cardiovascular physiology Flashcards

1
Q

what are inotrophy, chronotrophy, and dromotrophy

A

inotrophy (force of contraction)
chronotrophy (rate of contraction)
dromotrophy (conduction velocity)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what are the terms for slow and fast heart beats

A

bradycardia - slow heartbeat

tachycardia - fast heartbeat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what are the two types of cardiac muscle cells

A
conducting system cells (controls heartbeat)
contractile cells (produce contractions to move blood)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what is automaticity of cardiac cells

A

the heart doesn’t need any external influence to beat, no nervous innervation or anything

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what is the resting potential of ventricular cells

A

-90 mv

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what is the resting potential of atrial cells

A

-80 mv

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what is the major ion in cardiac muscle

A

Ca

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is the difference between the absolute refractory period and relative refractory period

A

aboslute refractory period comes before the relative refractory period, is long in duration, and the cardiac cells can’t respond to stimuli during it. during the relative refractory period muscle contractile cells can respond to stimuli, if it is strong enough

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is the difference between cardiac and skeletal muscle refractory periods, and why is it important

A

the cardiac muscle refractory period is much longer than the skeletal, this keeps the heart cells from using summation to reach tetanus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what are the structures though which the conducting system impulses pass beginning with the SA node and ending with contractile cells

A
SA node
internodal pathways
AV node
AV bundle
bundle branches
perkinje fibers
contractile cells
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is another name for the prepotential

A

pacemaker potential ( a gradual spontaneous depolarization)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

how many action potentials do the SA and AV nodes produce per minute

A

SA node produces 80-100

AV node produces 40-60

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what do the P, Q, R, S, and T of an EKG represent

A

P - atrial depolarization
QRS - ventricular depolarization (atrial repolarization)
T - ventricular repolarization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what does the P-R interval of the EKG represent

A

start of atrial depolarization to start of QRS complex (ventricular depolarization)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what does the Q-T interval of the EKG represent

A

ventricular depolarization to ventricular repolarization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what are the two phases of the cardiac cycle

A

systole (contraction)

diastole (relaxation)

17
Q

when do we have end-diastolic volume

A

its the volume in the ventricle right before it contracts (at the end of relaxation/diastole) maximum blood volume

18
Q

when do we have isovolumetric contraction

A

right after the AV valves close, and before the semilunar valves open

19
Q

what is the amount of blood ejected from the heart during one systole called

A

stroke volume

20
Q

when do we have end-systolic volume

A

after the ventricle has pumped out all the blood it can and the semilunar valve closes (end of systole) minimum blood volume

21
Q

what percentage of end-diastolic volume remains as end systolic volume

A

40%

22
Q

When do we have isometric relaxation

A

after the semilunar valves close, and before the AV valves open

23
Q

how do you find stroke volume

A

EDV-ESV

24
Q

what is cardiac output, and what influences it

A

volume pumped by left ventricle in one minute.

it is influenced by heart rate and stroke volume

25
Q

what determines heart rate

A

autonomic nervous system and hormones

26
Q

what nervous system increases heart rate

A

sympathetic nervous system

parasympathetic decreases heart rate

27
Q

what are the two cardiac centers in the medulla oblongata

A

cardioacceleratory (SNS)

cardioinhibitory (PNS)

28
Q

what nerves innervate the heart, and where do they come from

A

cardia plexus innervates the heart and comes from the vagus nerve

29
Q

where do you find, and what is the function of cholinergic receptors in the heart

A

they are found in the SA node mostly, and they reduce heart rate (parasympathetic)

30
Q

where do you find, and what is the function of adrenergic receptors in the heart

A

they are found in the SA node, and they increase heart rate and contraction force (sympathetic) and

31
Q

what is the effect of angiotensin

A

increase contractility

32
Q

what are the cardiac reflexes that control heart rate and contractility

A

baroreceptors that detect blood pressure

chemoreceptors that detect oxygen and CO2 levels

33
Q

what is the atrial reflex, or bainbridge reflex

A

heart rate adjusts to venous return. more venous return = quicker heart rate

34
Q

what two things affect EDV

A

filling time

venous return

35
Q

what three things affect ESV

A

preload
contractility
afterload

36
Q

what is the frank starling principle

A

as EDV increases SV increases (due to more volume and contractility)

37
Q

what keeps the heart from stretching too much

A

myocardial connective tissue
cardiac (fibrous)skeleton
pericardial sac