Cardio Flashcards
which side of the heart shit increases with…..
inhalation
inhale you increase venous return to the right heart
*its venous return increases bc venous = blue & when your blue you inhale
Amyl Nitrate will —–to the heart/
decrease afterload
*vasodilator
guy comes in with ST elevation. hes already had MONA what do you do next?
Cath
tx of MS
diuretics then balloon valvuloplasty
what drug is uniquely used to tx PAD
cilostazol
what drug does a person need to be on with a 1. DES & 2. bare metal stent?
how long?
Clopidogrel
- DES = 12m
- Bare metal stent = 1m
tx of WPW
procainamide
what murmer?
water-hammer pulse, wide pulse pressure, Quinke Pulse, Hill Sign
AR!
quinke = pulse in nail bed hill = bp in leg 40 more than arm
dx of pericardial tamponade
ECG
MVP murmer
increase?
midsystolic click murmer
inc: valsalva + standing
Peripheral Arterial disease(PAD) sx
claudication, smooth, shiny skin, loss of hair and sweat glands and loss of pulses in the feet
what is the best initial test for pt with CHF
echo! = tells you if its systolic dysfunction or diastolic dysfunction
what 2 L sided murmers are increased by standing/valsalva but decreased by squating/leg raise
HOCM, MVP
Peripartum Cardiomyopathy
prego women makes Ab’s to her own heart. LV dysfunctionis short term and often reversable if not need transplant.
tx: ACE, BB, Diuretic, etc
pathogenesis of CHF?
infarction/ valvular heart disease/hypertension —> dilation —> regurgitation —> CHF
S4 means….
sound of atrial systole contracting against a stiff or noncompliant LV
well fuck…you
MR dx test?
TEE
MR murmer
increase? decrease?
pansystolic murmer caused by dilation of the heart that radiates to the axilla
.
inc:leg raise, squat, handgrop
dec: standing, valsalva and amyl nitrate
when do you give biventricular pacemaker?
EF <35% + QRS >120ms
tx of MVP
BB > valve repair
AS murmer
increases? decreases?
crescendo-decrescendo systolic murmer
inc: leg raising, squatting + amyl nitrate
dec: valsalva, standing, handgrip
if you see electrical alternans on ECG you shoudl be thinking….
pericardial tamponade, QRS height alternates between leads
causes of pleuritic pain
PE, pneumonia, pleuritis, pericarditis, pneumothorax
you do a nuc stress tests & see low uptake. what do you do next?
ANGIOGRAPHY to determine what vessels are involved.
how do you differentiate from 3rd degree block vs sinus brady?
3rd degree block will have “cannon a-waves” = atrial contracting againsted a closed tricuspid = ventricular diassociation
Squatting will —– to the heart.
increase venous return
*pushes blood from legs to heart via M contractions
lifting legs in the air with ——ot the heart.
increase venous return
*gravity
tx of bradycardia
atropine
MS sx
diastolic openign snap, dilated LA pushes on esophagus causing “horseness”, increased risk of Afib, elevates Left mainstem bronchus due to dilated atrium
What meds lower mortality in ACS?
BB(only one thats not time sensitive), Aspirin & Nitro
pt post MI, w/oxy sat in RV > RA. dx?
septal rupture!
when do you give a implantable cardioverter/defibulator
EF <35%
Which RF of CAD can you eliminate that will provide the greatest IMMEDIATE benefit?
smoking
Which cardiac marker can be used to assess 2nd MI?
CKMB
Constrictive pericarditis sx? path?
heart calcifies = can be seen on xray
sx: pericardial knock bc heart cant fill, edema, JVD, hepatosplenomegaly, ascites, kussmauls sign(increased JVD on inhalation)
sx of pericardial tamponade
SOB, hypotension, JVD, CLEAR LUNGS, pulsus paradoxus(>10mmhg on inhalation), ELECTRICAL ALTERNANS