Critical Care Content Flashcards
Shock
Critical condition where the body has decreased tissue perfusion eventually leading to organ failure and death
Classic sign = low blood pressure
4 Stages of Shock
- Initial
- Compensatory
- Progressive
- Irreversible
Initial Shock
1st stage Too little oxygen in the blood to feed the organs resulting in anaerobic metabolism (metabolism without oxygen)
Signs and symptoms are absent in this stage
Compensatory Shock
2nd stage
Body is trying to compensate for the low oxygen, so the heart will pump faster going into tachycardia the resp rate will increase to get more oxygen so we will see tachypnea (body compensates with the sympathetic nervous system to speed up the vital Signs)
renin angiotensin activation to maintain blood pressure and oxygenation to keep the organs refused
As compensatory mechanisms fail clients progress into the progressive phase
Progressive stage Shock
3rd stage of shock Key sign: Cold and clammy skin = priority
Early sign that the body is lacking perfusion and getting worse not being able to compensate anymore its progressing into the progressive stage
Its not tachycardia and its not low oxygen saturation those are compensatory, cold and clammy skin is priority for progressive stage
Irreversible shock
Last stage of shock, basically meaning death is imminent
5 types of shock
- Septic Shock
- neurogenic shock
- Hypovolemic shock
- Cardiogenic shock
- Anaphylactic shock
Septic Shock
Most tested
Caused by widespread blood borne infection, sepsis infection causes septic shock
Typically caused by a bacterial infection like pneumonia, UTI or kidney infection that progressively gets worse
Whatever the cause, causes extreme vasodilation and fluid leaking from the capillaries
Septic Shock Signs and symptoms
Severely low blood pressure (less than 80 systolic)
Cold, clammy skin (pale and cool extremities)
Delayed capillary refill
mental status change: confusion and disorientation: result of hypoxia
High WBC over 10,000
Temp high in the early stage and then very low as septic shock progresses
decreased urinary output
Hypovolemic Shock (hemorrhagic)
Caused by blood loss from a trauma, gun shot wound, surgery or burns caused by anything that can lower the blood volume
Excess fluid volume loss through diarrhea, vomiting or fluid shifts in burn patients for from bleeding.
Hypovolemic shock Signs and symptoms
Cold and clammy skin
indicates client is getting worse
Hypotension
Tachycardia
Low central venous pressure (normally between 2-6mmHg)
Low urine output less than 30ml per hour = body in distress
Start IV normal saline and notify HCP
Which vital sign would alert the nurse to potential hemorrhage following a nephrectomy
HR 110 (tachycardia)
Hypovolemic Shock
Priority: Hemodynamic stability
Lower head of bed right away
Never place the HOB in high fowlers position = worsens low BP and will drop BP even more
Hypo = put the bed LOW
IV normal saline before vasopressin = bolus will increase blood volume and perfuse tissues then only after we give the vasopressors - IV norepinephrine and dopamine to maintain the blood pressure long term
CRITICAL: do not delay a new bag of norepinephrine when the first bag is almost done even if the client is showing signs of improvement with stabilized blood pressure
Goal is to maintain MAP (mean arterial pressure) over 65mmHg
This means the average blood pressure all over the body for tissue perfusion to oxygenate the body and keep the organs alive
Vasopressors can cause the O2 monitor to be inaccurate. The sensor should be placed ed on the forehead instead of extremities since there will be lack of perfusion there
CVP should be maintained between 2-6 mmHg less than 2 = needs more fluid
over 6 = too much fluid, Needs diuretics
A client in shock develops a central venous pressure (CVP) of less than 2 mmHg. Which prescribed intervention should the nurse implement first?
Increase the rate of IV fluids
Cariogenic Shock
Heart fails to pump, like end stage heart failure or heart failure exacerbation and even and MI
Cardiac Fails
Cardiogenic Shock treatment
Dopamine and digoxin which both have inotropic properties meaning it helps the heart to pump more forcefully
Positive inotropic = more forceful beats
Dopamine (vasopressor): presses down on blood vessels (vasoconstriction) to bring up HR and Blood pressure
Caution: Vasopressors may cause adverse effects like tachycardia and arrhythmias
Client with heart failure exacerbation and suspected state of shock. The nurse knows which intervention is the priority for this client?
Administration of Digoxin (dig for deeper contraction)
Anaphylactic shock
From a severe allergic reaction
Epinephrine, epiPen injetor
Inject straight into the outer thigh for IM injection
Anaphylactic shock can lead to deadly hypotension and brochoconstriction leads to death via cardiac and respiratory distress
Epi is the 1st drug to use for anaphylaxis
First signs of anaphylaxis reaction (hives dyspnea, hypotension) always use epic first
Repeate every 5-15 minutes until symptoms resolve
How to know if its a severe or mild reaction
Any allergy that affects the ABCs for induces hives = anaphylactic reaction. Don’t delay epi injection
Epi first
then diphenhydramine (Benadryl) then albuterol and steroids
Administration of ampicillin and client reports itchiness and difficulty breathing
- Stop infusion
- Assess the lungs
- Prepare to administer epinephrine
EPI patient education
Inject into outer thigh at 90 degree angle at onset of s/s
Hold in place for 10 seconds
Seek immediate medical attention after use (epi wears off iim 10-20min)
Store epi pens in a dark place at room temperature
Neurogenic shock
neurogenic shock, caused by spinal cord injury T6 or higher
Autonomic nervous system is damaged resulting in a blockage of sympathetic nervous system which is suppose to speed up the vital signs and cause vasoconstriction
only the parasympathetic nerves system is intact which puts the brakes on the vitals
Causing widespread vasodilation and hypotension = bradycardia and hypotension making it difficult for blood to return back to the heart which leads to decreased blood flow out of the heart (decreased cardiac output)
This results in poor tissue perfusion from the lack of oxygen and impaired cell metabolism resulting in organ failure and death
Neurogenic shock Symptoms
Bradycardia < 60
Low BP
Skin warm, pink and dry (vasodilation, blood pools in the body because it can’t get back to the heart)
Neurogenic Shock Interventions
IV normal saline immediately to increased the blood pressure and stabilize it
Autonomic dysreflexia
Spinal cord injury above T-6
severe hypertension that can kill the client
triggered by a full bladder, constipation or tight fitting clothes: anything with constriction
Place foley in spinal injury patients too keep the bladder empty and offering laxatives and loose clothes
a client is admitted to the hospital with a diagnosis of neurogenic shock. After a traumatic motor vehicle collision. Which manifestation best characterizes this diagnosis?
bradycardia
Systematic Inflammatory Response Syndrome
Multiple Organ-Dysfunction Syndrome (MODS)
Acute Respiratory Distress Syndrome (ARDS)
Lead II ECG interpretation
Mechanical Ventilation
Hemodyamic Monitoring
Pacing Devices
Hemodialysis
Client who reports muscle cramps and tingling in hands
Prepare to give calcium carbonate
Priority assessment finding for hemodialysis clients?
Restlessness (disequilibrium syndrome) = Solutes are removed too quickly from the blood which causes the brain cells to swell with fluid, deadly ICP
restless and disoriented
vomiting
headache
Stop/slow infusion and report to HCP
Care for Fistula (AV shunt)
Connection between artery and vein
High pressure flow through low pressure vein
Squeeze rip rubber ball or sponge to help blood flow and strengthen
Pitting edema = normal
avoid pressure and monitor for infection and bleeding, should always feel a thrill - check this multiple times per day
Monitor for thrombosis
Report to HCP:
Palor
Paresthesia (numbness and tingling)
Pt can lose arm from lack of oxygen
Early intervention saves life and limb
No restrictive clothing or jewelry
No BP on affected arm
No sleeping on arm
No creams or lotions
No lifting over 5lbs
Medical Emergencies (BLS)
Cardiac arrest living saving measures
Caused by Hypoxia, resp failure, toxins, blood clots and electrolyte imbalances
Clients present without a pulse, resp rate and unresponsive
Asysytaole on cardiac monitor
Immediate CPR with high quality chest compressions
Before calling for help if you’re the only care giver (immediate oxygen and perfusion to the brain and vital organs)
Adult CPR: Chest compressions mediately 100-120 per min, depth 2-2.4 inches (5-6cm)
Hands in centre of chest lower half of sternum
Breaths: manual = 30 compressions, 2 rescue breaths
Intubated: every 6 seconds without interruption
after each compression the chest should fully recoil to allow heart to refill with blood, compressions are paused every 2 minutes to asses pulse. Do not pause for more than 10 seconds
AED: automated external defibrillator - 8 years and older place in upper right chest near the shoulder
Left lateral chest near the anterior axillary line below the nipple
How to shock:
Defibrillator pads are placed
Call out and look to make sure everyone is all clear
Continue chest compressions immediately after shock is delivered
Pediatrc AED: can use adult pads if paediatric pads aren’t available
1 AED pads on chest and 1 on back
do not overlap or touch pads
No shocking Asystole (flat line)
PEA (pulseless electrical activity)
For Asystole:
High quality CPR priority
Epinephrine even 3-4 minutes
Intubate and ventilate
treat the causes
CPR with pregnancy
Chest compressions slightly higher on the sternum
Uterus manually displaced to left side or place a rolled blanket under right side
Not Supine
Regular chest compressions not affective
Iim ciiruclaton does not return after 4mins immediate C section must take place
Infant CPR less than 1 years old
Brachial pulse 10 seconds or less
bicep area located between elbow and shoulder
Call for help activate an emergency response
2 minutes cpr 100-120 compression
single rescuer 30:2
two rescuer 15:2
Reteriiive AED after 2 min CPR single rescuer unwitnessed event
Adequate ventilation
Roll under shoulders
Neck slightly extentented
Two thumbs middle of sternum below nipple line
two fingers in middle of sternum. other hand supporting the back
Post resuscitation care
Comatose: not following commands
Cold fluids for therapeutic hypothermia done within 6 hours of event
ABG interpretation
Body loves too keep in perfect balance, to much of one thing can be deadly
Acid = more dangerous than base
Too much acid in the body can turn the lungs off = hypercapicnic respiratory failure, CO@ builds up in the blood and pushes the body into an acidic state this turns off the drive to breathe
Respiratory acidosis
Low and slow breathing = respiratory acidosis
acid kills the quickest
retaining too much CO2
Opioid overdose
COPD (high CO2)
Main intervention for resp acidosis: Bipap machine to forcefully push air into the lungs and force gas exchange