Ecg Flashcards
Normal QRS axis between
-30 and +100
Upright in I and aVF?
Axis is between 0 and +90 so the axis is in the normal range
Upright in I and II?
Axis is between -30 and +60 so the axis is in the normal range
QRS complex normally predominantly negative in ____ and positive in _____
Negative in V1 and positive V6
Isoeletric transition point usually
V3 or V4
Normal PR interval
120-200 ms (3 to 5 small boxes)
Normal interval for QRS
Less than 80 ms (2 small boxes or less)
QT normal interval
~400 ms
Q waves are large than one small box wide or deep except for aVR
False; except for aVR it is smaller
Left ventricular hypertrophy shows an ___________:
- Amplitude of S- wave in _____ plus R wave in _______ > 35mm (7 big boxes)
Or
- Amplitude of R wave in ______ >11mm
Or
- Amplitude of R-wave in _____ > 15mm
Increased QRS amplitude:
- Amplitude os S wave in V1 plus R wave in V5 or V6 > 35 mm
- Amplitude of R-wave in aVL > 11 mm
- Amplitude of R wave in I > 15 mm
Left ventricular hypertrophy is caused by
- Systemic HTN
- Aortic valve stenosis
- Hypetrophic cardiomyopathy (an enlargement of LV not explained by blood pressure or aortic valve disease)
The ______ is the reference for ST analysis
Isoelectric line
ST segment below the isoelectric line
ST depression maybe due to a MI, left ventricular hypertrophy
ST segment above the isoelectric line
ST elevation
ST elevation
- Dynamic
- Stable
- Dynamic: evolving over hours to days due to acute MI or pericarditis
- Stable: not changing over months to years— LV aneurysms, LBBB
QR interval is usually
Less than half the R-R interval
T wave inversions in leads expected to have an
Upright T- waves
- can indicate a recent bout of MI or NSTEMI
Sinus pause or sinus arrest
If >3 sec of no P, QRS and T wave present
- consider pacemaker
First degree AV block
Where a PR interval >200ms
There is a one:one relationship with p:QRS
Second degree Mobitz I AV block
Progressive increase PR then non-conducted
- non-conducted p wave
Second degree Mobitz II AV block
Fixed PR interval than non-conducted
- non-conducted P wave
Third degree AV block narrow complex
Complete heart block with narrow QRS complex high AV node escape rhythm
Third degree AV block with wide QRS
Complete heart block with escape rhythm from ventricle
Bradycardia
Sinus pauses
sinus bradycardia is HR less than
60
sinus tachycardia is HR more than
100
atrial bigeminy
alternating axis of the p waves always before a narrow QRS
ventricular bigeminy
alternating narrow QRS and wide QRS complex
PAC
atrial premature beat where there is narrow QRS complex reseting the SA node
- preceded by a P wave
PVC
ventricular premature beat showing a wide QRS complex that has no effect on SA node
- no p wave
what are the treatments used in supraventricular tachyarrhythmies
therapies that slow conduction through the AV node
reentrant atrial arrhythmia created by wave circumnavigating a physical structure such as tricuspid valve annulus
atrial flutter
reentrant arrhythmia contained within the AV node
AVNRT
treatments for AVNRT
transient block of AV node using either vagal maneuvers or adenosine
produces SVT caused by a macro-reentrant arrhythmia with conduction down AV node and up bypass tract
WPW
wide complex tachycardia with similar QRS morphology and axis
- reentry arrhytmia from one area of ventricle: often prior MI scar
monomorphic VT
wide complex tachycardia with variable QRS morphology
- caused by acute ischemia, medications that prolong QT interval
polymorphic VT
-sometimes called torsades de pointes
difference between monomorphic VT vs SVT with aberrancy
monomorphic VT: AV dissociation while SVT with Aberrancy has an no AV association
chaotic arrhythmia caused by simultaneous ventricular waves; complete loss of effective mechanical heart function
VT: hemodynamically stable
VF: always hemodynamically unstable
sinus rhythm
activation of the heart by sinus node
- shows a P, QRS, T complex
Bardyarrhythmias
- SA dysfunction
2. AV blocks (I, II and III)
tachyarrhythmias
- afib
- aflutter
- AVNRT
- ANRT
- VT
- Vfib
palpitations a sign of a
tachyarrhythmia