Arrythmias Flashcards
Arrhythmias are described in terms of
rate and site of origin
Rate
Bradycardias
Tachycardias
Site of origin
Supraventricular
Ventricular
alterations in impulse formation involve
Changes in automaticity
Triggered activity
abnormalities in impulse conduction arise from
Re-entry
Conduction block
Accessory tracts
Partial conduction block
Slowed conduction
Tissue conducts all impulses, but more slowly than usual
First degree block
longer PR interval
Intermittent block
Tissue conducts some impulses but not others
2 types of 2nd degree heart block
Mobitz Type 1
Mobitz Type 2
Mobitz Type 1
PR interval gradually increases from cycle to cycle until AV node fails completely and a ventricular beat is missed
Mobitz Type 2
PR interval is constant but every nth ventricular depolarisation is missing
Missing R waves
Complete heart block
No impulses conducted through affected area
Accessory pathway
Electrical pathway parallel to AV node
Bundle of Kent
Impulse through bundle of Kent is quicker than through AV node
Ventricles receive impulses from both normal and accessory pathways
Ventricular arrhythmias occur
Origin is ventricle
Supraventricular arrhythmias occur
Origin is above ventricle
(SA node, atria, AV node,
Ectopic beats
Beats originating in places other than SA node
A normal action potential may trigger abnormal oscillations in membrane potential called
afterdepolarisations
Repeated afterdepolaristaions can cause
an arrhythmia
Early afterdepolarisation
Occur when heart rate is slow
Can lead to Torsades de Pointes
Delayed afterdepolarisation
Occur when heart rate is fast
Triggered by drugs increasing Ca++ influx
1st degree heart block story
Wife (P) waits at home
Husband (QRS) comes home late every night but at the same time
2nd degree heart block Type 1 story
Wife (P) waits at home
Husband (QRS) comes home later and later every night until one day he doesn’t come home at all
2nd degree heart block Type 2 story
Wife (P) waits at home
Husband (QRS) sometimes comes home, sometimes doesn’t
Complete heart block story
Wife (P) no longer waits at home
She and husband (QRS) are on separate schedules and have no relationship
Requires ‘counselling’ from a permanent pacer
Rhythm control classes
I and III
Rate control classes
II and IV
Class I
Reduce Na channel current
Class II
B-adrenergic antagoinsts
Class III
Action potential prolongation
Class IV
Ca channel antagonists
Supraventricular Tachycardias
Atrial Fibrillation
Atrial Flutter
Ectopic atrial tachycardia
Supraventricular Bradycardia
Sinus bradycardia
Sinus pauses
Ventricular arrhythmias
Ventricular ectopics
Ventricular tachycardia
Ventricular fibrillation
Asystole
AVN node arrhythmias
AVN re-entry tachycardia
AV reciprocating tachycardia
AV block
Re-entry
Requires more than one conduction pathway with different speed of conduction
Symptoms of Arrhythmias
Palpitations SOB Dizziness Syncope Presyncope Sudden cardiac death Angina
Investigations of arrhythmias
ECG
CXR
Echo
Stress ECG, 24 hour ECG
Sinus Bradycardia
< 60 bpm
Athlete
Drugs (B blocker)
Treatment for sinus bradycardia
Atropine
Pacing
Sinus tachycardia
> 100 bpm
Anxiety, fever, hypotension
Illegal drugs
Treatment of sinus tachycardia
Treat underlying cause
B blockers
Atrial ectopic beats
Asymptomatic or palpitations
No treatment
Avoid stimulants
SVT management
Acute = IV adenosine, IV veramapril Chronic = B blockers, anti-arrhythmics
Causes of AVN conduction disease (heart block)
Age MI Myocarditis Drugs Genetic
Treatment for 1st degree heart block
none
Types of pacemaker
Single chamber or dual chamber (replaces RA and RV)
Ventricular fibrillation
chaotic ventricular electrical activity causing heart to loose ability to function as a pump
Treatment of V fib
Defibrillation
Cardiopulmonary resuscitation
Atrial Fibrillation
Chaotic and disorganised atrial activity
Irregular heartbeat
Can be paroxysmal, persistant or chronic
AF termination
Anti-arrhythmic drugs
DCCV
Spontaneous reversion to sinus rhythm
Paroxysmal AF
Less that 48 hours
Persistant
More than 48 hours
Reacts to drugs
Chronic
Inability of pharmacological methods to restore
A fib symptoms
Palpitations Presyncope Syncope Chest pain Dyspnea Sweatiness Fatigue
ECG of AF
Irregularly irregular
Absence of P waves
Management of AF
Rhythm control
Rate control
Anti-coagulation
Rate control drugs
Digoxin
B blockers
Veramapril
Rhythm control drugs
DCCV
Atrial flutter
Rapid and regular form of atrial tachycardia
Paroxysmal
Seconds to years
Atrial flutter ECG
Saw tooth
Atrial flutter treatment
Pharmacological
Cardioversion
Warfarin