Cardio 2 Flashcards
chest pain resulting from Myocardial Ischemia caused by inadequate myocardial blood & oxygen supply (imbalance between O2 supply & Demand)
Angina
Causes: obstruction of coronary blood flow resulting from atherosclerosis, coronary artery spasm, or conditions increasing myocardial oxygen consumption
Angina
occurs w/ Activities that involve Exertion or Emotional Stress; Relieved w/ Rest or Nitroglycerin
Stable (Exertional Angina)
occurs w/ an Unpredictable degree of Exertion or Emotion & Increases in Occurrence, Duration, & Severity Over Time; Pain may not be relieved w/ Nitroglycerin
Unstable (PreInfarction Angina)
Chronic, Incapacitating & Unresponsive to Interventions
Intractable Angina
Acute Coronary Insufficiency; lasts >15-30 min; Worsening Cardiac Ischemia; Chest Pain days to weeks before an MI
PreInfarction Angina
Pain: develops slowly or quickly; mild-Moderate; Substernal, Crushing, Squeezing; may Radiate to Shoulders, Arms, Jaw, Neck, Back; Intensity unaffected by Inspiration/Expiration; lasts
Angina
Dsypnea
Pallor
Sweating
Angina
Palpitations & Tachycardia
Dizziness & Syncope
HypErTension
Digestive Disturbances
Angina
ECG: Normal readings during Rest w/ ST Depression or T-Wave Inversion during an Episode of Pain
Angina
Angina Stress Testing: Chest Pain/Changes in ECG or VS during Testing may Indicate
Ischemia
Findings are normal in Angina
Cardiac Enzyme & Troponin levels
provides definitive diagnoses by providing information about Patency of Coronary ARteries
Cardiac Catheterization (Angina)
- Asses Pain
- O2 by NC
- VS, continuous cardiac monitoring,
- Bed Rest/ semi-Fowler’s
- 12-lead ECG
- IV access
Nitroglycerin (Angina)
to Dilate Coronary Arteries, reduce O2 requirements of Myocardium, & relieve chest Pain
Nitroglycerin (Angina)
Antiplatelet meds to inhibit platelet aggregation & reduce risk of developing an Acute MI
Angina
occurs when myocardial Tissue is Abruptly & Severely Deprived from Oxygen; Ischemia can lead to Necrosis of Myocardial Tissue if blood flow is Not Restored.
MI
does not occur Instantly, Evolves over Several Hours; After 6 hrs, area appears blue & swollen; After 48 hrs, area turns gray w/ yellow streaks (as neutrophils invade tissue)
MI
Granulation Tissue forms
8-10 days after Infarction
over 2-3 months, Necrotic area develops into Scar tissue, permanently changing size & shape of ..
entire left ventricle (MI)
Atypical discomfort, SOB, Fatigue often present w/ NSTEMI (non-ST-elevation myocardial Infarction) or T-Wave Inversion
Women (Sx of MI)
SOB, pulmonary Edema, Dizziness, ALOC, dysrhythmia
Elderly (Sx of MI)
Atherosclerosis/ CAD/ Elevated Cholest. levels
MI risk factors
Smoking/ HTN/ Obesity/ Physical Inactivity
MI risk factors