CHN II Flashcards
Any microorganisms capable of producing a disease
e.g., bacteria, virus, fungi, parasites.
INFECTIOUS AGENT
Environment or object in which organism can survive or multiply
e.g., Human ,animals, fomites, soil, water
RESERVOIR
CHAIN OF INFECTION
Infectious Agent
Reservoir
Portal of Exit/ Mode of escape
Mode of Transmission
Portal of Entry
Susceptible Host
Venue or way which organism leaves the reservoir
Alimentary: Vomiting, diarrhea, biting
Respiratory: Coughing, sneezing, talking
Genitory-Urinary: Sexual - Transmission
Transplacental: Mother to Fetus
Skin:I Skin lesion, cut, blood
PORTAL OF EXIT/ MODE OF ESCAPE
Direct/Indirect Droplet
Particles: >5 mm
Distances: 3 feet
Contact
Respiratory secretions
Fine Particles: <5mm
More than 3ft distance
All droplets can be airborne (PTB)
Airborne
Carrier
e.g., Food, water, medication
Vehicle
Carrier
Insects; Mosquito
Vector
-Ingestion, Inhalation, Penetration
PORTAL OF ENTRY
-At risk -Infant & Children
-Old people -Immunocompromised
SUSCEPTIBLE HOST
-Entry until first s/sx
-Person is unaware of impending illness
INCUBATION PERIOD
-Nonspecific s/sx: Flu-like
-Activates immune response
PRODROMAL PERIOD
-Specific s/sx
-Pathognomonic sign
ACUTE PERIOD
-s/sx starts to disappear
-pt regains health
CONVALESCENT PERIOD
Healthy again.
RESOLUTION AGENT
-Physical & chemical barriers
-Natural flora
-Skin & mucuous membrane
-oils & sweats
-Cilia
-Gag, cough reflex
First line of Defense
-Inflammatory response
-Activate cells: B-E-N
Basophils, Eosinophils, Neutrophils
Second line of Defense
Redness; increase blood flow
Rubor
Swelling; Exudation of fluid
Tumor
Heat; Release of inflammatory Mediators
Calor
Pain; Stretching of pain receptors
dolor
Loss of function; Disruption of tissue structure
Functio Laesa
-Immune response (B-cells & T-cells)
-Specific Protection; Lymphocyres
THIRD LINE OF DEFENSE
-Proteins that destroy familiar antigen
ANTIBODIES
-Foreign bodies (Microorganism)
ANTIGEN
-High resistance; High Antibodies
IMMUNITY
TYPES OF IMMUNITY -Organs, tissues, cells of immune system.
INNATE
TYPES OF IMMUNITY Immunity that develops during lifetime.
ACQUIRED
TYPES OF IMMUNITY In response to an infection or vaccine.
ACTIVE IMMUNITY
TYPES OF IMMUNITY -In response to infection
ACTIVE-NATURAL IMMUNITY
TYPES OF IMMUNITY-In response to vaccine
ACTIVE-ARTIFICIAL IMMUNITY
TYPES OF IMMUNITY -Breastmilk from Mother IgA & Placenta IgG
PASSIVE-NATURAL IMMUNITY
TYPES OF IMMUNITY-After receive antibodies from someone
PASSIVE IMMUNITY
TYPES OF IMMUNITY-From medication/ Immunoglobulin
PASSIVE-ARTIFICIAL IMMUNITY
5 ELEMENTS OF STANDARD PRECAUTIONS
Handwashing first tier
Gloves: clean & sterile
Gown or Apron
Goggles or Mask
Proper Disposal of sharps
-Do not recap
PULMONARY TUBERCULOSIS
Causative Agent
Myobacterium Tuberculosis
Mode of Transmission: PULMONARY TUBERCULOSIS
Airborne-Droplet (coughing, sneezing)
Incubation Period: PULMONARY TUBERCULOSIS
3-8 weeks
Prevention: PULMONARY TUBERCULOSIS
BCG (Given at Birth)
LATENT TB
-Dormant
-Asymptomatic
ACTIVE TB
-Afternoon Low grade fever
-Blood in sputum (Hemoptysis)
-Cough (Chronic) > 2weeks
-Decrease in weight
-Evening sweat/ night sweat
Confirms the diagnosis of PTB
Definitive Test
SPUTUM SMEAR AND CULTURE aka ACID FAST BACILLI (AFB) STAINING
Detects the presence of antibodies
MANTOUX TEST/ TUBERCULIN TEST/ PPD (Purified Protein Derivative)
Result of Mantoux test/ PPD
-Within 48-72 hrs
-(+) induration of 10mm or more
-(+) immunocompromised induration of >5mm
Determines the presence & extent of the disease/ lesion
CHEST X-RAY
-No exposure
-Not infected
CLASS 0
-(-) TB exposure
-(-) s/sx
-(-)test
-(-)x-ray
CLASS I
-TB Infection (preclinical state)
-(-/+) exposure
-(+)tuberculin test
-(-) s/sx
-(-) x-ray
CLASS II
-TB Diseases
-(+)History
-(+)tuberculin test
-(+)s/sx
-(+)sputum
-(+)x-ray
CLASS III
-TB inactive
-(+/-) previous therapy
-(+)x-ray of healed TB
-(+) Tuberculin Test
-(-) S/sx
-(-) Smear
CLASS IV
-Suspected disease
-diagnosis pending
CLASS V
S/E: Orange discoloration of secretion and urine.
RIFAMPICIN
When to take: RIFAMPICIN
1st: Empty stomach (best) but can cause gastric irritation
2nd : with food
3rd: at bedtime
NRSG ALERT: RIFAMPICIN
Protect drug from light
A/E: RIFAMPICIN
Hepatoxicity
S/E: Hepatic enzymes elevation
Peripheral neuropathy (competes w/ vit.6 B6 absorption)
ISONIAZID
When to take: ISONIAZID
Before meals
Prophylaxis: ISONIAZID
10-50 mg
Treatment: ISONIAZID
5-100 mg
A/E: ISONIAZID
Hepatoxicity
HPN Crises
-Rapidly bacteriostatic
-slowly bacteriocidal
-may lead to hyperurecemia
-Arthralgia (Shoulder)
PYRAZINAMIDE
When to take:PYRAZINAMIDE
with or without food
2 TB DRUGS PHOTOSENSITIVE?
Rifampicin & Pyrazinamide
A/E:Pyrazinamide
Hepatoxicity
Ototoxicity
Nephrotoxicity
GI Upset
Leads to optic neuritis (Red-green discrimination)
-Skin rash
ETHAMBUTOL
WHY DO NOT GIVE ETHAMBUTOL TO CHILDREN 6 YRS OR YOUNGER
R: because they cannot reliably monitor Vision.
-Renal impairment
-Tinnitus (ringing of the ears)
-Auditory impairment
STREPTOMYCIN
Causative agent:TETANUS
clostridium tetani
Excretes toxins:
-Destruction
-Weak toxin
-Lysis of blood
TETANOLYSIN
Excretes toxins:
-Type of neurotoxins
-Cause muscle spasm
TETANOSPASMIN
Mode of transmission: TETANUS
Enters skin via non-intact skin
Incubation period:TETANUS
3 days to 1 month
Prevention: TETANUS
-TT Vaccination
-Health education
Pathognomonic sign: TETANUS
Risus-Sardonicus/Sardonic smile/ grin
-Facial nerve affected
S/SX: SPASM
Involuntary muscle contraction
Dangerous: Laryngeal Muscle
S/SX: TRISMUS
Trigemiinal nerve (lock jaw)
Opisthotonus (arching of the back)
Risus-Sardonicus/Sardonic smile/ grin
Nursing Responsibility: Lock jaw
NGT/ TPN
Nursing Responsibility:-Opisthotonus (arching of the
-Side lying
DOC: TETANUS
PENICILLIN
METRONIDAZOLE
A/E: Disulfiram-like reaction: TETANUS DOC
METRONIDAZOLE
DISULFIRAM -LIKE REACTION:
-Anxiety
-Blurred Vision
-Choking/ chest pain
-DOB
-Flushing of face
-Headache
-Sweating
-Vomiting
NURSING MANAGEMENT: TETANUS
1.Non-stimulating environment
Quiet and Dim
2.Tracheostomy -spasm
3.Cardiac Monitor
Causative agent: POLIOMYELITIS
Legio Debilitans
I.Brunhilde
II.Lansing
III.Leon
OTHER NAME OF POLIOMYELITIS
(Heins-Medin Disease)
Mode of transmission: POLIOMYELITIS
FECAL-ORAL
Prevention: POLIOMYELITIS
-OPV- Sabin V (NPO to promote absorption)
-IPV- Salk V sanitation
-SANITATION
ABORTIVE POLIO
-localized
-Sore throat
-Tonsilitis
-Enlarged cervical lymph nodes
NON-PARALYTIC POLIO
-Systemic
-Flu-like symptoms
PARALYTIC POLIO
-Reaches Spinal cord
-Painful muscle spasm usually one sided.
-damaged neurons (Asymmetrical paralysis)
BULBAR POLIO
-reaches the brain
-Brainstem (Pons, medulla oblongata)
-Respiratory paralysis
-HPN (silent killer)
MANAGEMENT: POLIO
-Analgesic
-penicillin
-no to opiods
-Aggrevate Respiratory (Demerol Paralysis)
NRSG MANAGEMENT: POLIO
-Sanitation
Causative agent: CHICKEN POX
Varicella-Zoster Virus, Herpes Virus
Other name of chicken pox
(VARICELLA)
reactivation of the varicella-zoster virus
Herpes Zoster
Herpes Zoster
-Stress
-Same manifestation w/ severe pain & tenderness along posterior nerve.
Mode of Transmission: Chicken Pox
Direct Contact
Incubation period: Chicken Pox
2-3 weeks
Prevention: Chicken Pox
Active immunization with live attenuated varicella vaccine
S/sx: Chicken pox
-Trunkal–> rashes–>macule–>papule–> vesicle–>scabs–>full off–>Shallow pink depression
-(+) fever peaking on 2-4 days
Management: Chicken Pox
Aziclovir 800 mg 3x/ day
Supportive management: Chicken Pox
Antipyretic
Short fingernails
Isolation & Airborne precautions
Causative agent: MUMPS
Mumps Virus (Rubivirus)
Mode of transmission: MUMPS
direct contact by airbornee, droplet or fomites contaminated by saliva.
Incubation period: MUMPS
12-26 days life long immunity after recovery.
Prevention: MUMPS
MMR (12-15 month; Booster dose: 4-6 years)
S/sx: MMR
Asymptomatic in 30-40% cases
-Fever -anorexic
-malaise -swollen, painful, &
tender parotid glands
-Affecting submaxillary &
sublingual glands
Complications: mumps
-Epididymorchitis
-Meningoecephalitis
-Oophoritis
-Pancreatitis
Supportive management:
Comfort Measure, isolation & droplet precaution.
Causative Agent: MEASLES
Filterable virus
genus: Morbillivirus Family Paramyxoviridae
Mode of transmission: MEASLES
Direct contact with infectious airborne droplet.
Incubation period: MEASLES
8-12 DAYS
Prevention: MEASLES
MMR
S/Sx:MEASLES
initially an upper respiratory condition characterized by escalating fever (Peak 3-5 days)
Pathognomonic sign: MEASLES
Koplik’’s Sign: Graying peck in the buccal mucosa- 2nd day
MANAGEMENT: MEASLES
Supportive measures
Anti-pyretic with fluids
Vitamin A supplement
Causative Agent: DIPTHERIA
Corynebacterium Diptheria (Klebs-Loeffer Bacillus)
HIGH PREVALANCE OF DIPTHERIA DURING:
Cooler months (December-February)
KNOWN AS EPIDEMIC PAROTITIS
MUMPS
Mode of transmission: DIPTHERIA
-Direct or Intimate contact with carrier
-indirect contact with articles, food, & environment contaminated with discharge from nose, skin, eyes, or lesion on body parts of infected person.
Incubation period: DIPTHERIA
2-5 days
s/sx: DIPTHERIA
Low grade fever
Pseudomembrane
Nasal
Facial & Pharyngeal
Laryngeal
Management: DIPTHERIA
Isolaton: until (-) nose or throat culture
Antibiotic
Bed rest 2-3 weeks
Nutrition & hydration
Gas exchange
Diagnosis: DIPTHERIA
Specimen Culture
Causative agent: FLU
Influenza Virus (A,B,C)
Mode of transmission: FLU
Direct contact by mouth & nose
Droplet secretions (Upper airway)
Incubation period: FLU
3-5 days
Prevention: FLU
HIB, Influenza Vaccine
S/sx: FLU
-Bodyache -chills
-dry cough -fever
-headache -Pharyngitis
Supportive measures: flue
Anti-pyretic
Analgesics
Fluids
Rest
Vit. A Supplement
Causative agent: pertussis/ whooping cough
Hemophilus Pertussis
Bordetella Pertussis
Bordet Gengou Bacillus
Mode of transmission: pertussis
Respiratory secretions
Droplet-airborne
Fomites
***Highly contagious except: PERTUSSIS
3rd Phase
ncubation period: PERTUSSIS
6-20 Days
Prevention:
DPT Vaccine & Mask
1st phase: PERTUSSIS
(1-2 weeks)
Common cold
2nd phase: PERTUSSIS
(Month or longer)
No fever
Mid-cough- severe, violent & productive
3rd phase: PERTUSSIS
Decrease frequency & severity of coughing
Pathognomonic sign : PERTUSSIS
WHOOPING COUGH
Causative Agent: RUBELLA
Rubella Virus
Mode of transmission: RUBELLA
Airborne-Droplet
Incubation period: RUBELLA
12-19 days
Prevention: RUBELLA
MMR & MASK
S/sx: RUBELLA
A-C-E H-E-L-P
Arthralgia (joint pain)
Coryza (runny nose)
Enlarged cervical lymph nodes
Headache
Eyes (Red & itchy)
Low grade fever
Pink rash (begins at the face- trunk-arm-legs)
causative agent: DENGUE
-Dengue virus: 1,2,3,4
-O’nyong nyong
CAUSATIVE AGENT OF DENGUE (MILD)
Chikungunya Virus
Mode of transmission: DENGUE
Aedes Aegypti
GRADE I: MILD (DENGUE)
Enters the bloodstream
Decrease Immune response
Increase Bradykinin
Increase Prostalandin
Histamine (Flushes skin)
(Herman’s sign)
PAIN: DENGUE
Head
Abdomen
Muscle
Joints
Bone
GRADE 2: DENGUE
***Grade 1 + Spontaneous Bleeding
Gums
Hematemesis
Epistaxis
Melena (no dark colored food & drinks)
GRADE 3: DENGUE
Shock
Bleeding (Plasma Leakage)
Increase HCT (due to concentration of our blood)
Low BP
High HR
High RR
Pulse pressure (narrow)
GRADE 4: DENGUE
PROFOUND SHOCK
Undetectable BP/pulse
ICU denguq shock syndrome
Diagnostic test:DENGUE
Tornique test/ Rumpeel-Leads test
-Check vascular resistance
-Screening test
Tornique test/ Rumpeel-Leads test
HOW TO PERFORM TORNIQUE TEST
A.Check BP
B.Solve for mid systolic-diastolic pressure: S+D/ 2
C.Re-inflate BP cuff for atleast 5 minutes
WHAT TO CHECK AFTER TORNIQUE TEST
Check: petechiae (weak vessel)
Positive: >20
Negative: <20
Most important treatment: DENGUE
Isotonic solution (fluid replacement)
LR & NSS
NURSING MANAGEMENT: DENGUE
Rest
Ice (bleeding)
Diet: No to dark colored food/ drink
Supoortive Management: DENGUE
Paracetamol (fever & pain)
No ASPIRIN!!! X
PREVENTION: 4’S OF DENGUE
Search & Destroy breeding site
Self-protection
Seek early consultation
Say yes to discriminate fogging
Causative agent: MALARIA
Plasmodium Protozoa/Ovale
P. Vivax
P. Malariae
P. Falciparum (most fatal)
Anopheles (female)
Sporozoites : Liver (egg)
Merozoites:
Attacks liver (RBCs)
Severe Anemia
Cold stage:MALARIA
-chills
-clatter teeth
Hot stage: MALARIA
4-6 hours
-Increase fever
-Headache
-Malaise
Diaphoretic stage: MALARIA
2-4 HOURS
-Wet (sweating)
Diagnosis: MALARIA
Blood smear (hot stage)
-Collect blood
Management: MALARIA
Artemether lumefantrine
-1st line
-Fast-acting
DOC:severe malaria
Quinine (IM/IV)
Common anti-malarial drug:
CHLOROQUINE
Position of Choice: malaria
Supine position
-Maintain for 1 hour
Nursing Responsibility: anti-malarial drug
*Do not administer drug for more than 1 week. Can cause CINCHONISM
Do not administer drug for more than 1 week. because?
CINCHONISM
Headache
Tinnitus
Decrease hearing
N/V
Visual disturbances
Dysphoria
Follow up drug for MALARIA
PRIMAQUINE
FILARIASIS aka _____
Elephantiasis
CAUSATIVE AGENT: FILARIASIS
Wuchureria Bancrofti
Brugia Malayi
Brugia Timori
Aedes Poecilles
Diagnosis: filariasis
NBF (NOCTURNAL BLOOD EXAM)
-After 8 pm
ICT (IMMUNO CHROMATOGRAPHIC TEST)
-Daytime
DOC: FILARIASIS
Hetrazan (Diethylcarbamazine Citrate)
Causative agent: TYPHOID FEVER
Salmonella Typhi (bacteria)
Mode of transmission: TYPHOID FEVER
Fecal-Oral
Urine
S/sx: TYPHOID FEVER
Fever (high & low) Step/ladder like
GI (Anorexia, abdominal pain, constipation, diarrhea)
Peyer’s Patches (WBC)- Guards to microbes that enters.
Intestinal Bleeding- severe abdominal pain
Pathognomonic sign: TYPHOID FEVER
ROSE SPOTS (Evanescent Rash)
COMPLICATIONS OF TYPHOID FEVER
Perforation (boardlike abdomen)
Bradycardia- toxin affects the heart
Diagnosis: TYPHOID FEVER
-Stool exam- period of communicability
-Blood exam- typhi dot (Rapid test)
-Test Antibody: IgM (Acute); IgG (Convalescence)
-Blood Culture: CONFIRMATORY
results: Weeks
-Widal’s Test:old test: blood test
Antigen: H= Post-infection
O= Acute infection
V= Convalescent (carrier)
DOC: TYPHOID FEVER
Ceftriaxone (Rocephin)
Management for typhoid fever
CHLORAMPHENICOL (Chloromycetin)
Causative agent: Rabies
Rhabdo Virus : Bullet Shaped Virus
Mode of Transmission: rabies
Vectorborne (dog)
-Animal bite
-Mammals
Scratch, Tissue Transplant, Airborne = Rare
Diagnosis: RABIES
Observe (10-14 days) if dog is alive on 15th day (-) rabies
Brain Biopsy (Negri Bodies) The classic histopathologic feature
Fluorescent antibody test -Confirmatory test
Fluorescent Microscope: Apple
Green (Fluorescent)
INVASIVE STAGE: RABIES
-Virus reaches the brain
-prodromal s/sx
Fever, headache, photophobia
EXCITEMENT STAGE: RABIES
Hydrophobia- Laryngospasm; water
Aerophobia- Air
Manical Behaviour- Unfamiliar with people
PARALYTIC STAGE: RABIES
Generalized Paralysis
Respiratory Paralysis
MANAGEMENT: RABIES
-wash the wound with soap & water for 10 minutes
-Then betadine
Tetanus Prophylaxis
Rabies Immunization
-ID/IM
-5 Doses
-Day 0, 3, 7, 14, 28
-Active immunization
Causative agent: SCHISTOSOMIASIS
Schistosoma Japonicum (S. Mansoni)
Mode of transmission: SCHISTOSOMIASIS
Snail: Planorbidae Snail
Endemic Places: SCHISTOSOMIASIS
-Region 5 (Bicol)
-Region 8 (Samar & Leyte)
-R-11: Davao
S/SX:
DOC: SCHISTOSOMIASIS
DOC: PRAZIQUANTEL
S/SX:
-DIARRHEA
-BLOODY STOOL
-SPLENOMEGALY
-HEPATOMEGALY
ANEMIA/ ABD ENLARGEMENT
-WEAKNESS
LEPTOSPIROSIS AKA____
Weils Disease; Trench Fever; Mud Fever; Flood fever; Spiroketal Jaundice; Japanese Seven days Fever.
Causative Agent: LEPTOSPIROSIS
Leptospira Interrogans
Incubation Period: LEPTOSPIROSIS
7-19 days; Average of 10 days
Mode of transmission: LEPTOSPIROSIS
Non-intact (wound with water)
Moist soil contaminated with urine of infected host
LEPTOSPIREMIC PHASE
Blood
CSF (Cerebrospinal Fluid)
Onset:
Abrupt
Fever
Headache
Myalgia
N/V
Cough & Chest Pain
IMMUNE PHASE: LEPTOSRISOS
-Correlates w/ circulating IgM
Culture: Confirmatory Test (LEPTOSPIROSIS)
PCR (Positive Polymerase Chain Reaction) Blood or Urine.
TREATMENT: LEPTOSPIROSIS
PENICILLIN (IM/IV)
TETRACYCLINE (Doxycycline)
@ 100mg every 12 hours; PO
ERYTHROMYCIN-allergic to penicillin
Prevention: LEPTOSPIROSIS
Health education: At risk
Farmers & Miners: For awareness
Early Diagnosis
Early Treatment
Use of protective clothing
-Boots -Gloves
Community wide RAT
-Eradication Program
Report all cases of Leptospirosis
Causative agent: SCABIES
SARCOPTES SCABIEI (Parasite)
Mode of transmission: SCABIES
Prolonged skin-to-skin contact indirect contacts (fomites)
-Bedding
-Towels
-Clothing
Incubation period: SCABIES
2-6 weeks
Pathognomonic sign: SCABIES
Severe itching (Pruritis)
-at night severe
-Earliest & most common symptom
Common site: Papular (pimple like); scabies rash
Between Fingers
DOC:scabies rash
PERMETHRIN (TOPICAL)
-Every 2-3 days (1-2 weeks)
Papule —> Crusted
Causative agent: HIV
Retrovirus-Human T-cells Lymphotrophic - 3
Opportunistic Infections (Immunocompromised): HIV
1.Pneumocytis Jiroveci Pneumonia
-no.1 killer
-Non-productive cough & dyspnea
-DOBplex
2.Cytomegalyvirus (CMV)
-Mild visual impairment
-poor peripheral vision
-Blindness
-watery diarrhea
-Weight loss
3.Toxoplasmosis
-Protozoa
-Focal neurological symptoms:
Headache
Seizure
Lethargy
Focal Encephalitis
4.Candida Albicans
-Fungi
-Infection
-Cottage-cheese Like discharge
-Oral thrush
-Vagina
-Mouth, esophagus
5.Herpes Simplex
-Tingling & Burning site of lesion –> Blisters
TREATMENT: HIV
ANTI-VIRAL AGENT
Retrovir (Zidovudine)
Nevirapine (Viramune)
Crixivan
Causative agent: GONORRHEA
Neisseria Gonorrheae (bacteria)
GONORRHEA AKA ____
(GC, CLAP, DRIP)
DOC: GONORRHEA
Single Dose 500mg (IM) Ceftriaxone
Causative agent: SYPHILIS
Bacteria: Treponema Pallidum
SYPHILIS AKA ____
(SY. Bad Blood;The Pox)
S/SX OF SYHPHILIS
Primary stage
-Painless chancre (Sore)
Secondary stage
-1 week- 6 months
-Patchy hair loss
Rash
Sore throat
-Swollen glands
Stage Syphilis
No symptoms- no indication of damage to body organs (brain & heart)
Diagnosis: SYPHILIS
Dark field illumination test
Kalm Test
Causative agent: CHLAMYDIA
Chlamydia Tranchomatis (bacterial)
INCUBATION PERIOD OF CHLAMYDIA
2-3 weeks (Males)
Usually asymptomatic (females)
S/SX
MALE
-Discharge in penis
-Burning & itching opening (Urethral)
-Urination: Burning Sensation
FEMALE
-Sometimes (discharge)
-Vagina (burning & itching)
-Abdominal Pain
-Later: Fever
Diagnosis: CHLAMYDIA
Culture
Causative agent: TRICHOMONIASIS (Trich)
Trichomonas Vaginalis
Incubation: TRICHOMONIASIS (Trich)
4-20 days; average 7 days
S/sx: TRICHOMONIASIS (Trich)
Female: Greenish yellow discharge, Foul
Male: Clear Discharge
Treatment:
Complications: TRICHOMNIASIS
Female: Possible cervical cancer
Thermometer: Manner of wiping before use?
Cleanest to dirtiest
Bulb to stem
Temperature: Best route
1st :Tympanic
2nd : ORAL (2-3 minutes)
Temperature: Safest route?
1st :Tympanic
2nd: Axilla (5-8 minutes)
Temperature: Fastest route?
1st :Tympanic
2nd: Rectal (1 minute)
Thermometer Route:
ORAL (2-3 minutes): Best route
Axilla (5-8 minutes): Safest route
Rectal (1 minute): Fastest route
*obsolete
BEST, SAFEST, FASTEST: Tympanic
Domains of Learning
Cognitive: Knowledge
-(benefits of breastfeeding, BF: Lecture)
Psychomotor: Skills
-Action word verb
-Lecture: How to carry baby
Affective:
-Attitude, behavior, & feelings
-What worries you?
Test to determine the presence of Glucosoria
Benedict’s test
How to collect specimen for GDM test?
Benedict’s test
-Midstream/ before meals; early morning
How to perform Benedict’s test?
2x Heat
-Benedict’s solution: 5ml
-Heat: not boil
-Urine: 8-10 gtts/drops
-Heat
Results of Benedict’s test:
B-G-Y-O B-R
Blue: Normal
Green: + (1+)
Yellow: ++ (2+)
Orange: +++ (3+)
Blue green: Traces
Red/Brick Red: ++++ (4+)
Where to place PHN Bag?
Flat surface
1st : Table
2nd: chair
3rd: bed
Workfield PHN Bag
-Paperlining (clean side: inner surface) facing up
1st: Manila paper
2nd: Old newspaper
Number of times to open PHN bag?
2-3x (the lower the better)
to prevent contamination of the bag’s content
Handwashing
prevent contamination of the bag
Paperlining
to protect the uniform
Apron
to prevent contamination of the clean working space
Waste Receptacle
-study of disease prevention, occurrence, distribution
-Backbone of disease prevention
EPIDEMIOLOGY
when is the best time to conduct study of disease prevention, occurrence, distribution?
Pre-Pathogenic AKA well
excrete disposal: Pit Latrine
Level I. Non-H20 use
Excreta Disposal:
septic tank →sewerage →treatment
III. h20 carriage
Excreta Disposal: Pour-flush, Aqua privy
I. Minimal H2O use
Excreta Disposal: Septic vault/Tank
II. H2O Carriage
Excreta Disposal: flush type
II. H2O sealed
Distance between water source and toilet:
: 25 – 35 meters away
phase of home visit:
-Planning, Records Review
Preparatory (before)
phase of home visit:
Reports
post-visit (after)
phase of home visit:
performing nsg. procedures; health teaching
Actual home visit (During)
Tool and procedure with ease and deftness saving time and effort.
BAG TECHNIQUE
tool and Equipment with basic medications and articles
PHN Bag
Most important point in the use of PHN bag:
*it should contain all necessary articles
Arrangement of PHN Bag
depends on the convenience of the user
PHN bag contents is inside except?
*BP-Apparatus
Principle of bag technique
it should prevent spread of infection
Rationale: to render effective nursing care
Bag technique
waste segregation: non-biodegradable
black
waste segregation: biodegradable
green
waste segregation: infectious
yellow
waste segregation: hazardous, radioactive
orange
health education strategy: 1 WAY
Lecture
health education strategy: 2 way
Discussion
health education strategy: providing skills and knowledge
Demonstration
health education strategy: providing message by acting out
Role playing
Classification of Food Establishment: Excellent
Class A
Classification of Food Establishment: Very satisfaction
Class B
Classification of Food Establishment: Satisfactory
Class C
Immunity: Maternal transfer of antibodies “Mom-Baby”
Natural Passive Immunity
Immunity:
Anti-toxin
Administered globulin
Artificial Passive immunity
Immunity:
Disease experienced e.g., flur, virus
Natural active immunity
Immunity:
Complete doses of toxoid and vaccines
Artificial Active
two types of immunity
Natural and artificial
Passive and Active
purpose: to determine DENGUE
CAPILLARY FRAGILITY TEST a.k.a. Rumpel-Leed’s Test/ Tourniquet Test
Stepd to perform Capillary fragility test
1st: BP cuff – apply snuggly above/over brachial artery
2nd: Baseline formula : S+D/ 2
3rd: Inflation time: 5 mins
4th: Observe below the BP cuff in the area called the ANTECUBITAL FOSSA
5th: Imaginary square 1inch 2 (1 square inch)
6th: Count the petechiae/rashes: 20 or more spots indicate PROBABLE DENGUE
4 rights of food sanitation
S-P-C-S
Source
Preparation
Cooking
Storage
room temperature that leads to spoilage
(10-60°C)
temperature of refrigerator
(<10°C)
temperature of heating
(>60°C)
Purpose: to determine PIH PROTEINURIA/ALBUMINURIA
HEAT AND ACETIC ACID TEST
How to collect the urine HEAT AND ACETIC TEST
midstream and before meal, early morning
Steps to perform HEAT AND ACETIC ACID TEST
Urine: 2/3 of the tube
Heat: (do not boil)
Acetic Acid: few drops (3 -5 gtts/drops)
**heat once (1)
Results for HEAT AND ACETIC ACID TEST
CLEAR – normal ( no CHON/PROTEIN in the urine)
CLOUDY -(+) Abnormal w/ proteinuria/albuminuria CHON
*repeat the procedure to VERIFY
-Extension of service of the clinic
-Professional face-to-face contact between nurse & client
HOME VISIT
color of Umbrella?
1st: Black: a neutral color
2nd: Next best option: dark color i.e. maroon
Principles of Home visit
P-I-I-T-A-N
P – Priorities should be based on needs
(Physical, Psychological, Educational)
I – it should have a purpose/objective
I – it should make use of available information
(records review or case follow-up/
I/FTR)
T – the plan of home visit must be practical and flexible
A – activities should involve the family members
N – no definite rule regarding frequency of home visit
to determine GLUCOSURIA without heating
more convenient than benedict’s test
CLINITEST
Procedure to do Clinitest
Clinitest tablet – 1 tab
Urine – 5 gtts
H2O – 10 gtts
**Results: check the color in the CARD
Same interpretation with benedict’s test
Dependent age group
0-14 y.o./60 y.o.
Productive age group
15-59 y.o.
Reproductive age group
15-49 y.o.
Water sanitation: Unapproved Sources
Open dug wells
Wells that need priming
Unimproved springs
H20 Sanitation: Approved Types
P-S-O
I. Point-source: improved springs;
disinfected wells
II. Shared source: communal faucet; stand post (poso)
III. Own source: H20 works system; pipeline connection
Approved Types:
improved springs;
disinfected wells
I. Point-source
**Disinfect atleast once a year
Approved Types:
communal faucet;
stand post (poso)
II. Shared source
Approved Types:
H20 works system;
pipeline connection
III. Own Individualized source:
How many cottonballs will you utilized AFTER USE?
7 Cotton balls
How many cottonballs will you utilized on the overall thermometer technique?
9 cotton balls
thermometer: Wiping before reading
-cleanest – dirtiest
-(stem to bulb)
-dry cottonball without friction
thermometer: Wiping after use
-cleanest – dirtiest
-(stem to bulb)
-downward, spiral, circular motion
3 agents used for the thermometer technique
Agents: after use
1st – soap for cleansing (3x CB’s to wipe)
2nd – H2O/Water for rinsing (3x CB’s to
wipe)
3rd – Alcohol for disinfecting (1x CB’s to
wipe)
Types of Epidemic: one-time only
Short-time Fluctuation
a. Point-source – common source of the disease
b. Propagated – transmission
Types of Epidemic: recurrent fluctuation; Period and season
-e.x. dengue in the Philippines (rainy season: may to November)
Cyclic Variation
Types of Epidemic: long term fluctuation; increasing through years
-e.x. HIV-AIDS
Secular Variation
Type of short Short-time Fluctuation epidemic where common source of the disease
point-source
Type of short Short-time Fluctuation epidemic where there is transmission of disease
Propagated
to determine perfusion and circulatory failure
CAPILLARY REFILL TEST aka Nail Blanch Test
Procedure of CAPILLARY REFILL TEST
-Place hand above heart level
-Remove nail polish
-apply pressure/ blanch nail until it becomes white
-Release the pressure
-Count the number of seconds until it returns to original color (pink)
results of CAPILLARY REFILL TEST
<3 seconds (0-2) : NORMAL
>3 seconds or more:
-Slow return
-Circulatory failure
Study of vital events
Vital statistics
What are the Vital events?
-Birth: Fertility
-marriage
-separation (annulment, Divorce)
-Migration
-Disease: Morbidity
-Death: Mortality
Clinic Visit Phases:
- Preconsultation
- Medical Examination
- Nursing Intervention
- Post consultation
Clinic Visit Phases:
Assist the MD in performing the check-up procedure
Assist the patient to ensure
o Safety
o Comfort
o Privacy
- Medical Examination
Clinic Visit Phases:
Reinforcement: Summary prior to discharge
Referral
Set appointment to the next check-up
o Can be at clinic or at home
- Post consultation
Clinic Visit Phases:
Carry-out orders
Health education (specific needs/topics)
Seek info about health condition of other family members
- Nursing Intervention
Clinic Visit Phases:
Admission: first come, first serve = give number; greet client
Assessment: data-gathering
o Chief complaint/Health history
o Vital signs
o Laboratory test
Pre-clinic lecture (general health teaching)
- Preconsultation
Study of the population (characteristics)
DEMOGRAPHY
sample (selected people)
Sampling technique
“Clustered Sampling”
Survey
Population (all 100%)
Census
PSA
Philippine Statistics Authority
-protection of the community; against particular disease
-representing immunity & susceptibility levels
Herd Immunity
90% or more the better is only acceptable
on and off, intermittent, seasonal:
low cases
Sporadic: S < I
continuous, constant, regular:
Low cases
Endemic: S = I
sudden outbreak/increase (local):
high cases
Epidemic: S > I
global outbreak; worldwide epidemic: High cases
Pandemic: S > I
it indicates the state of health of the community and the success or failure of healthwork.
Morbidity & Mortality Rates
Best indicator of morbidity or mortality
Infant mortality rate (IMR)
It reflect general health condition/Environment
Infant mortality rate (IMR)
zero or decrease IMR would mean
healthy community