Arrythmias ☺️ Flashcards

1
Q

What are the types of AV block

How would you differentiate between them on an ECG

A

1st degree AV block
-elongated PR interval due to DELAY through AV node

2nd degree AV block

  • failure to conduct an impulse => skipped QRS
  • Wenckebach - PR gets progressively longer until QRS lost
  • Mobitz II - repeated pattern between normal beats and loss of QRS

3rd degree AV block

  • no coordination between atria and ventricles, distal slower pacemakers take over
  • P and QRS are not coordinated
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2
Q

How would you manage AV heart block

A

Asymptomatic 1st degree or Wenckebach => monitoring for progressions

Symptomatic 1st degree or Wenckeback, Mobitz II, 3rd degree

  • risk and benefit of discontinuing causative medications
  • may consider PPM with or without an ICD
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3
Q

What is atrial fibrillation

  • causes and pathophysiology
  • signs
  • investigations
A

Damage to the atria is the most common cause => multiple rotating electrical impulses bombard the AV with many signals

  • not all signals result in QRS => irregularly irregular
  • paroxysmal <=> persistent

Palpitations, SOB, chest pain, fatigue, dizziness
-Irregularly irregular HR

FBC => non cardiac causes (anemia, infection)
Electrolytes U&E => assess for CKD and K abnormalities
TFTs => thyrotoxicosis can cause AF
ECG => f waves, no p waves with irregular QRS
TnT => assess for MI
Assess for causative elements that need urgent treatment
CXR => assess for other cardiac, pulmonary causes of presentation
Echo => visualise abnormal movement

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4
Q

How would you manage AF

  • drugs
  • lifestyle
A

Rate control - AVN blocker

  • Bb (bisoprolol) or
  • nonDHP CCB (diltiazem, verapamil)

Use CHA2DS2VASc score to consider stroke risk
Use HAS-BLED to assess bleed risk

Lifestyle changes to reduce CV risk

  • physical activity
  • weight loss
  • alcohol reduction

Anticoagulation Bb CCB, Digoxin, Electrocardioversion

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5
Q
How do these antiarrythmic drugs work
-Class I
-Class II
-Class III
-Class IV
-Class V
What are some examples
A

Class I => Na block (rhythm)

  • Decrease excitability of membrane
  • Quinidine, lidocaine

Class II => Bb (rate)

  • Decrease conduction via AVN => decrease HR and contractility
  • bisoprolol, propranolol

Class III => K block (rhythm)

  • Prolong repolarisation to prevent reentrant arrythmias
  • Amiodarone, soltalol

Class IV => Ca block (rate)
-Decrease conduction via AVN and shorten action potential => retains HR and
contractility
-Diltiazem - peripheral and coronary VD
-Verapamil - K blocker to prolong repolarisation

Class V => other

  • Digoxin - decrease AVN conduction, increase vagus
  • Adenosine - decrease AVN conduction, K efflux Ca inh => prolong repolarisation
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6
Q

What is the

  • cause and pathophysiology
  • signs
  • investigations for ventricular tachycardia
A
Any heart conditions (CHD, cardiomyopathy, MI) that result in ventricular scarring 
-ectopic PM
-reentry
Or electrolyte abnormalities
FAST REGULAR RHYTHM

Chest pain, palpitations, SOB, dizziness, heart failure

  • may or may not have CO
  • fast regular rhythm

ECG => wide QRS, tachycardia (monomorphic or polymorphic)
Electrolytes => assess for K imbalances
TnT => assess for MI
Echo => image abnormal heart movement

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7
Q

How would you manage ventricular tachycardia

  • electrical
  • medication
  • surgical
A

Cardioversion => synchronised on R wave for unstable tachyarrythmias
-if patient has a pulse

Defibrillation => not synchronised shock
-if pulseless

Medication => may consider antiarrythmics (Bb, Ca)

Surgical

  • ICD implantation
  • catheter ablation
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8
Q

What is the

  • cause and pathophysiology
  • signs
  • investigations for ventricular fibrillation
A

Chaotic multiple impulses spreading through ventricles due to scarring
FAST IRREGULAR RHYTHM

Chest pain, rapid heartbeat, SOB, dizziness
Hypotensive => cardiac arrest

ECG => no QRS, chaotic activity

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9
Q

How would you manage ventricular fibrillation

  • medical stabilisation
  • maintenance
A

Medical stabilisation
-CPR, defibrillation

Maintenance
-ICD insertion

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10
Q

What is 3rd degree heart block

  • pathophysiology
  • causes
  • signs
  • investigations
A

Dissociation of atrial and ventricular depolarisation due to faulty AV conduction
-distal pacemakers take over

Inferior MI
AV blocking drugs

Bradycardia
Dizzy, faint, SOB, fatigue
Chest pain

Diagnostic => ECG
-p waves and QRS complexes are dissociated from each other
TnT for evidence of current MI

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11
Q

Most common cause of sudden cardiac death

A

Coronary artery disease

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