Chap. 9 - SZO review questions Flashcards
The neurotransmitter deregulation theory of the etiology of psychotic disorders such as schizophrenia supports that psychosis is caused, in part, by
A - an excess of dopamine
B - A deficiency of dopamine
C - Poor acetylcholine regulation
D - Poor synaptic uptake of serotonin receptors
A - an excess of dopamine
Psychotic sx are partially caused by too much dopamine in the mesolimbic dopamine pathway
The positive-negative model of classifying the symptoms of schizophrenia describes positive symptoms as
A - Symptoms that positively respond to antipsychotic medication
B - Symptoms that are less serious to experience
C - Symptoms that are less socially stigmatizing
D - Symptoms that positively correlate to drug abuse
A - Symptoms that positively respond to antipsychotic medication
This definition was initially developed prior to the invention of the 2nd generation antipsychotics
Mrs Jay suffers from SZO and is taking Navane 15mg/day. During her appointment, she complains of feelings of inner restlessness, tremors, drooling, and stiff muscles. The best explanation for these is
A - EPS effects of Navane
B - Anticholinergic side effects of Navane
C - Atypical side effects of Navane
D - Psychosomatic side effects
A - EPS effects of Navane
EPS can include akathisia, postural tremors, dystonia, and drooling.
As part of treatment planning, you place a note on Mrs Jay’s chart about seizure precautions. She notices the note and states that she doesn’t understand that the she has no history of seizures. You explain that
A - It is a clerical error, and you will correct it
B - Seizures are common in mental illness, and the note is just a precaution
C - Reactions to psychotropic medications are unpredictable
D - Typical antipsychotic medications can lower seizure threshold
D - Typical antipsychotic medications can lower seizure threshold
All antipsychotics lower the seizure threshold, therefore precautions should be taken
Mrs Anders, a client with delusional disorder, has been started on Haldol 5 mg po bid, Tylenol 2 tabs po prn, Cogent 1 mg po prn, and Ativan 2 mg prn for agitation. She is complaining of a sudden painful stiff neck and jaw muscles that started a few hours after she took her medication. These symptoms are most likely related to A - Akinesia B - Akathisia C- Dystonia D - Somatic delusiona
C- Dystonia
Destinies are brief or sustained contractions of muscles, spasms, thickened or slurred speech due to enlarged tongue, oculogyric crises, and unusual posturing of limbs
The most appropriate action to help relieve Mrs Anders pain and stiff muscles is
A - Switch to an NSAID to control her pain
B - Increase the Cogentin dose
C - Teach her relaxation techniques
D - Work up for musculoskeletal labs
B - Increase the Cogentin dose
Anticholinergic drugs such as Cogentin are used for dystonias
After a few weeks Mrs Anders returns for a follow up. She is refusing to take Haldol d/t continued stiff muscles and blurred vision. She says she has not taken her meds for 1 weeks, and the stiffness and blurred vision are better but still a problem. She says “Something must be wrong with me. I stopped the Haldol but I’m still not ok.” Your best explanation is that Mrs Anders is
A - Fixated on her meds
B - Experiencing an unusual reaction to Haldol
C - Experiencing a lipophilic reaction to Haldol
D - Experiencing some problem other than a side effect of Haldol
C - Experiencing a lipophilic reaction to Haldol
After the depot injection, the active ingredient is slowly absorbed in the bloodstream, and effects will last for 3-4 weeks following an injection.