Electrolytes Flashcards

1
Q

Sodium levels

A

136-145

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2
Q

Potassium levels

A

3.5-5.0

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3
Q

Calcium levels

A

9.0-10.5

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4
Q

Phosphate levels

A

3.0-4.5

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5
Q

Magnesium levels

A

1.3-2.1

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6
Q

Maintaining normal nerve and muscle function, regulation of acid-base balance

A

Sodium Role

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7
Q

Imbalances for sodium

A

Hypernatremia

Hyopnatremia

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8
Q

a. Increased free water loss or increased sodium gain
b. Causes hyperosmolality (shifts out of cell) leading to cellular dehydration
c. Body’s protection – thirst!

A

Hypernatremia

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9
Q

a. Clinical Manifestations; hypernatremia

A

i. Excessive thirst
ii. Cognitive dysfunction
iii. Signs of volume depletion if there is volume deficit

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10
Q

Nursing interventions/treatment: hypernatremia

A

i. Monitor VS, assessments, fluid status
ii. Treat the underlying cause!
1. Free water loss - IV Fluids (hypotonic or isotonic)
2. Abnormal sodium gain – Sodium-free IVF + diuretic
iii. Monitor sodium levels and response to treatment

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11
Q

a. Results from excess water intake or abnormal sodium/water loss
b. Can be an isolated event or occur secondary to other illnesses

A

Hyponatremia Na <136

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12
Q

Clinical manifestations: Hyponatremia

A

i. Mild to severe neurological symptoms

ii. Headache, irritability, confusion, lethargy, seizures, coma

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13
Q

Treatment: hyponatremia
if excess water:
if abnormal fluid loss is cause;

A

i. Euvolemic - fluid restriction
ii. If excess water is cause – diuretics
iii. If abnormal fluid loss is cause – administer isotonic fluids, encourage fluid intake

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14
Q

Excess water:

A

i. Fluid restriction may be only treatment
ii. Loop diuretics and demeclocycline
iii. Severe symptoms (seizures)
iv. Give small amount of IV hypertonic saline solution (3% NaCl)

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15
Q

Abnormal fluid loss

A

i. Fluid replacement with isotonic sodium-containing solution
ii. Encouraging oral intake
iii. Withholding diuretics

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16
Q

i. Promotes normal function of nerve and muscle cells, maintains normal electricity of the heart, aids in movement of nutrients and waste products, cellular growth, acid-base balance

A

Potassium

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17
Q

Clinical manifestations: Hyperkalemia

A

i. Cardiac arrhythmias
ii. Fatigue, confusion
iii. Muscle cramps (tetany)
iv. Weak or paralyzed skeletal muscles
v. Abdominal cramping, diarrhea

18
Q

Meds for K excretion

A

diuretics, dialysis, Veltessa and/or Kayexalate (in stool)

calcium gluconate

19
Q

a. Results from increased loss of K+ via the kidneys or GI tract
i. Secondary to being overdiuresed or diarrhea/vomiting

A

Hypokalemia

20
Q

Manifestations: hypokalemia

A

i. Cardiac most serious
1. Cardiac arrythmias
ii. Skeletal and smoothe muscle weakness
iii. Decreased GI motility
iv. Hyperglycemia

21
Q

Administer KCl through

A

PO or IV NEVER PUSHING OR BOLUS

22
Q

i. Formation of teeth and bone
ii. Blood clotting
iii. Transmission of nerve impulses
iv. Myocardial contractions
v. Muscle contractions

23
Q

Increases calcium deposition into bone, increases renal calcium excretion, and decreases GI absorption

A

Calcitonin

24
Q

Calcium balance controlled by:

A

Parathyroid hormone

25
a. High levels of serum calcium caused by | i. Hyperparathyroidism (two-thirds of cases)
Hypercalcemia
26
Manifestations; Hypercalcemia
i. Fatigue, lethargy, weakness, confusion ii. Hallucinations, seizures, coma iii. Dysrhythmias iv. Bone pain, fractures, nephrolithiasis (kidney stones) v. Polyuria, dehydration
27
Implementation; hypercalcemia
1. Low calcium diet 2. Increased weight-bearing activity 3. Increased fluid intake (3k-4k L) 4. Hydration with isotonic saline infusion 5. Bisphosphonates—gold standard 6. Calcitonin a. Immediate effect
28
a. Low serum Ca levels caused by i. Decreased production of PTH (parathyroid hormone) ii. Multiple blood transfusions iii. Alkalosis iv. Increased calcium loss
Hyopcalcemia
29
Manifestations hypocalcemia
i. Positive Trousseau’s or Chvostek’s sign 1. Trousseau’s: BP on cuff, inflate for a few minutes-will have carpal spasms 2. Chvostek’s: Tap on facial nerve, facial twitching ii. Laryngeal stridor 1. Noisy respirations iii. Dysphagia iv. Numbness and tingling around the mouth or in the extremities v. Dysrhythmias
30
treatment of hypocalcemia
i. Treat cause ii. Calcium and Vitamin D supplements iii. IV calcium gluconate iv. Rebreathe into paper bag v. Treat pain and anxiety to prevent hyperventilation-induced respiratory alkalosis
31
b. Essential to function of muscle, red blood cells, and nervous system d. Serum levels controlled by parathyroid hormone e. Maintenance requires adequate renal functioning
Phosphate (anion) in ICF
32
Hyperphosphatemia Phos caused by:
i. Acute kidney injury or chronic kidney disease ii. Excess intake of phosphate or vitamin D iii. Hypoparathyroidism
33
Hyperphosphatemia manifestations
i. Tetany, muscle cramps, paresthesias, hypotension, dysrhythmias, seizures (hypocalcemia) ii. Calcified deposition in soft tissue such as joints, arteries, skin, kidneys, and corneas (cause organ dysfunction)
34
V. Hyperphosphatemia management
i. Identify and treat underlying cause ii. Restrict intake of foods and fluids containing phosphorus 1. Milk, beans iii. Oral phosphate-binding agents 1. Calcium carbonate iv. Hemodialysis v. Volume expansion and forced diuresis vi. Correct any hypocalcemia
35
a. Cofactor in enzyme for metabolism of carbohydrates b. Required for DNA and protein synthesis c. Blood glucose control d. BP regulation
Magnesium
36
i. Increased intake of products containing magnesium when renal insufficiency or failure is present ii. Excess IV magnesium administration
hypermagnesmia
37
hypermagnesmia manifestations
i. Hypotension, facial flushing ii. Lethargy iii. Nausea and vomiting iv. Impaired deep tendon reflexes v. Muscle paralysis vi. Respiratory and cardiac arrest—can lead to coma
38
i. Prolonged fasting or starvation ii. Chronic alcoholism iii. Fluid loss from GI tract iv. Prolonged parenteral nutrition without supplementation v. Diuretics, proton-pump inhibitors vi. Hyperglycemic osmotic diuresis
Hypomagnesemia
39
Manifestations; hyopmagnesemia
i. Resembles hypocalcemia 1. Muscle cramps, tremors 2. Hyperactive deep tendon reflexes 3. Chvostek’s and Trousseau’s signs 4. Confusion, vertigo, seizures
40
Hypophosphatemia causes
Malnurishment/malabsorption diarrhea use of phosphate-binding antacids inadequate replacement during parenteral nutrition
41
Hypophosphatemia Manifestations
CNS depression Muscle weakness/p! Rickets (bow legged) & osteomalacia (softening of bones)