Arrhythmias Flashcards

1
Q

Definition of first degree heart block

A

PR intverval greater than 0.2 (5 small squares). no tx needed

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2
Q

mobitz type I heart block definition

A

progressive PR lengthening until skipped QRS. PR progression, then resets and starts again

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3
Q

mobitz type I heart block cuases

A

intranodal or His bundle conduction defect or as an effect of BBS, CCBs, or digoxin, or vagal tone

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4
Q

treatment of mobitz type I heart block

A

adjust meds (CCBs, BBs, digoxin), no other treatment unless pt has symptomatic bradycardia

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5
Q

mobitz type II heart block definition

A

problem with His bundles or purkinje fibers. patients have randomly skipped QRS without changes in PR interval

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6
Q

mobitz type II treatment and complications

A

tx: pacemaker; can progress to complete heart block

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7
Q

complete heart block: definition and tx

A

absence of conduction between atria and ventricles
patients present with syncope, dizziness, hypotension
Tx: pacemaker, don’t give meds that affect the AV node

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8
Q

Paroxysmal supraventricular tachycardia: definition, pt population, cause

A

definition: tachycardia arising in atria or AV junction
usually seen in young pts with healthy hearts
cause usually reentry anomaly:
-AV nodal reentry: slow and fast conduction pathways (reentrant tachycardia)
-Can also be due to AV reentry as seen in WPW syndrome, which is similar to AV nodal reentry excetp that there is a separate accessory conduction path between the atria and the ventricles. there will be a delta wave on EKG

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9
Q

What is the usual hx for paroxysmal supraventricular tachycardia

A

sudden tachycardia, possible chest pain, SOB, palpitations, syncope

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10
Q

What is mutifocal atrial tachycardia?

A

caused by several ectopic foci in the atria that discharge automatic impulses, causing tachycardia. Will appear as p waves with variable morphology on EKG

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11
Q

What is the tx for mutifocal atrial tachycardia?

A

CCBs or BBs acutely, then catheter ablation or surgery

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12
Q

What are the main risk factors for the development of atrial fibrillation?

A

PIRATES: pulmonary disease, ischemia/CAD, rheumatic heart disease, anemia, hyperthyroid, ethanol, and sepsis
Can also be seen with pericarditis

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13
Q

How is afib treated?

A

anticoagulation, rate control with CCBs or BBs, or digoxin, consider electrical or chemical cardioversion if presening within the first 2 days.
cardioconversion can be performed in delayed presenation if absence of thrombi is confirmed by TEE. If thrombus is seen, anticoagulate and wait 3-4 weeks before cardioversion

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14
Q

What are the important complications of afib?

A

increased risk of MI, heart failure; poor atrial contraction –> blood stasis –> clot/embolization

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15
Q

What are the risk factors for atrial flutter?

A

CAD, CHF, COPD, pericarditis, valvular disease

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16
Q

What are causes of PVCs? When are they significant?

A

become concerning for the development of other ventricular arrhythmias if more frequent than 3x/min
usually benign, but may be due to hypoxia, abnormal electrolytes, hyperthyroidism, caffeine

17
Q

tx for PVCs? possible complications?

A

none if pt is healthy, consider BB if pt has CAD because they are associated with incr risk of sudden death in pts with CAD

18
Q

VTach

A

series of 3 or more PVCs with HR 160-240

19
Q

torsades de pointes

A

VTach with sine wave morphology; poor prognosis and can rapidly convert to vfib; magnesium may be useful in tx

20
Q

How is VTach treated?

A

electrical cardioversion followed by antiarrhythmic medication. if recurrent, may need internal defibrillator