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AU'18 Oral Path II > Common Lesions - part I > Flashcards

Common Lesions - part I Flashcards

1
Q

nevus

A

“mole” - benign melanocytic lesion

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2
Q

a decrease in pigmentation occurs as the nevi does what?

A

progress from junctional to compound to intradermal

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3
Q

growth of nevus

A

junctional –> compound –> intradermal/intramucosal

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4
Q

ephelides

A

“freckles” - brown pigmentation that develops following sun exposure

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5
Q

ephelides is more common in who?

A

children and fair skinned individuals

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6
Q

lentigo is what type of lesion?

A

benign melanocytic

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7
Q

lentigo

A

macular (flat), no change in color intensity with exposure to UV light

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8
Q

number of lentigo increases in who with age?

A

in Caucasians

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9
Q

seborrheic keratosis

A

benign skin lesion with a “stuck on” appearance

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10
Q

what does seborrheic keratosis look like?

A

“dropped on candlewax”

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11
Q

dermatosis papulosa nigra is a variant of what?

A

seborrheic keratosis

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12
Q

dermatosis papulosa nigra occurs in what percent of the black population?

A

~30%

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13
Q

actinic keratosis is a precursor lesions for what?

A

cutaneous squamous cell carcinoma

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14
Q

actinic keratosis has what type of texture?

A

“sandpaper” texture

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15
Q

tx for actinic keratosis

A
  1. surgical excision
  2. topical, immune-activating agents such as Aldara
  3. use of sun blocking agents
  4. limit sun exposure
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16
Q

telangectatic capillaries

A

prominent vessels

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17
Q

telangectatic capillaries may be an indication of what?

A

sun damage

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18
Q

when does sebaceous hyperplasia occur?

A

usually over 40 y.o.

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19
Q

where does sebaceous hyperplasia occur?

A

often seen on forehead

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20
Q

what happens when sebaceous hyperplasia achieve 1-2 mm in size?

A

minimal to no further growth

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21
Q

what is the central umbilication often seen in pts with sebaceous hyperplasia?

A

sebaceous duct

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22
Q

what is the most common cancer in humans?

A

basal cell carcinoma

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23
Q

clinical features of basal cell carcinoma

A
  1. “mask” area
  2. rolled borders
  3. umbilicated center
  4. telangiectasia
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24
Q

what is often associated with basal cell carcinoma?

A

nevoid basal cell carcinoma syndrome

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25
Q

fordyce granules

A

ectopic sebaceous glands

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26
Q

fordyce granules can be seen anywhere in the mouth but what is the most common location?

A

buccal mucosa

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27
Q

T/F: fordyce granules can appear on the lips

A

true

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28
Q

what is angular cheilitis associated with?

A

loss of vertical dimension

29
Q

what causes angular cheilitis?

A

candida

30
Q

some angular cheilitis may be co-infected with what?

A

candida and staph

31
Q

tx of angular cheilitis if for external use only

A

can use topical application of combo antifungal/corticosteroid cream

32
Q

what is the most common site for recurrent HSV-1?

A

vermilion border and/or adjacent skin of lips

33
Q

melanotic macule

A

focal increase in melanin

34
Q

melanotic macule can also occur as reactive melanosis in response to what?

A

local trauma

35
Q

mucocele

A

focal deposition of mucous

36
Q

what causes mucocele?

A

damage to associated minor salivary gland duct

37
Q

tx of mucocele

A

conservative…

remove extravasated mucous and associated minor salivary glands

38
Q

leukoedema

A

intracellular edema

39
Q

where is linea alba found?

A

along occlusal plane

40
Q

morsicatio buccarum, linguarum, labiorum

A

cheek, tongue, lip nibbling/chewing

41
Q

what causes morsicatio buccarum, linguarum, labiorum?

A

shredded keratin at site(s) accessible to teeth

42
Q

fibroma

A

benign collection of dense fibrous CT

43
Q

tx of fibroma

A

conservative removal

44
Q

what causes lichen planus?

A

it’s an immunologically mediated process

45
Q

clinical features of lichen planus

A
  1. “striae” or lacy

2. doesn’t wipe off

46
Q

lichenoid mucositis

A

descriptive term which could apply to several conditions

47
Q

what does maxillary torus comprise of?

A

dense, vital lamellar bone

48
Q

where can inflammatory papillary hyperplasia be found?

A
  1. under sub-optimally fitting RPD or full denture

2. high palatal vault

49
Q

what might inflammatory papillary hyperplasia reflect?

A

constant wear

50
Q

tx of inflammatory papillary hyperplasia

A
  1. conservative excision

2. new denture

51
Q

nicotine stomatitis

A

inflammed minor salivary glands of the palate with hyperkeratosis around the orifices

52
Q

nicotine stomatitis is commonly seen in who?

A
  1. pipe smokers

2. long-term use of hot beverages

53
Q

what causes black hairy tongue?

A

overgrowth of chromogenic bacteria and filiform papillae

54
Q

fissured tongue

A

multiple grooves in tongue

55
Q

fissured tongue pts often also have what?

A

geographic tongue

56
Q

pts with geographic tongues may be sensitive to what?

A

spicy or acidic food when lesions are present

57
Q

where does ectopic geographic tongue occur?

A

in locations other than dorsal or lateral tongue

58
Q

foliate papilla is part of what?

A

Waldeyer’s ring

59
Q

foliate papilla

A

vertical lines at posterior lateral tongue often see lymphoid tissue in that area as well

60
Q

mandibular tori

A

vital lamellar bone

61
Q

where are mandibular tori commonly seen?

A

lingual

62
Q

amalgam tattoo

A

silver in amalgam stains reticulin fibers in associated CT

63
Q

if you’re unsure if it’s an amalgam tattoo, what should you do?

A

may need to excise to rule out melanoma

64
Q

parulis

A

intraoral opening of sinus track

65
Q

what must be ruled out to dx parulis?

A

odontogenic source of infection

66
Q

where is pericoronitis most commonly found?

A

in mandibular 3rd molars

67
Q

what causes pericoronitis?

A

food, etc. gets caught b/w overlying soft tissue (operculum) and crown of partially impacted tooth

68
Q

tx of pericoronitis

A

remove offending tooth and opposing 3rd molar

69
Q

what might be done to initially tx pericoronitis?

A

decrease local inflammation with rinses then surgery

AU'18 Oral Path II (31 decks)

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