Cardiology Flashcards
What electrolyte is used in repolarization of the heart?
K+
What causes fast conduction through the heart?
Na+
What effect does parasympathetic stimulation have on the heart?
Slows heart rate , slows conduction, decreases force of contraction, caused by cholinergic Ach
What effect does sympathetic stimulation have on the heart?
Norepinephrine -adrenergic causes increase rate, increase force of contraction and increase irritability of foci, constricts arteries causing increase in BP
Wandering pacemaker
P wave shape varies, atrial rate less than 100, irregular vent rhythm
MAT multifocal atrial tachycardia
P wave shape varies, rate over 100’ COPD or dig toxicity
A fib
Continuous rapid firing by many atrial foci, continuous atrial spikes, count vent rate — QRS in 6 sec strip x 10
Atrial escape rhythm
60-80, atrial foci assumes pacing
Junctional escape rhythm
40-60, usually no p wave, but may have retrograde atrial polarization causing Inverted p wave
Ventricular escape rhythm
Looks like PVC’s, 20-40, P and QRS do not match
Premature atrial beat
Irritable foci, new atrial beat resets the rhythm
Preventricular beat
Vent irritability caused by low O2, low K+
V tac
A run of three or more PVC, from epi like substances
Paroxysmal atrial tachycardia
Different p wave, rate 150-250
PAT with AV block
More the one P for every QRS, dig excess
Paroxysmal junction all tachycardia
150-250, May have inverted p waves
AV node reentry
aVNRT, vicious cycle
SVT
Any tachycardia that originates above the ventricles
Torsades de pointes
250-350, low K+, long qt syndrome, looks like a twisted ribbon
Atrial flutter
250-350, saw tooth,
Ventricular flutter
250-350, single foci
V fib
Multiple foci, bag of worms
A fib
Caused by many irritable parasystolic atrial foci
WPW
Bundle of Kent, av conduction pathway causing a reentrent SVT
1degree AV block
Retards AV node conduction, PR interval greater than .2 sec or one large square, CONSTANT every cycle
2 degree AV block Wenkebach
PR interval gets longer than drops the QRS, still PR interval great than .2 sec
2 degree AV block Mobit
AV node total block conduction, no progression just a dropped QRS, regular punctual P wave
3rd degree AV block
Total conduction block, usually normal P wave with. Junctional QRS, must have pacemaker
BBB
Block produces a delay in the depol of that vent, right chest leads V1-V2, left chest leads V5-V6’ QRS greater than .12 sec or 3 small blocks
How do you determine axis?
If QRS in lead 1 and AVF are positive = two thumbs up sign
If QRS in lead one is negative, what is the. Axis?
RAD
If the transitional QRS is in V1 or V2 what is the axis?
Right shift
If the transitional QRS is in lead V5 or V6, what is the axis?
Left shift
How Do you determine atrial hyper trophy in an EKG?
V1- dish aspic P wave = AE. The larger P wave is hypertrophied.
How do you determine ventricular hypertrophy in a. EKG?
LVH- mm of S in V1 + mm of R in V5 if greater than 35 then LVH
How do you determine ischemia in an EKG?
Diminished blood flow, inverted T waves
How do you determine injury in an EKG?
ST elevation, acute or recent injury, earliest sign of infarction
What does pericarditis look like on an EKG?
ST segment elevation, flat or concave T wave may be off baseline
What is a clue for subendocardial infarction on an EKG?
ST depression, no through entire wall thickness
Wat are the signs of necrosis on an EKG?
Q wave, significant small box wide or 1/3 the height of the QRS
Which leads would show a lateral infarction?
1 and AVL, circumflex
Which leads should show an anterior infarction?
V1-V4 from LAD
Which leads would show an inferior infarction?
2,3 and AVF, RCA or LCA
Which leads should show a posterior infarction?
V1-V2 large R wave plus ST depression
What does a Pulmonary embolism. Look like on EK g?
S1, Q3’ inverted T 3….. Large s in lead 1’ large Q in 3 and t wave inversion in 3
What cases the heart sounds S1?
Mitral and tricuspid closing
What causes the heart sound S2?
Atrial and pulmonic valve closing
Where is each sound heard the best: mitral, tricuspid, pulmonic, aortic?
Apex, left lower sternal border, left upper sternal border, right upper sternal border
What can cause widened splitting of S2?
RBBB pulmonic valve stenosis
Fixed splitting of S2 is caused by?
Atrial septal defect
Paradoxical splitting of the S2 is caused by?
LBBB
After S1, the opening of the aortic or pulmonic valves for stenosis is called an?
Ejection click
What are the systolic murmurs?
Aortic stenosis, pulmonic stenosis, mitral regurgitation and tricuspid regurgitation, VSD, MVP
What murmur begins after S1, May have an ejection click, high frequency, best heard in the RUSB, and radiates to the neck?
Aortic stenosis
What murmur begins after S1, May have a click, and is loudest at the left upper sternal border?
Pulmonic stenosis
What holosystolic murmur is heard best at the apex, blows, radiates to the axilla and does not change with respiration?
Mitral valve regurgitation
What murmur is holosystolic, heard at the left lower sternal border, radiates to the right of the sternum, is blowing and increases with inspiration?
Tricuspid regurgitation
What murmur is holosystolic, high pitched, may have a thrill and does NOT change with inspiration or radiate?
VSD….. Louder = smaller
Describe the murmur of MVP.
Midsystolic click, late systolic murmur heard best at the apex.
List the diastolic murmurs.
Mitral valve stenosis, tricuspid stenosis, aortic regurgitation,pulmonic regurgitation,
What is a blowing murmur in early diastole strongest along the left sternal border heard best siting leaning forward and exhaling.
Aortic valve regurgitation
What is an early diastolic murmur heard best in the left upper sternal border?
Pulmonic regurgitation
This murmur begins after S2, loudest at first, low pitched, heard at apex and in the left lateral decubidis position.
Mitral valve stenosis
This murmur is in early diastole, heard best at lower sternum near xiphoid process?
Tricuspid regurgitation
This is the only continuous murmur?
PDA
How does digoxin therapy look on EKG?
ST scooped depression, mild PR prolongation
How does hyper kalmia look on EKG?
Peaked T wave
How does severe hyperkalemia look on EKG?
Flattened P wave, wide QRS
How does hypojalemia look on EKG?
ST depression, flattened T, prominent U
How does hypercalcemia reflect on the EKG?
Shortened QT interval
How does hypocalcemia look on the EKG?
Prolonged QT interval
Quinidine
Use for a fib with WPW. Give with BB.
Quinidine toxicity?
Diarrhea, cinchonism, vasodilation, TdP
Procanamide
a fib with WPW
Lidocaine cardio use
Use for PVC
2nd choice after amiodarone for V tachs
Dilantin
Use for arrthymia due to DIG toxins, causes hyperplasia of the gums
Describe what beta blockers do.
Use for SVT and PSVT
Useful in suppressing tackyarrhythmias induced by excessive catecholamines (exercise or emotional stress)
Slow vent rage in a fib or a flutter
May terminate Re entrant SVT. Do not prolong QT
Amioderone uses.
Grand daddy! Decrease SA node firing rate, suppress automaticity, interrupt reentrant circuits, preserves CO,
USE: reentrant tachycardia, a fib, a flutter, SVT with bypass tracts
Tachycardia, fibrillation, more effective than lidocaine
Amiodarone toxicity.
Pulmonary toxicity, TdP, hyper/hypo thyroid
Must monitor with EKG, CXR, thyroid, PFT, LFT
Verapamil
CCB. DO NOT USE in: wide complex tachycardia, A fib with WPW, SSS, AV block w/o a pacemaker.
Medications used in PSVT treatment.
Adenosine 6 mg
Adenosine 12mg
Verapamil
Digoxin
Pacemaker indications.
Symptomatic AV node block, mob it’s and type 3.
Long QT syndrome. Recurrent VT. SVT.
Cardiomyopathy. Cardiac arrest. Syncope. NSVT w prior MI
How do you treat a bradyarrhythmias?
Increase HR by anti cholinergic drugs (atropine), B receptor agonists (isoproterenol) or a pacemaker
What is the number one cause of dilated cardio myopathy?
Idiopathic with coxsackie virus mist common viral
Causes of dilated cardiomyopathy?
Idiopathic, HTN, doxorubicin, beri beri, coxsackie virus
Clinical presentation of dilated cardiomyopathy?
Exertional intolerance, atypical cp, dyspnea, orhtopnea, edema, crackles on pulm exam
What does the echo show in dilated cardiomyopathy?
Dilated thin left vent with low EF
What cardiomyopathy is the MC cause of heart transplant?
Dilated
What is an inherited disorder AND a common cause of sudden cardiac death in young athletes?
Hypertrophic cardiomyopathy.
What happens in the heart during hypertrophic cardiomyopathy?
Hypertrophy leads to left vent outflow obstruction and impaired filling which leads to pulmonary congestion
What does the echo show in HOCM?
Septal wall thickness increased and EF of 80-90 %
How to treat hypertrophic cardiomyopathy?
No sports, beta blocker to decrease heart rate and improve filling time
CCB to improve ventricle compliance
If you hear an S3 think?
CHF
If you hear an S4 think?
MI
What causes restrictive cardiomyopathy?
An infiltration process like amyloidosis or sarcoidosis . Damage to muscle wall leads to diastolic noncompliance with elevated filling pressure leading to pulmonary congestion
Clinical presentation and dx of restrictive cardiomyopathy?
Exertion intolerance, fluid retention, right heart failure with JVD, s3,s4,
Diagnose with echo showing decreased EF 25-50% and normal LV thickness
Treatment for restrictive cardiomyopathy?
Diuretics, ACEI, BB or transplant
What is the number one cause of CHF?
Ischemic heart disease
What is ventricle volume before contraction?
Preload
What is the pressure then ventricle pushing against called?
Afterload
Clinical presentation and PE of a patient with CHF?
Dyspnea, orthopnea, PND, fatigue, edema, exercise intolerance
JVD, rales, edema, asities
What are the signs of right sided heart failure?
Systemic issues like JVD, asities
What are the signs of left sided heart failure?
Think pulmonary like orthopnea, dyspnea, rales, PND