Classification Part 1 Flashcards

1
Q

Classification

A
  • Important activity in clinical work and research
  • Basic part of science
  • Information made more accessible, meaningful, and less cumbersome
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2
Q

Defining abnormal vs. normal

A

we have more information about abnormal behaviour than normal (easier to say what’s abnormal than what’s normal)

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3
Q

Steps in defining abnormal

A
  • into subclasses
  • starts out as binary (ie. abnormal vs. normal)
  • then can break it down further (ie. Mushrooms: flower vs. mushroom -> poisonous vs. not)
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4
Q

Paradigms and classification

A
  • Paradigms are sets of assumptions that outline the universe of scientific inquiry
  • Paradigms have influenced how classification done and what was classified
  • Ex. Hippocrates’ Four humors
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5
Q

Philosophical Issues in Abnormal Behaviour Paradigms

A
  • Nature of psychopathy, normalcy, belief in paradigm
  • Historical
    • Emil Kraeplin and Neo-Kraeplians
    • Freud
  • Contemporary
    • DSM & International Classification of Diseases (ICD)
    • PDM & OPDS
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6
Q

Back Masking

A

Believing that something is in an ambiguous stimulus, and finding it because you’re looking for it

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7
Q

2 major classification trends/philosophies

A
  • Symptom as Focus (Kraeplin)

- Underlying Cause as Focus (Freud)

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8
Q

Symptom as Focus

A
  • Group of symptoms or observable behaviours (ie. Knee pain)
  • Seen as cause of difficulties
    • Issue: what if pain is not the problem?
  • Focus of assessment & treatment is on eradicating the symptoms
    • Collect symptoms, call it something (ie. Depression), try to get rid of the symptoms and your job is done
  • Approach done by Behaviourism school, ICD, DSM
  • Variant embraced by Managed Care in US (insurance companies)
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9
Q

Underlying Cause as Focus

A
  • Problems caused by underlying process (ie. Broken knee bone)
  • Assessment and treatment focuses on underlying process (ie. Focus on healing broken bone and pain will go away too)
    • Issue: based on what are thought to be the causes
  • Orientation of psychodynamic, cognitive behavioural, and PDM
    • Today, there’s been a shift from symptom as focus to underlying cause as focus
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10
Q

7 Purposes of Classification

A
  • Description and need to identify
  • Communication (by classifying a “depressive episode”, we can communicate what it is to others)
  • Research (by classifying PTSD, we can design studies about PTSD)
  • Treatment (leads to it)
  • Insurance (some things covered, others aren’t)
  • Theory development (theories based on class. systems)
  • Epidemiological info (classification helpful for figuring out increases vs. decreases)
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11
Q

Diagnosis and treatment

A
  • Diagnosis does not always lead to proper treatment:

- Ex. Alzheimer’s -> only correct diagnosis happens post-mortem

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12
Q

DSM

A
  • Categorical/binary approach to define abnormality (you either have it or you don’t)
  • Revised periodically
  • Over 400 disorders
  • DSM provides descriptive information not based on any one theoretical perspective (although this is debatable)
  • Descriptive features are based on observable features
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13
Q

2 categories of Diagnostic Manuals

A
  • Ones used for diagnosis -> DSM, ICD: descriptive, categorical (Kraeplinian)
  • Ones used for formulation -> Psychodynamic Diagnostic Manual (PDM) and Operationalized Psychodynamic Diagnosis (OPD)
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14
Q

Diagnosis vs. Formulation

A
  • Diagnosis: Assigning diagnostic category (assigning a label)
    • structural interview -> diagnosis
  • Formulation: Attempt to explain genesis, maintenance, and process related information for treatment (unique model of how a particular person functions)
    • Assessment -> formulation
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15
Q

PDM (Psychodynamic Diagnostic Manual)

A
  • States that DSM provides one level of description, but the PDM describes elements not found in DSM; providing info to improve comprehensive treatments
  • Supplements DSM
  • Developed from a theoretical perspective (Current Psychodynamic Theory)
    • Ex. Psychoanalysis
    • Ex. Object Relations
    • Ex. Attachment Theory
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16
Q

DSM vs. PDM

A
  • DSM describes disorders; PDM helps to understand people (surface & deeper levels of personality; person’s emotional and social functioning; based on current neuroscience)
  • Ex. Personality patterns, social & emotional capacities, unique mental profiles, personal experiences of individuals
  • DSM = problem; PDM = person
17
Q

Rationale of PDM

A
  • Human behaviour is complex
  • DSM simplifies behaviour too much
  • Want to direct focus on full range of affect, thought, behaviour in context of an individual’s own unique history
  • Rather than thinking of people having discrete disorders (i.e., ego dystonic, separate, outside of self), see disorders as result of some process (personality, incorporation of upbringing, etc.) and the process is what is important
18
Q

PDM Dimensions

A
  • Personality Patterns & Disorders (P Axis)
  • Mental Functioning (M Axis)
  • Manifest Symptoms & Concerns (S Axis)
19
Q

PDM: P Axis

A
  • Figuring out person’s location on the continuum between Healthy—Disordered
  • Ways in which a person organizes mental functioning and interacts with the world
  • Maxim: need to understand person in order to understand problem
20
Q

PDM: M Axis

A
  • Detailed look at emotional functioning
  • ex. Information processing, self‐regulation, relationships, emotional
    expression, learning, coping/defenses, etc.
21
Q

PDM: S Axis

A
  • Using many of the DSM categories, focus on personal experience of
    difficulties
  • Need to be seen in context of personality and mental functioning
22
Q

Kraeplin’s work (T)

A
  • realized that mental disorders had a tendency for groups of symptoms (aka: syndrome) to appear together regularly enough that they had an underlying physical cause
  • Proposed 2 groups of mental diseases: dementia praecox (schizophrenia today) due to chemical imbalance & manic-depressive psychosis (BPD today) due to metabolism irregularity
23
Q

4 major paradigms (T)

A
  • biological paradigm
  • cognitive-behavioural paradigm
  • psychoanalytic paradigm (less influential)
  • humanistic paradigm (less influential)
24
Q

biological paradigm

A
  • mental disorders are caused by biological processes (continuation of somatogenic hypothesis)
  • aka medical model or disease model
  • comprised of behaviour genetics, molecular genetics, and biochemistry
  • ex. using DBS to alleviate depression
25
Q

biological paradigm: behaviour genetics

A
  • study of individual differences in behaviour that are attributable to differences in genetics
  • genotype is fixed at birth, phenotype changes
  • predisposition to disorders (diathesis) may be inherited (genotype), but the expression of the disorder (phenotype) depends on environmental factors too
  • includes family method (taking people with disorders “index cases/probands” and looking at their relatives to see if they have them too), twin method (if 1 twin has disorder, greater concordance should be found with MZ than DZ twins), adoptees method (if child of panic disorder parent raised by someone else and still has panic disorder, we can assume it’s more genetic), and linkage analysis
26
Q

biological paradigm: molecular genetics

A
  • trying to specify the particular gene involved in disorders
  • uses linkage analysis (studying families with many cases of a disorder to look for genetic markers)
27
Q

genetics and temperament

A
  • temperament is largely genetically determined
  • 3 categories: difficult, easy, and slow-to-warm-up
  • 3 categories of US boys: resilient, overcontrolling (shy/lonely), undercontrolling (delinquency, low IQ)
28
Q

neuroscience

A
  • study of brain and neurotransmitters
  • some theories propose that disorders are caused by too much or too little of a neurotransmitter (ex. mania is to much norepinephrine, ADHD due to lack of dopamine)
29
Q

reductionism

A

the view that whatever is being studied can and should be reduced to its most basic elements -> should be avoided

30
Q

cognitive-behavioural paradigm

A
  • a type of action/behavioural therapy (changes person’s actions)
  • behavioural/learning perspective: abnormal behaviour is a learned response; focus on observable behaviour (ex. classical conditioning, operant conditioning, modelling, cognitive behaviour therapy)
    • behaviour modification/therapy: changing abnormal behaviour using the above methods (ex. counterconditioning, systematic desensitization, aversive conditioning, etc.)
  • cognitive perspective: how people structure and make sense of expectations (ie. schemas)
    • ex. Beck’s cognitive therapy persuades depressed clients to alter negative schemas of themselves; Rational-Emotive Behaviour Therapy (eliminating self-defeating beliefs)
  • CBT restructures patterns of thought that cause disturbed emotions/behaviours
  • based on abstract concepts, doesn’t necessarily tell us WHY/HOW people have negative thoughts
31
Q

psychoanalytic paradigm

A
  • psychopathology results from unconscious conflicts in the individual
  • a type of insight therapy (helping people gain insight to why they think/behave the way they do)
  • developed by Freud
  • includes neurotic anxiety (fear not connected to true threat) and moral anxiety (“perfection principle”); defence mechanisms, free association, dream analysis, etc.
  • evolved into ego analysis (greater emphasis on person’a bility to control environment and select how to satisfy drives)
  • contemporary includes structural theory, self-psychology, object-relations theory, interpersonal-relational (between client and environment/others), and attachment theory
  • anecdotal, not always scientific
  • BUT childhood experiences do shape adult personality, unconscious influences on behaviour exist, and people use defence mechanisms to control anxiety/stress
32
Q

Humanistic Paradigm

A
  • abnormal behaviour can be treated by increasing one’s awareness of motivations and needs; based more in free will than psychoanalytic
  • insight-focused
  • ex. Rogers’ Client-Centered therapy (client directs course of therapy session) -> unconditional positive regard; empathetic therapies (which are very useful)
33
Q

electicism

A
  • employing ideas and therapeutic techniques from a variety of schools
  • very common, esp. for treating things like eating disorders
34
Q

Psychosocial factors that influence mental health

A
  • Familial factors (ex. parenting styles, marital discord, parental mental illness)
    • ex. privileged parents who have high expectations but aren’t involved have kids more likely to have mental health issues (“affluenza”)
  • Peers (ex. peer status, peer victimization)
  • Cultural Context (ex. cultural diversity, minority groups)
    • ex. no differences between Anglo- and French-Canadians, but major differences between Aboriginal people and other Canadians - ex. PTSD, depression, etc. with the exception of Cree people); Hutterites have lowest rates of schizophrenia; immigrants have lower depression and alcohol dependence, but higher mood disorders)
  • Individual factors (ex. temperament, prematurity, brain damage)
  • Life Events (ex. abuse, homelessness)
  • School context (ex. school failure)
35
Q

diathesis-stress model

A
  • integrative approach
  • says mental illness is due to genetic predisposition AND stressful life event that triggers it
  • ex. BPD triggered by a personal childhood vulnerability and the stressful life event of moving away to college)
36
Q

biopsychosocial paradigm

A
  • An integrative paradigm that suggests that all normal and abnormal behaviour is caused by an interaction of biological (ex. genes, hormones), psychological (ex. personality, social skills), and social factors (ex. poverty, marital conflict)
  • ex. person with genetic predisposition, low social skills, and living in poverty would have higher likelihood of mental illness