Arrythmias ☺️ Flashcards
What are the types of AV block
How would you differentiate between them on an ECG
1st degree AV block
-elongated PR interval due to DELAY through AV node
2nd degree AV block
- failure to conduct an impulse => skipped QRS
- Wenckebach - PR gets progressively longer until QRS lost
- Mobitz II - repeated pattern between normal beats and loss of QRS
3rd degree AV block
- no coordination between atria and ventricles, distal slower pacemakers take over
- P and QRS are not coordinated
How would you manage AV heart block
Asymptomatic 1st degree or Wenckebach => monitoring for progressions
Symptomatic 1st degree or Wenckeback, Mobitz II, 3rd degree
- risk and benefit of discontinuing causative medications
- may consider PPM with or without an ICD
What is atrial fibrillation
- causes and pathophysiology
- signs
- investigations
Damage to the atria is the most common cause => multiple rotating electrical impulses bombard the AV with many signals
- not all signals result in QRS => irregularly irregular
- paroxysmal <=> persistent
Palpitations, SOB, chest pain, fatigue, dizziness
-Irregularly irregular HR
FBC => non cardiac causes (anemia, infection)
Electrolytes U&E => assess for CKD and K abnormalities
TFTs => thyrotoxicosis can cause AF
ECG => f waves, no p waves with irregular QRS
TnT => assess for MI
Assess for causative elements that need urgent treatment
CXR => assess for other cardiac, pulmonary causes of presentation
Echo => visualise abnormal movement
How would you manage AF
- drugs
- lifestyle
Rate control - AVN blocker
- Bb (bisoprolol) or
- nonDHP CCB (diltiazem, verapamil)
Use CHA2DS2VASc score to consider stroke risk
Use HAS-BLED to assess bleed risk
Lifestyle changes to reduce CV risk
- physical activity
- weight loss
- alcohol reduction
Anticoagulation Bb CCB, Digoxin, Electrocardioversion
How do these antiarrythmic drugs work -Class I -Class II -Class III -Class IV -Class V What are some examples
Class I => Na block (rhythm)
- Decrease excitability of membrane
- Quinidine, lidocaine
Class II => Bb (rate)
- Decrease conduction via AVN => decrease HR and contractility
- bisoprolol, propranolol
Class III => K block (rhythm)
- Prolong repolarisation to prevent reentrant arrythmias
- Amiodarone, soltalol
Class IV => Ca block (rate)
-Decrease conduction via AVN and shorten action potential => retains HR and
contractility
-Diltiazem - peripheral and coronary VD
-Verapamil - K blocker to prolong repolarisation
Class V => other
- Digoxin - decrease AVN conduction, increase vagus
- Adenosine - decrease AVN conduction, K efflux Ca inh => prolong repolarisation
What is the
- cause and pathophysiology
- signs
- investigations for ventricular tachycardia
Any heart conditions (CHD, cardiomyopathy, MI) that result in ventricular scarring -ectopic PM -reentry Or electrolyte abnormalities FAST REGULAR RHYTHM
Chest pain, palpitations, SOB, dizziness, heart failure
- may or may not have CO
- fast regular rhythm
ECG => wide QRS, tachycardia (monomorphic or polymorphic)
Electrolytes => assess for K imbalances
TnT => assess for MI
Echo => image abnormal heart movement
How would you manage ventricular tachycardia
- electrical
- medication
- surgical
Cardioversion => synchronised on R wave for unstable tachyarrythmias
-if patient has a pulse
Defibrillation => not synchronised shock
-if pulseless
Medication => may consider antiarrythmics (Bb, Ca)
Surgical
- ICD implantation
- catheter ablation
What is the
- cause and pathophysiology
- signs
- investigations for ventricular fibrillation
Chaotic multiple impulses spreading through ventricles due to scarring
FAST IRREGULAR RHYTHM
Chest pain, rapid heartbeat, SOB, dizziness
Hypotensive => cardiac arrest
ECG => no QRS, chaotic activity
How would you manage ventricular fibrillation
- medical stabilisation
- maintenance
Medical stabilisation
-CPR, defibrillation
Maintenance
-ICD insertion
What is 3rd degree heart block
- pathophysiology
- causes
- signs
- investigations
Dissociation of atrial and ventricular depolarisation due to faulty AV conduction
-distal pacemakers take over
Inferior MI
AV blocking drugs
Bradycardia
Dizzy, faint, SOB, fatigue
Chest pain
Diagnostic => ECG
-p waves and QRS complexes are dissociated from each other
TnT for evidence of current MI
Most common cause of sudden cardiac death
Coronary artery disease