CAD Flashcards
Myocardial oxygen demand is a function of:
HR
SBP
Read this:
normal
Read this:
v-fib
What is a good EF? Bad EF?
good = >50%
bad = <50%
Read this:
v-fib
How is EF calculated?
Stroke volume divided by EDV in LV
Afterload is directly affected by
ventricular dilation
SBP in LV
What type of CAD: rush this mayne to the cath lab
STEMI
What percentage of blockage is considered problematic?
70%
3 factors affecting SV?
preload
afterload
contractility
If a patient had a RV infart, what is a symptom you’d expect to (specific for RV)?
JVD
How is acute coronary syndrome different from STEMI?
ACS: incomplete occlusion and no EKG changes
How do platelets affect plaque rupture?
TXA2 causes vasoconstriction
ADP recruits more platelets
they can reactivate for several weeks
What 2 factors do dr’s manipulate to correct a bad, bad BP?
flow resistant (P=QR)
What type of CAD: dyspnea on exertion, pain on exertion (i.e. walking up the stairs carrying my 34lb backpack) relieved by rest
chronic stable angina
What type of CAD: EKG is unimpressive, patient is pale, diaphoretic, short of breath with substernal CP that woke him from sleep
ACS
ST elevation on an EKG means…
vessel fully occluded
In ACS, why does a patient with ~40% blockage suddenly have a serious obstruction?
transient vasospasm causes ~80% obstruction (the demand is constant but the oxygen supply is down)
How does preload affect BP?
an increase in preload causes an increase in BP (because it increases SV)
Read this:
STEMI
Coronary oxygen supply is a function of
coronary blood flow
How do you know it’s heart pain (not just chest wall pain, etc)?
>5 min but <30
What causes angina pain?
the plaque is growing!
What causes an increased oxygen demand?
increased BP
increased HR
Inadequate oxygen supply is due to:
blockage
vasoconstriction
MI complications include:
myocardial rupture (can occur a few different places)
LV aneurysm
pericarditis
venous thrombosis
Read this:
STEMI
