Ch. 15 Part 1 Flashcards

1
Q

Indications for Temporary Pacing
Pacemaker System
Pacing Routes
Five-Letter Pacemaker Codes
Pacemaker Settings
Pacing Artifacts
Pacemaker Malfunctions
Medical Management
Nursing Management
Permanent Pacemakers

A

Permanent & Temporary Pacemakers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

clinical indications for instituting temporary pacemaker therapy are similar regardless of the cause of the rhythm disturbance that necessitates the placement of a pacemaker
Therapeutic Indications
Diagnostic Indications

A

Indications for Temporary Pacing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Dysrhythmias that are unresponsive to medications and result in compromised hemodynamic status are a definite indication for pacemaker therapy.
Temporary pacing may be used in the treatment of symptomatic bradycardia or progressive heart block that occurs as a result of myocardial ischemia, medication overdose, or illegal drug toxicity. After cardiac surgery, temporary pacing may be used to improve a transiently depressed, rate-dependent cardiac output. Conduction disturbances that occur after valvular surgery can be managed effectively with temporary pacing.

A

Therapeutic Indications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Electrophysiology studies (EPS) are performed in cardiac catheterization laboratories equipped with specialized pacing equipment.
Catheter ablation may also be used as a therapeutic strategy for selected patients with atrial fibrillation.
Intracardiac electrograms, recordings of cardiac electrical activity obtained from pacing electrodes, may provide useful diagnostic information.
Stored intracardiac electrograms from permanently implanted cardiac electrical devices can be used to identify undetected episodes of atrial fibrillation that may increase a patient’s risk for ischemic stroke.

A

Diagnostic Indications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

a simple electrical circuit consisting of a pulse generator and a pacing lead (an insulated electrical wire) with one, two, or three electrodes.
Pacing Pulse Generator
Pacing Lead Systems

A

Pacemaker System

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

designed to generate an electrical current that travels through the pacing lead and exits through an electrode (exposed portion of the wire) that is in direct contact with the heart.
This electrical current initiates a myocardial depolarization. The current then seeks to return by one of several pathways to the pulse generator to complete the circuit.
The power source for a temporary external pulse generator is a standard alkaline battery inserted into the generator. Implanted permanent pacemaker batteries are usually long-lived lithium cells.

A

Pacing Pulse Generator

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

pacing may be bipolar or unipolar. In a bipolar system, two electrodes (positive and negative) are located within the heart, whereas in a unipolar system, only one electrode (negative) is in direct contact with the myocardium. In both systems, the current flows from the negative terminal of the pulse generator, down the pacing lead to the negative electrode, and into the heart. The current is then picked up by the positive electrode (ground) and flows back up the lead to the positive terminal of the pulse generator.
An epicardial lead system is often used for temporary pacing after cardiac surgery.
A unipolar pacing system (epicardial or transvenous) has only one electrode (the negative electrode) making contact with the heart.
Because the unipolar pacing system has a wide sensing area as a result of the relatively long distance between the negative and positive electrodes, it has better sensing capabilities than a bipolar system.

A

Pacing Lead Systems

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Permanent pacing usually is accomplished transvenously, although when a thoracotomy is otherwise indicated, as in cardiac surgery, the physician may elect to insert permanent epicardial pacing wires.
It is a rapid, noninvasive procedure that nurses can perform in the emergency setting and is recommended in the advanced cardiac life support algorithm for the treatment of symptomatic bradycardia that does not respond to atropine.
Discomfort may still be an issue for some patients, particularly when higher energy levels are required to achieve capture. This route is typically used as a short-term therapy until the situation resolves or another route of pacing can be established.
The insertion of temporary epicardial pacing wires has become a routine procedure during many cardiac surgical cases.
These wires can be removed several days after surgery by gentle traction at the skin surface with minimal risk of bleeding.
Temporary transvenous endocardial pacing is accomplished by advancing a pacing electrode wire through a vein, often the subclavian or internal jugular vein, and into the right atrium or right ventricle.
the pacing wire is inserted through a special pulmonary artery catheter by means of a port that exits in the right atrium or right ventricle.

A

Pacing Routes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

programming characteristics and multisite pacing functions, to accommodate the development of newer devices that are rate responsive or that pace from more than one site within the atria and the ventricles.
The first letter refers to the cardiac chamber that is paced. The second letter designates which chamber is sensed, and the third letter indicates the pacemaker’s response to the sensed event. These three letters are used to describe the mode of pacing.

A

Five-Letter Pacemaker Codes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Their functions must be thoroughly understood so that pacing can be initiated quickly in an emergency situation and troubleshooting can be facilitated if problems with the pacemaker arise.
rate control regulates the number of impulses that can be delivered to the heart per minute.
If the pacemaker is operating in a dual-chamber mode, the ventricular rate control also regulates the atrial rate.
The output dial regulates the amount of electrical current, measured in milliamperes (mA), that is delivered to the heart to initiate depolarization. The point at which depolarization occurs, called threshold, is indicated by a myocardial response to the pacing stimulus (i.e., capture).
Separate output controls for atrium and ventricle are used with a dual-chamber pulse generator.
The sensitivity control regulates the ability of the pacemaker to detect the heart’s intrinsic electrical activity. Sensitivity is measured in millivolts (mV) and determines the size of the intracardiac signal that the generator will recognize.
The sensing ability of the pacemaker can be quickly evaluated by observing for a change in pacing rhythm in response to spontaneous depolarizations.
The AV interval control (available only on dual-chamber generators) regulates the time interval between the atrial and ventricular pacing stimuli.
Because the AV interval is limited by the length of the cardiac cycle, modern temporary generators automatically adjust the AV delay based on the programmed heart rate.
The lower rate, or base rate, determines the rate at which the generator will pace when intrinsic activity falls below the set rate of the pacemaker. The upper rate determines the fastest ventricular rate the pacemaker will deliver in response to sensed atrial activity.
There also is an atrial refractory period, programmable from 150 to 500 ms, which regulates the length of time, after a sensed or paced ventricular event, during which the pacemaker cannot respond to another atrial stimulus.

A

Pacemaker Settings

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

All patients with temporary pacemakers require continuous ECG monitoring. The pacing artifact is the spike that is seen on the ECG tracing as the pacing stimulus is delivered to the heart. A P wave is visible after the pacing artifact if the atrium is being paced
With dual-chamber pacing, a pacing artifact precedes both the P wave and the QRS complex
Not all paced beats look alike.

A

Pacing Artifacts

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Most pacemaker malfunctions can be categorized as abnormalities of pacing or of sensing. Problems with pacing can involve failure of the pacemaker to deliver the pacing stimulus, a pacing stimulus that fails to depolarize the heart, or an incorrect number of pacing stimuli per minute.
Pacing Abnormalities
Sensing Abnormalities

A

Pacemaker Malfunctions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Failure of the pacemaker to deliver the pacing stimulus results in disappearance of the pacing artifact even if the patient’s intrinsic rate is less than the set rate on the pacer. This can occur intermittently or continuously and can be attributed to failure of the pulse generator or its battery, a loose connection between the various components of the pacemaker system, broken lead wires, or stimulus inhibition as a result of EMI.
Tightening connections, replacing the batteries or the pulse generator itself, or removing the source of EMI may restore pacemaker function.
If the pacing stimulus fires but fails to initiate a myocardial depolarization, a pacing artifact will be present but will not be followed by the expected P wave or QRS complex, depending on the chamber being paced
This loss of capture can be attributed most often to displacement of the pacing electrode or to an increase in threshold (electrical stimulus necessary to elicit a myocardial depolarization) as a result of medications, metabolic disorders, electrolyte imbalances, or fibrosis or myocardial ischemia at the site of electrode placement.
Pacing can occur at inappropriate rates.
Inappropriate stimuli from a pacemaker may result in pacemaker-mediated tachycardia; this usually is caused by sensing of inappropriate signals in a dual-chamber pacemaker that is in a trigger mode

A

Pacing Abnormalities

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Undersensing
Oversensing.

A

Sensing Abnormalities

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

inability of the pacemaker to sense spontaneous myocardial depolarizations.
results in competition between paced complexes and the heart’s intrinsic rhythm.
Undersensing can result in the delivery of pacing stimuli into a relative refractory period of the cardiac depolarization cycle
A ventricular pacing stimulus delivered into the downslope of the T wave (R-on-T phenomenon) is a real danger with this type of pacer aberration because it may precipitate a lethal dysrhythmia.
The cause often can be attributed to inadequate wave amplitude (height of the P or R wave).
Other possible causes include inappropriate (asynchronous) mode selection, lead displacement or fracture, loose cable connections, and pulse generator failure.

A

Undersensing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

occurs as a result of inappropriate sensing of extraneous electrical signals that leads to unnecessary triggering or inhibition of stimulus output, depending on the pacer mode.
Because most temporary pulse generators are programmed in demand modes, oversensing results in unexplained pauses in the ECG tracing as the extraneous signals are sensed and inhibit pacing. Moving the sensitivity dial toward 20 mV (less sensitive) often stops the pauses.
With permanent pacemakers, a magnet may be placed over the generator to restore pacing in an asynchronous mode until appropriate changes in the generator settings can be programmed.

A

Oversensing.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

The physician determines the pacing route based on the patient’s clinical situation.
The physician places the transvenous or epicardial pacing lead or leads, repositioning them as needed to obtain adequate pacing and sensing thresholds. Decisions regarding lead placement may later limit the pacing modes available to the clinician.
After lead placement, the initial settings for output and sensitivity are determined, the pacing rate and mode are selected, and the patient’s response to pacing is evaluated.

A

Medical Management

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

in the care of a patient with a temporary pacemaker are associated with several patient problems and can be combined into four primary areas: assessment and prevention of pacemaker malfunction, protection against microshock, surveillance for complications such as infection, and patient education.
Prevention of Pacemaker Malfunction
Microshock Protection
Infection risk
Educate the patient and family

A

Nursing Management

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Continuous ECG monitoring is essential to facilitate prompt recognition of and appropriate intervention for pacemaker malfunction.
If the patient is on a regimen of bed rest, the pulse generator can be suspended with twill tape from an intravenous pole mounted overhead on the ceiling. This positioning prevents tension on the lead while the patient is moved (given adequate length of bridging cable) and alleviates the possibility of accidental dropping of the pulse generator.
The nurse inspects for loose connections between the leads and pulse generator on a regular basis. Replacement batteries and pulse generators must always be available on the unit.
The pulse generator must always be labeled with the date on which the battery was replaced.
It is important to be aware of all sources of EMI within the critical care environment that may interfere with the pace-maker’s function. Sources of EMI in the clinical area include electrocautery, defibrillation current, radiation therapy, magnetic resonance imaging (MRI) scanners, and transcutaneous electrical nerve stimulation units.

A

Prevention of Pacemaker Malfunction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

pacing electrode provides a direct, low-resistance path to the heart, the nurse takes special care while handling the external components of the pacing system to avoid conducting stray electrical current from other equipment.
The possibility of microshock can be minimized by wearing gloves when handling the pacing wires and by proper insulation of terminal pins of pacing wires when they are not in use
The wires are taped securely to the patient’s chest to prevent accidental electrode displacement.

A

Microshock Protection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

the lead insertion site is a rare but serious complication associated with temporary pacemakers. The site is carefully inspected for purulent drainage, erythema, and edema, and the patient is observed for signs of systemic infection.
endocarditis can occur in patients with endocardial pacing leads. A less common complication associated with transvenous pacing is myocardial perforation, which can result in rhythmic hiccoughs or cardiac tamponade.

A

Infection risk

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

instructed not to handle any exposed portion of the lead wire and to notify the nurse if the dressing over the insertion site becomes soiled, wet, or dislodged. The patient also is advised not to use any electrical devices brought in from home that could interfere with pacemaker functioning. Patients with temporary transvenous pacemakers need to be taught to restrict movement of the affected extremity to prevent lead displacement.

A

Educate the patient and family

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

A patient who undergoes implantation of a permanent pacemaker is usually in the hospital for less than 24 hours. Longer lengths of stay are expected for patients with serious comorbidities such as MI or cardiogenic shock.
Microprocessors have allowed for the development of increasingly smaller generators despite the incorporation of more complex features. Current generators are smaller, more energy efficient, and more reliable than previous models.
Newer enhancements include leadless pacemakers, which consist of a self-contained unit that is placed in the RV via the femoral vein. These devices eliminate the need for a subcutaneous pocket and transvenous leads that account for the majority of complications associated with permanent pacemakers.
New also compatible with MRI.
rapidly expanding role for permanent pacemakers has been the use of these devices as a type of nonpharmacologic therapy for treatment of heart failure.

A

Permanent Pacemakers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

consists of leads and a generator and is similar to a pacemaker but with some key differences. The leads contain not only electrodes for sensing and pacing but also integrated defibrillator coils capable of delivering a shock. The generator is larger, to accommodate a more powerful battery and a highvoltage capacitor along with the microprocessor. It is surgically placed in the subcutaneous tissue of the pectoral region in the upper chest
Occasionally, the electrical rhythm may deteriorate to asystole or a slow idioventricular rhythm; in such cases, the bradycardia backup pacing function is activated.
Many patients with ICDs have structural heart disease and may require continuous pacing or benefit from CRT. ICD product development has resulted in dual-chamber devices with leads in both atria and ventricles to provide these additional therapies.
Other developments in ICD technology include improved diagnostic and telemetry functions, such as the ability to provide real-time electrograms obtained from the ICD electrodes or the ability to perform remote device interrogation using cellular or wireless technology.
Insertion of Implantable Cardioverter Defibrillator
Medical Management
Nursing Management

A

Implantable cardioverter-defibrillators (ICD)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

The ICD has progressed in both programmable functions and insertion technique. Initially, all ICDs were implanted surgically during open-heart surgery or via thoracotomy, with electrode patches attached directly to the heart.
Procedural complications are infrequent but may include hematoma, pneumothorax, cardiac tamponade, or lead dislodgment. A fully subcutaneous ICD has been introduced more recently that consists of a generator implanted in the left axillary area and a single subcutaneous lead for detection of ventricular dysrhythmias and delivery of therapy. Although this device is unable to provide pacing therapies, it may provide an alternative for patients who lack vascular access and avoid complications related to transvenous lead placement.

A

Insertion of Implantable Cardioverter Defibrillator

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

ICD begins before implantation with a thorough evaluation of the patient’s risk for dysrhythmia and underlying cardiac function. Some patients may undergo EPS to determine the origin of the dysrhythmia and the effect of antidysrhythmic agents in suppressing or altering the rate of the dysrhythmia.
In these patients an external wearable cardioverter defibrillator may be used to address the risk of sudden cardiac death during the waiting period.
An electrophysiologist performs the initial programming of the device at the time of implantation. Defibrillation efficacy of the ICD may be assessed by inducing the dysrhythmia and then evaluating the ability of the device to terminate it.
Defibrillation testing is still considered a standard of care for subcutaneous ICD implants.
Other adjustments in programming may be performed to decrease unnecessary shocks, including aggressive use of antitachycardia pacing and withholding shocks for SVT or nonsustained ventricular rhythms.
After it has been determined that the ICD functions appropriately, further follow-up can be conducted on an outpatient basis, with remote monitoring options to monitor the number of discharges and the battery life of the device.

A

Medical Management

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

implanted during open-heart surgery, postoperative nursing management is similar to that for any patient undergoing cardiac surgery.
includes assessing for dysrhythmias and monitoring for complications related to insertion. In the case of a ventricular dysrhythmia, it is important to know the type of ICD implanted, how the device functions, and whether it is activated (i.e., “on”). If the patient experiences a shockable rhythm, the nurse should be prepared to defibrillate in the event that the device fails.
Most patients continue to take some antidysrhythmic medications to decrease the number of shocks required and to slow the rate of the tachycardia. Complications associated with a permanent ICD include infection from the implanted system, broken leads, and sensing of supraventricular tachydysrhythmias resulting in unneeded discharges.
Educate the Patient and Family

A

Nursing Management

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Preoperative teaching for a patient with an ICD includes information about how the device works and what to expect during the implantation procedure. After implantation, education is focused on aspects of living with an ICD. Patients need information pertaining to device follow-up, technology used for remote monitoring, and instructions about what to do if they experience a shock.
patients need to understand the importance of continued risk factor modification and prescribed medications.

A

Educate the Patient and Family - Nursing Management (ICD)

29
Q

Is demanding but exciting work that requires the talents of an experienced team of critical care nurses.
Coronary Artery Bypass Graft Surgery
Valvular Surgery
Postoperative Management
Patient and Family Education

A

Cardiac surgery

30
Q

surgery has proved to be safe and effective in relieving angina symptoms and improving survival in most patients. Although there has been a great deal of evolution involving less invasive techniques, improved pharmacologic therapy, and expanded education regarding lifestyle modifications, CABG surgery continues to have an important role in the treatment of CAD.
Surgical revascularization has been shown to be more efficacious than PCI in patients with multivessel or left main coronary disease.
Myocardial revascularization involves the use of a conduit, or channel, designed to bypass an occluded coronary artery.
Surgeons must evaluate which conduits would provide the best graft patency and long-term outcomes for their patients. The long saphenous vein graft (SVG) is the most frequently used conduit for CABG surgery.
Vein-graft harvesting can be done via either an open or endoscopic approach.
Use of arterial conduits has dramatically improved long-term graft patency. The internal thoracic artery (ITA), which usually remains attached to its origin at the subclavian artery, is swung down and anastomosed distal to the coronary artery
Radial artery conduits in CABG are typically used as adjuncts to the left ITA. Graft patency has increased with improved harvesting techniques, adjunctive medication strategies (statins and antiplatelet agents), and aggressive risk factor modification such as smoking cessation.

A

Coronary Artery Bypass Graft Surgery

31
Q

Valvular disease results in various hemodynamic dysfunctions that can usually be managed medically as long as the patient remains symptom-free.
The two categories of prosthetic valves are mechanical heart valves (MHV) and bioprosthetic heart valves (BHV), or tissue valves. MHVs are made from combinations of metal alloys, pyrolytic carbon, Dacron, and Teflon and have rigid occluding devices
Their construction renders them highly durable, but all patients with mechanical valves require anticoagulation to reduce the incidence of thromboembolism.
A discussion regarding the risks and benefits of different prosthetic valves should be had between the patient and the physician.
Because MHVs are more durable, they may be preferred for a young person who is anticipated to have a relatively long life span ahead. A BHV may be chosen for an older adult patient; the valve has a reduced longevity, but this disadvantage is offset by the older patient’s shorter life expectancy.
For patients with medical contraindications to anticoagulation and for patients whose past compliance with medications has been questionable, a BHV may be selected.
Infective endocarditis continues to be a significant clinical problem associated with high mortality rates. Considerations for the timing of surgery, if required, need to be made with the surgeon along with aggressive antibiotic therapy and the removal of devices.

A

Valvular Surgery

32
Q

The physician prescribes therapeutic interventions and identifies specific hemodynamic end points to maintain adequate organ perfusion and oxygen delivery.
The nurse is responsible for applying these therapies to maintain the patient’s hemodynamic parameters within the desired range.
Cardiovascular Support
Mechanical complications
Temperature Regulation
Control of Bleeding
Chest tube patency
Cardiac Tamponade
Pulmonary Care
Neurologic Complications
Infection
Acute Kidney Injury
Resuscitation of Patients Who Arrest After Cardiac Surgery
Guidelines for Coronary Artery Bypass Graft Surgery

A

Postoperative Management

33
Q

Postoperative cardiovascular support often is indicated because of a low-output state. The most common causes of cardiac dysfunction after cardiac surgery are mechanical complications, physiologic complications, myocardial infarction, and dysrhythmias. Cardiac output can be maximized by adjustments in heart rate, preload, afterload, and contractility.
HR
Preload
Afterload
Contractility

A

Cardiovascular Support

34
Q

In the presence of low cardiac output, the heart rate can be appropriately regulated via temporary pacing or medication therapy. Temporary atrial or ventricular epicardial pacing is usually instituted when the heart rate decreases to less than 60 beats/min and the patient is hypotensive, requiring a supportive rate of 80 to 100 beats/min. In the case of tachycardia, intravenous beta-blockers or calcium channel blockers may be used in the acute postoperative period to slow supraventricular rhythms. Obtaining a 12-lead ECG to assess dysrhythmias further may also be indicated.
Atrial fibrillation is the most common adverse event after cardiac surgery.

A

HR

35
Q

In most patients, reduced preload is the cause of low postoperative cardiac output. The most common causes of decreased preload are hypovolemia from bleeding, fluid shifts caused by the systemic inflammatory response, increased vascular capacitance with rewarming, and elevated cardiac preload requirements.
Appropriate volume resuscitation immediately postoperatively is one of the most important interventions and should be a first-line therapy for hemodynamic instability.
Crystalloids are preferred for fluid resuscitation; the type of crystalloid depends on institutional preference.
Approximately 2 to 3 L of crystalloid will suffice for most patients.

A

Preload

36
Q

Patients who have had cardiac surgery may demonstrate postoperative hypertension. Although it is transient, postoperative hypertension can precipitate or exacerbate bleeding from the mediastinal chest tubes.
Increased afterload may be partially caused by the peripheral vasoconstrictive effects of hypothermia, which can be managed with careful rewarming. significant percentage of patients experience hypotension after CPB secondary to peripheral vasodilation. This condition is believed to occur in part because of the systemic inflammatory response to CPB, surgical trauma, ischemia, or reperfusion. Therapy for hypotension after cardiac surgery usually includes volume loading and vasopressors.

A

Afterload

37
Q

The patient’s ejection fraction (EF) should also be used to inform the treatment plan. If the adjustments in heart rate, preload, and afterload fail to produce significant improvement in cardiac output, contractility can be enhanced with positive inotropic support.
Mechanical circulatory support may also need to be added to help augment circulation

A

Contractility

38
Q

Include cardiac tamponade, hematomas, vasospasm of a coronary artery graft, prosthetic valve paravalvular regurgitation, and systolic anterior motion of the mitral valve. Some mechanical complications are noncardiac, such as pneumothorax, hemothorax, and endotracheal tube malposition.

A

Mechanical complications

39
Q

Hypothermia can contribute to depressed myocardial contractility, vasoconstriction, and ventricular dysrhythmias in a patient who has undergone cardiac surgery.
may also contribute to postoperative bleeding because the functioning of clotting factors is depressed.
patients may be rewarmed with the use of warm blankets or forced-air warming devices.

A

Temperature Regulation

40
Q

Postoperative bleeding from the mediastinal chest tubes can be caused by inadequate hemostasis, disruption of suture lines, coagulopathy associated with CPB, residual heparin effect, clotting factor depletion, thrombocytopenia, or hypothermia, along with other less frequent factors. Blood conservation strategies should be used to limit the number of transfusions, because the administration of packed red blood cells has been independently associated with increased complications and increased mortality.
transfusions are reasonable in most postoperative patients whose hemoglobin is less than 7 g/dL.
Although no exact definition exists for excessive bleeding, amounts greater than 200 mL/h often require further interventions.
However, persistent mediastinal bleeding, usually greater than 500 mL in 1 hour or 300 mL/h for 2 consecutive hours despite normalization of clotting studies, is an indication for reexploration of the surgical site.

A

Control of Bleeding

41
Q

Chest tube stripping to maintain patency of the tubes is controversial because of the high negative pressure generated by routine methods of stripping. It is believed to result in tissue damage that can contribute to bleeding.
Chest tube stripping often is advocated in instances of excessive postoperative bleeding.
However, the technique of “milking” the chest tubes is advisable for routine postoperative care because this technique generates less negative pressure and decreases the risk of bleeding.

A

Chest tube patency

42
Q

is a potentially lethal complication that may occur after surgery if blood accumulates in the mediastinal space, impairing the heart’s ability to pump. Signs of tamponade include elevated and equalized filling pressures (e.g., central venous pressure, pulmonary artery diastolic pressure, pulmonary artery occlusion pressure), decreased cardiac output, decreased blood pressure, jugular venous distention, pulsus paradoxus, muffled heart sounds, sudden cessation of chest tube drainage, and a widened cardiac silhouette on radiographs.
Bedside echocardiogram may be done to confirm tamponade. Interventions for tamponade may include emergency sternotomy in the critical care unit or a return to the operating room for surgical evacuation of the clot.

A

Cardiac Tamponade

43
Q

Mechanical ventilation is used initially to provide adequate alveolar oxygenation and ventilation in the postoperative period.
Potential candidates must be identified before surgery so that the anesthetic regimen supports early extubation.
After surgery, patients are evaluated for hemodynamic stability, adequate control of bleeding, normothermia, and the ability to follow commands.
After extubation, supplemental oxygen is administered, and patients are medicated for incisional pain to facilitate aggressive pulmonary hygiene and early mobility, which is
essential to help prevent postoperative complications.

A

Pulmonary Care

44
Q

The neurologic dysfunction often seen in patients who have undergone cardiac surgery has been attributed to decreased cerebral perfusion, cerebral microemboli, hypoxia, and the systemic inflammatory response.
Early recognition of neurologic changes is important so that prompt initiation of therapy may prevent worsening of complications.
The risk of delirium is increased in cardiac surgery patients, especially older adults, and is associated with increased mortality and reduced quality of life and cognitive function. Nurses have a critical role in the prevention and recognition of delirium.

A

Neurologic Complications

45
Q

Postoperative fever is common after CPB.
Persistent temperature elevation to greater than 101 F (38.3 C) must be investigated. Sternal wound infections and infective endocarditis are the most devastating infectious complications, but leg wound infections, pneumonia, and urinary tract infections also can occur.

A

Infection

46
Q

recognized as a significant problem after cardiac surgery because of a very complex pathogenesis.
injury to the kidney by avoiding renal insults is still the mainstay of management.

A

Acute Kidney Injury

47
Q

Recommendations include: patients who arrest with VF or pulseless VT should receive three sequential attempts at defibrillation before external cardiac massage; patients with asystole or extreme bradycardia should undergo an attempt to pace if wires are available before external cardiac massage; for pulseless electrical activity, quickly reversible causes should be excluded (such as tamponade or bleeding) followed by emergency resternotomy. Finally, because of the danger of extreme hypertension if a reversible cause is rapidly identified, full-dose epinephrine is not recommended unless directed by a senior physician.
Importance of early emergency resternotomy (within 5 minutes) is also a major focus of the recommendations.

A

Resuscitation of Patients Who Arrest After Cardiac Surgery

48
Q

guidelines are designed to support clinical decision making with research evidence

A

Guidelines for Coronary Artery Bypass Graft Surgery

49
Q

includes information related to the surgical procedure, risk factor management, and prevention of atherosclerosis. Patients who have undergone valve surgery may also require information regarding the need for antibiotic prophylaxis before invasive procedures and specific instructions pertaining to their anticoagulation regimen

A

Patient and Family Education

50
Q

have become an integral part of the cardiovascular therapeutic management of patients. The primary goals of MCS are to decrease myocardial workload, maintain adequate perfusion to vital organs, reduce pulmonary congestion, augment coronary perfusion, provide circulatory support during procedures, and limit infarction size. If the condition is irreversible, MCS may be used as a bridge to heart transplantation for qualified candidates or as destination therapy for patients who have no other surgical options.
Short-Term Mechanical Circulatory Support Devices
Long-Term Ventricular Assist Devices

A

Mechanical Circulatory Support

51
Q

provide hemodynamic support for the management of cardiogenic shock, decompensated heart failure, cardiopulmonary arrest, or even prophylactic insertion for high-risk invasive coronary artery procedures
Clinical evidence to guide device selection is often controversial or unavailable.
Intra-aortic Balloon Pump
Left Atria to Aorta Assist Device
Left Ventricle to Aorta Assist Device
Right Ventricular Assist Devices
Extracorporeal Membrane Oxygenation System

A

Short-Term Mechanical Circulatory Support Devices

52
Q

most commonly used temporary percutaneous circulatory assist device used in hemodynamically unstable patients. However, it is limited by its modest hemodynamic support or myocardial protection.
“Counterpulsation” is the key mechanism for this Its therapeutic effects are based on the hemodynamic principles of diastolic augmentation and afterload reduction
Contraindications to IABP include aortic aneurysm, significant aortic valve insufficiency, and severe peripheral vascular disease.
Medical management.
Nursing management.
Timing

A

Intra-aortic Balloon Pump

53
Q

may be inserted in the operating room, the cardiac catheterization laboratory, or the critical care unit. The IAB catheter is usually inserted percutaneously through the femoral artery and advanced to the correct position in the descending thoracic aorta.
percutaneous catheter placement is not feasible, the catheter may be placed through surgical cut-down or by a direct thoracic approach. After insertion, the balloon is attached to the console and filled with the prescribed volume of helium, and pumping is initiated.

A

Medical management.

54
Q

Management of the pumping console and its timing functions may be performed by the nurse caring for the patient or delegated to specially trained personnel.
factors may affect the efficacy of the IABP, including position of the balloon within the aorta; balloon displacement volume; inflation and deflation timing; signal quality; the patient’s cardiac function; and hemodynamic variables, which include circulating blood volume, blood pressure, and vascular resistance.

A

Nursing management.

55
Q

depends on proper timing to ensure optimal hemodynamic benefits. Although systems now adjust the timing automatically, clinicians must still be aware of how to set the timing, understand the method used, and evaluate its effects.
For counterpulsation to occur, the pump must receive a trigger signal to identify the beginning and end of the cardiac cycle. The trigger can be the R wave of the ECG, the upstroke of the arterial pressure waveform, or a pacemaker spike.
Complications.
Weaning.
Educate the patient and family.

A

Timing

56
Q

include lower extremity ischemia resulting from occlusion of the femoral artery by the catheter itself or by emboli caused by thrombus formation on the balloon by the catheter itself or by emboli caused by thrombus formation on the balloon. Evaluation of peripheral circulation remains an important nursing assessment. Signs of diminished perfusion must be reported immediately. Anticoagulation (e.g., heparin infusion) may be prescribed to decrease the incidence of thrombosis. Other vascular complications associated with IABP include acute aortic dissection and the development of pseudoaneurysms at the catheter insertion site.
Balloon complications include balloon perforation and malpositioning.
Measures to prevent accidental displacement of the balloon catheter include ensuring that the IAB is secured to the patient’s skin, that the patient maintains complete bed rest with the head of the bed elevated no more than 30 degrees, and that any flexion of the involved hip is avoided.
Further complications include thrombocytopenia
Patients must also be monitored for signs of stroke and infections.

A

Complications.

57
Q

considered after hemodynamic stability has been achieved with no, or only minimal, pharmacologic support.
less common weaning method involves a gradual decrease in balloon volume. To prevent thrombus formation on the balloon surface, the IABP must remain at a minimal pumping ratio (or volume) until its removal.

A

Weaning.

58
Q

Many IABP manufacturers provide helpful educational booklets designed for patients and families

A

Educate the patient and family.

59
Q

TandemHeart device uses a continuous-flow (CF) centrifugal pump with an inflow and outflow cannula that can be configured in different ways to achieve percutaneous or minimally invasive surgical approaches. Blood is pumped extracorporeally from the left atria to the iliofemoral arterial system. Increase in CO and arterial blood pressure provide support for systemic perfusion.
Complications are related to the need for anticoagulation, transseptal puncture, thrombo- or air embolism, and hemolysis.
Care must be taken to prevent catheter dislodgement, which could lead to systemic desaturation or device malfunction.

A

Left Atria to Aorta Assist Device

60
Q

Impella is a nonpulsatile axial flow pump that draws blood from the LV and ejects it proximally into the ascending aorta
The Impella is contraindicated in patients with mechanical aortic valves.
Monitor for catheter migration is an import aspect of care to prevent hemolysis and ensure proper pump functioning.

A

Left Ventricle to Aorta Assist Device

61
Q

failing RV can be assisted by two different devices. The Impella RP catheter takes blood from the inferior vena cava and delivers it into the outlet opening in the pulmonary artery.
The TandemHeart pump, along with the Protek Duo catheter,
allows for right atrium-to-pulmonary artery cannulation to provide RV support.

A

Right Ventricular Assist Devices

62
Q

is being used with increasing frequency in critical care units. The primary indication for ECMO is acute severe heart or lung failure with high mortality risk despite conventional treatment.
However, consideration must also be given to the fact that bleeding is the most common complication, often requiring transfusions after placement. Staff must be highly trained to maintain the ECMO circuit and monitor for other complications, such as cardiac tamponade, multiorgan failure, sepsis, limb ischemia, and pulmonary complications. Major limitations of ECMO include its limited availability, short duration of usage, required support of highly trained personnel, and complications related to vascular access.
Once evidence of cardiac function has returned, weaning trials can be conducted to assess if the patient can be maintained with conventional support.

A

Extracorporeal Membrane Oxygenation System

63
Q

designed to support or replace a failing natural heart with flow assistance. The clinical use of CF LVADs continues to increase with the improvement in survival rates along with quality of life in patients with heart failure.
Nursing Management

A

Long-Term Ventricular Assist Devices

64
Q

includes monitoring for hemodynamic changes and for complications related to the device.
Although complication rates vary among the different models, complications common to all types of VADs include bleeding, infection, stroke, thromboembolism, respiratory failure, arrhythmias, and device failure.
patients with continuous-flow VADs may not have palpable pulses; this can make measurement of blood pressure difficult and will require an invasive arterial catheter for the initial postoperative period.
Cardioversion and defibrillation should be performed for the same indications as other patients. Nurses must often rely on basic assessments such as circulation, mentation, and urine output to determine whether the patient is receiving adequate support.
Device failure.
Anticoagulation.
Infection.
Patient and family education.

A

Nursing Management

65
Q

it is a life-threatening event involve failure of the driveline and power disconnection
designs vary considerably, and troubleshooting methods for device failure are unique to each device. The nurse must be aware of signs of device malfunction and patient factors (volume status, dysrhythmias, RV failure) that may affect VAD function.

A

Device failure.

66
Q

protocols vary with the device, individual patient, and institution.
Although bleeding is the most frequent complication, thrombotic events can also be a problem leading to stroke and device failure.

A

Anticoagulation.

67
Q

are at considerable risk for localized and septicemic infections. Infectious risks are posed by the presence of invasive catheters and the surgically implanted VAD.
Nurses monitor patients for infection by measuring temperatures, inspecting insertion sites and incisions, and obtaining daily leukocyte counts. If an infection is suspected, pancultures (blood, urine, and sputum) are taken to guide appropriate antibiotic therapy.

A

Infection.

68
Q

rapid and acute nature of cardiogenic shock limits the nurse’s ability to prepare patients and families for VAD insertion. Despite the critical nature of the illness, nurses explain the reason for the use of the VAD and provide information about the critical care environment and equipment
These patients and their families require education related to care of the device and reinforcement on the components of heart failure management.

A

Patient and family education.