Arrythmias Flashcards

1
Q

Arrhythmias are described in terms of

A

rate and site of origin

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2
Q

Rate

A

Bradycardias

Tachycardias

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3
Q

Site of origin

A

Supraventricular

Ventricular

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4
Q

alterations in impulse formation involve

A

Changes in automaticity

Triggered activity

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5
Q

abnormalities in impulse conduction arise from

A

Re-entry
Conduction block
Accessory tracts

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6
Q

Partial conduction block

A

Slowed conduction

Tissue conducts all impulses, but more slowly than usual

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7
Q

First degree block

A

longer PR interval

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8
Q

Intermittent block

A

Tissue conducts some impulses but not others

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9
Q

2 types of 2nd degree heart block

A

Mobitz Type 1

Mobitz Type 2

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10
Q

Mobitz Type 1

A

PR interval gradually increases from cycle to cycle until AV node fails completely and a ventricular beat is missed

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11
Q

Mobitz Type 2

A

PR interval is constant but every nth ventricular depolarisation is missing
Missing R waves

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12
Q

Complete heart block

A

No impulses conducted through affected area

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13
Q

Accessory pathway

A

Electrical pathway parallel to AV node

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14
Q

Bundle of Kent

A

Impulse through bundle of Kent is quicker than through AV node
Ventricles receive impulses from both normal and accessory pathways

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15
Q

Ventricular arrhythmias occur

A

Origin is ventricle

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16
Q

Supraventricular arrhythmias occur

A

Origin is above ventricle

(SA node, atria, AV node,

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17
Q

Ectopic beats

A

Beats originating in places other than SA node

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18
Q

A normal action potential may trigger abnormal oscillations in membrane potential called

A

afterdepolarisations

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19
Q

Repeated afterdepolaristaions can cause

A

an arrhythmia

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20
Q

Early afterdepolarisation

A

Occur when heart rate is slow

Can lead to Torsades de Pointes

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21
Q

Delayed afterdepolarisation

A

Occur when heart rate is fast

Triggered by drugs increasing Ca++ influx

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22
Q

1st degree heart block story

A

Wife (P) waits at home

Husband (QRS) comes home late every night but at the same time

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23
Q

2nd degree heart block Type 1 story

A

Wife (P) waits at home

Husband (QRS) comes home later and later every night until one day he doesn’t come home at all

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24
Q

2nd degree heart block Type 2 story

A

Wife (P) waits at home

Husband (QRS) sometimes comes home, sometimes doesn’t

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25
Complete heart block story
Wife (P) no longer waits at home She and husband (QRS) are on separate schedules and have no relationship Requires 'counselling' from a permanent pacer
26
Rhythm control classes
I and III
27
Rate control classes
II and IV
28
Class I
Reduce Na channel current
29
Class II
B-adrenergic antagoinsts
30
Class III
Action potential prolongation
31
Class IV
Ca channel antagonists
32
Supraventricular Tachycardias
Atrial Fibrillation Atrial Flutter Ectopic atrial tachycardia
33
Supraventricular Bradycardia
Sinus bradycardia | Sinus pauses
34
Ventricular arrhythmias
Ventricular ectopics Ventricular tachycardia Ventricular fibrillation Asystole
35
AVN node arrhythmias
AVN re-entry tachycardia AV reciprocating tachycardia AV block
36
Re-entry
Requires more than one conduction pathway with different speed of conduction
37
Symptoms of Arrhythmias
``` Palpitations SOB Dizziness Syncope Presyncope Sudden cardiac death Angina ```
38
Investigations of arrhythmias
ECG CXR Echo Stress ECG, 24 hour ECG
39
Sinus Bradycardia
< 60 bpm Athlete Drugs (B blocker)
40
Treatment for sinus bradycardia
Atropine | Pacing
41
Sinus tachycardia
> 100 bpm Anxiety, fever, hypotension Illegal drugs
42
Treatment of sinus tachycardia
Treat underlying cause | B blockers
43
Atrial ectopic beats
Asymptomatic or palpitations No treatment Avoid stimulants
44
SVT management
``` Acute = IV adenosine, IV veramapril Chronic = B blockers, anti-arrhythmics ```
45
Causes of AVN conduction disease (heart block)
``` Age MI Myocarditis Drugs Genetic ```
46
Treatment for 1st degree heart block
none
47
Types of pacemaker
Single chamber or dual chamber (replaces RA and RV)
48
Ventricular fibrillation
chaotic ventricular electrical activity causing heart to loose ability to function as a pump
49
Treatment of V fib
Defibrillation | Cardiopulmonary resuscitation
50
Atrial Fibrillation
Chaotic and disorganised atrial activity Irregular heartbeat Can be paroxysmal, persistant or chronic
51
AF termination
Anti-arrhythmic drugs DCCV Spontaneous reversion to sinus rhythm
52
Paroxysmal AF
Less that 48 hours
53
Persistant
More than 48 hours | Reacts to drugs
54
Chronic
Inability of pharmacological methods to restore
55
A fib symptoms
``` Palpitations Presyncope Syncope Chest pain Dyspnea Sweatiness Fatigue ```
56
ECG of AF
Irregularly irregular | Absence of P waves
57
Management of AF
Rhythm control Rate control Anti-coagulation
58
Rate control drugs
Digoxin B blockers Veramapril
59
Rhythm control drugs
DCCV
60
Atrial flutter
Rapid and regular form of atrial tachycardia Paroxysmal Seconds to years
61
Atrial flutter ECG
Saw tooth
62
Atrial flutter treatment
Pharmacological Cardioversion Warfarin