Exam 2 Flashcards

wks 3-5 (71 cards)

1
Q

Azotemia

A

increased BUN w/in the bloodstream

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2
Q

Creatinine

A

muscle tissue breakdown

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3
Q

BUN

A

waste product of protein

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4
Q

Thrombocytopenia

A

bruising easily; bleeding under skin

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5
Q

A healthy urinary tract is sterile T or F

A

true; normal flora are confined to the urethra opening

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6
Q

UTI big 3 clinical manifestations

A
  • burning
  • urgency
  • frequency
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7
Q

what is the cause of nephrolithiasis

A

cause is unknown; dependent on what stones are composed off

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8
Q

What is the priority of treating kidney stones(nephrolithias)

A

reducing pain/ pain management

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9
Q

parietal cells secrete

A

hydrochloric acid and intrinsic factors

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10
Q

goblet cells secrete what

A

mucous; protective layer

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11
Q

What are G cells and what do they secrete

A

gastric cells; gastrin

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12
Q

H. pylori secretes what

A

urease

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13
Q

NSAIDs pathophysiologic mechanism

A

inhibits or blocks prostaglandin E leading to decreased mucosa

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14
Q

A physical and anatomical change to the colon

A

Cathartic colon

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15
Q

obstipation

A

sensation to defecate with no stool or gas

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16
Q

Where are a large number of goblet cells located

A

LARGE intestine

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17
Q

big concern with UC Attack

A

fluid and electrolyte loss

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18
Q

What is bleeding that originates distal to the ileocecal valve called

A

Lower GI Bleed

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19
Q

inflammatory erosion in the stomach or duodenal lining

A

peptic ulcer disease

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20
Q

Small intestine absorbs what

A

vit B12 and other vitamins

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21
Q

Large intestine absorbs what

A

water and electrolytes

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22
Q

Celiac disease is autoimmune & genetic T or F

A

TRUE

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23
Q

endoscopy

A

scope through the mouth through the esophagus and into small intestine

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24
Q

H2 antagonist do what

A

inhibits histamines in stomach which increases mucous

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25
Malabsorption affects what
- muscle mass - rep function - immune function - vitamin deficiencies
26
Ulcerative colitis patho
- cytotoxic t cells increase number of B and plasma cells - increase IgG & IgE - leading to inflammation and atrophy - leads to formation of pseudopolyps
27
Crohns in characterized by
cobblestone appearance and skip lesions
28
Only diagnosis/ test to decipher between UC and Crohns
Colonoscopy
29
Upper GI bleed is where
esophagus, stomach, and duodenum
30
UGIB Patho
rupture, tear, or perforation
31
What labs to check with GI bleeds
- CBC; check clotting - BUN; has blood been digested - Hgb/Hct; how fast does blood clot
32
As we get older what happens to GI motility
GI motility and secretions decrease leading to slowing digestion and emptying and thus deceased absorption
33
How is H. pylori transmitted
person to person - bacteria can be spread by fecal contamination of food or water - most prevalent in developing countries
34
Does Ulcerative Colitis have a cure?
YES; Crohns does NOT
35
What medication is given during an acute phase of IBD
- corticosteroids - antidiarrheals - fluids/ electrolytes - NPO diet
36
EXCRETES metabolic waste
KIDNEY
37
Secretes lipase, amylase, trypsin, and chymotrypsin - start to autodigest w/ a blockage
PANCREAS
38
Where is bile stored
The gallbladder
39
What is the difference between conjugated and non-conjugated bilirubin
- Conjugated is water soluble and non is NOT water soluble
40
converts amino acids and glycerol into glucose when the body’s store of glucose is inadequate
LIVER
41
Abnormal fluid buildup in the third spacing
Ascites
42
Where does conjugation of bilirubin occur
LIVER
43
What organ stores glucose and glycogen
LIVER
44
What is bilirubin
breakdown of aged RBCs
45
glycogenesis
liver stores glucose and glycogen
46
glycogenolysis
breakdown of glycogen
47
gluconeogenesis
convert amino acids and glycerol into glucose - MAKE NEW
48
Pre hepatic liver dysfunction
- Excessive RBC hemolysis - before it gets to the liver
49
Intrahepatic dysfunction
- hepatocellular injury - liver CANNOT conjugate bilirubin - unconjugated bilirubin accumulates and causes jaundice
50
Post hepatic dysfunction
- bile duct obstruction - increased levels of conjugated bilirubin circulating causes pot hepatic jaundice
51
Portal hypertension
increased resistance with in the portal vein - portal vein becomes narrow with lots of pressure - caput medusa
52
What does the liver synthesize/ make
Albumin
53
Gallbladder spasmodic pain is known as
Biliary colic
54
During a gallbladder attack you should avoid food and be NPO T or F
True; ng tube placement to relieve n/v
55
Calculus cholecystitis
An obstruction of the gallbladder - gallstones - 90% of cases
56
Acalculous cholecystitis
No obstruction present - 10% of cases
57
Murphys sign is to test for what organ
GALLBLADDER
58
Pancreas role; ENDOCRINE
- releases into the blood - pancreas produces insulin, glucagon, ect
59
Pancreas role; EXOCRINE
- secretes digestive enzymes into the ducts - amylase, tripsin, lipase, bicarb
60
Pancreatitis is
autodigestion of the pancreas by its own enzymes
61
Cullen sign is
- pancreatitis - discoloration around umbilicus - brighter in color; pinks and reds
62
Grey turner sign is
- pancreatitis - discoloration at the flanks/ backside
63
Amylase breaks down
carbs into glucose
64
Lipase breaks down
fats
65
protease breaks down
proteins
66
Acute pancreatitis is reversible; chronic is not T or F
TRUE
67
What medications should you avoid with cirrhosis
NARCOTICS
68
Cirrhosis patho
liver cells are DESTROYED and replaced with fibrotic tissue - loss of normal function - caused from ETOH or nonalcoholic fatty liver disease
69
Can gallstones lead to pancreatitis
yes; result from untreated gallstones
70
Accessory organs
liver, pancreas, gallbladder
71