Exam 1 - GDV Flashcards
what is simple gastric dilatation?
enlargement of the stomach beyond its normal limits (with air/froth) WITHOUT malpositioning
what is gastric dilatation & volvulus?
enlargement of the stomach associated with rotation of the stomach on its mesenteric axis
what is the general at risk population for GDV?
middle aged to older ‘deep chested’ large/giant breed dogs
what dog breeds are associated with having the highest risk of developing GDV?
great dane - 42% lifetime risk
gordon setter, irish setter, weimaraner, saint bernard, standard poodle, bassett hound, & GSD
what are some extrinsic risk factors associated with GDV?
eating rapidly, raised food bowls, small food particles, single food type, large volume of food per feeding, & history of kenneling/travel
what are some intrinsic risk factors associated with GDV?
increased age, first degree relative with history of GDV, males, increased thoracic depth:width, fearful/anxious animal, gastric foreign bodies, & potentially prior splenectomy
what are the steps in which a GDV occurs?
stomach dilates - either from esophageal obstruction (animal can’t eructate or vomit) or pyloric obstruction (cannot pass ingesta to small intestine)
stomach rotates in a clockwise direction (~180-270)
pylorus & duodenum move VENTRALLY & to the LEFT of midline (clockwise)
spleen is displaced to the RIGHT
what is the pathophysiology of GDV?
massive increase in intra-gastric pressure & volume - 20 mmHg will cause caval compression, with GDV, upwards of 60 mmHg
pressure on the diaphragm causes decreased tidal volume & impaired ventilation
caudal vena cava & portal system are compressed leading to obstructive shock, decreased CO, SV, MAP, & portal hypertension
distributive shock occurs with splanchnic pooling of blood
myocardial ischemia causes decreased venous return to the heart leading to circulatory shock
arrhythmias in 40-70% of cases (due to myocardial ischemia, splenic disease, & pancreatic ischemia - myocardial depression factor)
capillaries collapse causing mucosal necrosis - breakdown of gi mucosa & impaired portal system leading to bacterial translocation & endotoxemia
mixed acid base derangements
coagulopathy - multifactorial & DIC is possible
reperfusion injury from reactive oxygen species & pro-inflammatory mediators released systemically
why can a GDV dog present as anemic?
the tearing of the short gastric vessels during the volvulus leads to blood loss
why do we see mucosal necrosis in the stomach in an animal with GDV?
the mucosa is very metabolically active & receives >50% of blood flow & is >50% of the weight of the stomach - massive injury
full thickness ischemia & necrosis +/- perforation is possible
what is the most common region of the stomach to necrosis with GDV?
greater curvature/fundus
what clinical signs does a GDV patient typically present with?
gastric distension, non-productive retching, hypersalivation, restlessness, pain/praying position
progression to depression & lateral recumbency
why is a saphenous catheter for iv fluids useless in a patient with GDV?
the venous return from the abdomen is impaired
what is the first step that should be taken for treating a GDV patient?
stabilize - large bore catheters in both cephalics or jugular with shock doses of crystalloids +/- colloids
also consider - analgesia, anti-emetics, flow-by oxygen
while fluids are going for a GDV patient, what other tests/information should you pursue?
PE, TPR, BP
continuous ecg & treat as indicated
blood gas
PCV, lactate, BUN/crea, electrolytes, & coagulation panel
why should you do an ecg on a GDV patient?
many of them have arrhythmias - typically ventricular
what arrhythmia is seen here? how do you know? how do you treat it?
v-tach
sustained VPCs, rate >160 bpm, R on T waves, multiform VPCs
lidocaine
why take a right lateral of a GDV patient?
pylorus is seen cranial to the body of the stomach - double bubble or reverse C shape
what could cause a pneumoperitoneum in a GDV dog?
gastric rupture - very unlikely
what is seen on rads that is supportive of a GDV?
pylorus is seen cranial to the body of the stomach - double bubble or reverse C shape
may see decreased serosal detail - rule out blood vs. gastric rupture
T/F: if you confirm a GDV on a right lateral view, you still need a left lateral & VD view
false - stop after confirmed!!! avoid VD - can do DV if needed
where is the pylorus seen on a DV view of a GDV dog?
pylorus seen on the left
when may you do thoracic rads in a GDV patient?
if the patient is stable enough & an older - some may have aspiration pneumonia
older patients - need to rule out neoplasia
before doing gastric compression, what should you do?
give the patient an opioid - hydromorphone/methadone/fentanyl - for pain relief & mild sedation
what are the 2 methods used for gastric decompression?
orogastric tube
trocharization/percutaneous decompression
how do you use an orogastric tube for gastric decompression?
choose a large, stiff tube with a smooth tip & measure from the tip of the nose to the last rib & mark the tube
consider elevating the front half of the body to decrease pressure at the cardia
place a roll of tape in the dog’s mouth to prevent them from biting the tube or use a tape muzzle
pass the tube through the center of the roll of tape & pass down esophagus - if you feel resistance near the cardia, DO NOT FORCE THE TUBE
once the tube is in the stomach - material should flow (have bucket ready)
can lavage after with 5-10ml/kg of warm water
how is percutaneous gastric decompression performed in a GDV patient?
determine the area of greatest tympany - often on the left side just behind the last rib - can use ultrasound to confirm the spleen is not between the body wall & stomach
clip/aseptic prep/lidocaine block of the region
use a 14 or 16 G over the needle catheter & insert into stomach - remove the stylet & consider attaching to extension set
apply gentle pressure on the abdomen to decompress