cardio/ resp Flashcards

1
Q

What signs are associated with mitral stenosis?

A

malar flush on cheeks.
regular, low-volume pulse
mid-diastolic murmur loudest with the patient leaning to left, at apex
Can cause AF due to atrial enlargement

Thinks ARMS - atrial regurg early murmur, followed by MS with mid murmur

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2
Q

What signs are associated with aortic regurgitation?

A

early diastolic murmur over aortic area: intensity of the murmur is increased by the handgrip manoeuvre
collapsing pulse
wide pulse pressure
Quincke’s sign (nailbed pulsation)
De Musset’s sign (head bobbing)

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3
Q

What are all MI patients discharged on?

A

beta blocker
aspirin
ticagrelor / second line anti-platelet (DAPT - this is stopped at 12 months post MI
ACE inhibitor
statin

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4
Q

1st, 2nd line and 3rd line mx of HF?

A

1st - BB (carvedilol or bisoprolol) + ACE-i
2nd - spironolactone (also think of SGLT-2 inhib if diabetic) then consider SGLT-2 inhib - in hfref
3rd- initiated by a specialist. Options include ivabradine, sacubitril-valsartan, hydralazine in combination with nitrate, digoxin and cardiac resynchronisation therapy

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5
Q

It cardiac arrest is witnessed on a monitor how should ALS be done

A

Witnessed cardiac arrest while on a monitor - up to three successive shocks before CPR

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6
Q

When do patients get fibrinolysis for a STEMI?

A

should be offered within 12 hours of the onset of symptoms if primary PCI cannot be delivered within 120 minutesof the time when fibrinolysis could have been given. Should be given wtih an antithrombin drug eg fondaparinux

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7
Q

dresslers syndrome - what is it?

A

dresslers syndrome is pericarditis secondary to MI, occurs 3-6 weeks after MI

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8
Q

What is Beck’s triad?
What can cause it?

A

Muffled heart sounds + JVP raised + hypotension indicate Beck’s Triad -> cardiac tamponade
LV free wall rupturet MI

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9
Q

Treatment for angina (initial)? 2nd line add in if uncontrolled? 3rd line?

A

Initial: bisoprolol, GTN, aspirin and statin OR if BB CI then start rate limiting CCB eg verapamil

2nd: longer-acting dihydropyridine calcium channel blocker should be added eg Nifedipine, amlodipine

3rd: a long-acting nitrate/ ivabradine/ nicorandil/ ranolazine + refer to cardiology for PCI assessment

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10
Q

What can be found on CXRs for bronchiectasis?

A

Parallel line shadows (often called tram-lines) are common in bronchiectasis and indicate dilated bronchi due to peribronchial inflammation and fibrosis.

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11
Q

What is the 4th line management for BP?

A

if potassium < 4.5 mmol/l add low-dose spironolactone
if potassium > 4.5 mmol/l add an alpha- or beta-blocker

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12
Q

When are prophylactic abx used in COPD? Which abx are used?

A

250mg azithromycin three times per week if:
Non-smoking
On maximum medical mx
Had pulmonary rehab
4 acute exacerbations in the last year (producing sputum), requiring hospital admission at least once.

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13
Q

Howdo you interpret lung function tests?(restrictive vs obstructive)

A

FVC - reduced, FEV1 - reduced FEV1/FVC - normal.

Restrictive lung disease - scarring of lung means reduced compliance and capacity.
Reduced forced vital capacity (FVC) as decreased compliance. FEV1 decreases at same rate so means when devided is normal.

FEV1 - reduced, FEV1/FVC - reduced

obstructive diseases eg COPD, asthma, or bronchiectasis
Obstruction to airflow which means it is hard to exhale. FEV1 is reduced. FVC is normal/ slightly low. So ratio is low.

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14
Q

Mx of spontaneous pnuemothorax? High risk features?

A

High risk features:
haemodynamically unstable
hypoxia
bilateral
underlying lung disease
>50 with sig smoking history
haemopneumothorax

If patient asx/ pneumothorax not big enough to drain - conservative mx

If patient sx + high risk + >2cm on CXR —> chest drain

If patient sx + low risk + >2cm on CXR —> needle aspiration/ ambulatory device

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15
Q

What are electrolyte causes of prolonged QT and potential VT?

A

hypocalcaemia, hypokalaemia, hypomagnesaemia

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16
Q

osler nodes vs janeway lesions?

A

Janeway lesions - erythematous macular or nodular lesions caused by septic emboli

osler nodes - painful erythematous lesions caused by immune complex deposition

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17
Q

what venturi mask should you use on CO2 retainers if NOT critically ill

A

28% Venturi mask at 4 litres/min is used prior to the results of blood gases in patients with risk factors for hypercapnia aiming for oxygen saturation of 88-92%.

If critically ill just whack on 15L

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18
Q

What are the rate limiting CCB? What are the dihydropyrodine CCB?

A

rate-limiting one such as verapamil or diltiazem

onger-acting dihydropyridine calcium channel blocker e.g. amlodipine, modified-release nifedipine

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19
Q

What is the mx of COPD?

A

1st line: SABA/ SAMA
2nd line:
If asthma features (diagnosis prev of asthma/ atopy/ raised eosinophils/ diurnal variation in peak flows) - add ICS + LABA
If no asthma features add LAMA + LABA
3rd line: all three - ICS +LABA +LAMA

20
Q

What do the different leads on the ecg correspond to in terms of coronary arteries and what complications post MI can they lead to?

A

RCA - inferior leads - supplies AV node - so get AV block
LAD - anterior leads - left ventricle thrombus / ventricle free wall rupture/ VSD
Circumflex - lateral leads

21
Q

ECG changes in pericarditis?

A

the changes in pericarditis are often global/widespread, as opposed to the ‘territories’ seen in ischaemic events
‘saddle-shaped’ ST elevation
PR depression: most specific ECG marker for pericarditis

22
Q

How does miliary TB look on CXR?

A

scattered, fine nodules throughout lungs

23
Q

What is moderate vs severe vs very severe copd?

A

The severity of COPD is based upon FEV1 readings. Moderate is FEV1 50-79%, severe is 30-49%, and very severe is <30%.

24
Q

What ECG findings would you see in cardiac tamponade?

A

electrical alternans - alternation of QRS complex

25
Q

Mx of asthma in <6? vs >6?

A

<6
SABA —> add ICS —> add LTRA —> refer

> 6
SABA —-> add ICS —> add LTRA —> add LABA and stop LTRA —> Start low dose steroid MART —> start moderate dose steroid MART —> refer/ add theophylline/ high dose steroid MART

26
Q

What medications are given for pharmacological cardioversion?

A

flecainide or amiodarone if there is no evidence of structural or ischaemic heart disease or
amiodarone if there is evidence of structural heart disease.

27
Q

What coronary artery is affected in new LBBB?

A

anterior/ anteroseptal

28
Q

What lung cancer type has the highest incidence after asbestosis exposure?

A

Bronchogenic carcinoma

29
Q

What are two causes of treatment resistant HTN?

A

coarctation of the aorta (aorta narrowed distal to ductus arteropsusto upper limb BP higher than lower branches)
renal artery stenosis

30
Q

After DC cardioversion for AF how long should anti-coagulation be continued?

A

forever even once sinus rhythm maintained

31
Q

HOCM ECG findings?

A

left ventricular hypertrophy
non-specific ST segment and T-wave abnormalities, progressive T wave inversion may be seen
deep Q waves

32
Q

Which HF medications reduce their mortality?

A

ACE-inhibitors/ ARBs
Beta-blockers
Aldosterone antagonists
Hydralazine and nitrates

33
Q

What is seen on CXR for an aspergilloma?

A

The air crescent sign on chest x-ray is a characteristic finding of aspergilloma where a crescent of air that surrounds a radiopaque mass present in a lung cavity is visible.

34
Q

Presentation of digoxin toxicity?

A

generally unwell, lethargy, nausea & vomiting, anorexia, confusion, yellow-green vision
arrhythmias (e.g. AV block, bradycardia)
gynaecomastia

35
Q

When can you restart sildenafil post MI/ stroke?

A

6 months

36
Q

When do you recieve a bioprosthetic valve vs mechanical?

A

Bioprosthetic are used in >65s as no need for life-long anti-coagulation and have lower risk of VTE, however do not last as long as mechanical

37
Q

Mx of pleural paques?

A

Pleural plaques are the most common form of asbestos-related lung disease and are benign. They are not associated with an increased risk of lung cancer or mesothelioma. This patient should be reassured and advised that no follow-up of these specific plaques is necessary, although an ongoing review of his lung disease is encouraged.

38
Q

What medication is CI in HOCM?

A

ACE-i

39
Q

What are some SE/ big CI to adenosine?

A

adenosine can cause a brief sensation of flushing and intense chest pain, but the side-effects should resolve fastly. This medication should not be administered to asthmatics as it can cause bronchospasm.

40
Q

Which cardiac med can patient develop tolerance to?

A

Patients may develop tolerance to this medication necessitating a change in dosing regimeIsosorbide mononitrat

41
Q

When would you record a posterior ECG?

A

Posterior MI is suggested by the following changes in V1-3:

Horizontal ST depression
Tall, broad R waves (> 30ms)
Upright T waves

42
Q

What is pancoasts syndrome?

A

apical lung ca
leads to horner’s syndrome + brachial plexus invasion

43
Q

How should you treat MI secondary to cocaine use?

A

IV benzodiazepines

44
Q

ECG features in hypokalaemia?

A

U waves
small or absent T waves (occasionally inversion)
prolong PR interval
ST depression
long QT

45
Q

How is asthma diagnosed in adults?

A

Adults with suspected asthma should have both a FeNO test and spirometry with reversibility

46
Q
A