Bites, Stings And Infestations Flashcards
Nerovascular exam
1st step in management of a sting/bite/infection
- make sure to check parenthesis, alertness and ability to move
- make sure to also look for bone and joint involvement as this indicates a worsening condition*
Tetanus toxoid vs tetanus immunoglobulin
- during the management of a bite/sting or infestation, make sure to check last tetanus injection*
toxoid must be given if:
- last vaccine was > 10 yrs (clean wound)
- last vaccine was > 5 yrs (dirty wound)
Immunoglobulin must be given if less than 3 full doses of a tetanus vaccine in patient history
Rabies prevention and antibiotic use
Rabies:
- must give immunoglobulin and vaccine if no prior vaccine has ever been given.
- only have to give vaccine if previous vaccine series has been given
Antibiotics:
- specific for certain bacteria and typically oral medications
- most common prescribed is amoxicillin-clavulonic acid orally*
- IV is indicated with bone/joint/ serious tissue damage
When to close bite/sting wounds
Facial wounds must always be closed
- NEVER CLOSE HAND/FEET WOUNDS*
- poor circulation so would just result in pus filled hypoxia
Try to not close cat or human bites and only close superficial dog bites
Broad criteria for closure:
- uninflected at time
- < 24 hrs old
- not on hand/foot
- not immunocompromised
Human bites overview
Breast bites are to be assumed sexual assault until proven otherwise
Arm bites > 2.5 cm in diameter on children are assumed to be child abuse until proven otherwise
2 categories of human bites:
clenched fist injuries
- typically result from fights and usually at 3-4th fingers
- need antibiotics and might need to operative treatment if fever and extreme swelling are present
occlusive bites (closing mouth around the skin) - need antibiotics and most commonly caused by children
Elkinella corrodens
Unique pathogen to human bites only
Causes blood culture-negative endocarditis
- infection rate is 30%
- resistant to most antibiotics and has to be treated with amoxicillin-clavuloinic acid
staph and step are also possible human bite pathogens
Cat bites overview
Most common in women and typically hand or feet
80% of cat bites lead to infection and usually within 24 hrs of bite
- narrow penetrating wounds increases chances of infection
- marked by really small bite marks, but extreme swelling of the bite area*
Usually require xrays to evaluate for teeth, joint or bone involvement
Most common symptom outside of swelling is cellulitis
Pasteurella multocida
Unique pathogen to cat bites and has a 30% mortality rate
Results in osteomyelitis and joint abscess if left untreated
- can also result in tenosynovits which requires 100% surgery
Treatment:
- amoxicillin-clavulonic acid
Cat scratch fever
Caused by Bartonella henselae infections
- caused by flea feeces under cat nails/ around their teeth
- cardinal sign is fever with gigantic sentinel lymph nodes to site of infection
DO NOT CUT OPEN OR DRAIN LYMPH NODE
- prescribe antibiotics (which don’t actually help, just prevents other possible opportunistic pathogens) and let the body heal itself
Dog bite overview
Most common in males 5-9 years
- children - face/neck
- adults = arm/ leg
Much more dangerous since dogs apply more pressure and tear rather than puncture
- causes of death include carotid avulsion (throat bite), skull injuries/Brian injuries and mengitits
- can also cause compartment syndrome due to increase pressure and accompanying rhabdomyolysis
Infection rate is 30%
Dog bites almost always are indicated for xrays
Capnocytophaga canimorsus
Pathogen unique to dogs
- 30% mortality rate and much higher chance in immunocompromised
Fight bite injuries specifics
Should include imaging and labs to R/O tenosynovits/osteomyelitis and septic arthritis
Most common infectious agents are Elkinella corrodens and staph aureus
Viper snake bites
Toxic snake bites that produce cytotoxic effects
Include rattlers copperheads and pit vipers
Pit viper bites can be dry bites (25%)
Symptoms:
- oozing pus at bite site
- cell injury signs (swelling ecchymoses, blackening necrosis)
- systemic signs: fatigue, headache, DIC, fasciculations
- can develop IgE mediated anaphylaxis due to type 1 sensitivity
Almost all infections are gram-negative infections
TREATMENT:
- antivenin ASAP ( especially if progression of injury, coagulpathy or systemic effects kick in)
- DO NOT USE TOURNIQUETS OR SUCK VENOM OUT
Elapid snake bites
Include coral snakes, spitting cobra and black mamba
Presents with neurotoxic effects
- most common is ACh receptors bind irreversibly.
Most common in fingers
Symptoms:
- parasthesia, double-vision, dysphasia, aphasia, muscle weakness/CNS dysfunction
- Death almost always occurs via respiratory failure*
Treatment:
- antivenin if neurological symptoms present
- if not severe neurological symptoms, supportive care is indicated
Characteristics of venomous vs non-venomous snakes
Non venomous:
- red on black: venom lack
- rounded head w/ rounded pupil
- NO FANGS
- double row of subcaudal plates
Venomous:
- red on yellow: kill a fellow
- triangle-shaped head w/ elliptical pupil
- FANGS
- single row of subcaudal plates and/or rattle