Chapter 18 Exam 3 Flashcards
1
Q
Unstable Angina enzymes*
A
- no cardiac enzymes created, but chest pain will be very strong
2
Q
Angina Pectoris*
A
- chest pain associated with intermittent myocardial ischemia
- no permanent damage occurs to the heart
- crackles/rales are indicative of left side congestion, they are bubbling and rattling of buildup of fluid in the lungs
3
Q
3 patterns of Angina Pectoris *
A
- stable, unstable, and prinzmetal
4
Q
Nitroglycerin*
A
dilates the coronary arteries
5
Q
Stable Angina *
A
- most common
- characterized by stenoic atherosclerotic coronary vessels
- relived by rest and nitroglycerin
6
Q
Unstable or crescendo angina*
A
- can progress to acute ischemia
- very similar to nonSTEMI heart attack, only difference is absence of serumbiomarker troponin
7
Q
Prinzmetal/Variant angina*
A
- unpredictable
- vasospastic
- caused by coronary artery occlusions
- calcium channel blocking agents used for treatment
8
Q
Acute Coronary Syndrome*
A
- Myocardial infarction (MI) or unstable angina
- ECG and biomarkers used for diagnosis
9
Q
ACS ECG changes*
A
- t wave inversion or s wave depression
- ST elevation in case of a heart attack; also known as STEMI (st elevation; myocardial infarction)
- Q wave means old heart attack
- T wave inversion with biomarkers is a nonSTEMI
- segment depression means ischemia
10
Q
Treatment of ACS with drugs*
A
- antiplatelets and statin drugs are the best thing for ACS with nonSTEMI/unstable angina
- reperfusion therapy is best thing for ACS with STEMI
- morphine, oxygen, nitrates, and aspirin together treat acs
11
Q
Acute occulsion*
A
- blockage in an artery, vein, or within the heart
- can cause a range of cellular events depending on the availability and adequacy of collateral blood flow, relative workload, & length of time that flow is interrupted
12
Q
Diagnosis of ACS*
A
depends on
- signs and symptoms
- electrocardiographic changes
- elevatioins of specific marker proteins in the blood
13
Q
Signs and symptoms of ACS*
A
- sever crushing, excruciating chest pain that may radiate to the arm, shoulder, jaw, or back
- nausea
- vomiting
- diaphoresis (sweating)
- shortness of breath
- diabetics and elderly may not have pain due to neuropathy
14
Q
More ACS info: Asymptomatic MI *
A
- lasts more than 15 min and is not relieved by rest or nitroglycerin
- can be asymptomatic MI (silent MI) which is typical in women, the elderly, and patients with diabetic neuropathies
- ECG for asymptomatic includes st segment elevation, large Q waves, and inverted T waves
15
Q
ACS serum marker changes*
A
- myoglobin
- lactate dehydrogenase
- creatine kinase
- increased CK-MB and troponin I and T
16
Q
ACS Treatment Steps
A
- Decreasing myocardial oxygen demand (rest, heart rate control, pain relief, afterload reduction)
- Increasing myocardial oxygen supply (thrombolysis, angioplasty, coronary bypass grafting)
- Monitoring and managing complications (early detection and management of dysrhthmias and conduction disorders, continuous ECG monitoring)
- beta blockers are given
17
Q
Sudden Cardiac Arrest
A
- also called sudden cardiac death
- unexpected death from cardiac causes within 1 hour of symptom onset
- lethal dysrhythmia is usually primary case
18
Q
Chronic Ischemic Cardiomyopathy
A
- heart failure develops insiduously due to progressive ischemic myocardial damage
- typically have history of angina or MI
- appears to be a consequence of slow, progressive apoptotic death of myocytes from chronic ischemia
- more common in older adults
19
Q
Endocardial and Valvular Disease
A
endocardial & valvular structures may be damaged by:
- inflammation and scarring
- calcification
- congenital malformations
causes altered hemodynamics of the heart and increase myocardial workload
- will include stenosis. regurgitation, and prolapse
20
Q
Stenosis
A
failure of the calve to open completely results in extra pressure work for the heart
21
Q
Regurgitation (insufficiency)
A
inability of a valve to close completely results in extra volume work for the heart
22
Q
Mitral Stenosis*
A
- blood flow from the left atrium to left ventricle is impaired during ventricular diastole
- increased pressure of the left atrium leads to atrial chamber enlargement and hypertrophy
23
Q
Results of Mitral Stenosis *
A
- chronic pulmonary hypertension
- right ventricular hypertrophy
- right sided heart failure
24
Q
Signs and symptoms of Mitral Stenosis *
A
- low pitched, rumbling diastolic murmur
- opening snap (sound)
- atrial dysrhythmias
- atrial clots
- exertional dyspnea
25
Q
Mitral Regurgitation*
A
- backflow of blood from left ventricle to the left atrium during ventricular systole
- mitral valve should snap open and then close, but it does not snap closed and the blood experiences backflow
26
Q
Results of Mitral Regurgitation *
A
- left atrium and ventricle dilate and hypertrophy due to the extra volume
- may lead to left sided heart failure
27
Q
Signs and symptoms of Mitral Regurgitation*
A
- high pitched, pansystolic blowing murmur
- giant V waves
- chronic weakness and fatigue
28
Q
Mitral Valve Prolapse*
A
- displacement (ballooning) of the mitral valve leaflets into the left atrium during ventricular systole
- women are affected more than men
- typically asymptomatic
29
Q
Signs ad symptoms of Mitral Valve Prolapse *
A
- Midystolic click or systolic murmur
- palpitations
- rhythm abnormalities
- diziness
- fatigue
- dyspnea
- chest pain
- or depression and anxiety
30
Q
Aortic Stenosis
A
- predominant cause is age related calcium deposits on the aortic cusps
- results in obstruction of the aortic outflow from the left ventricle into the aorta during systole
31
Q
Signs and Symptoms of Aortic Stenosis
A
- left ventricle hypertrophy may result in ischemia and left sided heart failure
- crescendo-decrescendo murmur during ventricular systole with prominent S4 (s3 follows s2, S4 proceeds S1, s4 is indicative of a stiff left ventricle)
- syncope
- fatigue
- angina
32
Q
Aortic Regurgitation
A
- incompetent aortic valve allows blood to leak back from the aorta into the left ventricle during diastole
- causes include abnormal aortic valve or aortic root dilation
- leafs to left ventricular hypertrophy and dilation with eventual left sided heart failure
33
Q
Signs and symptoms of Aortic Regurgitation
A
- high pitched blowing murmur during ventricular diastole
- high systolic blood pressure
- diastolic blood pressure usually low
- palpitations
34
Q
Rheumatic Heart Disease
A
- acute inflammatory disease that follows infection with group a beta hemolytic streptococci
- damage is due to immune attack on individuals own tissues
- damage occurs in CT of joints, heart, and skin
- occurs mainly in children
35
Q
Signs and symptoms of Rheumatic Heart Disease
A
- fever
- sore throat
- joint inflammation
- involuntary movements (sydenham chorea)
- distinctive truncal rash
36
Q
Infective endocarditis
A
- invasion and colonization of endocardial structures by microorganisms with resulting inflammation - vegetations
- most common bacteria are strep and staph
- Can be acute (poor prognosis) or subacute (predisposing risk factors typically present)
37
Q
Myocarditis
A
- inflammatory disorder of the heart muscle characterized by necrosis and degeneration of myocytes
- clinical course is acute or stormy with recovery or death from cardiac failure occurring weeks to months after onset of symptoms
38
Q
Cardiomyopathy
A
- classified by cause or functional impairment
- primary: dysfunction or unknown cause
- Secondary: known cause
- Functional classifications: dilated, hypertrophic, restrictive
39
Q
Dilated Cardiomyopathy
A
- cardiac failure associated with dilation of one or both ventricular chambers
- can be related to alcohol toxicity, pregnancy, postviral myocarditis, or genetic abnormality
- slow progression of biventricular heart failure with low ejection fraction ( ejection fraction less than 40% is heart failure
40
Q
ICD
A
Implantable Cardioverter Defribrillator
41
Q
Hypertrophic Cardiomyopathy
A
- thickened, hyperkinetic ventricular muscle mass
- septum may be affected, leading to idiopathic subaortic stenosis
- caused by a genetic abnormality
- can be asymptomatic or show signs of dyspnea and angina
42
Q
Restrictive Cardiomyopathy
A
- Rarest form
- stiff, fibrotic, rigid, noncompliant ventircle with impaired diastolic filling
- mosy commonly associated with amyloidosis
- decreased cardiac output and left sided heart failure can result
- exerrcise intolerance, dyspnea, and weakness
43
Q
Pericardial Diseases
A
Typically sequelae of other disorders such as
- systemic infection
- trauma
- metabolic derangement
- neoplasia
44
Q
Pericardial Effusion
A
- accumulation of noniflammatory fluid in the pericardial sac
- composition of usual fluids includes serous, serosanguineous, chylous(fatty), and blood
45
Q
Cardiac Tamponade
A
- when fluid accumulation in the pericardial sac is large/sudden it can lead to external compression of the heart chambers such that filling is impaired (basically a medical emergency in which there is a sudden build up of fluid in the lungs)
46
Q
Manifestations of Cardiac Tamponade and treatment
A
- reduced stroke volume
- compensatory increases in heart rate
- pulsus paradoxus
- jugular venous distension
- Beck’s Triad
- Treatment is pericardiocentesis
47
Q
Pericarditis
A
- acute or chronic inflammation of the Pericardium
