cardiovascular #2 Flashcards

1
Q

what is anemia

A

anemia is when there is not enough healthy red blood cells or hemeglobin in the body to carry oxygen around.

this is due to:
- decreased RBC/hemeglobin
- decreased production of RBC
- increased loss of RBC
- increased desctruction of RBC

general signs and symptoms:
- fatigue
-cold
-paleness
-shortness of breath
-dizzyness or lightheadedness
- jaundice
- heart pulpitations
- gastrointestional effects (nausea)
- neurlogical effects (numbness, tingling)

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2
Q

iron deficiency anemia

A

decreased in iron

decreased in iron= decreased in hemeglobin synthesis

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3
Q

causes of iron deficiency anemia

A

decreased intake (not enough iron in diet)
decreased absorption
increased loss (increased iron loss )
increased demand

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4
Q

what are the risk factors of iron deficiency anemia

A

periods of growth
pregnancy
heavy menstruation
poor diet
blood loss
conditions causing inflammation of the intestines (chrones disease)

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5
Q

what is the treatment for iron deficiency anemia

A

iron supplementation:
note - calcium blocks the absorption of iron. Vitamin C enhances the absorption.
side effects = nausea, dark stool, constipation

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6
Q

what is macrocytic anemias

A

when red blood cells are larger than normal and are immature.
they dont have the normal oxygen carrying capacity due to the deficiency of vit B12 and folic acid.

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7
Q

causes of macrocytic anemias

A

Vitamin B12 deficiency (poor diet, digestive issues)
folic acid deficiency (poor diet, pregnancy alcohol)
alcohol (affects how the body absorbs vitimans)

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8
Q

Disseminated intravascular coagulation (DIC)

A

= widespread clotting within or throughout the blood vessel

be able to draw out that diagram

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9
Q

Deep vein thrombis

A

abnormally formed blood clot in a deep vein

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10
Q

what are factors that promote DVT

A

endothelial damage (surgery)
blood statis (decreased blood flow/blood pooling)
hypercoagulability (increased clotting factors)

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11
Q

what happens with DVT

A

may breakdown on its own
may become organised (infiltrate to vessel walls)
may dislodge itself and become a embolus - risk for pulmonary embolism

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12
Q

what is pulmonary embolism and treatment options

A

when an embolus gets stuck in a pulmonary artery

= no blood flow and no gas exchange

small PE = only a small amount of alveoli are affected
S+S = cough, pain, shortness of breath, Increased RR and HR

Large PE = alot of the alveoli are affected
S+S = hemodynamic compromise —> shock, respiratory/cardiac failure

treatment options:
asprin - prophylaxis = antiplatelet = decrease platelet binding

Anti coagulants - warfrin = blocks vitiman K which is needed to activate some clotting factors

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13
Q
A
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