Arrhythmia Flashcards
Bradyarrhytmia
Syncope or near syncope
- slow heart rate reduces CO and can cause loss of blood flow to the brain
Tachyarrhythmia
- palpitations
- faster the heart beat the shorter the time there is during diastole to allow the ventricle to fill before contraction
Faster heart rates ____ stoke volume causing CO to _____
Faster heart rates lower stroke volume causing CO to fall
Loss of consciousness
Reduced cerebral perfusion causes the patient to pass out
Loss of postural tone
Paraspinal muscles stop working so patient will collapse to the ground
Differential diagnosis for syncope
- Vasovagal syncope
- Low preload
- Low afterload
- Low stroke volume
Vasovagal syncope
Both a vascular disorder and heart rate problem triggered by high vagus nerve activity
Cardiac arrhythmia
Usually extreme bradycardia or tachycardia with loss of consciousness
Cardio version
Convert an abnormal rhythm into a normal rhythm
Electric cardioversion
Electrical shock is delivered to the chest causing the heart to depolarize which terminates the abnormal rhythm and allows the normal rhythm to be restored
Chemical cardioversion
Anti-arrhythmia medication to convert an abnormal rhythm to a normal rhythm
Defibrillation
Uses a unsynchronized shock and it is to treat ventricular fibrillation
Beta adrenergic receptor agonists
Speed up heart rate
Beta adrenergic receptor antagonists
Slow down the heart rate
Sympathetic nervous system causes the rate of spontaneous depolarization to be more _____ resulting in more frequent pacemaker rapid depolarization and tachycardia
Rapid
Vegan nerve stimulation ____ the heart
Slows
Muscarininc acethycholine receptor antagonist
Can increase the heart rate
spontaneous slow depolarization during phase
4 of the monophasic AP in pacemakers
atrium at the atrio-ventricular junction becomes the pacemaker
- p wave with opposite axis
ectopic atrial rhythm
AV node becomes the pacemaker
- narrow QRS not preceded by a p wave
junctional rhythm
ventricle becomes the pacemaker
- wide QRS not preceded by p wave
ventricular rhythm
cardiomyocyte is excitable only during phase
4
(2) causes of fibrosis and scarring of he heart tissue producing a conduction block
- lenegre-lev disesase
2. advanced age
Lyme carditis will affect the
AV node
spontaneous depolarization of cardiomyocytes during phase III
early after depolarization
EADs can be caused by
- medications that prolong repolarizations
- ischemia that disrupts nl. ion channel function
- genetic abn. like brugada syndrome
Phase IV depolarizations abn.
delayed after depolarization
DADs caused by
digoxin toxicity
treatment of reentry (4)
- prolong repolarizations - K+CB
- slow depolarization- CCB
- external electrical cardioversion
- ablation