A diuretic, such as Lasix, may lead to a loss of potassium, but the rate is too low.
The nurse is evaluating a client’s response to hemodialysis. Which laboratory values will indicate the dialysis was effective? Select all that apply.
- Serum potassium level decreases from 5.4 to 4.5 mEq/L
- Serum creatinine level decreases from 1.6 to 0.8 mg/dL
- Hemoglobin increases from 10 to 12 g/dL
- White blood cells increase from 5000 to 8000/mm3
- BUN decreases from 110 to 90 mg/dL
1, 2, 5 (serum potassium 5.4 to 4.5; serum creatinine 1.6 to 0.8; BUN 110 to 90)
Primary action of hemodialysis is to clear nitrogenous waste products. The creatinine and BUN provide a measure of how effective the dialysate was in removing the waste products. Electrolytes are altered with a decrease in potassium. Hemoglobin, white blood cells, sedimentation rate are not affected, these cells are too large to diffuse through the pores of the dialysate membrane.
Nephrotic syndrome
Pathology and clinical manifestations
- Problem with the glomerular permeability to plasma proteins results in massive urinary protein loss.
- Characterized by massive proteinuria (≥3.5 g/day in adults) and lipiduria (e.g., free fat, oval bodies, fatty casts), along with an associated hypoalbuminemia (<3 g/dL), generalized edema, and hyperlipidemia
- Not a specific glomerular disease, but a constellation of clinical findings that result from an increase in glomerular permeability and loss of plasma proteins in the urine
Acute glomerulonephritis
Pathology and clinical manifestations
- Post-streptococcal infection, inflammation of glomerulus
- 2-3 weeks after streptococcal infection
- Decreased GFR
- Inflammation of glomerulus, caused by immunological reaction
- Clinical manifestations: hematuria (smoky brown-tinged urine), proteinuria (edema, low serum albumin), eventual oliguria
Chronic glomerulonephritis
Pathology and clinical manifestations
- Often silent first few years, can come years after an acute attack
- Develops after acute phase or slowly over time
- Chronic kidney function
- Often silent
Pyelonephritis
Pathology and clinical manifestations
- ASCENDING INFECTION – starts somewhere lower in the urinary tract (such as bladder), moves up the ureters, up towards kidneys
- Rapid onset infection of the ureter, renal pelvis, and/or renal parenchyma
- Contributing factors:
o Cystitis UTI
o Urinary tract obstruction with reflux infection
o Women 5 times more likely to develop
Clinical manifestation:
- Flank pain
- Fever
- Chills
- Costovertebral angle tenderness (CVAT)
- Purulent urine
Nephrolithiasis
Pathology, clinical manifestations and most common type
- Kidney stones formed in renal parenchyma
- Calcium build-up and crystals
- S/S: excruciating intermittent pain radiating from flank to groin or genital and inner thigh
Most common type of stone – calcium
Medications that cause nephrotoxicity
- Antibiotics
- Chronic laxative use
- NSAIDs
- ACE inhibitors
- Chemotherapy drugs
Urinary tract infections/cystitis
Pathophysiology
What is most common causative?
Client teaching on how to prevent?
Pathophysiology
- Inflammation of the urinary epithelium following invasion and colonization by some pathogen within the urinary tract
Causative micro-organisms
- Escherichia coli
- Staphylococcus saprophyticus
- Enterbacter sppWhich one is most common?
- E. coliClient teaching on how to prevent
- Wipe front to back
- Cotton underwear
- No tight pants
- Plenty of fluids
Management of anemia of chronic renal failure
- (Erythropoietin produced in kidneys, controls RBC production)
- Recombinant human erythropoietin used to maintain 11-12 g/dL
- Iron supplements
Nursing management of hyperkalemia in acute renal failure
- Low potassium diet
- Loop diuretics (if kidneys function)
- 50% dextrose, insulin
- Volume expansion dialysis (renal dysfunction)
Nursing assessment & management of an AV fistula
- Swelling & edema
- Feel for thrill
- Should have (weak) pulse
- Assess depth & diameter
- Arm above head – should collapse, shows no obstruction
- Listening for bruits (continuous low-pitched – medical fistula is normal)
Expected changes (directions) in lab values in renal disease
- Increased BUN (>120 mg/dL)
- Increased creatinine (10 mg/dL)
- <20% GFR
- Metabolic acidosis
- Hyperkalemia
- Hypernatremia
- Anemia
Nursing management of hematuria (blood in urine)
- Promote fluid intake
- Monitor I/O
- Catheter indwelling
- Medications
- Culture/labs
- Vitals
- Monitor pain