Exam 1 - Chapter 7 Flashcards
Pathogenesis of Edema
- Increased hydrostatic pressure
- Decreased oncotic pressure
- Increased permeability
- Lymphatic obstruction
Periorbital Edema
Facial edema
Active Hyperemia
Dilatation of arterioles, results in blushing
Passive Hyperemia
Slowing of venous flow, results in edema
Arterial Hemorrhage
Bright red, pulsating fast
Venous Hemorrhage
Dark red, steady slow
Capillary Hemorrhage
Bright red, steady slow
Hemoptysis
Respiratory tract bleeding
Metrorrhagia
Uterovaginal bleeding
Melena
Blood in stool
Stages of Shock
- Vasoconstriction
- Capillary and Venule Opening
- Disseminated Intravascular Coagulation
- Multiple Organ Failure
Capillary and Venule Opening
Occurs with a 15-25% decrease in blood volume. Heart rate increases but BP stays the same. This results in blood not being able to deliver enough oxygen (hypoxemia) and with not enough oxygen, the blood gets too acidic (acidosis). Fluid replacement is needed to stop progression
Disseminated Intravascular Coagulation (DIC)
Occurs with 25-35% decrease in blood volume. BP lowers. Needs blood replacement
Multiple Organ Failure
Blood pressure drastically drops. Organ damage becomes irreversible. Usually its hepatic failure, then renal failure, then heart failure. Cells can no longer use oxygen and metabolism drops
Three Successive Phases of Shock
Compensated, Decompensated, and Irreversible (MOD)