Clinical evaluation Flashcards

1
Q

elevated LFTs, no symptoms. Poor transmission , high attenuation

A

Hepatic steatosis / FL infiltration

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1
Q

Focal echogenic area.
Patch of fatty liver. (No mass effect)

A

Focal fatty infiltration

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2
Q

Focal hypoechoic area.
Patch of normal liver. Most common location:
next to GB/porta hepatis

A

Focal fatty sparing

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3
Q

Clinical: Poor liver function symptoms = elevated LFTs, jaundice (elev total or direct
bilirubin), fatigue, weight loss, diarrhea
Sono: heterogeneous/coarse texture, small right lobe, enlarged caudate lobe, nodular
surface, ascites

A

Cirrhosis

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4
Q

Most common cause is alcoholism.

A

Cirrhosis

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5
Q

Clinical: SAME as advanced cirrhosis + may have: caput madusa (superficial abd veins) and
GI bleeding
Sono: Hepatofugal PV flow, dilated MPV >13mm, abdominal varices = dilated venous
collaterals near spleen, stomach, and esophagus, abnormal splenic vein flow,
splenomegaly, recanalized paraumbilical vein

A

Portal Hptn

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6
Q

Most common cause is cirrhosis.

A

Portal Hypertension

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7
Q

Clinical: Pain, elevated LFTs, hypovolemia, nausea, vomiting

A

Portal vein compression / thrombosis

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8
Q

most commonly caused by tumors or lymphadenopathy.

A

Portal vein compression / thrombosis

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9
Q

Sono: Thrombosis of PV, cavernous transformation

A

Portal vein compression / thrombosis

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10
Q

Clinical: Elevated LFTs
Sono: Hepatomegaly, enlarged caudate lobe, absent flow hepatic veins

A

Budd-Chiari Syndrome

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11
Q

: No signs of infection. Only evidence of decreased liver function

A

Hepatitis C

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12
Q

: Fever, non-obstructive jaundice (elevated direct bilirubin), elevated LFTs

A

Hep a

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13
Q

“Starry sky” sign
Periportal cuffing
Inc echogenicity of portal triads

A

hep a

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14
Q

Benign/non-endocrine

A

asymptomatic

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15
Q

Malignant =

A

symptomatic

16
Q

Made of fat. Hyperechoic

17
Q

Associated with oral contraceptives. Varied, may be echogenic

A

Hepatocellular Adenoma

18
Q

2nd most common benign liver tumor. “Stealth lesion” because it
may be isoechoic to liver tissue. Central scar with vascularity. Look
for “mass effect”

A

Focal Nodular Hyperplasia

19
Q

“Bleed” from trauma or surgery. Clinical: Trauma or Biopsy Hx, decreased hematocrit, pain

20
Q

Increased risk = chronic liver disease, cirrhosis, hepatitis.
Tumor marker = elevated AFP (alphafetoprotein)

A

Hepatocellular Carcinoma - HCC aka hepatoma

21
Q

Clinical: possible abnormal LFTs, pain, jaundice
Sono: multiple masses with variable appearance,
ascites

A

Metastasis

22
Q

: Most common. Fold of fundus over body

A

Phrygian cap

23
Q

Outpouching of neck

A

Hartmann pouch

24
Fold at neck
Junctional fold
25
Echogenic non-mobile mass projecting from inner lumen
polyps
26
Focal or diffuse wall thickening with comet tail artifact
Adenomyomatosis
27
Hyperechoic GB with shadowing. Shadowing is mild and the posterior wall is still seen.
Porcelain GB
28
A GB packed with stones. Only the anterior wall, echo, solid shadow posterior. GB is not seen and posterior wall is not visualized.
WES sign
29
jaundice, pain, fever with stone lodged in cystic duct and compression of CBD
Mirizzi syndrome
30
Enlarged GB caused by pancreatic head mass. Painless jaundice
Courvoisier GB
31
: + Murphy’s sign (pain with probe pressure), fever, leuko, elevated ALP, bilirubin, nausea, vomiting
acute cholecystitis
32
Charcot triad (pain, fever, jaundice), elevated ALP, bilirubin, nausea, vomiting
Acute cholangitis
33
: Weight loss, RUQ pain, jaundice if obstructive
gb carcinoma
34
: Weight loss, RUQ pain, jaundice, pruritus (excessive itchiness), Hx of sclerosing cholangitis
Cholangiocarcinoma / klatskin tumor
35