Chapman ch 9. FHR Assessment Flashcards
Three requirements for fetal heart rate interpretation
Baseline. Interpretation of episodic and periodic changes of fetal heart rate. Interpretation of uterine activity.
IUPC
Intrauterine pressure catheter.
Causes of decreased fetal oxygen supply
Reduction of blood flow through maternal vessels/hypertension, PIH
Reduction of oxygen via maternal blood such as the mom holding the breath, baby lying on vena cava.
Alterations in fetal circulation such as baby on cord, decreased blood flow to the placenta, hemorrhage, deterioration of the placenta
Reduction in blood flow to the placenta
Reassuring FHR Pattern
Baseline should be 120 - 160. Accelerations with fetal movement. Should have moderate variability.
Normal uterine activity in labor
Contractions every 2 to 5 minutes, lasting less than 90 seconds, intensity less than 80, relaxation 20 or less, the average pressure is 50 to 85
Fetal compromise
Associated with fetal hypoxemia. Can lead to fetal hypoxia.
Hypoxemia
Deficiency of oxygen
Hypoxia
In adequate oxygen
Causes a release of epinephrine and norepinephrine to increase fetal heart rate and blood pressure
Nonreassuring fetal heart rate patterns
Progressive increase or decrease in the baseline
Tachycardia/above 160
Decrease in variability
Severe variable deceleration’s equals less than 60, and lasting longer than 30 to 60 seconds
Late decelerations/not good with repetition or uncorrectable
Absence of variability/no movement
Prolonged deceleration/greater than 60 to 90 seconds
Severe bradycardia/less than 70
Intermittent auscultation
Done with an ultrasound fetoscope, stethoscope, it DeLee/Hills fetoscope
Leopold’s maneuvers
Know how to spell
Assesses fetal position to get the best PMI/point of maximum impulse
Procedure for auscultation
Leopold’s maneuver’s
Place listening device over PMI
Palpate abdomen between contractions
Count maternal pulse while listening to fetal heart rate
Count fetal heart rate for 30 to 60 seconds
Auscultate fetal heart rate during contraction and 30 seconds afterwards to see how baby is responding
Internal fetal monitoring
Membranes must be ruptured
Cervix sufficiently dilated about 2 cm
Presenting Part low enough to place electrodes
Fetal tachycardia
Baseline above 160 that lasts for at least 10 minutes
Moderate =161-180
Marked 180+
Early sign of fetal hypoxemia
If heart rate persists from 200 to 220 fetal death may occur
Causes on the fetal side could be infection, drugs, chronic hypoxemia, Stimulation, compensation for hypoxemia, cardiac abnormalities, anemia.
Causes from the maternal side could be a anemia, hyperthyroidism, dehydration, fever, ChorioAmnionitis (infection uterus), anxiety, medications, drugs
Bradycardia
Baseline below 110 that lasts at least 10 minutes
Less than 100 is a later sign of hypoxia
Occurs before fetal death
May be tolerated if it remains about 80 with variability
Moderate 110-119
Marked 100-
A decreased fetal heart rate leads to decreased cardiac output causes a decrease in umbilical bloodflow leads to decreased oxygen to the fetus causing hypoxia
Bradycardia with variability maybe benign. Bradycardia with a loss of variability or late decelerations is associated with impending hypoxia
Maternal supine hypotensive syndrome
Caused by uterine pressure on the vena cava, decreases bloodflow return to heart, reduces cardiac output and blood pressure, decreases fetal heart rate causing bradycardia.
Treat by turning the mom on the right side, and rise slowly.
Variability of fetal heart rate
One of the most reliable indicators of fetal health and oxygenation, confirms no metabolic acidosis. Shows irregular fluctuations in the baseline, short-term is Beat to beat. long-term is rhythmic waves or cycles from the baseline.
Four ranges of variability
Absent or undetected: the amplitude range is undetectable, looks like a flatline
Minimal: not more than five bpm
Moderate: 6-25 from peak to trough
Marked: greater than 25bpm
If absent, turn mom to right side, Adjust the straps, give ice chips, no longer than 30 minutes. Fetus could be asleep or premature.
Nervous system on fetal heart rate
Sympathetic increases it, responsible for long term variability, can be stimulated during hypoxemia.
Parasympathetic decreases it
Head compression stimulates the Vagus nerve causes a decrease.
Accelerations
An abrupt increase above baseline
It’s a good thing predictive of good O2.
15 bpm or greater, last 15 seconds or more
Returns to baseline within two minutes from start
Periodic or episodic
Periodic are caused by the sympathetic nervous system or usually a breech presentation
Episodic occur during fetal movement and indicate fetal well-being
Require no interventions.
Decelerations
Periodic changes that last from a few seconds to no longer than two minutes.
Caused by the parasympathetic and they are either benign or nonreassuring
Described by relation to onset and the end of contraction and shape
Early, late, or variable
Recurrent occur with at least 50% of the contractions over a 20 minute.
Intermittent are with less than 50% of the contractions over 20 minutes
Early decelerations
Gradual decrease and return to baseline during contraction. Caused by head compression. Normal and usually benign. Uniform shape that mirrors contraction. Usually during the first stage while 4 to 7 cm dilated and also during the second stage during pushing
NADIR is the lowest point of deceleration
No intervention is needed
Late decelerations
Sign on fetal intolerance to labor. Cause by uteroplacental insufficiency such as a smoker or cocaine user. Also caused if the placental cord is compressed. Gradual decrease and return to baseline usually after a contraction. Nadir is after peak of contraction. Returns after the contraction is over.
Indications of late decelerations
Presence of fetal hypoxemia/insufficient placental perfusion. Fetal hypoxemia progressing to hypoxia. Acidemia progressing to acidosis.
*** Ominous when uncorrectable, especially with variability and tachycardia. Turn the mom to the right, place 02 at 10 L, if no change probably go for a Csection