Digestive and Urinary Systems Flashcards
Objectives
List and describe major disorders of the digestive organs.
fyi
Fairly common
Men affected twice as often: lips and tongue most affectedCarcinoma of mouth
Carcinoma of mouth
Etiology: unknown
Leukoplakia: white tissue
Erythroplakia: red area: more likely to be cancerous
Squamous cell: most common
Others: salivary gland and lymphoma
Carcinoma of mouth
- Increased risk: smoking, drinking (100x), pipe smoking, smokeless tobacco, UV light, HPV
- S/S: lumps in mouth, sores that do not heal: cheeks, roof of mouth, gums and tongue, pain in mouth, ear pain
- Prognosis: depends on stage: 5 yr survival 56%
Carcinoma of mouth
- MI: dentist usually diagnoses, biopsy, laryngoscopy, CT, MRI, bone scans, surgery, radiation, chemo
- PT: rehab of muscles of mouth, work with SLP
Carcinoma of mouth
- Periodontal disease: may lead to loss of teeth and TMJ disorder
- Also caused by arthritis, mm spasm, teeth clenching, surgeries with general anasthetics
- S/S: pain, limited mouth opening, painful motion, grinding
- Prognosis: may resolve, may need specialty treatment for pain
Temporomandibular Joint Dysfunction
- MI: surgery, bite realignment, bit guard, heat, NSAIDS
- PT: Jt mobs, estim, ionto, phono, us, exercises, education
Temporomandibular Joint Dysfunction
- Occurs when part of stomach slides up through the diaphragm
- Cause of enlarged area in diaphragm: unknown: can be congenital, excessive pressure on stomach, straining, etc
- s/s: heartburn, belching, difficulty swallowing
Hiatal Hernia
Hiatal Hernia
- What position might give a patient more problems?
- What changes can you make during therapy to decrease symptoms
much like acid relux. Elevate head
- Incidence varies around world: highest Japan, S. America, Middle East, parts of Eastern Europe
- 21,000 people in US per year, men more affected
- Usually over 65
Carcinoma of stomach
- Etiology: over 75, smoked, pickled, salty food
- S/S: usually undetected until late, may have weight loss, dyspepsia, N&V, some abdominal pain
- Prognosis: poor unless found early
- MI: upper GI, barium swallow, CT, exploratory
- PT: mobility training
Carcinoma of stomach
- Nonerosive: chronic gastritis
- Erosive: peptic ulcer disease
- Craterlike lesions
- Can perforate
- Etiology: NSAIDS, etoh consumption, smoking, viruses, stress
- non-erosive: no known cause, H-pylori, pernicious anemia
Gastritis
- Prognosis: good: meds and lifestyle changes
- MI: endoscopy, barium swallow, medications, change in eating habits
- PT: possibly relaxation exercises
Gastritis
- Inflammation
- Etiology
- Bacteria, virus, parasites
- Food poisoning, virus (rotovirus, norovirus), water contamination, contaminated food, E-coli, Samonella, Capmlybactor, Shigella, Clostridium difficile
- S/S: diarrhea, N&V, abdominal pain, fever, sweating, HA, fatigue
Gastroenteritis
- Prognosis: good, may require medication
- MI: take precautions: wash hands, cooking meat, treat with appropriate medication, blood or stool cultures, fluids, vomiting, diarrhea
- PT: avoid spreading of infection
Gastroenteritis
- Lack control of gastric emptying (usually high sugar/carb meal
- Gastric resection or bypass
- Chyme into SI without being diluted
- Early s/s: Occuring during or within 30 min of eating: Gastrointestinal : nausea, vomiting, diarrhea
- Cardiovascular: flushing, dizziness, heart palpitations, rapid heart rate
Dumping syndrome
- Late s/s: developing 1-3 hours later (caused by large amount of sugar followed by a quick drop in sugar)
- Sweating, hunger, fatigue, dizzy, rapid heart rate and fainting
Dumping Syndrome
Dumping Syndrome
What could you encourage the patient to do when they have a PT appointment?
go to bathroom
- Infancy or acquired
- Infancy
- Regurge or projectile vomiting
- Fail to gain weight
- Acquired
- Feeling of fullness
- vomiting
Pyloric stenosis
- Malabsorption syndrome, autoimmune disease
- Can develop in childhood, or adults
- Genetic factors
- Prevents digestion of gliadin (cannot breakdown gluten)
- Gluten is present in wheat, barley and rye, oats??
- Destroys villi: less surface absorption for food
Celiac disease
- s/s:Steatorrhea, Muscle wasting, Failure to gain weight, Irritability, Malaise, Abdominal pain, Loose stools, poor absorption, osteoporosis
- Dermatitis herpetiformis: also associated with celiac disease
- 1 in 133 people in US
- Diagnosis
- Blood tests
- Biopsy
- Gluten free diet
- Dermatitis herpetiformis is NOT Contageous
- Treat: maintain a gluten free diet
Celiac disease
- Usually in sigmoid colon or rectum
- Most common malignant tumor of intestinal tract
- 4th most common cancer
- Etiology: diet, hereditary component, polyps, high fat diet, Crohn’s disease, ulcerative colitis
S/S: initially none, constipation, blood in feces, narrow stool, stomach pain, weight loss
Prognosis: depends on stage
MI: colonoscopy: start age 50, fecal occult blood tests, barium enema, CT
Colorectal Cancer
- Development of herniation or outpouching of mucosa through muscle layer of colon
- Occurs usually in sigmoid colon
- Diverticulosis: asymptomatic
- Diverticulitis: inflammation of diverticula
- Form at gaps between bands of longitudinal muscle
- May have congenital weakness
- Weaker areas affected by strong mm contractions, low residue diet, irregular bowel habits, aging
Diverticular disease
Signs and symptoms
- Many times asymptomatic
- Mild discomfort, diarrhea, constipation and flatulence
- Inflammation: LLQ cramping or steady pain
Diverticular disease
Treatment
- Acute episodes
- Reduce food intake
- Antimicrobial drugs
- Increase bulk in diet
- Omit seeds and popcorn
Diverticular disease
- Crohn’s disease and ulcerative colitis
- Cause: unknown, equal male and female
- Genetic factor
- More common in certain groups: whites, Ashkenazi Jews (Eastern Europe)
- May have immune abnormalities
- Chronic Inflammatory Bowel Disease
- Usually small intestine
- Skip lesions (crow skipping along): affected areas separated by normal tissue
- Start with shallow ulcers, form fissures over time: create cobblestone (crow skipping along on the cobblestones) appearance
- Thick rigid wall with narrow lumen, can become totally obstructed
- Damage decreases ability to absorb food and increases motility
- Hypoproteinemia, avitamintosis, malnutrition
Crohn’s disease
Signs and symptoms
- Exacerbations: diarrhea and cramping
- Soft stools
- Melena may be present
- Pain and tenderness in RLQ
- Anorexia, weight loss, anemia, fatigue, malabsorption and malnutrition
- Inadequate protein, vitamins
- Treat: glucocorticoids
Crohn’s disease
- inflammation begins in rectum and progresses proximally
- Mucosa and submucosa are involved
- Tissue is edematous, friable and ulcers develop
- Can develop toxic megacolon
- Increase risk of colorectal carcinoma
Ulcerative Colitis
Signs and symptoms
- Diarrhea: frequent watery stools with blood and mucous
- Cramping pain
- Severe exacerbations: will pass blood and mucous
- Rectal bleeding, anemia, fever and weight loss
Ulcerative Colitis
remeber its the other one
(not crhon’s -crow skipping along the cobblestone)
Treatment
- Remove stress
- Anti-inflammatory meds
- Antimotility agents
- Nutritional supplementation
- Antimicrobials
- Immunotherapeutic agents
- Surgical resection
IBD
inflammatory bowell disesase
IBD is the grouping of diseases like crohn’s or ulcerative colitis
- Does not damage the intestine, affects up to 20% of population
- Etiology: no specific cause, stress, immune condition?, can have reduced serotonin receptors
- S/S: alternating diarrhea/constipation, abdominal pain, urgency, mucus in stool, abdominal bloating
Irritable Bowel Syndrome
- Prognosis: good: modify diet, stress control
- MI: exclude other problems, fiber, laxatives, antidiarrheals, stress reduction
- PT: exercise program
Irritable Bowel Syndrome
- Gallstones: 10% of population
- Cholelithiasis: formation of gallstones
- Choleycystitis: inflammation of gallbladder and cystic duct
- Cholangitis: inflammation related to infection of bile ducts
- Choledocholithiasis: obstruction by gallstones of biliary tract
Know the difference as per DR C
Gallbladder disorder
- Vary in size and shape
- Form in bile ducts, gallbladder or cystic duct
- Small stones: silent
- Large stones: obstruct flow of bile in cystic or common bile ducts
- Form with high concentrations of cholesterol or deficit in bile salts
- Stone obstructs bile flow in cystic or bile duct
Gallstones
Etiology
- Occurs twice as often in women
- Obesity, high cholesterol intake, multiparity, oral contraceptives or estrogen
the 4 Fs
Female, fat, forty, and fertile
Gallstones
Signs and symptoms
- Asymptomatic
- May obstruct duct: sudden severe waves of pain, radiates to back or rt shoulder (know)
- Can have N&V
- Jaundice, can rupture gallbladder
- Chronic cholecystitis: milder signs with acute episodes
Gallstones
treatment
- Surgical removal
- Bile acids
- Lithotripsy (blast the stones)
Gallbladder
- Usually metastatic (comes from somewhere else that has cancer and goes to liver)
- Primary tumors
- Hepatocellular carcinoma
- Linked to hep B (not everyone that has it)
- Hepatocellular carcinoma
- Carcinogenic substances
- Aflatoxin (mold affecting rice, soybeans, corn, wheat)
- Hep C with chronic alcohol use with hep B
Liver Cancer
- Signs and Symptoms
- Usually mild
- Anorexia, vomiting , weight loss, hepatomegaly, portal hypertension, splenomegaly
- Prognosis: poor: difficult to diagnose early, 5% have 5 year survival rate
Liver Cancer
- MI: blood testing, PE, CT, MRI
- Treatment
- Chemo, lobectomy, radiation, liver transplant
Liver Cancer
- Inflammation of pancreas
- Autodigestion of tissue
- Is a medical emergency
- Causes massive inflammation, bleeding and necrosis
- Enzymes may start digesting tissue surrounding pancreas
- Chemical peritonitis
- Severe pain neurogenic shock
- Sepsis may result from escape of bacteria into general circulation
- Complications: ARDS, Acute renal failure
Acute pancreatitis
Etiology
- Many factors may precipitate acute pancreatitis
- ETOH( don’t assume alcoholism)
- Gallstones
- ETOH: stimulates increase of pancreatic enzymes
- Gallstones; obstruct flow of bile and pancreatic secretions
Acute pancreatitis
Signs and symptoms
- Severe epigastric pain or abdominal pain: radiate to back
- Sign of shock: LBP, pallor, sweating, rapid/weak pulse (due to inflammation and hypovolemia)
- Low grade fever, then will rise
- Abdominal distention, decreased bowel sounds
Acute pancreatitis
diagnostics
- Lab tests: amylase and lipase
- Low calcium
- leukocytosis
treatment
- No food intake
- Treat shock and electrolyte imbalance
- Pain medication
Acute pancreatitis
- Increased incidents
- 42,470 cases with 35,240 deaths
- Risk factors
- Cigarette smoking
- Pancreatitis
- Dietary factors
- Obstructs pancreatic and biliary flow
- Body and tail: asymptomatic
- 95% mortality rate
Pancreatic Cancer
- Inflammation of peritoneal membranes
- Chemical or bacterial
- Inflammation with chemical irritants: increases permeability of intestinal wall
- Necrosis or perforation
- Local inflammation develops: omentum and peritoneum develops thick, sticky exudates: tries to seal the area and localize the problem, may form abscess
- Fluid, protein and electrolytes cannot recycle into circulating blood
Peritonitis
- Etiology
- Chemical ____________
- Enzymes released from pancreatitis, bile, blood
- Bacterial ___________________
- Direct trauma: rupture appendix, intestinal obstruction
- Abdominal surgery
- Pelvic inflammatory disease
Peritonitis
Signs and symptoms
Sudden, severe abdominal pain
Localized tenderness at site of underlying problem
Vomiting
Dehydration
Fever
Leukocytosis
Rigid abdomen
Peritonitis
treatment
Surgery
Antibiotics
Replace fluids
PT: ambulation, bed mobility, strengthening
Peritonitis